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1 f intravenous normal saline when they became hypotensive.
2 en patients who received procainamide became hypotensive.
3 of lactate values less than 2 mmol/L and not hypotensive.
4   Male KO mice, but not female KO mice, were hypotensive.
5       He became tachycardic, tachypneic, and hypotensive.
6 contrast, W/W(v) mouse LES was significantly hypotensive (11 +/- 2 mm Hg; N = 6; P < 0.05) with norma
7         Of 97 severe reactions 45 (46%) were hypotensive, 23 (24%) were hypoxemic, and 29 (30%) were
8 lgorithm on data from 563 Chinese women, 206 hypotensive, 357 hypertensive, with information on ethni
9 ow pathway play a central role in the ocular hypotensive action of adenosine A(1) agonists.
10 ed peripherally located CB1 receptors in the hypotensive action of cannabinoids.
11  study was designed to determine whether the hypotensive action of clonidine resulted from its stimul
12 rs and is in part responsible for the ocular hypotensive action of epinephrine.
13  that a significant proportion of the ocular hypotensive action of PGF2alpha in cats is mediated by E
14 establish how this effect is involved in the hypotensive action of prostaglandin analogues in vivo.
15 r, the mechanisms responsible for the ocular hypotensive action of this compound are not completely u
16                                         This hypotensive action results from an early reduction in aq
17 ion of c-fos expression in brainstem and its hypotensive action, and provide the first evidence that
18       These receptors may mediate the ocular hypotensive actions of 5HT2A agonists.
19 r-regulatory actions that buffer the extreme hypotensive actions of the peptides when released in sep
20 oncerning both the affinity for I1Rs and the hypotensive activity of 2-aryliminopyrrolidines.
21 nhuman primates, and its mechanism of ocular hypotensive activity was investigated.
22 s for a substantial proportion of the ocular hypotensive activity.
23                           Septic rats became hypotensive after 12 hrs, with a 24-hr mortality of 51.7
24                          In patients who are hypotensive after blunt abdominal trauma and not hemodyn
25       Although 59% potentially had an ocular hypotensive agent at 12 months, only 10% had such medica
26 ty with prostaglandin F(2 alpha), the ocular hypotensive agent bimatoprost (Lumigan; Allergan, Inc.,
27 show that 17-phenyl trinor PGE2 is an ocular hypotensive agent in cats.
28          Bimatoprost is a widely used ocular hypotensive agent to treat glaucoma.
29  who were using a single bottle of an ocular hypotensive agent, and objectively measured adherence wi
30 ug delivery device loaded with a proprietary hypotensive agent, DE-117, reduced intraocular pressure
31 st (BMP), which is a highly effective ocular hypotensive agent, is a PGF(2)(alpha) analogue that inhi
32 icacious and generally well-tolerated ocular hypotensive agent.
33 trong candidate for development as an ocular hypotensive agent.
34 y was to assess level of adherence to ocular hypotensive agents and to identify factors affecting adh
35                     Good adherence to ocular hypotensive agents is important to control intraocular p
36 s of three pharmacologically distinct ocular hypotensive agents on Cyr61 and CTGF gene expression.
37                      The mean (SD) number of hypotensive agents per eye had decreased from 0.7 (1.1)
38 the adherence level to the prescribed ocular hypotensive agents to be sub-optimal and is influenced b
39 of-of-concept study for this class of ocular hypotensive agents.
40 ts, 42.6 % were adherent to their prescribed hypotensive agents.
41                           However, they were hypotensive and bradycardic when compared with heterozyg
42 ocannabinoids are novel lipid mediators with hypotensive and cardiodepressor activity.
43            Furthermore, AKAP150-/- mice were hypotensive and did not develop angiotensin II-induced h
44                         Transgenic mice were hypotensive and exhibited a decreased pressor response.
45 uretic and diuretic peptide that is markedly hypotensive and functions via a separate guanylyl cyclas
46 A, an optically active tropane alkaloid with hypotensive and miotic activity isolated from the Chines
47 ine induced by gamma interferon/CXCL9 in the hypotensive and normotensive patients, respectively, aff
48 all Multiple Organ Dysfunction score between hypotensive and normotensive patients.
49    Our objective was to compare four groups, hypotensive and normotensive resuscitation of hemorrhage
50 quiring PN after CPB were significantly more hypotensive and required more vasopressive drugs during
51 may be reliable alternatives, only to detect hypotensive and therapy-responding patients.
