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1 MOG cross linking on oligodendrocytes and/or immune complex formation.
2 ice prevented proteinuria and reduced kidney immune complex formation.
3 eserve CD4(+) T cell memory independently of immune complex formation.
4 une and inflammatory responses via localized immune complex formation.
5 , oligoclonal CD4(+) T cell infiltrates, and immune complex formation.
6  by mediating kidney damage via antibody-DNA immune complex formation.
7 RA patients were also elevated, which led to immune complex formation.
8 get organs and acting as a nidus for in situ immune complex formation.
9 ssociated with adverse effects suggestive of immune complex formation.
10 cytosis of ERV-K102 envelope protein through immune complex formation.
11 f-tolerance with autoantibody production and immune complex formation.
12 signal transduction via NFkB, suggesting BCR-immune complex formation.
13 bility to undergo Fab-arm exchange and limit immune complex formation.
14 nd the consequence of subsequent ADA-induced immune complex formation, a translational approach was p
15 genic IgA production and IgA/IgA and IgA/IgG immune complex formation along with (ii) therapies to ma
16 f the AP in CAIA through binding to sites of immune complex formation and complement activation.
17 BAK1)/SUPPRESSOR OF BIR1-1 (StSOBIR1)-StBAK1 immune complex formation and inhibits StFLS2 kinase acti
18 es against aggregated misfolded protein with immune complex formation and kidney fibrosis.
19 hibitory KIR with cognate ligands, promoting immune complex formation and NK-cell cytotoxicity specif
20 l inflammation in SLE are closely linked via immune complex formation and systemic complement depleti
21             Notably, our results showed that immune complex formation and the activating Fc gamma rec
22 ata provide a mechanism explaining increased immune-complex formation and disease exacerbation during
23 er characterized by autoantibody production, immune-complex formation, and altered cytokine expressio
24 ntially injurious inflammatory reactions via immune complex formation associated with phagocytosis an
25 dy-mediated brain disease does not depend on immune complex formation but rather on antibody-mediated
26                                              Immune complex formation could explain the peculiar fate
27 elopment of antibodies to FCS indicates that immune complex formation could have occurred after the c
28 s on Fcgamma receptor and the requirement of immune complex formation for effector cell activations.
29 ximal tubule antigen-specific antibodies and immune complex formation has not been well characterized
30 ell-driven antibody production that leads to immune complex formation in glomeruli, complement activa
31 glomerular antigens, leading subsequently to immune complex formation in situ and complement activati
32 may be a promising approach to down-regulate immune complex formation in the target organ in individu
33              This high binding was linked to immune complex formation in vivo but not during in vitro
34 ody-secreting cells and prohibit detrimental immune complex formation in vivo.
35 nd allows the calculation of the kinetics of immune complexes' formation in a continuous-flow system
36 odels of pathogenesis usually assume in-situ immune-complex formation involving an as yet uncharacter
37       These data suggest that B. burgdorferi immune complex formation is a common process in active L
38                                              Immune complex formation occurred between individual fus
39 min to 3.5 h), different kinetic profiles of immune complex formation of defined geometry were docume
40              These data strongly suggest the immune complex formation of IgG4 and major cow's milk pr
41    In this article, we provide evidence that immune complex formation of serum IgA plays an important
42 odifications, namely aggregation, glycation, immune complex formation, proteoglycan complex formation
43           We also show that rADAMTS13 causes immune complex formation, which saturates the autoantibo
44 ntibody immobilization, analyte capture, and immune complex formation with a second biotinylated anti
45 ngly through effects on NK-cell trafficking, immune complex formation with MM cells, and cytotoxicity
46 e-mediated innate immune activation requires immune complex formation with naturally occurring IgG an