ライフサイエンスコーパス: hypoxemia

1 tic microembolism as the underlying cause of hypoxemia.

2 piratory system compliance and a significant hypoxemia.

3 nal endothelial cell injury) associated with hypoxemia.

4 copy following hypoventilation, may decrease hypoxemia.

5 ems accurate, especially for moderate-severe hypoxemia.

6 aryngoscopy (apneic oxygenation) may prevent hypoxemia.

7 d for oxygen and is especially vulnerable to hypoxemia.

8 cardia did not alter the prognostic value of hypoxemia.

9 mes were similarly increased after prolonged hypoxemia.

10 to safety process measures, and frequency of hypoxemia.

11 dent sympathetic activation and/or degree of hypoxemia.

12 ochemical indicators of vitamin A status and hypoxemia.

13 atients is frequently associated with severe hypoxemia.

14 fetus as growth restriction and progressive hypoxemia.

15 la may offer an alternative in patients with hypoxemia.

16 including the management of HPS with severe hypoxemia.

17 are similar to those of SCD mice but without hypoxemia.

18 tely and died by 4 months of age with severe hypoxemia.

19 he venular endothelium to the same extent as hypoxemia.

20 e control group received HFOV for refractory hypoxemia.

21 ble coughing, urticaria, edema, wheezing and hypoxemia.

22 ry disease (COPD) and chronic severe daytime hypoxemia.

23 in patients with severe ARDS and refractory hypoxemia.

24 e development of life-threatening refractory hypoxemia.

25 ked by impaired gas exchange and significant hypoxemia.

26 features were fever, cough, rhinorrhea, and hypoxemia.

27 s mismatching is an important determinant of hypoxemia.

28 consumption, they may decrease postoperative hypoxemia.

29 s such as emphysema, chronic bronchitis, and hypoxemia.

30 (39%) versus 38 (40%) exhibiting reversal of hypoxemia.

31 ion or recruitment of post-I activity during hypoxemia.

32 ccount for most, if not all, cases of silent hypoxemia.

33 cs are augmented with both acute and chronic hypoxemia.

34 critical illness severity and to preexisting hypoxemia.

35 ons to improve oxygenation in BD donors with hypoxemia.

36 lity of the hippocampus to brief episodes of hypoxemia.

37 ble to improve oxygenation in BD donors with hypoxemia.

38 associated with reduced lung compliance and hypoxemia.

39 may be afforded to those with HPS and severe hypoxemia.

40 ts was associated with a lower prevalence of hypoxemia (10.1% vs. 17.4%, p = .022) and a lower preval

41 sk group experienced more episodes of severe hypoxemia (2% vs 14%, p = 0.03).

42 uality evidence) with severe chronic resting hypoxemia, 2) a conditional recommendation against long-

43 in the presence of respiratory distress and hypoxemia (2C), use of physical examination therapeutic

44 ents with COPD with moderate chronic resting hypoxemia, 3) conditional recommendations for ambulatory

45 gnificantly higher in those with reversal of hypoxemia (32/69 versus 2/111, P < 0.001).

46 1.40-5.54]), tachycardia (2.99 [1.48-6.06]), hypoxemia (4.40 [2.03-9.51]), and inability to stand (3.

47 Although all cases presented with hypoxemia, 4 had respiratory copathogens or concomitant

48 low-quality evidence) with severe exertional hypoxemia, 4) a conditional recommendation for ambulator

49 e likely than those with normal CXRs to have hypoxemia (45% vs 26%), crackles (69% vs 62%), tachypnea

50 tly different, including fewer patients with hypoxemia (5.0% in the high level of PEEP group vs 13.6%

51 ommon intubation-related adverse events were hypoxemia (55 of 610 patients [9.0%]) and hypotension (3

52 patients [6.4%]) in the rocuronium group and hypoxemia (61 of 616 [9.9%]) and hypotension (62 of 616

53 transfusion, higher fluid balance, obesity, hypoxemia, acidosis, tobacco use, emergent hematoma evac

54 ates neurogenic pulmonary edema and reverses hypoxemia after brain death.