52  been ascribed to Allium extracts, including hypotensive and vasorelaxant activities.
53         Within 24 h post procedure he became hypotensive and was admitted to ICU in frank septic shoc
54          The third patient eventually became hypotensive and was treated with repeated sessions of re
55  13/3893 (0.3%) had severe reactions (6 were hypotensive) and 6 reported syncope.
56 in replacement when the ob/ob mice developed hypotensive ARF with a higher dose of LPS (0.5 mg/kg).
57 aring all patients or only patients who were hypotensive at enrollment.
58 r in conscious mice, alpha 1A/C KO mice were hypotensive at rest, with an 8-12% reduction of blood pr
59 2, which is a component of the counteracting hypotensive axis of RAS.
60 uring 10-min normovolemic baseline, bleed to hypotensive baseline, 10-min ITD-assisted breathing, 10
61        No rats were anemic, hypoglycemic, or hypotensive before CCI.
62 l exercise prescriptions remain unclear, but hypotensive benefits have been noted for mild to vigorou
63                                    There are hypotensive benefits to exercise training found across a
64 rtensive blood pressure high and genetically hypotensive blood pressure low mice.
65  ischemia compared with those with normal or hypotensive BP responses but are not more likely to have
66 e hypertensive BPH (blood pressure high) and hypotensive BPL (blood pressure low).
67  display dramatically enhanced and prolonged hypotensive, bradycardic, and sedative-hypnotic response
68 lternative to norepinephrine in managing the hypotensive brain dead donor.
69                       Hemodynamic stress via hypotensive challenge has been shown previously to cause
70                In some rats LC activation by hypotensive challenge was also examined.
71 cleus and LC neurons were still activated by hypotensive challenge.
72 tem would normally be expected to respond to hypotensive challenge; however, inflammation appears to
73 esponses parallel the sympathoinhibitory and hypotensive components of the Bezold-Jarisch reflex.
74        AVP deficiency may contribute to this hypotensive condition.
75 Ps, a protein kinase A inhibitor, attenuated hypotensive dilation (35+/-1 vs. 16+/-2% before and afte
76 hat the released uPA impairs hypercapnic and hypotensive dilation through an LRP- and ERK MAPK depend
77 tion of the gene responsible for orthostatic hypotensive disorder in these families may advance under
78                         Familial orthostatic hypotensive disorder is characterized by light-headednes
79 pressor effect of vasopressin replacement in hypotensive donors.
80 h treatment is usually initiated with ocular hypotensive drops, laser trabeculoplasty and surgery may
81  beta-adrenoceptor agonist, isoproterenol, a hypotensive drug that typically produces only water inta
82 e mouse eye that facilitates study of ocular hypotensive drugs and mouse models of glaucoma.
83 therapeutic effects of candidates for ocular hypotensive drugs.
84 leads to the best results in terms of ocular hypotensive effect and safety.
85 the mechanism(s) responsible for this ocular hypotensive effect has not been established.
86                        We defined the portal hypotensive effect in rat models of sinusoidal PHT and t
87 ned by activation of vagal afferents and the hypotensive effect may be secondary to a reduction of ca
88 d mice, O-1918 dose-dependently inhibits the hypotensive effect of abn-cbd but not the hypotensive ef
89                                          The hypotensive effect of alpha2 agonists was completely abs
90      The aim of this work was to analyse the hypotensive effect of amaranth protein/peptides on spont
91 ribution of TRPV(1) receptors to the in vivo hypotensive effect of anandamide is equivocal.
92 on of icatibant significantly attenuated the hypotensive effect of captopril (maximal decrease in mea
93 uggest a link between the estrogen-dependent hypotensive effect of chronically administered ethanol a
94 abolished the bradycardia and attenuated the hypotensive effect of endomorphin 1.
95 aneous pellet, 14.2 microg/day) restored the hypotensive effect of ethanol to a level similar to that
96 action is involved in the estrogen-dependent hypotensive effect of ethanol.
