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1 s validated by aptamer screening, ELISA, and immunofluorescence microscopy.
2 assays, genome editing, flow cytometry, and immunofluorescence microscopy.
3 ivation, as determined by flow cytometry and immunofluorescence microscopy.
4 e protein levels were quantified by confocal immunofluorescence microscopy.
5 ing P. gingivalis infection was confirmed by immunofluorescence microscopy.
6 , which all localize to the AZs, as shown by immunofluorescence microscopy.
7 intracellular localization in HeLa cells by immunofluorescence microscopy.
8 t, adherent cell types that are required for immunofluorescence microscopy.
9 chemical markers, histological staining, and immunofluorescence microscopy.
10 APC were demonstrated through histology and immunofluorescence microscopy.
11 using multicolor flow cytometry and confocal immunofluorescence microscopy.
12 as assessed by flow cytometric assays and by immunofluorescence microscopy.
13 Morphologic alterations are shown by using immunofluorescence microscopy.
14 ntent and capillarisation using quantitative immunofluorescence microscopy.
15 transcription-polymerase chain reaction and immunofluorescence microscopy.
16 lls, using deep transcriptome sequencing and immunofluorescence microscopy.
17 ficking, as demonstrated by Western blot and immunofluorescence microscopy.
18 iopsies were studied by Western blotting and immunofluorescence microscopy.
19 O-GlcNAc and ZASP in Western blotting and by immunofluorescence microscopy.
20 visualizes translation in cells via standard immunofluorescence microscopy.
21 s were assessed by western blot and indirect immunofluorescence microscopy.
22 he influence of the labeling density in STED immunofluorescence microscopy.
23 n was evaluated by phospho-flow analysis and immunofluorescence microscopy.
24 muscle actin expression was assessed through immunofluorescence microscopy.
25 rotein epitopes on root cap cell walls using immunofluorescence microscopy.
26 ntent and capillarisation using quantitative immunofluorescence microscopy.
27 ined using PCR, immunoblotting, and confocal immunofluorescence microscopy.
28 T cells were assessed using immunofluorescence microscopy.
29 TNO1 was found to localize to the TGN by immunofluorescence microscopy.
30 ferent developmental stages using RT-PCR and immunofluorescence microscopy.
31 utant were confirmed by PCR, sequencing, and immunofluorescence microscopy.
32 (GLUT4) trafficking, as assessed by means of immunofluorescence microscopy.
33 protein, as determined by flow cytometry and immunofluorescence microscopy.
34 orescence-activated cell sorter analysis and immunofluorescence microscopy.
35 )F4/80(+)NOS2(+) cells by flow cytometry and immunofluorescence microscopy.
36 uman and mouse kidneys by immunoblotting and immunofluorescence microscopy.
37 on of this receptor was verified by confocal immunofluorescence microscopy.
38 8 localization as a helical array of foci by immunofluorescence microscopy.
39 pilin PpdD, as shown by shearing assays and immunofluorescence microscopy.
40 As and cells were analyzed by immunoblot and immunofluorescence microscopy.
41 positive and 20 were IgG-negative by routine immunofluorescence microscopy.
42 tion (PCR) and sequencing, targeted PCR, and immunofluorescence microscopy.
43 GLUT4 protein expression using quantitative immunofluorescence microscopy.
44 ellular assembly by immunohistochemistry and immunofluorescence microscopy.
45 r exposure to CSF-1 for 2.5 min was shown by immunofluorescence microscopy.
46 s, were confirmed by electron microscopy and immunofluorescence microscopy.
47 MP co-receptor hemojuvelin was visualized by immunofluorescence microscopy.
48 rkers WT1 and synaptopodin, as determined by immunofluorescence microscopy.
49 and validated with the established method of immunofluorescence microscopy.
50 terocolitis and measured levels of S100A8 by immunofluorescence microscopy.
51 ild type by immunoblot analysis and confocal immunofluorescence microscopy.
52 ntent and capillarisation using quantitative immunofluorescence microscopy.
53 , as identified through Western blotting and immunofluorescence microscopy.
54 ified in isolated blood lymphocytes by using immunofluorescence microscopy after staining the phospho
57 long with several biochemical approaches and immunofluorescence microscopy analyses, we sought to inv
58 ected by the loss of K7 and western blot and immunofluorescence microscopy analysis revealed that the
60 X to induce microtubule polymerization using immunofluorescence microscopy and a cell-based tubulin p
61 reparations were used in vitro to assess, by immunofluorescence microscopy and Alizarin red staining,
62 cDNA and determined their localization using immunofluorescence microscopy and biochemical assays.
