ライフサイエンスコーパス: immune complex

1 on follicular dendritic cells in the form of immune complex.

2 borate to 'stabilize' the ternary pathogenic immune complex.

3 d were highly efficient in taking up OVA/IgG immune complexes.

4 the adaptive immune response, and removal of immune complexes.

5 lar patterns such as IgG- and IgM-containing immune complexes.

6 and misfolded proteins, forming circulating immune complexes.

7 e uptake and cycling of complement-opsonized immune complexes.

8 culating autoantibodies and the formation of immune complexes.

9 rheumatoid-factor B cells by DNA-containing immune complexes.

10 ibodies through immunization with sialylated immune complexes.

11 rsistently infected mice specifically lacked immune complexes.

12 from that employed during the endocytosis of immune complexes.

13 ith synthetic TLR agonists or RNA-containing immune complexes.

14 um levels of BAFF, antinuclear Abs (ANA) and immune complexes.

15 uggested the development of antigen-antibody immune complexes.

16 he low-affinity IgG receptor, CD16, by Ag-Ab immune complexes.

17 ous neutrophil stimulation with OVA/anti-OVA immune complexes.

18 nt C3 and fibronectin associated with IgA on immune complexes.

19 ith glomerular deposition of IgA1-containing immune complexes.

20 hepatic fibrosis and serum levels of ANA and immune complexes.

21 ation with immunoglobulin E (IgE)-containing immune complexes.

22 IgG3, which self-associates to form large immune complexes, accounts for more than 97% of the mous

23 cR- and complement-dependent effects of ACPA immune complexes (ACPA-IC).

24 Prior to this, DENV immune complexes activate spleen tyrosine kinase (Syk) w

25 This study provides evidence that DENV-2 immune complexes activate Syk to mediate elevated expres

26 LF were also elevated in C5a-induced and IgG immune complex ALI models, suggesting a common inflammat

27 MN), which is characterized by deposition of immune complexes along the glomerular basement membrane

28 nephropathy (MN) occurs due to deposition of immune complexes along the subepithelial region of glome

29 r, these studies demonstrate assembly of HIT immune complexes along VWF strings released by injured e

30 ation of BCR signaling in the absence of IgE immune complex and suggest that CD23 down-regulates BCR

31 (Horseradish Peroxidase) which binds to the immune complex and the response was observed using Hydro

32 features are not associated with circulating immune complexes and C3 deposition and are more pertinen

33 respond robustly to autoantigen/autoadjuvant immune complexes and could therefore participate in both

34 fM analyte because beads attached via single-immune complexes and DNA strands form tethers, and tethe

35 Our aim was to study the involvement of immune complexes and neonatal Fc receptor (FcRn) in the

36 due to release of HCV-Ags sequestered in HCV immune complexes and should not be used in any HCV-Ags,

37 ystemic lupus erythematosus, deposits of IgG-immune complexes and the activation of complement in the

38 ergens influenced the shape of the resulting immune complexes and the magnitude of effector cell acti

39 eveloping nephritis, despite the presence of immune complexes and the production of proinflammatory c

40 hesized that increasing the affinity between immune complexes and TRIM21 might markedly improve CD8 T

41 ory sites, where atherogenic factors such as immune complexes and/or pathogens can activate the endot

42 ctivation results in clearance of pathogens, immune complex, and apoptotic cells.

43 EMRMs have a higher capacity for scavenging immune complex, and are more sensitive to immune challen

44 Isolated neutrophils were incubated with immune complexes, and activation and release of NETs wer

45 munity may include IgE against autoantigens, immune complexes, and complement.

46 Human NK cells are activated by cytokines, immune complexes, and signals transduced via activating

47 GP patients had no circulating FH-containing immune complexes, and their anti-FH IgG had a weaker aff

48 al production, storage of viral particles in immune complexes, and viral persistence.

49 The inhibition of IRAK4 repressed SLE immune complex- and TLR7-mediated activation of human pl

50 against anti-TNF-alpha Abs, suggesting that immune complexes are a major determinant of this immuniz

51 ting Fcgamma receptors (FcgammaRs) that bind immune complexes are controlled by a single inhibitory r

52 In IgA nephropathy (IgAN), IgA immune complexes are deposited in the mesangium and driv

53 dentify specific target antigens in PAH lung immune complexes as a starting point toward resolving th

54 ross-reactivity for HT-2 and T-2 toxins, the immune complex assay is highly specific for HT-2 alone.