97                                          The hypotensive effect of inhaled Y-27632 on hypoxic PH was
98           In EP2-/- mice, the characteristic hypotensive effect of intravenous PGE2 infusion was abse
99 B2-receptor antagonist, had no effect on the hypotensive effect of kallistatin yet it abolished the b
100           These data indicate that the early hypotensive effect of latanoprost in the mouse eye is as
101                                  The initial hypotensive effect of spinal anaesthesia is caused by a
102 reported findings that ethanol abolishes the hypotensive effect of the alpha(2)-adrenoceptor agonist
103 he hypotensive effect of abn-cbd but not the hypotensive effect of the CB(1) receptor agonist (-)-11-
104            In addition to their sedative and hypotensive effect, their curare-like activity and struc
105 SC, at a dose selected for lack of long-term hypotensive effects at baseline).
106                        To compare the ocular hypotensive effects of 15-keto latanoprost (KL) with the
107 rdiovascular regulation, which contribute to hypotensive effects of acupuncture.
108 e cardioprotective, bronchoconstrictive, and hypotensive effects of adenosine.
109 c receptor (alpha2A-AR) is necessary for the hypotensive effects of clonidine and other sympathoinhib
110 irs the diuretic and natriuretic but not the hypotensive effects of D(1)-like agonist stimulation.
111 hypothesis that a greater attenuation of the hypotensive effects of losartan would be observed in rat
112 ons were independently lesioned, the chronic hypotensive effects of the AT(1) receptor blocker losart
113 the LHA or PAG with lidocaine attenuates the hypotensive effects produced by electrical stimulation o
114 no greater attenuation of losartan's chronic hypotensive effects than animals with lesion of either t
115                                          The hypotensive effects were observed within 3 wk after the
116 as associated with diuretic, natriuretic and hypotensive effects.
117 nal biochemical phenotype that exerts potent hypotensive effects.
118 ation related through negative inotropic and hypotensive effects.
119 this 28-day study was to evaluate the ocular hypotensive efficacy and safety of AR-13324 ophthalmic s
120                         The definition for a hypotensive episode is either a systolic blood pressure
121 n, corrected by renal artery stenting, and 1 hypotensive episode, which resolved with medication adju
122 values collected before, during, and after a hypotensive episode.
123                                              Hypotensive episodes can be managed with countermeasures
124                                         Many hypotensive episodes in the ICU are drug related and req
125  emphasize the importance of avoiding severe hypotensive episodes in this at-risk cohort.
126 hundred fifty-eight patients experienced 204 hypotensive episodes that were considered unintentional
127 al nephrectomies and documentation of severe hypotensive episodes were independent risk factors for s
128 re lability with severe hypertensive crises, hypotensive episodes, and orthostatic hypotension, makin
129 ons based on dynamic variables surrounding a hypotensive event is a new approach to predicting progno
130                              A single, acute hypotensive event lasting 30 mins did not aggravate the
131 ain swelling, pupillary abnormalities, early hypotensive events (before intensive care monitoring), a
132                                The number of hypotensive events was twice as high during treatments w
133       The primary endpoint was the number of hypotensive events, as defined by any of the following:
134                              The duration of hypotensive exposure and development of SSI was assessed
135 logistic regression; the association between hypotensive exposure and duration of hospitalization was
136 olume 7.5% NaCl adenosine, lidocaine, and Mg hypotensive fluid resuscitation from the rat to the pig.
137                     Here, we discovered that hypotensive folk medicines from a genetically diverse ra
138 o, which were widely used as ingredients for hypotensive food, showed a slight selectivity for the N-
139 ion and 417 of the remaining 1596 (26%) were hypotensive for age.
140                     Fixed-combination ocular hypotensives have multiple advantages, but triple-therap
141  were either volume expanded or subjected to hypotensive hemorrhage.
142                                              Hypotensive hemostasis is usually an effective means to
143                       This approach includes hypotensive hemostasis, minimizing fluid resuscitation,
144             Endotoxemia induced an immediate hypotensive, hyperdynamic, tachycardic state with progre
145                         The injury induced a hypotensive-hyperdynamic circulation; increases in pulmo
146 nary bypass just before rewarming commenced (hypotensive, hypothermic), after rewarming (hypotensive,
147  whether naloxone would augment tolerance to hypotensive hypovolemic stress (lower body negative pres
148 he multicenter PAMPer trial was performed on hypotensive injured patients from the scene.
149  subjects with UARS (7.2 +/- 0.84 mm Hg), or hypotensive insomniacs (7.4 +/- 1.1 mm Hg) (p < 0.01).