64 ins on intact cells was analyzed by confocal immunofluorescence microscopy and by a novel cross linki
65 es in histone acetylation were determined by immunofluorescence microscopy and chromatin immunoprecip
68 ence of intraerythrocytic bacteria by double-immunofluorescence microscopy and ex vivo gentamicin pro
73 biochemical assays and approaches, including immunofluorescence microscopy and FRET analyses, we demo
75 in beta and ADAM proteases could be shown by immunofluorescence microscopy and immunoprecipitation ex
76 ct the conformation of this polybasic motif, immunofluorescence microscopy and live cell imaging to i
78 alpha- and beta-cells using 3-D confocal and immunofluorescence microscopy and orthogonal analyses.
80 platelets, as assessed biochemically, and by immunofluorescence microscopy and proximity ligation.
82 on of tissue-associated bacteria, histology, immunofluorescence microscopy and scanning electron micr
83 he subcellular localization of protein p6 by immunofluorescence microscopy and show that, at early in
84 tion factories were visualized with confocal immunofluorescence microscopy and single-replicon analys
87 s heterogeneously detected in mESC nuclei by immunofluorescence microscopy and this result correlated
90 ors (VPAC1 and 2) were determined by RT-PCR, immunofluorescence microscopy and Western blot analysis.
92 es and in cultures of human outflow cells by immunofluorescence microscopy and Western blot, respecti
95 , and fibronectin were evaluated by indirect immunofluorescence microscopy and/or Western blot analys
96 Here using RNAi, endogenous epitope tagging, immunofluorescence microscopy, and 3D-structured illumin
97 including FACS analysis, time-lapse imaging, immunofluorescence microscopy, and co-immunoprecipitatio
98 g bioluminescence resonance energy transfer, immunofluorescence microscopy, and co-immunoprecipitatio
99 Green fluorescent protein (GFP) fusions, immunofluorescence microscopy, and cryo-electron tomogra
100 a combination of immunoelectron microscopy, immunofluorescence microscopy, and functional analysis,
101 Ps) was investigated by immunoprecipitation, immunofluorescence microscopy, and HSP knockout using sm
102 hagosomes was confirmed by Western blotting, immunofluorescence microscopy, and immunoelectron micros
103 eruli at light microscopy, one glomerulus at immunofluorescence microscopy, and one glomerulus at ele
105 analysis was evaluated with flow cytometry, immunofluorescence microscopy, and short tandem repeat p
106 y determined by yeast cell agglutination and immunofluorescence microscopy, and the results were corr
107 g [Hh] pathway transcription factor Gli3) by immunofluorescence microscopy; and cilia function using
109 biochemical, subcellular fractionation, and immunofluorescence microscopy approaches to elucidate CP
111 ir ability to bind LPL were assessed with an immunofluorescence microscopy assay and a Western blot a
112 s spectrometric identification, and confocal immunofluorescence microscopy assays, we found that MSec
113 essed Akt membrane translocation as shown by immunofluorescence microscopy but left the concentration
114 d trafficking of Galphas and XLalphas, using immunofluorescence microscopy, cell fractionation, and t
115 g bioluminescence resonance energy transfer, immunofluorescence microscopy, co-immunoprecipitation, a
120 lastoid cell lines, we previously showed how immunofluorescence microscopy could define the distribut
128 y in immature SOD1(G93A) spinal cords and by immunofluorescence microscopy detection of a longer pers
129 ut screens, we recently combined a classical immunofluorescence microscopy detection technique with f
131 nes for the report format, light microscopy, immunofluorescence microscopy, electron microscopy, and
135 evidence for the proposed mechanism includes immunofluorescence microscopy experiments showing co-occ
138 by using quantitative PCR, ELISA, histology, immunofluorescence microscopy, flow cytometry, and metha
139 in the gentamicin protection assays, double-immunofluorescence microscopy, flow cytometry, scanning
140 (0, 2 and 6 h) were analysed using confocal immunofluorescence microscopy for fibre type-specific IM
143 dney-biopsy specimens without IgG by routine immunofluorescence microscopy had IgG specific for Gd-Ig
145 essed by scanning electron microscopy (SEM), immunofluorescence microscopy, histochemistry and quanti
146 s followed by analyzing NSP4 localization by immunofluorescence microscopy identified the 61-83-amino
149 protein-fluorescent vancomycin 2D and 3D-SIM immunofluorescence microscopy (IFM) of cells at differen
150 tein 2D and 3D-SIM (structured illumination) immunofluorescence microscopy (IFM) showed that GpsB and
154 We constructed the system using confocal immunofluorescence microscopy images from the Human Prot
156 fied the presence of RPE65 in lamprey RPE by immunofluorescence microscopy, immunoblot and mass spect
157 Moreover, traditional analyses, including immunofluorescence microscopy, immunoblot, and microplat
158 and proteins were analyzed by using confocal immunofluorescence microscopy, immunoblotting, myelopero
159 ction was demonstrated on the graft sites by immunofluorescence microscopy in 9 of 10 biopsy samples
160 Using selective permeabilization indirect immunofluorescence microscopy in combination with glycos
163 combined Fura-2-based [Ca(2+)]i imaging with immunofluorescence microscopy in isolated split-opened d