55 When the noncompetitive immune complex assay was compared to the traditional com

56 that epitope mutability and accessibility to immune complex assembly are important attributes to cons

57 e, in most patients with C3 glomerulopathies/immune complex-associated membranoproliferative glomerul

58 une responses as well as in the clearance of immune complexes, autoreactivity to complement component

59 mmatory Syk-ERK signaling axis requires DENV immune complexes, because DENV-2 in the presence of sero

60 Formation of the immune complex between T cell and APC starts with format

61 glycolipid antigen presentation, and of the immune complex binding Fcgamma receptor III (CD16).

62 inding site may be in close proximity to the immune complex binding site in CD16.

63 C3d and CR2 also mediate immune complex binding to follicular dendritic cells.

64 Anti-CD16 mAb inhibited immune complex binding to sCD16, whereas it partially in

65 ively, have no influence on monomeric IgG or immune complex binding, but FcRgamma signaling is decrea

66 e extracellular domain of FcgammaRI, reduces immune-complex binding of FcgammaRI whereas monomeric Ig

67 hat are bound to pathogens enter the cell as immune complexes, binding of TRIM21 to Fc initiates down

68 of 10(4) molar excess of IgM or its soluble immune complexes, but it could be inhibited by addition

69 Replacement of antibody on immune complexes by antibody generated from GC-derived p

70 uptake of IgM and minimal endocytosis of IgG immune complexes by neutrophils was observed, in contras

71 vation threshold of innate effector cells to immune complexes by stimulating the up-regulation of the

72 vation threshold of innate effector cells to immune complexes by stimulating the upregulation of the

73 ion of idiopathic nephrotic syndrome with no immune complexes can improve the sensitivity to detect s

74 Small immune complexes cause type III hypersensitivity reactio

75 8 and 13 and major collagen genes, IgG4-rich immune complexes coating alveolar macrophages, and incre

76 4) in vitro after activation with zymosan or immune complexes, compared with wild-type (WT) neutrophi

77 tion for IPD changes (e.g., ABA, circulating immune complexes, complement activation, complete blood

78 We found that FDCs took up and retained self-immune complexes composed of ribonucleotide proteins, au

79 tiinfluenza antibodies by demonstrating that immune complexes composed of sialylated antihemagglutini

80 ake up and internalize donor cell-associated immune complexes composed of specific monoclonal antibod

81 RV144, pointed to mechanistic insights into immune complex composition that may underlie protective

82 se immunodeposits originate from circulating immune complexes consisting of anti-glycan antibodies bo

83 ntibodies specific for Fel d1 and exposed to immune complexes consisting of Fel d1-specific IgG antib

84 an autoimmune disease mediated by pathogenic immune complexes consisting of galactose-deficient IgA1

85 Immune complexes consisting of heparin, platelet factor

86 Immune complexes consisting of heparin, platelet factor

87 lly "super-activate" kidney macrophages when immune complexes contain a nucleic acid.

88 Anti-Ro60-positive SLE immune complexes contained Alu RNAs, and Alu transcripts

89 an autoimmune thrombotic disorder caused by immune complexes containing platelet factor 4 (PF4), ant

90 Immune complexes containing RNA induced OX40L expression

91 Immobilized immune complexes containing the rIgA1 and rIgA2 autoanti

92 and suggest that high levels of pre-existing immune complexes could limit the effectiveness of antibo

93 4Nb8 inhibits the classical pathway-mediated immune complex delivery to follicular dendritic cells in

94 with IgG, IgM, C1q, and C4d, consistent with immune complex dependent activation of the classical com

95 orly activate effector mechanisms, reveal an immune-complex-dependent, complement- and FcR-independen

96 glomerular capillaries are frequent sites of immune complex deposition and subsequent neutrophil accu

97 This activation can be caused by immune complex deposition or an acquired or inherited de

98 einuria, blood urea nitrogen, and glomerular immune complex deposition were also exacerbated when com

99 ory cytokines and autoantibodies, glomerular immune complex deposition, and evidence of kidney damage

100 markers of renal disease, enhance glomerular immune complex deposition, and modulate TLR7/9 cytokine

101 l nerve antigens, but additionally encompass immune complex deposition, cellular infiltration, and cy

102 lls caused autoantibody production and renal immune complex deposition.

103 ties, such as mesangial hypercellularity and immune complex deposition.

