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1 ified 178 of all 938 patients with permanent hypoparathyroidism.
2 ces the incidence of permanent postoperative hypoparathyroidism.
3 and 30 patients (15.0%) developed persistent hypoparathyroidism.
4 reserve their function and prevent permanent hypoparathyroidism.
5 rgical treatment for permanent postoperative hypoparathyroidism.
6 C) (residue 25 of PTH), which is linked with hypoparathyroidism.
7 perative recurrent laryngeal nerve palsy and hypoparathyroidism.
8 ant decrease in both immediate and permanent hypoparathyroidism.
9 oidism, while 2 patients developed permanent hypoparathyroidism.
10 equire tracheostomy, and 2 had postoperative hypoparathyroidism.
11 emia occurred in the setting of pre-existing hypoparathyroidism.
12 ptides, including drugs for osteoporosis and hypoparathyroidism.
13 s relevant to therapeutic strategies against hypoparathyroidism.
14 , potentially reducing the risk of permanent hypoparathyroidism.
15 rior to TT regarding transient postoperative hypoparathyroidism.
16 was the incidence of transient postoperative hypoparathyroidism.
17 hereas ADH1 is a major cause of non-surgical hypoparathyroidism.
18 nd a therapeutic target for osteoporosis and hypoparathyroidism.
19 y Ca(2+) loss, particularly in patients with hypoparathyroidism.
20 ting correction of both immunodeficiency and hypoparathyroidism.
21 spondylarthropathic changes of long-standing hypoparathyroidism.
22 ed delayed parathyroid AT to treat permanent hypoparathyroidism.
23 tive that results in a very low incidence of hypoparathyroidism.
24 eatening APS-I manifestations such as AI and hypoparathyroidism.
25  a PTH replacement therapy for patients with hypoparathyroidism.
26 id hormone 1-84 (rhPTH[1-84]) in adults with hypoparathyroidism.
27 ta might underestimate the rate of permanent hypoparathyroidism.
28 d to impaired PTH secretion, such as primary hypoparathyroidism.
29 h autosomal dominant and autosomal recessive hypoparathyroidism.
30 DR syndrome and not in one, such as isolated hypoparathyroidism.
31 included and 938 (12.5%) developed permanent hypoparathyroidism.
32 nidentified gene in the etiology of X-linked hypoparathyroidism.
33 hypoplasia, conotruncal cardiac defects, and hypoparathyroidism.
34 roband of an extensive kindred with isolated hypoparathyroidism.
35 pped to 6p23-24, as a candidate for isolated hypoparathyroidism.
36 ssociated with a high incidence of permanent hypoparathyroidism.
37 ns result in autosomal dominant and sporadic hypoparathyroidism.
38 sk for the later development of hypocalcemic hypoparathyroidism.
39 tor mutation in a child with severe sporadic hypoparathyroidism.
40 oidectomy virtually eliminates postoperative hypoparathyroidism.
41 ted RR, 0.1; 95% CI, 0.0-0.4), and permanent hypoparathyroidism (0% vs 1.8%) (weighted RR, 0.2; 95% C
42 ratio [RR], 0.4; 95% CI, 0.2-0.7), temporary hypoparathyroidism (2.2% vs 21.3%) (weighted RR, 0.1; 95
43 tegories of disease, risks were elevated for hypoparathyroidism (2.58; 1.35 to 4.92; p-value 0.004),
44  and transplant has a high rate of permanent hypoparathyroidism (22%).
45 uent (recurrent laryngeal nerve palsy 25.3%; hypoparathyroidism 6%).
46         Autosomal dominant familial isolated hypoparathyroidism (AD-FIH) is caused by a Cys --> Arg m
47 this receptor might cause autosomal dominant hypoparathyroidism (ADHP).
48 PS 1), which manifests in a classic triad of hypoparathyroidism, adrenal insufficiency, and candidias
49 udy evaluated the incidence of postoperative hypoparathyroidism after near-total (NTT) versus TT in G
50  Previous data suggest that the incidence of hypoparathyroidism after surgery for Graves disease (GD)
51 st-free, objective method for helping reduce hypoparathyroidism after thyroid surgery.
52 In this cohort study, the risk of persistent hypoparathyroidism after total or completion thyroidecto
53                        The risk of permanent hypoparathyroidism after total thyroidectomy was high an
54                                     Acquired hypoparathyroidism (AH) has been considered to result fr
55 ls from 12 families with autosomal recessive hypoparathyroidism and have investigated them for GCMB a
56  with consequent development of hypocalcemic hypoparathyroidism and hypercalciuria.