55 Randomized clinical trial of patients with hypoxemia after cardiac surgery at a single ICU in Brazi

56 Among patients with hypoxemia after cardiac surgery, the use of an intensive

57 bation and improve outcomes of patients with hypoxemia after cardiothoracic surgery.

58 ere 67% and 52% (P = .02), and for recurrent hypoxemia after oxygen supplementation were 38% and 18%

59 The hypoxemia after sildenafil administration in group B imp

60 ium further than either hemorrhagic shock or hypoxemia alone did.

61 Hypoxemia also triggered leukocytes adhesion to the venu

62 Nocturnal hypoxemia, an important pathophysiological feature of OS

63 t the immediate implementation of permissive hypoxemia and a comprehensive evaluation of its value in

64 To examine whether patients with acute hypoxemia and bilateral opacities treated with high-flow

65 association between markers of sleep-related hypoxemia and brain anatomy.

66 tients, MDK levels correlated with nocturnal hypoxemia and CM mitotic rate.

67 n is influenced by the severity of nocturnal hypoxemia and comorbid obesity has not been determined.

68 of dyspnea and fatigue, partially related to hypoxemia and compromised oxygen delivery.

69 ular, the dissociation between the degree of hypoxemia and Crs was characterized as a distinct ARDS p

70 dema) in the alveolar airspaces and leads to hypoxemia and death if not corrected.

71 Hypoxemia and enhanced ventilatory demands result, altho

72 vel to altitude, but this may expose them to hypoxemia and exacerbation of sleep apnea.

73 The association of hypoxemia and hemorrhagic shock did not amplify leukocyt

74 The association of hypoxemia and hemorrhagic shock did not further amplify

75 perglycemia and hypercarbia but inhibited by hypoxemia and hypoglycemia; hypocarbia had no appreciabl

76 These effects, as well as hypoxemia and hypotension, were prevented by 3-aminobenz

77 tein-rich pulmonary edema that causes severe hypoxemia and impaired carbon dioxide excretion.

78 atous optic neuropathy by creating transient hypoxemia and increasing vascular resistance.

79 ng transplant and is characterized by severe hypoxemia and infiltrates in the allograft.

80 cantly associated with the level of systemic hypoxemia and metabolic stress regardless of etiology.

81 ificantly associated with the level of fetal hypoxemia and metabolic stress.

82 Severe hypoxemia and overall immediate intubation-related compl

83 is associated with a decreased prevalence of hypoxemia and reduced rate of procedure-related complica

84 physiologic factors, such as propensity for hypoxemia and respiratory arousability.

85 l life support can lead to rapid reversal of hypoxemia and shock; however, it can also result in vary

86 thin and between patients, such as degree of hypoxemia and sleep fragmentation, that reflect differen

87 long-term survival relative to the degree of hypoxemia and the era in which LT was conducted.

88 he severity of lung injury and the degree of hypoxemia and/or the effects of ventilator settings on g

89 Refractory hypoxemia and/or uncompensated hypercapnia despite optim

90 a, and hypomagnesemia; 14 patients (52%) had hypoxemia, and 9 (33%) had oliguria, of whom 5 had anuri

91 We assessed Crs, degree of hypoxemia, and associated Crs-based phenotypic patterns

92 beneficial, such as pulmonary hypertension, hypoxemia, and cystic fibrosis.

93 rapy were associated significantly with LRD, hypoxemia, and death.

94 o mobilization were hemodynamic instability, hypoxemia, and dependency on venovenous extracorporeal m

95 ding hyperglycemia/hypoglycemia, hyperoxemia/hypoxemia, and hypercarbia/hypocarbia - conditions commo

96 ermeability pulmonary edema, severe arterial hypoxemia, and impaired carbon dioxide excretion.