165 17 SCID mice, the bacteria are detectable by immunofluorescence microscopy in neutrophils and macroph
166 nd its intracellular adaptor protein Dab2 by immunofluorescence microscopy in placental biopsies from
168 sucrose gradient centrifugation and indirect immunofluorescence microscopy indicated that most ROMK p
170 nuclear antigen (LANA) dots, as detected by immunofluorescence microscopy, indicating a higher viral
172 copy serration pattern analysis and indirect immunofluorescence microscopy knockout analysis are valu
175 d over wild-type), and quantitative confocal immunofluorescence microscopy localized over-expressed v
178 boratory has recently developed quantitative immunofluorescence microscopy methods to measure protein
179 studied by single DNA molecule analyses and immunofluorescence microscopy (molecular combing) showed
181 ride)-split human skin substrate by indirect immunofluorescence microscopy, not diagnosed epidermolys
182 eir validity via parabiosis and quantitative immunofluorescence microscopy of a mouse memory CD8 T ce
183 criteria are usually not sufficient, direct immunofluorescence microscopy of a perilesional biopsy s
184 ratinocyte cell membrane, detected by direct immunofluorescence microscopy of a perilesional biopsy,
185 interaction with the P protein, as shown by immunofluorescence microscopy of cells expressing differ
188 terize the SM-DHX9 interaction, we performed immunofluorescence microscopy of EBV-infected cells and
192 so requires HA trimerization, as revealed by immunofluorescence microscopy of IAV-infected cells and
198 Subcellular localisation was observed by immunofluorescence microscopy of transfected HEK293 cell
201 ns was confirmed by immunohistochemistry and immunofluorescence microscopy on clinical samples and ti
204 romatin has traditionally been studied using immunofluorescence microscopy or biochemical cellular fr
205 e are many third-party data sources, such as immunofluorescence microscopy or protein annotations and
207 plating homogenates onto MacConkey agar, and immunofluorescence microscopy performed using anti-LPS a
208 rase chain reaction, immunoblot analysis, or immunofluorescence microscopy; proliferation was measure
210 olution stimulated emission depletion (STED) immunofluorescence microscopy resolved individual NPCs,
221 ppocampi from chronically stressed rats, and immunofluorescence microscopy revealed redistribution of
235 n gut, ovaries, and Malpighian tubules where immunofluorescence microscopy reveals that AgAQP1 reside
238 functions before DacB in D-Ala removal, and immunofluorescence microscopy showed that DacA and DacB
240 vo and analysis of frozen tissue sections by immunofluorescence microscopy showed that red cells from
241 both cytoplasmic and membrane fractions, and immunofluorescence microscopy showed that septin 7 is ex
243 ption profiling results were corroborated by immunofluorescence microscopy showing increased expressi
250 on (based on gammaH2AX/53BP1 high-resolution immunofluorescence microscopy) that amifostine treatment
252 or flat mount preparations were subjected to immunofluorescence microscopy to detect blood vessels (i
253 We utilized quantitative PCR and double immunofluorescence microscopy to determine that both PPA
254 Here, we employed bioinformatic analysis and immunofluorescence microscopy to examine the physiologic
255 analysis, immunoblotting, HEK293 cells, and immunofluorescence microscopy to identify a histidine-co
256 ed complementary electron cryotomography and immunofluorescence microscopy to investigate the molecul
257 from human kidney tissue biopsy samples, and immunofluorescence microscopy to localize these cells.
259 re, we used a cell-based assay and automated immunofluorescence microscopy to screen 17,700 small mol
261 tion and inhibition tests were combined with immunofluorescence microscopy to show that the Wip1 gene
262 tions, or trypsin digestion and subjected to immunofluorescence microscopy to visualize vessels using
264 bule network was visualized in HeLa cells by immunofluorescence microscopy using Bimolecular Fluoresc
265 examined by immunohistology and quantitative immunofluorescence microscopy using lymphatic endothelia
267 of endogenous CLAMP in IECs was analyzed by immunofluorescence microscopy using total internal refle
268 nuclide-treated patients are quantifiable by immunofluorescence microscopy, using phosphorylation of
279 blotting, immunohistochemistry, and confocal immunofluorescence microscopy, we analyzed the cell surf
286 ermeability assay, FACS, cytokine assay, and immunofluorescence microscopy, we report that obese indi
288 SSP3-specific antibodies in conjunction with immunofluorescence microscopy, we showed that SSP3 is ex
289 g confocal and stimulated emission depletion immunofluorescence microscopy, we showed that VHC-I had
290 scopy, transmission electron microscopy, and immunofluorescence microscopy were performed to ascertai
291 lymerase chain reaction, immunoblotting, and immunofluorescence microscopy were used for expression s
293 inylation, immobilized lectins, and confocal immunofluorescence microscopy were used to characterize
297 luorescent protein (eGFP) fusion protein and immunofluorescence microscopy with anti-BetA antibodies,
300 tegrating transcriptomics and antibody-based immunofluorescence microscopy with validation by mass sp