150                In contrast, W/W(v) mice have hypotensive LES with unimpaired relaxation, suggesting t
151 betes, uveitis, retinitis pigmentosa, ocular hypotensive lipids, internal limiting membrane peeling,
152 geal manometry showed a high prevalence of a hypotensive lower esophageal sphincter (55%) and impaire
153 and ICU mortality (p = 0.76) associated with hypotensive mean arterial pressure readings (</=60 mm Hg
154 lial nitric oxide synthase and production of hypotensive mediators, nitric oxide and cyclic guanosine
155 nsion may respond to specific antagonists to hypotensive mediators.
156                     The initiation of ocular hypotensive medication among OHTS participants originall
157 f 77 patients; 59 eyes were receiving ocular hypotensive medication and had no previous history of gl
158 7% of control eyes were not receiving ocular hypotensive medication at 23 months.
159  we censored data after initiation of ocular hypotensive medication or glaucoma surgery of any kind.
160                                              Hypotensive medication use was tracked throughout the co
161                     Annual washout of ocular hypotensive medication was performed.
162 t and second eyes to the same topical ocular hypotensive medication.
163  >=20% in intraocular pressure without using hypotensive medication.
164 pnea-hypopnea index [AHI], 35) not receiving hypotensive medication.
165  and by 367 trabeculectomies (86%) including hypotensive medication.
166 g but not more than 21 mm Hg with or without hypotensive medication.
167 C >0.75), from 0.29 to 0.80 in patients with hypotensive medications (3 time points with ICCs >0.75),
168 .75), from -0.07 to 0.60 in patients without hypotensive medications (zero time points with ICC >0.75
169      With the development of potent arterial hypotensive medications for arterial hypertension, arter
170  to determine the response to topical ocular hypotensive medications has been recently debated.
171                                   Washout of hypotensive medications was repeated at 12 and 24 months
172 93), and the proportion of patients using no hypotensive medications was significantly higher at 24 m
173       After a washout of all prestudy ocular hypotensive medications, 756 eligible patients with elev
174 up in eyes treated with a mean of 0.9 ocular hypotensive medications, and 14.5 (3.4) mm Hg in the dou
175 up in eyes treated with a mean of 2.3 ocular hypotensive medications, and 25.7 (8.0) mm Hg in the dou
176 eyes and separately in eyes with and without hypotensive medications, and in eyes naive and non-naive
177 cess was stratified according to IOP, use of hypotensive medications, bleb needling, and resuturing/r
178      After a washout of all pre-study ocular hypotensive medications, eligible patients were randomiz
179 as an initial therapy or in conjunction with hypotensive medications.
180 eful addition to the armamentarium of ocular hypotensive medications.
181 d likely improve access to prescribed ocular hypotensive medications.
182        Commercially available topical ocular hypotensive medications.
183 SE than those with normal (n=19 770; 90%) or hypotensive (n=274; 1%) BP responses (32% versus 21% ver
184 course of their disease, some of the sickest hypotensive newborns become unresponsive to volume and p
185 (hypotensive, hypothermic), after rewarming (hypotensive, normothermic) just before discontinuation o
186  events (pulmonary hypertensive and systemic hypotensive) occurring within 10 min of protamine admini
187                               No orthostatic hypotensive or hypertensive reactions were observed.
188                 However, SNP did not improve hypotensive PAD after FPI in females and paradoxically c
189 steroid insufficiency should be suspected in hypotensive patients who have responded poorly to fluids
190 ds (sensitivity 100%, specificity 99.3%) and hypotensive patients with blunt abdominal trauma (sensit
191             The study group consisted of 128 hypotensive patients with blunt abdominal trauma who und
192 ation of patients with precordial wounds and hypotensive patients with blunt torso trauma, immediate
193                                           In hypotensive patients, 73% of respondents (108 of 149) re
194  minutes, from 32+/-32 to 20+/-17 minutes in hypotensive patients, and for craniotomy decreased from
195  base deficit) were significantly altered in hypotensive patients.
196          A possible function for this potent hypotensive peptide in the regulation of intraocular pre
197      Adrenomedullin is a recently discovered hypotensive peptide that is expressed in a variety of ce
198    Adrenomedullin (AM) is a newly discovered hypotensive peptide which is believed to play an importa
199                Adrenomedullin (AM), a potent hypotensive peptide, is produced in numerous tissues inc
200                                   Two potent hypotensive peptides, adrenomedullin (AM) and proadrenom
201       Indeed, alpha(1D)-AR KO mice display a hypotensive phenotype and are resistant to salt-induced
202 eta2-AdR expression, possibly leading to the hypotensive phenotype during the rest phase.