97 rs appear to be exaggerated polycythemia and hypoxemia, and lower and sluggish CBF compared to CMS pa

98 1 had lower respiratory tract disease (LRD), hypoxemia, and prolonged viral shedding compared with se

99 infected mice from weight loss, hypothermia, hypoxemia, and respiratory compromise.

100 IST had a lower proportion of comorbidities, hypoxemia, and viral detection and had more intense syst

101 ions are associated with SARS-CoV-2 viremia, hypoxemia, and worse outcome.

102 hypoxemia, or need for rescue therapies for hypoxemia; and days with use of vasopressors or sedation

103 >/=90% [moderate hypoxemia] or <90% [severe hypoxemia]) and 13 obese control subjects.

104 Severe hypoxemia (aOR 4.25, 95%CI 2.36-7.64), leukocytosis (aOR

105 ry end points were the occurrences of severe hypoxemia, apnea, and oxygen supplementation.

106 Obstructive sleep apnea (OSA) and nocturnal hypoxemia are associated with chronic kidney disease and

107 ce rate of ICU-acquired infection and severe hypoxemia are expected outcomes from RBC transfusion tha

108 determine the effect of postoperative IS on hypoxemia, arterial oxygen saturation (Sao2) level, and

109 ntrol of arterial oxygenation and permissive hypoxemia as candidate management strategies in hypoxemi

110 in subsets of patients with mild and severe hypoxemia as defined by PaO(2)/Fio(2).

111 recruitment of post-I activity during acute hypoxemia as predicted.

112 harm will aid the development of permissive hypoxemia as viable clinical strategy.

113 in the bag-mask ventilation group had severe hypoxemia, as compared with 45 patients (22.8%) in the n

114 te the same severity of illness and level of hypoxemia at admission, coronavirus disease 2019 patient

115 acute myocardial infarction who do not have hypoxemia at baseline is uncertain.

116 More than 40% had hypoxemia at rest (13%) or on exercise (29%), with pulmo

117 rtality, ICU-acquired infections, and severe hypoxemia) at day 30, we used marginal structural models

118 y deserve preference in patients with marked hypoxemia before endotracheal intubation.

119 yndrome in acute liver failure are scant and hypoxemia being a commonly encountered systemic complica

120 nt differences in frequency of postoperative hypoxemia between the control and test groups were found

121 horacentesis may be complicated by transient hypoxemia, bleeding, patient discomfort, reexpansion pul

122 iated with detectable SARS-CoV-2 viremia and hypoxemia (both P<0.001).

123 suggest that even a single brief episode of hypoxemia can markedly disrupt hippocampal maturation.

124 rtality, which was accompanied by attenuated hypoxemia, cardiopulmonary dysfunction, and pulmonary ed

125 e the clearance of Abeta from the brain, and hypoxemia characteristic of sleep-disordered breathing i

126 ardia, tachypnea, hypotension, and prolonged hypoxemia compared with controls.

127 oxygenation and reduced prevalence of severe hypoxemia compared with nonrebreathing bag reservoir fac

128 se to AngII was less in patients with severe hypoxemia compared with those with moderate hypoxemia (P

129 agic telangiectasia: iron deficiency impairs hypoxemia compensations by restricting erythropoiesis an

130 During hemorrhagic shock, the occurrence of hypoxemia considerably alters villous intestinal perfusi

131 The impact of hypoxemia correction on liver disease severity warrants

132 Higher levels of hypoxemia correlated with increased volume and thickness

133 Hypoxemia decreased RBCs velocity in intestinal villi bu

134 upplemental oxygen for patients with resting hypoxemia (defined as Spo2 <89%) improves survival.

135 econdary outcome was the incidence of severe hypoxemia, defined as an oxygen saturation of less than

136 The primary outcome was frequency of hypoxemia, defined as an Sao2 level of less than 92% wit

137 ectin-1:mIgG1 and Dectin-1:mIgG2a Fc reduced hypoxemia despite minimal effects on fungal burden in th

138 Hypoxemia developed in 62 patients (1.9%) in the oxygen

139 Hypoxemia developed in 69% of patients in the blinded ar

140 which are relevant for the understanding of hypoxemia development during extracorporeal CO2 removal.