104 er from hypersensitivity mechanisms, or from immune-complex deposition via anti-adalimumab antibodies

105 elected scFv was applied in serodiagnosis by immune complexes detection in serum samples from individ

106 However, ATGs can induce immune complex diseases, including serum sickness diseas

107 Application of OVA/IgG immune complexes during pregnancy boosted OVA uptake by

108 another function of IgG by showing that IgG immune complexes elicit distinct cytokine profiles by hu

109 Commensal-IgG immune complexes engaged gut-resident FcgammaR-expressin

110 organisms to FcR in mice i.n., with MAb-iFT immune complexes, enhances F. tularensis-specific immune

111 eceptor FcgammaRIIIa; when incorporated into immune complexes, Fc afucosylation enhanced production o

112 e that in contrast to internalization of IgG immune complexes, FcgammaRIIb-augmented internalization

113 tream inflammatory signaling and targets the immune complexes for proteasomal degradation.

114 es against aggregated misfolded protein with immune complex formation and kidney fibrosis.

115 Notably, our results showed that immune complex formation and the activating Fc gamma rec

116 s on Fcgamma receptor and the requirement of immune complex formation for effector cell activations.

117 ximal tubule antigen-specific antibodies and immune complex formation has not been well characterized

118 In this article, we provide evidence that immune complex formation of serum IgA plays an important

119 e-mediated innate immune activation requires immune complex formation with naturally occurring IgG an

120 bility to undergo Fab-arm exchange and limit immune complex formation.

121 nd allows the calculation of the kinetics of immune complexes' formation in a continuous-flow system

122 The shape of immune complexes formed between artificial allergens and

123 Here, we demonstrated that immune complexes formed between omalizumab and IgE can i

124 ach to isolate and identify TARK1-associated immune complexes formed during infection.

125 ammation in experimental CrGN resulting from immune complexes formed within the glomerular capillary

126 ntified and confirmed as highly expressed in immune complexes from 16 hereditary and idiopathic PAH v

127 Herein, we show that increased amounts of immune complexes generated in mice persistently infected

128 ncubation step promotes the formation of the immune-complex, generating a 51.7-fold sensitivity enhan

129 mportant extrahepatic manifestations include immune complex glomerular disease, accelerated progressi

130 sults in increased systemic inflammation and immune complex glomerulonephritis, despite intact TLR si

131 rular C4d staining in 18 biopsy specimens of immune-complex GN, 30 biopsy specimens of C3 GN, and 13

132 types, each with specific disease entities: immune-complex GN, pauci-immune GN, antiglomerular basem

133 of the immunoassay was introduced by an anti-immune complex (IC) antibody binding the primary antibod

134 val of both subendothelial and subepithelial immune complex (IC) deposits.

135 A role for FcgammaRIIIa activation from immune complex (IC) ligation and sublytic terminal compl

136 Immune complex (IC) vaccines have been successfully used

137 Upon ex vivo stimulation with IgG immune complex (IC), neutrophils up-regulated expression

138 acrophages isolated from inflamed tissues in immune complex (IC)-associated diseases, including SLE a

139 ental effects of neutrophils using models of immune complex (IC)-mediated inflammation in mice immuno

140 in skin biopsies of patients suffering from immune complex (IC)-mediated vasculitis (ICV).

141 llmarks of the disease are the appearance of immune complexes (IC) containing autoreactive Abs and TL

142 matosus (SLE), where nucleic acid-containing immune complexes (IC) drive inflammation.

143 TLR ligands in the presence of high-density immune complexes (IC).

144 transgenic mice with IgG2a(a) anti-chromatin immune complexes (ICs) activated RF B cells in a BCR- an

145 RF in the role of antibody to immune complexes (ICs) appears to be involved in activat

146 ion is complexed to Abs, and these resulting immune complexes (ICs) are a prominent feature of chroni

147 riven immune responses that are modulated by immune complexes (ICs) are known to inhibit IFN-gamma-de

148 ose EVs are responsible for the formation of immune complexes (ICs) containing anti-MASP SP IgGs in p

149 I (Fc-gammaRIII) implies that IgG and ASNase immune complexes (ICs) could directly induce hypersensit

150 Apoptotic debris, autoantibody, and IgG-immune complexes (ICs) have long been implicated in the

151 The ligation of FcgammaRIIIa by immune complexes (ICs) in human CD4(+) T-cells produced

152 mmatory disease characterized by deposits of immune complexes (ICs) in organs and tissues.

