ライフサイエンスコーパス: hypoventilation

1 losum, congenital heart defects, and central hypoventilation.

2 or control over secretions, airway spasm, or hypoventilation.

3 in a syndrome resembling congenital central hypoventilation.

4 ied within 5 minutes of seizure onset due to hypoventilation.

5 illator settings that would lead to alveolar hypoventilation.

6 iated with impaired diaphragm activation and hypoventilation.

7 nsitivity and life-threatening sleep-related hypoventilation.

8 d to have evolved for proactive avoidance of hypoventilation.

9 contrast two orphan disorders of late-onset hypoventilation.

10 f patients who had both hyperventilation and hypoventilation.

11 sias, and frequent autonomic instability and hypoventilation.

12 r airway obstruction leading to hypoxemia or hypoventilation.

13 ovements, 86% autonomic instability, and 23% hypoventilation.

14 yskinesias, 69 autonomic instability, and 66 hypoventilation.

15 ctal oxygen desaturation is a consequence of hypoventilation.

16 7-41) with captive bolt guns was followed by hypoventilation.

17 ry severity score was 25-32) was followed by hypoventilation.

18 riers may be at risk for developing alveolar hypoventilation.

19 OX2B gene and evidence of nocturnal alveolar hypoventilation.

20 y Ins, and expire shortly after birth due to hypoventilation.

21 eathing and may possibly improve symptoms of hypoventilation.

22 Air breathing in CH rats induced hypoventilation, a 12% increase in ABP, no change in mRA

23 psychiatric symptoms, seizures, and central hypoventilation, a paraneoplastic immune-mediated syndro

24 more, KO mice exhibited transient but marked hypoventilation after acute base loading.

25 ificant postventilator apneas and postapneic hypoventilation also occurred even when end-tidal CO(2)

26 e caudal MR simultaneously produces enhanced hypoventilation and a 51% decrease in the CO(2) response

27 y during sleep could attenuate sleep-related hypoventilation and also negatively impact sleep and cli

28 Central hypoventilation and apnea accompany generalized status e

29 acking AMPK-alpha1 and AMPK-alpha2 exhibited hypoventilation and apnea during hypoxia, with the prima

30 nd thereby aids the clinician in identifying hypoventilation and apnea in the sedated patient at an e

31 deficiencies in AMPK expression precipitate hypoventilation and apnea, even when carotid body affere

32 onset Obesity with Hypothalamic Dysfunction, Hypoventilation and Autonomic Dysregulation (ROHHAD), is

33 rove safety by enabling early recognition of hypoventilation and by reducing the risk of oversedation

34 Ala/+) mutation predisposed pups not only to hypoventilation and central apneas, but also to obstruct

35 eous marriage who presented with respiratory hypoventilation and died 10 days and 4 years later, resp

36 The syndrome is characterized by hypoventilation and inability to regulate breathing to m

37 Carotid body denervation (CBD) causes hypoventilation and increases the arterial PCO2 set-poin

38 ) is characterized by life-threatening sleep hypoventilation and is caused by PHOX2B gene mutations,

39 hibition of the PBel(CGRP) neurons may avoid hypoventilation and minimize EEG arousals.

40 rons accompanies REM sleep and is a cause of hypoventilation and obstructive sleep apnea in humans.

41 adapt and control ventilation to ameliorate hypoventilation and restore normocapnia regardless of th

42 on of TRPM7 in the CB improved sleep-related hypoventilation and that the respiratory effects of CB T

43 m (CNS) leptin levels or activity may induce hypoventilation and the Pickwickian syndrome in some obe

44 emia, which is likely to be a consequence of hypoventilation and ventilation-perfusion mismatching of

45 ted oxygen desaturation was a consequence of hypoventilation and whether factors such as seizure loca

46 onset obesity with hypothalamic dysfunction, hypoventilation, and autonomic dysregulation (ROHHAD) an

47 ent who presented with congenital hypotonia, hypoventilation, and cerebellar histopathological altera

48 The effects of sedation, hypoxia, hypoventilation, and changes in intrathoracic pressure c

49 ilibrium, dysarthria, dysphagia, and central hypoventilation, and died 2 months and 6 months, respect

50 rial carbon dioxide levels (PaCO2), alveolar hypoventilation, and increased cardiorespiratory morbidi

51 lation, leading to hypotension, bradycardia, hypoventilation, and potentially respiratory arrest.

52 erized by weight loss, parkinsonism, central hypoventilation, and psychiatric disturbances.

53 ry stress incontinence, sleep apnea, obesity hypoventilation, and type II diabetes mellitus.

54 ystem leading to sleep-disordered breathing, hypoventilation, and weakness of the expiratory and insp

55 nd present a respiratory phenotype including hypoventilation, apnea, and a diminished ventilatory res

56 hat of the adults, although dysautonomia and hypoventilation are less frequent or severe in children.

