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1 , suggesting that preoptic AgRP2 neurons are hypophysiotropic.
2 T expression by the ventral tanycytes in the hypophysiotropic area suggests a role of tanycyte-mediat
3 minals establish synaptic relationships with hypophysiotropic CRH and TRH neurons, we propose that CA
4                                              Hypophysiotropic CRH neurons serve as the origin of the
5  be accomplished by GLP-1's direct action on hypophysiotropic CRH neurons.
6 rticular, serves as a potential regulator of hypophysiotropic DA neurons and reproduction.
7 tary, and explored its role in regulation of hypophysiotropic dopamine (DA) neurons in the catfish, C
8 hey have also been proposed to have a direct hypophysiotropic effect on pituitary gonadotropin releas
9       In addition to CRF being the principal hypophysiotropic factor driving the HPA axis, it is a po
10 ulted in the selective expression of ChR2 in hypophysiotropic GnRH neurons.
11 eems to rule out this peptide as a classical hypophysiotropic hormone and, furthermore, PrRP is less
12        The present findings suggest that the hypophysiotropic LHRH-synthesizing neurons may be innerv
13  activated cells corresponded principally to hypophysiotropic neurons expressing corticotropin-releas
14 ional activation of the CRF and AVP genes in hypophysiotropic neurons follows distinct time courses t
15 -adrenal (HPA) axis activation by inhibiting hypophysiotropic neurons in the paraventricular hypothal
16 the activation of pro-TRH gene expression in hypophysiotropic neurons via either a mono- and/or multi
17 g the gene expression of both CRH and TRH in hypophysiotropic neurons, and CART-containing axon termi
18 eural-specific cytoskeletal protein, and two hypophysiotropic neuropeptides.
19 lase (TH)-immunoreactive neurons in NPPa are hypophysiotropic, none of the TRH-immunoreactive neurons
20  incerta dopaminergic neurons (which are not hypophysiotropic) or RFRP-3 neurons were labeled with th
21 at corticotropin-releasing factor (CRF), the hypophysiotropic peptide that initiates endocrine respon
22 itionally, dual-label ISHH demonstrated that hypophysiotropic PVH cells coexpress Y1-R and pro-thyrot
23 transcript (CART) has an important action on hypophysiotropic thyrotropin-releasing hormone (TRH) and
24 y also be contained in fibers that innervate hypophysiotropic thyrotropin-releasing hormone (TRH) neu
25 strate that CART has a stimulatory effect on hypophysiotropic TRH neurons by increasing pro-TRH gene
26 a major source of the CART-IR innervation of hypophysiotropic TRH neurons.
27 (3)-mediated negative-feedback regulation by hypophysiotropic TRH neurons.
28 ggesting that CART may serve as a marker for hypophysiotropic TRH neurons.
29 uoro-Gold resulted in the labeling of 40% of hypophysiotropic tuberoinfundibular dopaminergic (tyrosi