141 that trigger white matter injury, transient hypoxemia disrupted SPN arborization and functional matu

142 nical syndrome characterized by a refractory hypoxemia due to an inflammatory and high permeability p

143 rs in sepsis patients with severe refractory hypoxemia due to ARDS (2C); prone positioning in sepsis-

144 ve sleep apnea is primarily characterized by hypoxemia due to frequent apneic episodes and fragmentat

145 of advanced liver disease, characterized by hypoxemia due to intrapulmonary vascular dilatations.

146 cted at limiting the duration or severity of hypoxemia during brain development may mitigate disturba

147 cted at limiting the duration or severity of hypoxemia during brain development may mitigate disturba

148 warning system for hypoxemia, would decrease hypoxemia during endoscopy.

149 r data suggest that the main determinants of hypoxemia during endotracheal intubation may be related

150 = 0.03) and subjects with any two events of hypoxemia during sedation, maintenance or recovery were

151 ge in body mass index, severity of nocturnal hypoxemia, E/E', and sex were independently associated w

152 hyperalgesia in volunteers, while nocturnal hypoxemia enhanced morphine analgesic potency in childre

153 Mean percentages of recorded time with hypoxemia for the least and most affected 10% of infants

154 hanisms, physicians caring for patients with hypoxemia free of dyspnea are operating in the dark, pla

155 As hypoxemia frequently occurs simultaneously with hemorrha

156 Mice were randomly assigned to hypoxemia group in which we decreased inspired oxygen fr

157 or height, unresponsiveness, deep breathing, hypoxemia, grunting, and the absence of cough.

158 Transplant recipients with more severe hypoxemia had increased risk of death after liver transp

159 that high-altitude adaptation in response to hypoxemia has different underlying mechanisms between me

160 ; 95% CI, 2.33-3.28; p < 0.01) and of severe hypoxemia (hazard ratio, 1.29; 95% CI, 1.14-1.47; p < 0.

161 , since he developed progressive dyspnea and hypoxemia, he was admitted to our hospital.

162 Obstructive sleep apnea causes intermittent hypoxemia, hemodynamic fluctuations, and sleep fragmenta

163 he primary outcome was the total duration of hypoxemia (hemoglobin oxygen saturation [Spo2] <90%) per

164 ressure level of 40 mm Hg during 30 minutes, hypoxemia-hemorrhagic shock group in which PaO2 was decr

165 opped in a synergistic manner (69% +/- 3% in hypoxemia-hemorrhagic shock group vs 94 +/- 2 in hemorrh

166 in total lung capacity, resulting in chronic hypoxemia, hypercapnia, and increased erythropoietin syn

167 e high risk for death associated with severe hypoxemia, hypercapnia, or both not responding to maximi

168 e high risk for death associated with severe hypoxemia, hypercapnia, or both not responding to maximi

169 physiologically detrimental conditions (e.g. hypoxemia, hypercarbia, acidosis, hypothermia, hypervole

170 Every blood gas derangement (hypoxemia, hyperoxemia, hypocapnia, hypercapnia, and aci

171 ery are reviewed, relative to the threats of hypoxemia, hyperoxia and mechanical lung injury.

172 The management of hypoxemia in critically ill patients is challenging.

173 vel related strategies for the management of hypoxemia in critically ill patients.

174 ation-perfusion mismatch is a major cause of hypoxemia in cystic fibrosis.

175 on (PAH) is commonly associated with chronic hypoxemia in disorders such as chronic obstructive pulmo

176 ve signals all noninvasively-measured during hypoxemia in healthy volunteers include four signals mea

177 ere selected that explored the physiology of hypoxemia in healthy volunteers or critically ill patien

178 iation is related to the degree of nocturnal hypoxemia in OSA.