57 s, sudden infant death syndrome, and central hypoventilation are relatively frequent conditions in th

58 The mechanisms that link obesity and hypoventilation are unknown, but thought to involve depr

59 ted respiratory acidosis because of alveolar hypoventilation, as well as profound breathing instabili

60 sities and durations of exercise a sustained hypoventilation, as well as reduced systemic pressure an

61 ecreased level of consciousness, and central hypoventilation associated with ovarian teratoma (OT) an

62 alysis of ACSF in or near to the RTN, causes hypoventilation but has no significant effect on the CO(

63 o2 - Paco2 remained essentially stable after hypoventilation but increased significantly after induci

64 and CO2 production, indicating a reversal of hypoventilation by stimulation of central respiratory co

65 Hypoventilation can occur in REM sleep and progress into

66 tributions of airway obstruction and central hypoventilation could not be determined because airway f

67 d (ACSF) in or near to the caudal MR, causes hypoventilation (decrease in the ratio of minute ventila

68 The hypoventilation during induction in the control group an

69 We hypothesize that capnography could detect hypoventilation during induction of bronchoscopic sedati

70 Compared to placebo, significant hypoventilation during the fentanyl trial was indicated

71 We also question whether the transient hypoventilation elicited by CB denervation means that th

72 other comorbidities, including sleep apnea, hypoventilation, gastroesophageal reflux, degenerative j

73 tilation (group "Hyper," n = 4); 48 hours of hypoventilation (group "Hypo," n = 4); 24 hours of hypov

74 re randomized to: starting bronchoscopy when hypoventilation (hypopnea, two successive breaths of at

75 rs and adverse events including hypotension, hypoventilation, ileus, and coma.

76 bserved in 38%, normoventilation in 29%, and hypoventilation in 46%, with a 13% overlap of patients w

77 RATIONALE: Regional hypoventilation in bronchoconstricted patients with asth

78 fy forebrain sites underlying seizure-evoked hypoventilation in humans.

79 carotid bodies improves the obesity-induced hypoventilation in mice.

80 at was normal on MRI correlated with central hypoventilation; in another case, hyperactivity in the c

81 The hypoventilation is attributed to reduced CB afferent act

82 In addition, alveolar hypoventilation is discussed in children with congenital

83 e induction and start bronchoscopy following hypoventilation may decrease hypoxemia without compromis

84 Understanding the cause of this peri-ictal hypoventilation may lead to preventative strategies.

85 sedation and starting bronchoscopy following hypoventilation, may decrease hypoxemia.

86 opnea, whereas reduced ventilatory drive and hypoventilation narrowed the DeltaPET(CO2) and increased

87 Significant hypoventilation occurred during the induction and start

88 The magnitude of hypoventilation on return to sleep was not affected by t

89 hyperventilation followed by more prolonged hypoventilation on return to sleep.

90 d reinstates normal breathing in the case of hypoventilation or apnea.

91 target range in 91% of cases and because of hypoventilation or hyperventilation in 9%.

92 al recommendations were available in case of hypoventilation or hyperventilation.

93 several hours is necessary for recurrence of hypoventilation or other complications.

94 all three rat strains, CBD elicited eupnoeic hypoventilation (PaCO2 +8.7-11.0 mmHg) 1-2 days post-CBD

95 henylalanine (PCPA) mimicked seizure-induced hypoventilation, partially occluded the postictal decrea

96 After 60 mins of hypoventilation, Pico2 - Paco2 decreased to 14.2+/-1.1 a

97 roup "Hypo-Baseline," n = 4); or 24 hours of hypoventilation plus 24 hours of hypoventilation plus EC

98 ntilation (group "Hypo," n = 4); 24 hours of hypoventilation plus 24 hours of normoventilation (group

99 24 hours of hypoventilation plus 24 hours of hypoventilation plus ECCO(2)R (group "Hypo-ECCO(2)R," n

100 ssive parenchymal derecruitment and alveolar hypoventilation, potentially aggravating systemic hyperc

101 Hypoventilation preceded OAAS < 4 by 96.5 +/- 88.1 secon

102 omboembolism, preoperative vena cava filter, hypoventilation, pulmonary hypertension), and age>or=45

103 geal amyloid included radiculopathy, central hypoventilation, recurrent subarachnoid haemorrhage, dep

104 ened with hyperventilation and narrowed with hypoventilation, regardless of the stimulus and whether

105 We report a patient manifesting severe hypoventilation resulting from diaphragmatic paresis sec

106 however, she became dyspneic and manifested hypoventilation secondary to muscle weakness.

