1  ability of this mutant strain to bind human immunoglobulin D.

2 ells stimulated with dextran-conjugated anti-immunoglobulin D (anti-IgD) antibodies (anti-Ig-dex), a

3 depletion levels by 26 weeks posttransplant; immunoglobulin D(+)CD27(-)-naive B cells predominated.

4 tenuation of BCR responsiveness by promoting immunoglobulin D expression in anergic B cells.

5 1/GFP knock-in mice could be subdivided, and immunoglobulin D(H)-J(H) rearrangements, as well as tran

6                                          The immunoglobulin D (IgD) antibody class was, for many year

7 that include CD80 and CD86 up-regulation and immunoglobulin D (IgD) down-regulation.

8                                              Immunoglobulin D (IgD) is an enigmatic antibody isotype

9                                        Human immunoglobulin D (IgD) occurs most abundantly as a membr

10 esponse; after immunization with antibody to immunoglobulin D (IgD), CD1-deficient mice produced IgE.

11 uency of gammaHV68 infection was observed in immunoglobulin D (IgD)-negative B cells, which was stabl

12 ation of cells characterized as CD19(hi) and immunoglobulin D negative, a phenotype that is consisten

13 rus was able to establish latency in surface immunoglobulin D-negative (sIgD(-)) B cells; by 6 months

14 gammaHV68 is largely confined to the surface immunoglobulin D-negative subset of B cells.

15 receptor associated periodic syndrome; hyper immunoglobulin D syndrome; neonatal onset multisystemic

16 on of CD22, Fcgamma receptor IIb, CD79b, and immunoglobulin D that are down-regulated by cell trigger