153 Deposition of immune complexes (ICs) in tissues triggers acute inflamm

154 have indicated that nucleic acid-containing immune complexes (ICs) induce B cell and dendritic cell

155 dendritic cell (FDC) reservoir of HIV-bound immune complexes (ICs) is unknown.

156 therapeutic target for diseases in which IgG immune complexes (ICs) mediate inflammation, such as hep

157 Activation of neutrophils through immune complexes (ICs) plays a central role in the patho

158 IgG immune complexes (ICs) promote autoimmunity through bind

159 dary exposure of the immune system to an Ag, immune complexes (ICs) that contain the unprocessed Ag a

160 the mAb had formed immunogenicity-enhancing immune complexes (ICs) with nucleosomal Ags during cell

161 egulatory M2b macrophages in the presence of immune complexes (ICs), but the role of the specific sub

162 caused by immunoglobulin G (IgG)-containing immune complexes (ICs), but the underlying mechanisms ar

163 In this study, we identify a role for IgG-immune complexes (ICs), FcgammaRs, and BAFF during the f

164 dividuals and stimulated with RNA-containing immune complexes (ICs), herpes simplex virus (HSV) or th

165 redominantly in the form of gp120-anti-gp120 immune complexes (ICs).

166 permutated B cell antigen receptors (BCR) on immune complexes (ICs).

167 ansfer and uptake of allergen-containing IgG immune complexes (Ig-ICs) by gut dendritic cells (DCs).

168 t fragments with minimal or no deposition of immune complexes/Ig; the C3 is derived from activation o

169 ted GN results from glomerular deposition of immune-complexes/Ig and C3; the C3 is derived from activ

170 IgE-immune complexes (IgE-ICs) have been shown to enhance an

171 ow free IgE versus precomplexed IgE-allergen immune complexes (IgE-ICs) target the 2 IgE receptors Fc

172 w that FcgammaRI, an immune receptor for IgG immune complex (IgG-IC), is expressed in a subpopulation

173 )-dependent transfer of maternal IgG and OVA immune complexes (IgG-IC) via breast milk and induction

174 egrade apoptotic debris contained within IgG-immune complexes (IgG-ICs) and promotes recycling and th

175 ctor mechanisms may be useful for inhibiting immune complex immunopathology.

176 te effector mechanisms might protect against immune complex immunopathology.

177 ng GADA emergence, and to monitor GADA-GAD65 immune complexes in blood samples.

178 n can take place directly on IgA1-containing immune complexes in circulation and/or after their depos

179 o tissue damage, we investigated whether IgA immune complexes in plasma and synovial fluid of RA pati

180 storage in liver, and (e) fewer circulating immune complexes in plasma.

181 ough deposition of immunoglobulin-containing immune complexes in premalignant tissue and Fcgamma rece

182 initiate the inflammatory response to small immune complexes in the kidney.

183 f autoantibodies to dsDNA, and deposition of immune complexes in the kidney.

184 subdomains that participate in keeping ZAR1 immune complexes inactive.

185 Immune complex increased pKal activity, which led to HK

186 psilonRII-mediated signaling by allergen-IgE immune complexes increased IFN-gamma production in B cel

187 portion of IgG (FcgammaRIIA), a receptor for immune complexes, independently of thrombin.

188 Asparaginase immune complexes induce Fc-gammaRIII-dependent hypersens

189 Immunization of mice with IgE-immune complexes induced glycan-specific anti-IgE autoan

190 revious data have shown that during LPS-/IgG immune complexes-induced ALI, the DNA binding activities

191 together, C/EBPgamma suppresses LPS- and IgG immune complexes-induced pro-inflammatory mediators' pro

192 n some other cell engaged by IgG1-amastigote immune complexes induces IL-10 from T cells.

193 These immune complexes initiate an inflammatory response resul

194 We were also able to detect immune complexes involving GAD65 and GADA.

195 " Taking into account that the deposition of immune complexes is a major event in acute inflammation

196 determine whether ALI induced by LPS and IgG immune complexes is affected by C/EBPgamma.