179 corporeal membrane oxygenation) to alleviate hypoxemia in patients unable to maintain reasonable oxyg

180 oes not appear to lead to increased rates of hypoxemia in patients undergoing ambulatory upper endosc

181 med on these 25 subjects to examine cerebral hypoxemia in specific regions (periventricular white mat

182 Nocturnal hypoxemia in subjects at high risk for OSA was associate

183 ric results, whereas there was more profound hypoxemia in the PH group.

184 entrations; each 10% increase in duration of hypoxemia increased BNP by 9.6% (95% confidence interval

185 using the apnea-hypopnea index (AHI) and the hypoxemia index (percent sleep time below 90% oxyhemoglo

186 For categories of increasing severity of the hypoxemia index, the corresponding relative hazards were

187 The severity of nocturnal hypoxemia influences the magnitude of renal, but not the

188 inority of patients with ARDS and refractory hypoxemia, institution of these therapies may be conside

189 Permissive hypoxemia is a concept that is untested clinically and r

190 Hypoxemia is a feared complication of acute liver failur

191 Hypoxemia is a risk factor for long-term neuropsychologi

192 Arterial hypoxemia is associated with cerebral and retinal venous

193 Hypoxemia is common during endotracheal intubation of cr

194 Hypoxemia is common in diabetes, and reflex responses to

195 Opioid-induced ventilatory depression and hypoxemia is common, severe, and often unrecognized in p

196 are consistent with an injury model in which hypoxemia is exacerbated by endotoxin-mediated failure o

197 Worsening of hypoxemia is frequent during low-flow extracorporeal CO2

198 Hypoxemia is mainly due to intrapulmonary shunt, whereas

199 Hypoxemia is the most common complication during trachea

200 Severe hypoxemia is the most common serious adverse event durin

201 Persistent hypoxemia is the principal reason lungs from otherwise e

202 The aim of permissive hypoxemia is to minimize the possible harms caused by re

203 apy for the management of isolated nocturnal hypoxemia is uncertain.

204 current arousals from sleep and intermittent hypoxemia) is common among older adults.

205 ppy hypoxia but more precisely termed silent hypoxemia-is especially bewildering to physicians and is

206 clude species that tolerate acute or chronic hypoxemia like deep-diving mammals and high-altitude inh

207 d in reduced carotid arterial O2 saturation (hypoxemia), lung pathology, pulmonary edema, reduced lun

208 Lipoplexes induced no weight loss, hypoxemia, lung dysfunction, pulmonary edema, or pulmona

209 fits of attempting to fully reverse arterial hypoxemia may be outweighed by the harms associated with

210 Their vulnerability to hypoxemia may contribute to behavioral phenotype and cog

211 Hypoxemia may contribute to long-term working memory dis

212 Since intermittent hypoxemia may underlie cardiovascular sequelae of sleep

213 r other interventions targeted at mitigating hypoxemia, may be inadequately appreciated.

214 ison of HFNC and helmet NIV in patients with hypoxemia.Methods: Fifteen patients with hypoxemia with

215 lusive categories of ARDS based on degree of hypoxemia: mild (200 mm Hg < PaO2/FIO2 </= 300 mm Hg)

216 The severe hypoxemia, moderate volume loss, and perfusion patterns

217 sk intolerance (n = 11, 30%), and refractory hypoxemia (n = 1, 2.7%).

218 ratory distress syndrome use some measure of hypoxemia, neither the Berlin definition nor recently pr

219 rotective factor of the occurrence of severe hypoxemia (odds ratio, 0.146; 95% CI, 0.01-0.90; p = 0.0

220 -venous extracorporeal membrane oxygenation, hypoxemia (odds ratio, 1.68; 95% CI, 1.09-2.57) and mode

221 improve survival among patients with severe hypoxemia on conventional mechanical ventilation.

222 dex, higher d-dimer, and greater severity of hypoxemia on ICU admission.