203 nage from peripheral tissues and thus with a hypotensive phenotype.
204        Phenylephrine decreased impairment of hypotensive pial artery dilation after fluid percussion
205  in spontaneously breathing normotensive and hypotensive pigs to increase blood pressure and enhance
206 impedance threshold device (-12 cm H(2)O) in hypotensive pigs, systolic blood pressure (mean +/- sem)
207 d pressure profiles validated the beneficial hypotensive properties of these prodrugs.
208                 CD-NP also demonstrates less hypotensive properties when compared with B-type natriur
209 phrine are equally effective in treating the hypotensive pulmonary donor in this rodent model.
210 axis cases (9.2%) and were more common after hypotensive reactions and with pre-existing lung disease
211 reated with a NO synthase inhibitor remained hypotensive relative to the wild type.
212 lly or specifically in endothelial cells are hypotensive, resistant to angiotensin II-induced hyperte
213 eversed the HS-induced pressor response to a hypotensive response (from 121 +/- 3 to 68 +/- 2 mmHg),
214 e depend on the spleen; (d) PAF mediated the hypotensive response and its action in the spleen; and (
215                                         This hypotensive response appears to be due to the inhibition
216 tant finding is that statins exert an ocular hypotensive response in an organ-culture perfusion model
217 ulfate (OSCS) derivative that could elicit a hypotensive response in pigs following a single high-dos
218 tive B(1)R agonist produced a dose-dependent hypotensive response in the knockout mice only.
219 duce vasodilation ex vivo, produces an acute hypotensive response in wild-type mice.
220 reduction of IOP in rabbits, with the ocular hypotensive response lasting for more than 48 hrs.
221 re performed on three patients with systemic hypotensive response pattern on captopril renography.
222 ery stenosis in three patients with systemic hypotensive response pattern on captopril renography.
223                                 The systemic hypotensive response pattern seen on captopril renograph
224 716A (3 mg/kg i.v.), which also inhibits the hypotensive response to anandamide or 2-AG.
225                              The predominant hypotensive response to bolus intravenous injections of
226                 One patient in Group 1 had a hypotensive response to dipyridamole and was exluded fro
227                                          The hypotensive response to isoproterenol, however, is signi
228                            MPD decreased the hypotensive response to levodopa.
229 o lower blood pressure because we observe no hypotensive response to moxonidine in alpha(2A)-AR-null
230 nock-in mouse was substantively deficient in hypotensive response to nitroglycerin compared with wild
231  the IC50 values had no effect on the ocular hypotensive response to PGF2alpha.
232  outflow facility associated with the ocular hypotensive response to the adenosine agonist cyclohexyl
233 uction in blood pressure in all fetuses; the hypotensive response was greatest in fetuses studied nea
234                   In TRPV(1)(+/+) mice, this hypotensive response was preceded by a transient, profou
235 P, while significantly inhibiting the ocular hypotensive response.
236 ography is described that is due to systemic hypotensive response.
237  explainable by an overly exuberant systemic hypotensive response.
238  ADR SPNs (by 2.9+/-0.5 spikes/s; n=19) with hypotensive responses (DeltaMAP=33.2+/-5.3 and 26.4+/-5.
239 ium diet, they show increased but incomplete hypotensive responses to acute treatment an ET(A)-antago
240 ide (NO) in the gracile nucleus modifies the hypotensive responses to electroacupuncture (EA) stimula
241 mpared with placebo, alcohol potentiated the hypotensive responses to LBNP, particularly at -40 mm Hg
242                                (2) HA-evoked hypotensive responses were mediated by PAG neuronal acti
243 d Mg (n = 8) or 4 mL/kg 7.5% NaCl (n = 8) at hypotensive resuscitation and 0.9% NaCl +/- adenosine an
244 enosine and lidocaine, adenocaine) and Mg on hypotensive resuscitation and coagulopathy in the rat mo
245 Hg for 90 minutes, followed by 60 minutes of hypotensive resuscitation and infusion of shed blood.
246                                              Hypotensive resuscitation is gaining clinical acceptance
247                             Normotensive and hypotensive resuscitation to mean arterial pressures of
248                  Fluid volume infused during hypotensive resuscitation was 40% less in the 7.5% NaCl-
249                             At 60 minutes of hypotensive resuscitation, treatment with 7.5% NaCl + ad
250 nction and reduces fluid requirements during hypotensive resuscitation, whereas a second AL infusion
251 pparent when used for either normotensive or hypotensive resuscitation.