197 tion of modified forms of LDL in circulating immune complexes (LDL-ICs) was associated with cardiovas

198 cgammaRs), which transduce interactions with immune complexes, leading to a variety of cellular outco

199 free serum ABA levels decreased, circulating immune complex levels increased, and complement activati

200 d: large scroll-like immunotactoid deposits, immune complex-like deposits, and randomly arranged fibr

201 of-principle, immune sandwich assay in which immune complexes link micron-size beads via DNA tethers

202 ssays applied for detection of GAD65 and its immune complexes may thus enable improved diagnosis and

203 Leukocytoclastic vasculitis (LCV) is an immune-complex mediated vasculitis characterized by neut

204 Ptpn22(-/-) mice were protected from immune complex-mediated arthritis, induced by the transf

205 This finding suggests that inflammation in immune complex-mediated CrGN is predominantly intravascu

206 n CD20-expressing tumors, as well as reduced immune complex-mediated cross-presentation to T cells.

207 nderlying cause as a complement-mediated and immune complex-mediated disease.

208 Immune complex-mediated diseases, such as systemic lupus

209 sm should be important for allergy and other immune complex-mediated diseases.

210 gated the role of FcRn in the development of immune complex-mediated glomerular disease in mice.

211 uced higher anti-dsDNA IgG Abs and developed immune complex-mediated glomerulonephritis, compared wit

212 dependent inflammation in a model of in situ immune complex-mediated glomerulonephritis.

213 B cells is sufficient for the development of immune complex-mediated glomerulonephritis.

214 mmarize, C4d serves as a positive marker for immune complex-mediated GN but is absent or minimally de

215 Immune complex-mediated GN results from glomerular depos

216 All specimens of immune complex-mediated GN, except two specimens of IgA

217 increased in the inflammatory environment of immune complex-mediated GN.

218 ory roles particularly in the context of IgG immune complex-mediated inflammation.

219 plement-mediated C3 glomerulopathy (C3G) and immune complex-mediated membranoproliferative GN (IC-MPG

220 There were 23 patients with immune complex-mediated MPGN and available eye examinati

221 nts with pathology-confirmed complement- and immune complex-mediated MPGN who had available ophthalmo

222 diated MPGN when compared with patients with immune complex-mediated MPGN.

223 n SLE, develops elevated antinuclear Abs and immune complex-mediated nephritis along with other manif

224 mportant role for BTK in the pathogenesis of immune complex-mediated nephritis, and BTK inhibition as

225 Proliferative GN is classified as immune complex-mediated or complement-mediated (C3 glome

226 echanisms, C/EBPgamma might inhibit LPS-/IgG immune complexes-mediated inflammation via alleviating C

227 The presence of circulating immune complexes of IgA bound to beta2-glycoprotein I (B

228 lt from glomerular deposition of circulating immune complexes of IgG bound to galactose-deficient IgA

229 t glomerulopathy after rigorous exclusion of immune complexes of other cause, particularly recurrent

230 titer plate immunoassay, individual sandwich immune complexes of the cancer marker prostate-specific

231 Ib) that, upon multivalent binding of IgG in immune complexes or on opsonized targets, mediate respon

232 cally injected antibodies and form localized immune complexes outside the Sertoli cell barrier.

233 reviously identified target antigens for the immune complexes, PLA2R (identified in 2009) and THSD7A

234 Combined, these data indicate that IgG immune complexes potentiate inflammation by human microg

235 plement and FcR independent and results from immune complex precipitation in glomerular capillaries,

236 oinflammatory lipids, circulating cytokines, immune complexes, proinflammatory metals and toxic chape

237 at SAP is essential when autoantibody-driven immune complexes promote inflammation but is not require

238 ADAMTS5 processing of immune complex proteins reduced binding to cultured mesa

239 esolution structure of a type III RNA-guided immune complex provides new insights into the mechanisms

240 On stimulation with immobilized immune complexes, Ptpn22(-/-) neutrophils manifested red

241 macrophages detect and scavenge circulating immune complexes "pumped" into the interstitium via tran

242 Antibody on these immune complexes regulates antigen accessibility by shie

243 (IgG)-opsonized particles and polyvalent IgG immune complexes, respectively.

244 Additionally, in vitro stimulation with immune complexes resulted in significantly less CXCL-1 a

245 Incubation of neutrophils with immune complexes resulted in the production of reactive

246 ce and formation of nuclear autoantigens and immune complexes resulting in inflammation of multiple o

247 Ribonucleoprotein immune complexes (RNP ICs), inducers of NETosis, require

248 2 (10) Fab reaction was reported by anti-HT2 immune complex single-chain variable fragment of antibod

249 een extensively investigated in LPS- and IgG immune complexes-stimulated acute lung injury.