223 ympathetic activation and a direct effect of hypoxemia on the myocardium have been proposed, the latt

224 preterm infants may experience intermittent hypoxemia or bradycardia for many weeks after birth.

225 n to intestinal venules compared with either hypoxemia or hemorrhagic shock alone.

226 rt is increasingly being deployed for severe hypoxemia or hypercapnic acidosis refractory to conventi

227 transient worsening of mismatch by episodic hypoxemia or hypotension also reproducibly triggers PIDs

228 depression or airway obstruction leading to hypoxemia or hypoventilation.

229 anagement of isolated fast breathing without hypoxemia or other clinical signs of illness in term you

230 the routine management in case of persistent hypoxemia or respiratory deterioration.

231 erienced significant metabolic derangements, hypoxemia, or exposure to sedating medications that may

232 ia, pneumothorax, severe atelectasis, severe hypoxemia, or need for rescue therapies for hypoxemia; a

233 tatus (mean nocturnal SaO2, >/=90% [moderate hypoxemia] or <90% [severe hypoxemia]) and 13 obese cont

234 icantly reduce the duration of postoperative hypoxemia over 48 hours.

235 were intraoperative complications, including hypoxemia (oxygen desaturation with Spo2 <=92% for >1 mi

236 The presence of moderate hypoxemia (oxygen saturation </=96%; LR, 2.8 [95% CI, 2.

237 , however, depending on baseline severity of hypoxemia (P = 0.0003), with harm increasing with PaO2/F

238 hypoxemia compared with those with moderate hypoxemia (P = 0.001) and obese control subjects after 3

239 showed low Dl(CO) (30% [12-79%]) and severe hypoxemia (Pa(O(2)) 56 [38-99] mm Hg).

240 n patients with COPD who have severe resting hypoxemia (Pao(2) </=55 mm Hg or Spo(2) </=88%) (Grade:

241 The relationships between hypoxemia (PaO2 < 60mm Hg), normoxia (PaO2 60-100mm Hg),

242 ated patients admitted to the ICU with acute hypoxemia (PaO2/FIO2 </= 300) and bilateral opacities.

243 44 [54%] vs 135 [72%]; p=0.006), more severe hypoxemia (PaO2/FIO2: 165+/-73 mm Hg vs 199+/-79 mm Hg;

244 eing considered for allocation but exhibited hypoxemia (partial pressure of oxygen in arterial blood

245 Severe hypoxemia percentages in the blinded and open arms were

246 bariatric surgery, the severity of nocturnal hypoxemia predicted Angptl4 levels in subcutaneous adipo

247 CI, 1.13-4.56), but not obstructive apnea or hypoxemia, predicted incident atrial fibrillation.

248 Supplemental oxygen in patients with resting hypoxemia prolongs life, and other advanced treatments a

249 Hypoxemia, pulmonary edema, and levels of BALF alveolar

250 Episodes of hypoxemia (pulse oximeter oxygen saturation <80%) or bra

251 CPAP corrected OSA and hypoxemia (RDI: 42 +/- 4 vs. 4 +/- 1 h(-1), P < 0.001; d

252 ct on the occurrence rate of peri-intubation hypoxemia (relative risk, 0.98; 95% CI, 0.68-1.42; 0.3%

253 unit patients) were characterized by severe hypoxemia (requiring high levels of inspired oxygen and

254 Tachypnea and hypoxemia resolved faster in older children (P = 0.0001)

255 re indicated in cases of fluid retention and hypoxemia, respectively.

256 opulmonary resuscitation results in profound hypoxemia, respiratory acidosis, and significantly worse

257 eatening situations associated with profound hypoxemia, respiratory acidosis, or hemodynamic compromi

258 markers of obstructive sleep apnea severity (hypoxemia, respiratory disturbances, and sleep fragmenta

259 ertrophy and thickening were associated with hypoxemia, respiratory disturbances, and sleep fragmenta

260 on to lower respiratory tract disease (LRD), hypoxemia, respiratory failure, and overall and influenz

261 After WLS, there was rapid onset of profound hypoxemia resulting in acute pulmonary hypertension and

262 s of overwhelming pulmonary inflammation and hypoxemia, resulting in high morbidity and mortality.