57                                Post-surgical hypoparathyroidism and hypocalcemia are known to occur a
58                                The resulting hypoparathyroidism and hypocalcemia can range from asymp
59 ntribute to the previously reported postburn hypoparathyroidism and hypocalcemia.
60                                    Permanent hypoparathyroidism and laryngeal nerve injury were not o
61 ions of 10p (one patient diagnosed as having hypoparathyroidism and three as DGS) and one patient wit
62      To describe the course of postoperative hypoparathyroidism and to assess whether genotype is ass
63                                    Permanent hypoparathyroidism and unintentional recurrent nerve par
64 ociated with potential complications such as hypoparathyroidism and vocal cord paralysis in a small p
65 er the second surgery, 2 (25%) had permanent hypoparathyroidism, and 1 (12.5%) developed inoperable c
66 erized by chronic mucocutaneous candidiasis, hypoparathyroidism, and adrenal insufficiency, but patie
67  like autoimmune adrenal insufficiency (AI), hypoparathyroidism, and chronic mucocutaneous candidiasi
68 nifests as a triad of adrenal insufficiency, hypoparathyroidism, and chronic mucocutaneous infections
69  in T cell-mediated deficiency, hypocalcemic hypoparathyroidism, and conotruncal cardiac defects.
70 ongenital disorder characterized by athymia, hypoparathyroidism, and heart defects.
71 and disease-specific), vocal fold paralysis, hypoparathyroidism, and hemorrhage/hematoma.
72 n the definition and treatment of persistent hypoparathyroidism, and use of uniform evidence-based tr
73 congenital malformations, thymic hypoplasia, hypoparathyroidism, and/or cardiac defects.
74                      Hyperparathyroidism and hypoparathyroidism are clinically important disorders af
75 e two kindreds exhibiting X-linked recessive hypoparathyroidism are indeed related and that an identi
76            Skeletal changes of long-standing hypoparathyroidism are irreversible.
77 s, our results, which expand the spectrum of hypoparathyroidism-associated GCMB mutations, help eluci
78 t-effective compared with the usual care for hypoparathyroidism at a large, academic institution.
79 cessive disorder characterized by autoimmune hypoparathyroidism, autoimmune adrenocortical failure, a
80  half-life of parathyroid hormone (PTH), can hypoparathyroidism be diagnosed intraoperatively when re
81 ength of stay (OR, 0.98; 95% CI, 0.98-0.99), hypoparathyroidism/calcium disorder (OR, 1.0; 95% CI, 0.
82                                   Congenital hypoparathyroidism can be severely debilitating for pati
83                               Long- standing hypoparathyroidism can cause spondyloarthropathic change
84 emia type 1 (ADH1) is a rare genetic form of hypoparathyroidism caused by gain-of-function (GoF) vari
85 me of the deafness associated with the human hypoparathyroidism, deafness and renal anomaly (HDR) syn
86 ur work indicates that hearing loss in human hypoparathyroidism, deafness and renal anomaly (HDR) syn
87 Human GATA3 haploinsufficiency leads to HDR (hypoparathyroidism, deafness and renal dysplasia) syndro
88 A3 haploinsufficiency also causes human HDR (hypoparathyroidism, deafness, and renal dysplasia) syndr
89                                          The hypoparathyroidism-deafness-renal (HDR) dysplasia syndro
90                        The rate of immediate hypoparathyroidism decreased from 37% (95% CI, 27%-48%)
91     We report on the incidence of persistent hypoparathyroidism, defined as the need for active vitam
92  achieve durable correction of the patients' hypoparathyroidism due to parathyroid graft rejection.
93 ions included seven cases (17%) of permanent hypoparathyroidism; five (71%) of these occurred in grou
94                                              Hypoparathyroidism following thyroid surgery is a seriou
95 1 HDR probands and 14 patients with isolated hypoparathyroidism for GATA3 abnormalities.
96  study (REPLACE), we recruited patients with hypoparathyroidism (>/= 18 months duration) aged 18-85 y
97 caemic (subclinical) hyperparathyroidism and hypoparathyroidism have a low risk of progression to ove
98 and exhibiting X-linked recessive idiopathic hypoparathyroidism have been described.
