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1 ates intercellular adhesion of coelomocytes (immunocytes).
2 nd led to hypersensitive and hyperresponsive immunocytes.
3 ctions between infected epithelial cells and immunocytes.
4 oducing beta-cells are destroyed by invading immunocytes.
5 raft mucosa undergoes repopulation with host immunocytes.
6 ith aberrant regulation of keratinocytes and immunocytes.
7 ay cells have been proposed to be the sponge immunocytes.
8 relevant direction for attraction of mucosal immunocytes.
9 mitantly reduce repopulation by autoreactive immunocytes.
10 s (n = 8), did not contain such NKR positive immunocytes.
11 kin by injection of autologous blood-derived immunocytes.
13 on in xenografts composed of human PHKCs and immunocytes abolished psoriasiform hyperplasia and infla
14 a(2+)-permeable cation channel essential for immunocyte activation, insulin secretion, and postischem
16 in the distribution and function of splenic immunocytes and a significant reduction in suppressive a
17 s that direct cell- cell interaction between immunocytes and airway smooth muscle may also modulate a
18 pathologic interplay between skin cells and immunocytes and can result in disfiguring cutaneous lesi
19 sses that include inflammatory activation of immunocytes and macrophages, spillage of intracellular c
20 ow clear that molecular interactions between immunocytes and microbes are mediated largely by Toll-li
23 ive resources for the 3D genome structure of immunocytes and sheds insights into the order of genome
24 lls depends on interactions with other local immunocytes and, importantly, subtypes of VAT mesenchyma
26 sease, continuously fed by a mixed influx of immunocytes, and thus susceptible to evolve over time in
29 ur current understanding of how these unique immunocytes arise, traffic to various sites, and may or
31 ctional trafficking of lymphocytes and other immunocytes begins as soon as the vascular clamp is rele
32 Antiviral responses, neuroinflammation and immunocyte blood-brain barrier (BBB) trafficking follow
33 ated during HCV infection, and LPA activates immunocytes, but whether this contributes to immune acti
34 lerotic changes in the absence of detectable immunocytes by acting on VSMCs to potentiate growth-fact
35 nchymal cells are separated from circulating immunocytes by the endothelium, which is targeted by mic
38 mammary gland to prepare for lactation, yet immunocyte changes that accompany this rapid remodeling
40 sults from a blockade of the infiltration of immunocytes containing beta-endorphin and the consequent
42 in gene and chromatin structure within skin immunocytes could provide key insights into the pathogen
43 modulation of the keratinocyte phenotype by immunocyte-derived cytokines, in which induction of CDw6
44 that promise site-specific actions affecting immunocyte differentiation and proliferation are feasibl
45 that promise site-specific actions affecting immunocyte differentiation and proliferation are now fea
49 activation of various cell types, including immunocytes (eg, macrophages and T cells), smooth muscle
50 ion or function of Sp-Eph results in rounded immunocytes entering ectoderm but not adopting a dendrit
51 nflammation or persistent changes in mucosal immunocytes, enterochromaffin and mast cells, enteric ne
57 e the breadth and underpinning of changes in immunocyte gene expression due to genetic variation in m
60 The results of functional in vitro assays on immunocytes (haemocytes) of the Mediterranean mussel Myt
63 ermine the role of I kappa B-alpha deficient immunocytes in the pathogenesis of the skin disease in a
64 airway epithelium, airway smooth muscle, and immunocytes in the respiratory mucosa, suggesting potent
66 tocompatibility complex class II+, or CD11b+ immunocytes in the skin mesenchyme was increased, and es
67 lly, video microscopic tracing of GFP-tagged immunocytes in the skin of mouse ears reveals that motil
69 iferation, we discovered that intraepidermal immunocytes, including both CD4 and CD8+ T cells, expres
71 lti-layered capsule, which reduced allograft immunocyte infiltrates by enhancement of apoptotic death
73 d MC903-induced itch, epidermal hyperplasia, immunocyte infiltration, and resulted in lower transcrip
78 and acanthosis and introduce targeting nerve-immunocyte/KC interactions as potential psoriasis therap
79 are important in the development of several immunocyte lineages and modulating the immune response.
85 umor metabolic activity and tumor associated immunocytes may be a critical driver of improved clinica
86 m and host immune cells and demonstrate that immunocytes may influence the ability of C. albicans to
87 anding of how KCs communicate with microbes, immunocytes, neurons, and other cells to form an effecti
88 -alpha-syn and vasoactive intestinal peptide immunocytes or natural Tregs administered to MPTP mice a
90 nal mucosa is capable of developing a mature immunocyte population and that exposure to luminal stimu
93 y to develop a mucosal immune system with an immunocyte population similar to that of native small in
94 by unfavorably altering effector:suppressor immunocyte ratios and upregulating PD-1 expression on CD
95 t, we prevented treatment-induced peripheral immunocyte recruitment and, surprisingly, largely ablate
99 tation (ITx) on the basis of altered mucosal immunocytes, rejecting and rejection-free ITx allografts
102 parallel with immunophenotyping of cutaneous immunocytes revealed a distinct dichotomy in atopic derm
104 ller cell activity was observed, and splenic immunocytes secreted copious quantities of IFN-gamma.
105 d defined novel phenotypic markers for these immunocytes (see the related article beginning on page 2
106 automated system and strategy for screening immunocyte-solid tumor interactions, enabling the discov
110 r lavage fluids after challenge, and splenic immunocytes that secreted IL-5 but not gamma interferon
111 driven by proinflammatory cytokines from the immunocytes, the functional role of keratinocytes in the
113 n indirect communicator with other cutaneous immunocytes to enhance defense and potentially contribut
114 by the ability of pathogenetic blood-derived immunocytes to induce secondary activation and disordere
115 invasive labeling and subsequent tracking of immunocytes, to investigate pancreatic infiltrate dynami
116 and HBD2 in mucosal inflammation to include immunocyte trafficking and killing of microbes with the
120 re combined immunodeficient mice, autologous immunocytes were injected into dermis, and the resultant
121 servations in which autologous blood-derived immunocytes were injected into PN skin engrafted onto SC
122 e pancreatic islets by a mixed population of immunocytes, which results in the impairment and eventua