263 Intriguingly, postnatal hypoxemia, ROS scavenging, or inhibition of DDR all prol

264 bance index [RDI] > 15 h(-1)) with nocturnal hypoxemia (SaO2 < 90% for >12% of the night) were studie

265 not demonstrate any effect on postoperative hypoxemia, Sao2 level, or postoperative pulmonary compli

266 The primary end point was the occurrence of hypoxemia; secondary end points were the occurrences of

267 lure Assessment, duration of coma, delirium, hypoxemia, sepsis, education level, hospital type, insur

268 erfusion mismatch in the model, the reported hypoxemia severity in early COVID-19 patients is not rep

269 OSA) were higher in OSA and correlated with hypoxemia severity.

270 ortive care, in particular the correction of hypoxemia, should be evaluated in clinical trials to add

271 adherence, major complications, and rates of hypoxemia (SpO2 <90%).

272 25 h(-1)), stratified according to nocturnal hypoxemia status (mean nocturnal SaO2, >/=90% [moderate

273 management) were similar in mild and severe hypoxemia subsets as defined by PaO(2)/Fio(2) ratios.

274 ients with critical illness characterized by hypoxemia such as acute respiratory distress syndrome.

275 saturations and a lower incidence of severe hypoxemia than those receiving no ventilation.

276 However, the burden of hypoxemia (the time spent with oxygen saturation <90%) s

277 In patients with moderate-to-severe hypoxemia, the expired tidal volume above 9.5 mL/kg pred

278 Secondly, we describe permissive hypoxemia: the acceptance of levels of arterial oxygenat

279 Some measures of hypoxemia to guide 'need' for transfusion have potential

280 o evaluate adherence to process measures and hypoxemia trends over time.

281 Finally, we chemically induce hypoxemia via hemolytic anemia resulting in HIF stabiliz

282 nsing is essential to avoid life-threatening hypoxemia via hypoxic pulmonary vasoconstriction (HPV) w

283 Occurrence of severe hypoxemia was 20.6% vs 17.6% (risk ratio, 1.17; 95% CI,

284 However, reversal of hypoxemia was a powerful predictor of lung utilization r

285 rease in Kupffer cell phagocytosis following hypoxemia was also prevented by Wortmannin or YC-1 treat

286 When hypoxemia was associated to hemorrhagic shock, it decrea

287 Hypoxemia was associated with an increased mortality in

288 Worsening hypoxemia was associated with initiation of rescue thera

289 In vivo, hypoxemia was more severe in Cx40-/- mice than in wild-t

290 Severity of hypoxemia was not associated with the blood pressure or

291 ected myocardial infarction who did not have hypoxemia was not found to reduce 1-year all-cause morta

292 respiratory event index >=15, and nocturnal hypoxemia was quantified as percent sleep time with <90%

293 s 8 days (interquartile range, 5 to 10), and hypoxemia was the most frequent severity criterion for e

294 ower respiratory tract infection with severe hypoxemia were 0.5% and 1.0% (vaccine efficacy, 48.3%; 9

295 ired vasodilation and oxygen delivery during hypoxemia with advancing age.

296 ith hypoxemia.Methods: Fifteen patients with hypoxemia with Pa(O(2))/Fi(O(2)) < 200 mm Hg received he

297 scopy following hypoventilation may decrease hypoxemia without compromising patient tolerance.

298 intubation of critically ill adults prevents hypoxemia without increasing the risk of aspiration rema

299 Thus, an effective and rapid treatment for hypoxemia would be revolutionary.

300 n to function as an early warning system for hypoxemia, would decrease hypoxemia during endoscopy.