99  reported the association of papilledema and hypoparathyroidism, However, very rarely, case reports r
100 MC) and multisystem autoimmunity, most often hypoparathyroidism (HP) and adrenal insufficiency (AI).
101                                     X-linked hypoparathyroidism (HPT) has been mapped to a 988-kb reg
102      Endocrine disorders such as obesity and hypoparathyroidism, hypervitaminosis A, tetracycline use
103 pe 1 (APS-1), which includes hypothyroidism, hypoparathyroidism, hypoadrenalism, and hypogonadotropic
104                                              Hypoparathyroidism (hypoPT) is the most common complicat
105  parathyroid glands and exhibit a biological hypoparathyroidism, identifying Gcm2 as a master regulat
106 sease genes-including, for example, X-linked hypoparathyroidism in 3 Mb (6 cM) telomeric to Factor IX
107 d embryology and is responsible for isolated hypoparathyroidism in a subset of patients with this dis
108      Surprisingly, we observed correction of hypoparathyroidism in one patient after thymus transplan
109 nalysis there was a higher risk of permanent hypoparathyroidism in patients with parathyroid autotran
110               The reported incidence rate of hypoparathyroidism in the literature is highly variable.
111 04 cases and resulted in a 1.0% incidence of hypoparathyroidism in this series.
112                                              Hypoparathyroidism is a frequent concomitant clinical pr
113                                   Idiopathic hypoparathyroidism is a rare endocrine disorder with cli
114                                    Permanent hypoparathyroidism is a recognized complication of thyro
115                                   Idiopathic hypoparathyroidism is an extremely rare endocrinal disor
116                                              Hypoparathyroidism is associated with more severe infect
117                                              Hypoparathyroidism is characterized by hypocalcemia, hyp
118               The reported rate of permanent hypoparathyroidism is highly variable and mostly rely on
119                                              Hypoparathyroidism is one of the most common complicatio
120                            Postthyroidectomy hypoparathyroidism is typically diagnosed with low serum
121                                Postoperative hypoparathyroidism lasting 6 months or more should not b
122 stating disorder characterized by congenital hypoparathyroidism, mental retardation, facial dysmorphi
123 c sequencing of individuals with nonsurgical hypoparathyroidism (n = 169) and an in vitro functional
124 o intention-to-treat postoperative transient hypoparathyroidism occurred in 19% (20/103) patients aft
125       Persistent (>/=6 months) postoperative hypoparathyroidism occurred in 24% after <SPTX, 39% afte
126                                    Permanent hypoparathyroidism occurred in 3 (6%) of 50 patients in
127                                              Hypoparathyroidism occurred in 3%, 10%, and 22%, respect
128  can exacerbate GO and surgery can result in hypoparathyroidism or laryngeal nerve damage.
129                Unlike patients with acquired hypoparathyroidism, patients with these mutations had hy
130 cal harm was 2.12 to 5.93 cases of permanent hypoparathyroidism per 100 thyroidectomies and 0.99 to 2
131                                              Hypoparathyroidism post-thyroidectomy arises when all pa
132                                    Permanent hypoparathyroidism rates decreased from 32% (95% CI, 22%
133              When autofluorescence was used, hypoparathyroidism rates were comparable with those of h
134 tions can cause renal aplasia as part of the hypoparathyroidism, renal dysplasia, deafness (HDR) synd
135                                              Hypoparathyroidism results in impaired mineral homoeosta
136 ly, Gata3 loss of function is known to cause hypoparathyroidism, sensorineural deafness and renal dis
137 seases due to GATA-3 deficiency - such as in hypoparathyroidism, sensorineural deafness, and renal (H
138 Haplo-insufficiency of the GATA3 gene causes hypoparathyroidism, sensorineural hearing loss, and rena
139 clinical problems including cardiac defects, hypoparathyroidism, T-cell immunodeficiency and facial d
140 s, conotruncal cardiac defects, hypocalcemic hypoparathyroidism, T-cell mediated immune deficiency, a
141 ns were identified in patients with isolated hypoparathyroidism, thereby indicating that GATA3 abnorm
142           Therefore we expect this patient's hypoparathyroidism to be permanently cured.
143           Secondary endpoints were permanent hypoparathyroidism, transient recurrent laryngeal nerve
144                  The incidence of persistent hypoparathyroidism varied between 14.5% (calcium and act
145                  The high rate of persistent hypoparathyroidism warrants efforts to reduce this compl
146                           Median duration of hypoparathyroidism was 1.5 years, in 65% successful cess
147 vitamin D postoperatively, whereas permanent hypoparathyroidism was considered when there still was a
148                                    Immediate hypoparathyroidism was defined as the need for active vi
149                                    Permanent hypoparathyroidism was defined as treatment with calcium
150                  A total of 10 patients with hypoparathyroidism were enrolled consecutively over a 15
151   Ninety eight (45.2%) experienced temporary hypoparathyroidism, while 2 patients developed permanent
152                                 Treatment of hypoparathyroidism with PTH 1-34 reduces urine calcium e
153 ns physiologically meaningful variations for hypoparathyroidism) within 10 minutes.

 
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