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1 s strongly correlated with neurohormonal and immune abnormalities.
2  cell populations and predispose the host to immune abnormalities.
3 to AIDS, organ transplantation or congenital immune abnormalities.
4 systems are known to be affected causing non-immune abnormalities.
5 h disparities; and characterising drivers of immune abnormalities.
6 the treatment of patients with MDD featuring immune abnormalities.
7 ment strategy to compensate for these innate immune abnormalities.
8 f patients with pES, our findings of similar immune abnormalities across all patients with pES help e
9                        The identification of immune abnormalities allows for specific counseling of f
10 t is characterized by serologic and cellular immune abnormalities and is dependent on the presence of
11 ck of or defective hematopoietic stem cells, immune abnormalities, and disorders of the bone marrow m
12                                      In ASD, immune abnormalities are frequently observed, such as el
13 own gene defect had a similar characteristic immune abnormality as patients with defined genetic defe
14 fants may be capable of producing thymic and immune abnormalities, as suggested by previous reports o
15 odels of lupus has provided insight into the immune abnormalities associated with autoantibody produc
16   The data of this study provide evidence of immune abnormalities before motor onset of disease.
17 ciated with a series of in vitro and in vivo immune abnormalities consistent with lymphocyte hyperact
18 ment of barrier abnormalities and downstream immune abnormalities during the elicitation phase of mur
19 pic march." Controversy exists as to whether immune abnormalities, epidermal barrier defects, or both
20 he immune system, as evidenced by the severe immune abnormalities exhibited by patients bearing inact
21 exposed children, raising the possibility of immune abnormalities following exposure to maternal vira
22 d analysis appeared to show a progression of immune abnormalities from no cGVHD/L-aGVHD to L-aGVHD, w
23                                              Immune abnormalities have been described in some individ
24 position to developing cancer (thymomas) and immune abnormalities (impaired early thymocyte developme
25  and anxiety-like behaviors, suggesting that immune abnormalities in MIA offspring can contribute to
26 rointestinal tract and to define the mucosal immune abnormalities in patients with and without sympto
27 emerges as a therapeutic target in resetting immune abnormalities in RA.
28               Converging evidence implicates immune abnormalities in schizophrenia (SCZ), and recent
29 ormation will help to elucidate the cellular immune abnormalities leading to production of pathogenic
30 tory conditions suggests a common underlying immune abnormality leading to both conditions.
31 ison of these strains reveals a hierarchy of immune abnormalities, lung inflammation and fibrosis, wh
32 , identifying atopic patients with molecular immune abnormalities may be helpful for diagnostic, ther
33 st in investigating the impact of early-life immune abnormalities on later onset of psychosis.
34  cells, we tested the hypothesis that ocular immune abnormalities participate in PG phenotypes.
35        Melanosomal defects along with ocular immune abnormalities play a role in the propagation of p
36           This demonstrates that some of the immune abnormalities previously identified in DBA/2J mic
37 background AD might be explained by baseline immune abnormalities, such as increased TH2, TH17, and n
38 /TSP and underlie many of the characteristic immune abnormalities, such as spontaneous lymphocyte pro
39 rder (PTSD) is associated with endocrine and immune abnormalities that could increase risk for autoim
40 how that if immune biomarkers exist for such immune abnormality, they may be found in raised macropha
41 emotherapy associated with broad reversal of immune abnormalities together with fibroblast transition
42                          All of the cellular immune abnormalities were reversed by day 360 in abstine
43 er p21 Ras oncoproteins by causing selective immune abnormalities without general developmental defec