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1 asurements and transendothelial migration of immunocompetent cells.
2 hrough modulation of the CXCR4 coreceptor on immunocompetent cells.
3 ) ET-1 is produced by activated infiltrating immunocompetent cells.
4 us contribute to the lack of regeneration of immunocompetent cells.
5 isease characterized by many dysfunctions of immunocompetent cells.
6  alpha7 nicotinic acetylcholine receptors on immunocompetent cells.
7 lation, homing, retention, and activation of immunocompetent cells.
8 d the enhanced transendothelial migration of immunocompetent cells across the BBB.
9 ion and differentiation of antigen-triggered immunocompetent cells and extrinsic mechanisms mediated
10 er adolescence could affect microglia, other immunocompetent cells, and the neuroimmune environment o
11 investigated whether ERAP2 can trigger other immunocompetent cells, boosting their antiviral potentia
12 n could be generated in both mouse and human immunocompetent cell culture by the addition of even a s
13 hat pre-existing antigens are tolerogenic to immunocompetent cells generated thereafter.
14 genic potential of Cas9 in models of retinal immunocompetent cells: human microglia (IMhu) and ARPE-1
15  the identification of spatially correlating immunocompetent cells in a sub-group of six patients.
16 about a marked down-regulation of autologous immunocompetent cells in cytotoxicity reactions, partial
17 ng evidence has emphasized the importance of immunocompetent cells in determining the psoriatic pheno
18                       Microglia, the primary immunocompetent cells in the brain, are active neonatall
19       Hypothetically, microglia are the main immunocompetent cells in the immature CNS, and depending
20               Interaction of the toxins with immunocompetent cells is not exclusively dependent upon
21 ong others of (1) myocardial infiltration by immunocompetent cells not only because of an obesity-ind
22                          Microglia, the main immunocompetent cells of the brain, regulate neuronal fu
23 vides a biomarker for microglia, the primary immunocompetent cells of the brain.
24                            Microglia are the immunocompetent cells of the CNS, and their activation i
25  neuropathic pain by increasing IL-10 in the immunocompetent cells of the CNS.
26                       Microglia are the main immunocompetent cells of the mammalian central nervous s
27 A status) population of T1D patients, to the immunocompetent cells of the skin.
28 he ability to engraft human tumors and human immunocompetent cells successfully in severe combined im
29  differentially and selectively expressed in immunocompetent cells, such as B cells (CD20/MS4A1) and
30                             Here we identify immunocompetent cell targets of fgTiO(2) in humans, most
31                                              Immunocompetent cells that accumulate fgTiO(2) in vivo a
32                         Skin is replete with immunocompetent cells that modulate signaling pathways t
33 ic acetylcholine receptors (alpha7 nAChR) on immunocompetent cells to inhibit cytokine release in mac
34 is required for the efficient trafficking of immunocompetent cells to sites of inflammation.
35 mechanism responsible for the sensitivity of immunocompetent cells to TCDD may be directly associated
36     However, it was observed that allogeneic immunocompetent cells transplanted with the stem cells,
37 tiating immune responses by interacting with immunocompetent cells via class II MHC proteins.