107 ive noninvasive strategies for management of hypoventilation, sleep-disordered breathing, and cough i

108 ts suffering from this syndrome, therapeutic hypoventilation strategy designed to reduce mechanical d

109 f the terminal bowel) and congenital central hypoventilation syndrome (CCHS) (also known as NB-HSCR-C

110 ysregulation (ROHHAD) and congenital central hypoventilation syndrome (CCHS) are distinct in presenta

111 g exercise, children with congenital central hypoventilation syndrome (CCHS) demonstrate coupling of

112 Rationale: Congenital central hypoventilation syndrome (CCHS) is a rare autonomic diso

113 Rationale: Congenital central hypoventilation syndrome (CCHS) is characterized by life

114 The cause of congenital central hypoventilation syndrome (CCHS) is unknown, but a geneti

115 Congenital Central Hypoventilation Syndrome (CCHS) patients exhibit comprom

116 Congenital central hypoventilation syndrome (CCHS) typically presents in th

117 stem development, lead to congenital central hypoventilation syndrome (CCHS), a neurodevelopmental di

118 ble for the occurrence of Congenital Central Hypoventilation Syndrome (CCHS), a rare neurological dis

119 Like patients with congenital central hypoventilation syndrome (CCHS), our patient had a relat

120 en found in patients with congenital central hypoventilation syndrome (CCHS), the cardinal feature of

121 A) in awake children with congenital central hypoventilation syndrome (CCHS), who have absent or near

122 ption factor Phox2b cause congenital central hypoventilation syndrome (CCHS).

123 cluding neuroblastoma and congenital central hypoventilation syndrome (CCHS).

124 which are associated with congenital central hypoventilation syndrome (CCHS).

125 enital breathing disorder congenital central hypoventilation syndrome (CCHS).

126 utations, which cause the central congenital hypoventilation syndrome (CCHS, also known as Ondine's c

127 Rationale: Obesity hypoventilation syndrome (OHS) has been associated with

128 cy of NIV during similar episodes in obesity hypoventilation syndrome (OHS).

129 tion and management of patients with obesity hypoventilation syndrome (OHS).Methods: A multidisciplin

130 ents aged 15-80 years with untreated obesity hypoventilation syndrome and an apnoea-hypopnoea index o

131 In stable patients with obesity hypoventilation syndrome and severe obstructive sleep ap

132 mice provide a model for congenital central hypoventilation syndrome and suggest that Pbx3 mutations

133 Central congenital hypoventilation syndrome is caused by mutations of the g

134 Obesity hypoventilation syndrome is commonly treated with contin

135 verlap syndrome) and morbid obesity (obesity hypoventilation syndrome) increases the odds of mortalit

136 with insomnia, excessive sleepiness, obesity hypoventilation syndrome, and chronic obstructive pulmon

137 AD with disorders outside congenital central hypoventilation syndrome, further advancement will be ma

138 iscussed in children with congenital central hypoventilation syndrome, myelomeningocele, and Prader-W

139 Patients with obesity hypoventilation syndrome, previous bariatric surgery, co

140 1, Mowat-Wilson syndrome, congenital central hypoventilation syndrome, Shah-Waardenburg syndrome and

141 Additional comorbidities included obesity hypoventilation syndrome, sleep apnea syndrome, hyperten

142 ion, diabetes mellitus, sleep apnea, obesity hypoventilation syndrome, venous stasis ulcers, intestin

143 oreflex that characterize congenital central hypoventilation syndrome.

144 obably contributes to the congenital central hypoventilation syndrome.

145 including sleep apnea syndromes and obesity hypoventilation syndrome.

146 in children affected with congenital central hypoventilation syndrome.

147 rmal men are more vulnerable to load-induced hypoventilation than women, due to increased upper airwa

148 The alveolar hypoventilation that occurred during insufflation with v

149 2/6 hypertension, and all with sleep apnea, hypoventilation, type II diabetes mellitus, and urinary

150 Regional hypoperfusion but not hypoventilation typifies lung gas exchange in HF.

151 ze abnormalities, deafness, facial weakness, hypoventilation, vascular malformations of the internal

152 ng permissive hypercapnia, due to controlled hypoventilation, warrants investigation.

153 mpared to CTRL exercise (P < 0.001) and this hypoventilation was accompanied by an up to 10% lower ar

154 ndent outcome effect of hyperventilation and hypoventilation was assessed.

155 Hypoventilation was observed in 74.6% of the patients be

156 Both hyperventilation and hypoventilation were associated with worse outcomes in i

157 All 8 animals demonstrated central apnea and hypoventilation, which resulted in the death of 1 and co

158 e involvement of large contiguous regions of hypoventilation with substantial subresolution intraregi

159 insonism, depression, severe weight loss and hypoventilation, with brain pathology characterized by T