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1 n plasma; and (d) a novel empirical model of hypoxic pulmonary vasoconstriction.
2 icates pulmonary hypertension and attenuated hypoxic pulmonary vasoconstriction.
3 elium-dependent vasodilatation, and enhanced hypoxic pulmonary vasoconstriction.
4 er increases in [Ca(2+)](i) that cause acute hypoxic pulmonary vasoconstriction.
5 exacerbated by endotoxin-mediated failure of hypoxic pulmonary vasoconstriction.
6 l, to the pulmonary selectivity required for hypoxic pulmonary vasoconstriction.
7 ity in acute hypoxia, consistent with uneven hypoxic pulmonary vasoconstriction.
8 lar oxygen signal responsible for triggering hypoxic pulmonary vasoconstriction.
9 ponsible for the modulatory effects of pH on hypoxic pulmonary vasoconstriction.
10  arterial resistance is most consistent with hypoxic pulmonary vasoconstriction.
11 ient mice, sildenafil markedly blunted acute hypoxic pulmonary vasoconstriction.
12 tes for the K(+) channel isoforms initiating hypoxic pulmonary vasoconstriction.
13  receptor activity of epinephrine attenuated hypoxic pulmonary vasoconstriction.
14 of a combination of reduced blood volume and hypoxic pulmonary vasoconstriction.
15 emia may be explained in part by the loss of hypoxic pulmonary vasoconstriction.
16 fusion matching governed by the mechanism of hypoxic pulmonary vasoconstriction.
17 ays an influential role in the modulation of hypoxic pulmonary vasoconstriction.
18  decreases in Pao2/Fio2 ratio, and a loss of hypoxic pulmonary vasoconstriction (48 hrs, -90.5% vs. b
19                                              Hypoxic pulmonary vasoconstriction, a protective mechani
20 nce hypoxia, the functional response, termed hypoxic pulmonary vasoconstriction, activates a multitud
21  not understood how the integrated action of hypoxic pulmonary vasoconstriction affects oxygen transp
22                     In Group "E+OA" (n = 5), hypoxic pulmonary vasoconstriction after ALI was blocked
23                             We conclude that hypoxic pulmonary vasoconstriction and a profound catech
24          We studied the effects of sepsis on hypoxic pulmonary vasoconstriction and attempted to dete
25 that pergolide might have similar effects on hypoxic pulmonary vasoconstriction and Kv channels.
26      Therapy with supplemental oxygen alters hypoxic pulmonary vasoconstriction and modulates the Hal
27 mitochondrial metabolism by hypoxia to acute hypoxic pulmonary vasoconstriction and progression of pu
28 therapeutic option to counteract the loss of hypoxic pulmonary vasoconstriction and redistribute bloo
29                                              Hypoxic pulmonary vasoconstriction and responses to exog
30 by HbFe(II)NO-bearing RBCs may contribute to hypoxic pulmonary vasoconstriction and the rebound effec
31           The cellular mechanisms underlying hypoxic pulmonary vasoconstriction are not fully underst
32         AMPK has been reported to facilitate hypoxic pulmonary vasoconstriction but, paradoxically, i
33 ondria function as the O2 sensors underlying hypoxic pulmonary vasoconstriction by releasing reactive
34  In summary, at 3800 m both hypovolaemia and hypoxic pulmonary vasoconstriction contribute to the dec
35 lmonary microvessels and is the mechanism of hypoxic pulmonary vasoconstriction failure associated wi
36 provides evidence for a severe impairment of hypoxic pulmonary vasoconstriction following combined bu
37                                       Uneven hypoxic pulmonary vasoconstriction has been proposed to
38 iction matches perfusion to ventilation; (2) hypoxic pulmonary vasoconstriction homogenizes regional
39                                              Hypoxic pulmonary vasoconstriction (HPV) acts to optimis
40 sponsible for sensing hypoxia and initiating hypoxic pulmonary vasoconstriction (HPV) are unclear.
41  hemoglobin (Hb) and red blood cells augment hypoxic pulmonary vasoconstriction (HPV) by scavenging n
42                                              Hypoxic pulmonary vasoconstriction (HPV) directs blood f
43 lar analogue methazolamide prevent or reduce hypoxic pulmonary vasoconstriction (HPV) in dogs and hum
44 CA) inhibitors such as acetazolamide inhibit hypoxic pulmonary vasoconstriction (HPV) in humans and o
45                                              Hypoxic pulmonary vasoconstriction (HPV) is a physiologi
46                                              Hypoxic pulmonary vasoconstriction (HPV) is a physiologi
47                                              Hypoxic pulmonary vasoconstriction (HPV) is driven by th
48                                              Hypoxic pulmonary vasoconstriction (HPV) is inhibited by
49                    It has been proposed that hypoxic pulmonary vasoconstriction (HPV) is mediated via
50                                              Hypoxic pulmonary vasoconstriction (HPV) is unique to pu
51                                              Hypoxic pulmonary vasoconstriction (HPV) maintains blood
52                                              Hypoxic pulmonary vasoconstriction (HPV) normally optimi
53                                              Hypoxic pulmonary vasoconstriction (HPV) optimizes pulmo
54 binoid anandamide (AEA) is a key mediator of hypoxic pulmonary vasoconstriction (HPV) via fatty acid
55 tial to avoid life-threatening hypoxemia via hypoxic pulmonary vasoconstriction (HPV) which matches p
56                    This phenomenon is called hypoxic pulmonary vasoconstriction (HPV), and is respons
57  a potential site of O(2) sensing underlying hypoxic pulmonary vasoconstriction (HPV), but 2 disparat
58                Free hemoglobin (Hb) augments hypoxic pulmonary vasoconstriction (HPV), ostensibly by
59                             This phenomenon, hypoxic pulmonary vasoconstriction (HPV), preserves the
60                Sepsis and endotoxemia impair hypoxic pulmonary vasoconstriction (HPV), thereby reduci
61                Sepsis and endotoxemia impair hypoxic pulmonary vasoconstriction (HPV), thereby reduci
62             To assess the role of cPLA(2) in hypoxic pulmonary vasoconstriction (HPV), we measured th
63  perfusion, which is proposed to result from hypoxic pulmonary vasoconstriction (HPV).