252 s to phenylephrine and angiotensin II during hypotensive sepsis in conscious sheep.
253           Administration of clonidine during hypotensive sepsis reduced renal sympathetic nerve activ
254 were measured in fasted healthy (n = 10) and hypotensive septic (n = 6) adults by using a 6-h constan
255 vivo arginine and NO kinetics are lacking in hypotensive septic adults.
256  pressure underestimates central pressure in hypotensive septic patients receiving high-dose vasopres
257 n and the intravascular NO synthesis rate in hypotensive septic patients.
258                                  Conversely, hypotensive Sgk1 KO mice exhibited low WNK1 expression a
259 order characterized by transient episodes of hypotensive shock and anasarca thought to arise from rev
260                                   Refractory hypotensive shock was fatal in 55 of 115 patients treate
261 se ingesting other pesticides and often from hypotensive shock.
262  the prevalence of vasopressin deficiency in hypotensive solid organ donors without clinical evidence
263                Analyzing constituents of the hypotensive Sophora flavescens root, we found that the q
264 ng 60% of the blood volume and maintaining a hypotensive state.
265 essed by applying rapid pulses of hyper- and hypotensive stimuli to the neck via a customised collar.
266                                     During a hypotensive stimulus, cardiovascular homeostasis is main
267  between sexes and menstrual phases during a hypotensive stimulus.
268 s at rest and during a nitroprusside-induced hypotensive stimulus.
269  sensitivity and the sympathetic response to hypotensive stress are attenuated after antecedent hypog
270 -anesthetized rats before, during, and after hypotensive stress elicited by intravenous nitroprusside
271 50488 also attenuated tonic LC activation by hypotensive stress, an effect mediated by CRF afferents.
272 electively engaged during the termination of hypotensive stress.
273 ardiac events (1 myocardial infarction and 1 hypotensive supraventricular tachyarrhythmia), neither o
274                   There was a higher risk of hypotensive symptoms in the combination-therapy group th
275                   Our results indicated that hypotensive systolic noninvasive blood pressure readings
276 SETTING, AND PATIENTS: The 316 patients were hypotensive (systolic blood pressure=81 mm Hg) and tachy
277     While lacking renal actions, CNP is less hypotensive than the cardiac peptides atrial natriuretic
278                      Persistency with ocular hypotensive therapies has been found to be poor.
279 has been observed commonly with other ocular hypotensive therapies in this population.
280 n or of initiation or augmentation of ocular hypotensive therapy by 3 years, after repeated ranibizum
281 ut the potential risks of intensive arterial hypotensive therapy, particularly that administered in t
282  or the initiation or augmentation of ocular hypotensive therapy, through 3 years of follow-up.
283 CT, using BCC to improve adherence to ocular hypotensive therapy, to our knowledge is the first withi
284 of genetically diverse traditional botanical hypotensives, transcending plant genus and human cultura
285 inimizing sensory stimulation and hypoxic or hypotensive transients in stroke and brain injury would
286 rabecular meshwork outflow may be the future hypotensive treatment for glaucoma patients.
287 gical treatment is not feasible then medical hypotensive treatment may be a viable alternative.
288                           Two animals in the hypotensive treatment protocols died during the second a
289 and higher occurrences of deaths only in the hypotensive treatment protocols suggest that resuscitati
290 scontinuation and washout of existing ocular hypotensive treatment, patients who had intraocular pres
291 e significantly higher with normotensive vs. hypotensive treatment.
292 stained IOP elevation or the need for ocular hypotensive treatment.
293 h lactate levels (>10 mmol) with both of the hypotensive treatments and also with the HS90 treatment,
294 s. LR when compared with the normotensive or hypotensive treatments.
295 al levels of SNOs in red blood cells and are hypotensive under anesthesia.
296       Thus, the clinical picture was that of hypotensive ventricular tachycardia (VT).
297 ated over the first 7 days postinjury in the hypotensive versus normotensive patients.
298 ther felt nor visible on the ECG presents as hypotensive VT.
299 ere we report that P311-/- mice are markedly hypotensive with accompanying defects in vascular tone a
300                Septic rats became unwell and hypotensive, with a mortality of 64% at 48 hrs (0% in co

 
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