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1 ies had chronic heart failure resulting from idiopathic dilated cardiomyopathy.
2 pared with children who are transplanted for idiopathic dilated cardiomyopathy.
3 ) age was 51.2 (13.8) years, and 7 (70%) had idiopathic dilated cardiomyopathy.
4 more severe heart failure than children with idiopathic dilated cardiomyopathy.
5 imited evidence is available specifically on idiopathic dilated cardiomyopathy.
6 oppose the negative effects of ischemic and idiopathic dilated cardiomyopathy.
7 nts with endstage ischemic cardiomyopathy or idiopathic dilated cardiomyopathy.
8 domain (MVt) and correlates with hereditary idiopathic dilated cardiomyopathy.
9 rdial biopsy samples, 180 were identified as idiopathic dilated cardiomyopathy.
10 ing ventricular arrhythmias in patients with idiopathic dilated cardiomyopathy.
11 HF and from patients with HF attributable to idiopathic dilated cardiomyopathy.
12 ay improve cardiac function in patients with idiopathic dilated cardiomyopathy.
13 left ventricular efficiency in patients with idiopathic dilated cardiomyopathy.
14 1Met, was identified as the genetic cause of idiopathic dilated cardiomyopathy.
15 elderly persons who are at increased risk of idiopathic dilated cardiomyopathy.
16 B viruses are detected in about one-third of idiopathic dilated cardiomyopathies.
17 rt disease (152 myocytes per million) and in idiopathic dilated cardiomyopathy (131 myocytes per mill
18 ), of black ethnicity (18% vs. 2%), and have idiopathic-dilated cardiomyopathy (62% vs. 9%) (all p <
20 age 42+/-15.5 years; 64 patients (65.9%) had idiopathic dilated cardiomyopathy and 21 patients (21.6%
21 e studied 11 patients with LV failure due to idiopathic dilated cardiomyopathy and 7 control subjects
22 tion patients with ischemic heart disease or idiopathic dilated cardiomyopathy and compared with samp
24 ng in approximately 25% of familial cases of idiopathic dilated cardiomyopathy and in 18% of sporadic
25 s had severe chronic heart failure: four had idiopathic dilated cardiomyopathy, and three had ischemi
26 r in patients with ischemic heart disease or idiopathic dilated cardiomyopathy as the cause of heart
27 be present in the hearts of individuals with idiopathic dilated cardiomyopathy, as well as the hearts
28 lation in patients with heart failure due to idiopathic dilated cardiomyopathy but not in control sub
29 It shares many clinical characteristics with idiopathic dilated cardiomyopathy but occurs at a younge
30 sis of data from two case-control studies of idiopathic dilated cardiomyopathy carried out in Baltimo
31 hus examined changes in prognosis of a large idiopathic dilated cardiomyopathy cohort systematically
32 differentially upregulated in patients with idiopathic dilated cardiomyopathy compared with other fo
34 n may act as a trigger in the development of idiopathic dilated cardiomyopathy (DCM) among individual
35 function exists in patients with acute-onset idiopathic dilated cardiomyopathy (DCM) and to explore i
39 re obtained at transplant from patients with idiopathic dilated cardiomyopathy (DCM) not requiring LV
40 Abs) against cardiac myosin (CM) in clinical idiopathic dilated cardiomyopathy (DCM) remains controve
41 ta-adrenergic contractility in patients with idiopathic dilated cardiomyopathy (DCM) through nitric o
47 with ischemic cardiomyopathy (ICM, n=16) or idiopathic dilated cardiomyopathy (DCM, n=15) and nonfai
48 d by ischemic cardiomyopathy (ICM-MR) and by idiopathic dilated cardiomyopathy (DCM-MR) by use of rea
49 eart failure programs included patients with idiopathic dilated cardiomyopathy defined as left ventri
50 to dramatic improvement in the prognosis of idiopathic dilated cardiomyopathy during the past 3 deca
51 njury and in heart tissue from patients with idiopathic dilated cardiomyopathy, focal areas of intens
52 ssed genes in lymphocytic myocarditis versus idiopathic dilated cardiomyopathy (fold change >1.2; fal
58 four index patients initially diagnosed with idiopathic dilated cardiomyopathy (IDC) and to provide c
62 s to heart failure, patients with hereditary idiopathic dilated cardiomyopathy (IDC) were examined fo
63 left and right ventricular membranes from 14 idiopathic dilated cardiomyopathy (IDC), 8 primary pulmo
64 ons of hearts obtained from 36 subjects with idiopathic dilated cardiomyopathy (IDC), 8 subjects with
65 ult heart failure therapies to children with idiopathic dilated cardiomyopathy (IDC), prognosis remai
66 sus has emerged that with a new diagnosis of idiopathic dilated cardiomyopathy (IDC), the clinical sc
67 from primary pulmonary hypertension (PPH) or idiopathic dilated cardiomyopathy (IDC), we measured exp
71 = 19) and failing human left ventricles with idiopathic dilated cardiomyopathy (IDC; n = 31) or ische
73 VC) and postmyocarditis VT; in patients with idiopathic dilated cardiomyopathy (IDCM) and postmyocard
74 study was to determine whether patients with idiopathic dilated cardiomyopathy (IDCM) exhibit alterat
75 rdiomyopathy (FDCM) to that of children with idiopathic dilated cardiomyopathy (IDCM) has produced co
76 thyroid function and myocardial injuries in idiopathic dilated cardiomyopathy (IDCM) using cardiac m
77 rvival is worse for patients with ACM versus idiopathic dilated cardiomyopathy (IDCM) with continued
80 heart failure and rhythm disturbances due to idiopathic dilated cardiomyopathy identified two mutatio
82 lling World Health Organization criteria for idiopathic dilated cardiomyopathy, including negative co
88 yocardium were obtained from 9 patients with idiopathic dilated cardiomyopathy (mean duration of LVAD
89 ly proven lymphocytic myocarditis (n=16) and idiopathic dilated cardiomyopathy (n=32) to develop accu
92 ths after the procedure in two patients with idiopathic dilated cardiomyopathy, one of whom had a pac
93 duration, and an evaluation consistent with idiopathic dilated cardiomyopathy or myocarditis were en
98 d adults, morbidity and mortality rates from idiopathic dilated cardiomyopathy rise sharply with age
100 were isolated from a patient suffering from idiopathic dilated cardiomyopathy, suggesting that such
103 beta-blockers produce reverse-remodeling in idiopathic dilated cardiomyopathy, they partially revers
105 mural sites was performed in 6 patients with idiopathic dilated cardiomyopathy undergoing cardiac tra
111 inase activity falls to negligible levels in idiopathic dilated cardiomyopathy, whereas alpha 2-macro
113 221 children with myocarditis and 1583 with idiopathic dilated cardiomyopathy who were <18 years old