64  cells (PASMCs) has been proposed to mediate hypoxic pulmonary vasoconstriction (HPV).
65 eculated to contribute to Ca2+ influx during hypoxic pulmonary vasoconstriction (HPV).
66 ndria function as the O(2) sensor underlying hypoxic pulmonary vasoconstriction (HPV).
67  Red blood cells (RBCs) are known to augment hypoxic pulmonary vasoconstriction (HPV).
68 result in membrane depolarization leading to hypoxic pulmonary vasoconstriction (HPV).
69 ry vessels, which may explain the failure of hypoxic pulmonary vasoconstriction in acute respiratory
70 3) dipyridamole blunts the severity of acute hypoxic pulmonary vasoconstriction in children with exag
71 oxical findings may be explained by impaired hypoxic pulmonary vasoconstriction in infected lung regi
72            Moreover, Piezo1 knockout reduced hypoxic pulmonary vasoconstriction in isolated, perfused
73 nd combined contribution of hypovolaemia and hypoxic pulmonary vasoconstriction in limiting left vent
74 of the proximal region of the ETC attenuates hypoxic pulmonary vasoconstriction in rat lungs and bloc
75                           Pergolide enhanced hypoxic pulmonary vasoconstriction in the isolated perfu
76            Anorexigens were shown to enhance hypoxic pulmonary vasoconstriction, in part by inhibitin
77 tude include hyperventilation, polycythemia, hypoxic pulmonary vasoconstriction-increased intracellul
78 onal alveolar-capillary oxygen flux; and (3) hypoxic pulmonary vasoconstriction increases whole-lobe
79                                              Hypoxic pulmonary vasoconstriction inhibition by Escheri
80                                              Hypoxic pulmonary vasoconstriction is a local reflex in
81                                              Hypoxic pulmonary vasoconstriction is blunted during sep
82                                              Hypoxic pulmonary vasoconstriction is correlated with pu
83 thesis of the etiology of HAPE suggests that hypoxic pulmonary vasoconstriction is extensive but not
84 striction and attempted to determine whether hypoxic pulmonary vasoconstriction is influenced by pyri
85                                              Hypoxic pulmonary vasoconstriction is initiated by inhib
86                                              Hypoxic pulmonary vasoconstriction is the primary physio
87                                              Hypoxic pulmonary vasoconstriction is understood to be t
88 on of a uniform ventilation distribution (1) hypoxic pulmonary vasoconstriction matches perfusion to
89  addition to airway obstruction, the loss of hypoxic pulmonary vasoconstriction may help to explain w
90 hanism also may be an important component of hypoxic pulmonary vasoconstriction, memory, and circadia
91                                              Hypoxic pulmonary vasoconstriction occurred following WL
92 d and combined influence of hypovolaemia and hypoxic pulmonary vasoconstriction on the decrease in le
93                                 For example, hypoxic pulmonary vasoconstriction optimizes ventilation
94 n polyoxyethylene conjugate had no effect on hypoxic pulmonary vasoconstriction or pulmonary shunt.
95                        Both hypovolaemia and hypoxic pulmonary vasoconstriction play a role in the re
96 We found that dasatinib treatment attenuated hypoxic pulmonary vasoconstriction responses and increas
97 shunt or may worsen oxygenation by reversing hypoxic pulmonary vasoconstriction, therapy increasing v
98 arterial smooth muscle cells responsible for hypoxic pulmonary vasoconstriction to limit perfusion of
99                                              Hypoxic pulmonary vasoconstriction was assessed as chang
100                                              Hypoxic pulmonary vasoconstriction was assessed as the c
101                                    Moreover, hypoxic pulmonary vasoconstriction was attenuated by AMP
102                                              Hypoxic pulmonary vasoconstriction was repeatedly tested
103 asma volume was normalised to SL values, and hypoxic pulmonary vasoconstriction was reversed by admin
104 redictions of a computer lung model in which hypoxic pulmonary vasoconstriction was suppressed by end
105 greater resting hypoxaemia, they had similar hypoxic pulmonary vasoconstriction when compared to lowl
106     Inhaled nitric oxide was able to abolish hypoxic pulmonary vasoconstriction when insufflated into
107 triction; (ii) sympathoexcitation; and (iii) hypoxic pulmonary vasoconstriction with respect to alter
108 lation of ENO provided immediate relief from hypoxic pulmonary vasoconstriction without affecting sys

 
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