250 ssion in the lung tissue alleviates LPS-/IgG immune complexes-stimulated acute pulmonary damage throu

251 Moreover, we identified that IgE immune complex stimulation of FcepsilonRII activated int

252 Importantly, we show that IgG immune complexes strongly potentiate activation of prima

253 macrophages express FcgammaR that engage IgG immune complexes such as Ab-opsonized pathogens or cance

254 s brought about by FcgammaRI engagement with immune complexes such as the inhibition of IFNgamma and

255 The high amounts of immune complexes suppressed antibody-mediated cell deple

256 ntibodies complex with self-antigen and form immune complexes that accumulate in the glomeruli.

257 oduction, induced by nucleic-acid-containing immune complexes that activate endosomal Toll-like recep

258 ism for trans-endothelial transport of small immune complexes that activate strategically positioned

259 f" and microbial DNA into liquid crystalline immune complexes that amplify TLR9-mediated plasmacytoid

260 epends on the structure of Fc domains within immune complexes that are formed when IgGs bind to cogna

261 reover, the autoantibodies they produce form immune complexes that can activate myeloid cells and the

262 us, directly kill infected cells and produce immune complexes that can enhance host immunity to the v

263 cells only proliferate in response to IgG2a immune complexes that incorporate DNA, RNA, or nucleic a

264 Autoreactive B cell-derived antibodies form immune complexes that likely play a pathogenic role in a

265 d by antinuclear antibodies (ANAs) that form immune complexes that mediate pathogenesis by tissue dep

266 caffolds with immunoglobulin G (IgG) to form immune complexes that promote platelet activation.

267 llance by non-classical monocytes may detect immune complexes through CD16, orchestrating the inflamm

268 ination of direct platelet activation by HIT immune complexes through FcgammaRIIA and transactivation

269 ble dissociation of human antibodies-Ara h 6 immune complex, thus rendering Ara h 6 accessible for it

270 lymphocytes and resulted in reduced ANA and immune complex titers.

271 s the transport and delivery of bacteria and immune complexes to phagocytes, through a process known

272 blood-borne antigens and antigen-containing immune complexes to splenic FDC.

273 in the erythrocyte membrane is important for immune-complex transfer and clearance.

274 e systemic anaphylaxis, which may due to IgG immune complex triggered complement activation, anaphyla

275 Immune complex tubulointerstitial nephritis due to antib

276 The significance of alloimmune immune complex-type deposits in human transplants deserv

277 rafts, we found mesangial and subendothelial immune complex-type electron-dense deposits.

278 the affinity between Fc and TRIM21, Ags from immune complexes undergo enhanced cross-presentation on

279 e, FcgammaRIIB was required for Sendai virus immune complex uptake by splenic pDCs in vitro, and inte

280 Thus, pDCs bind viral immune complexes via FcgammaRIIB and prevent IFN-alpha p

281 f influenza virus as a model hapten, and the immune complex was injected to chickens.

282 ding of dimeric rsFcgammaR ectodomains to Ab immune complexes was affected by Ab subclass, presentati

283 rmed that the rapid clearance of these large immune complexes was associated with Fcgamma receptor-de

284 nd Y1 RNA complexed with SLE patient Abs and immune complexes was measured in a cross-section of 228

285 Defined IgE-allergen immune complexes were formed with human monoclonal aller

286 Lung immune complexes were isolated and PAH target antigens w

287 RF isotypes were measured with ELISA, and immune complexes were precipitated using polyethylene gl

288 es, and degranulation induced by immobilized immune complexes, were reduced in Ptpn22(-/-) neutrophil

289 hese autoantibodies trigger the formation of immune complexes, which are hypothesized to cause enhanc

290 Neutrophils were activated by IgA immune complexes, which suggests that neutrophils play a

291 bodies and circulating uromodulin containing immune complexes with glomerular deposition and kidney f

292 Alternatively, immune complexes with IgG1 or IgG4 were formed using pro

293 The BiSAb formed large immune complexes with IL-6 that can bind Fcgamma recepto

294 Immune complexes with subclasses IgG1 and IgG4 can in vi

295 Adenovirus immune complexes with the engineered Fc induced greater

296 By preforming immune complexes with TNF-alpha, an anti-infliximab resp

297 aturation of human dendritic cells (DC) than immune complexes with unmodified Fc and stimulated incre

298 tion of IgG, reducing pathogenic IgG and IgG immune complexes, with no anticipated effects on IgA, Ig

299 Immune complexes within glomerular capillary walls cause

300 might expect that CR2 ligation by C3d-bound immune complexes would promote development of SLE.