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1 DEIA groups, except for level of total serum immunoglobulin E.
2 mpaired in mastocytosis or allergen-specific immunoglobulin E.
3 r "aptamer" designed to bind specifically to immunoglobulin E.
4 d by T helper type 2 cytokines and increased immunoglobulin E.
5 high levels of interleukin (IL)-5, IL-4, and immunoglobulin E.
6 n 5 in the lung, and reduced serum levels of immunoglobulin E.
7 s to common allergens, which are mediated by immunoglobulin E.
8 rface expression by a 4-d preincubation with immunoglobulin E.
9 lergic inflammation, as measured by elevated immunoglobulin E.
10 rmalized circulating levels of cytokines and immunoglobulin E.
11 lycerides,.37; diastolic blood pressure,.21; immunoglobulin E,.63; lipoprotein(a),.77; and body-mass
12 of airway medication, blood eosinophils, and immunoglobulin E (adjusted OR [aOR], 3.56 [95% CI, 2.51-
13                                        Serum immunoglobulin E against eight common inhalant and six f
14  sensitization by specific serum antibodies (immunoglobulin E) against aero-allergens.
15  aggregation of Fc epsilon RI receptors with immunoglobulin E and antigen is mediated through the act
16                                     Specific immunoglobulin E and bronchial hyper-responsiveness were
17  asthma phenotypes, such as high total serum immunoglobulin E and bronchial hyperresponsiveness, have
18 tors, including interleukin-5 (IL-5), IL-13, immunoglobulin E and eosinophils.
19 of asthma in a manner that is independent of immunoglobulin E and eosinophils.
20                                  Total serum immunoglobulin E and FEV1 predicted levels were not asso
21                         We measured specific immunoglobulin E and G4 directed to recombinant A. fumig
22 -alpha-treated mice exhibited elevated serum immunoglobulin E and inhibition of the anticryptococcal
23                                              Immunoglobulin E and its interactions with receptors FcR
24 oded nanopores to detect single molecules of immunoglobulin E and the bioterrorist agent ricin, seque
25 h2 response was accompanied by production of immunoglobulin E and the sensitization of circulating ba
26 eutic approaches, including the targeting of immunoglobulin E and tumour necrosis factor alpha with b
27  a mutant deficient in delta-toxin, promoted immunoglobulin-E and interleukin-4 production, as well a
28 amers against a surface-immobilized protein (immunoglobulin E) and a solution-phase small molecule (b
29 with asthma, AHR, lung function, total serum immunoglobulin E, and blood eosinophil levels.
30 Th2 cell skewing was dependent on basophils, immunoglobulin E, and interleukin-4, but was independent
31 terleukin-5 (IL-5), IL-13, gamma interferon, immunoglobulin E, and mucus-secreting cells.
32           Aptamers for human alpha-thrombin, immunoglobulin E, and platelet-derived growth factor B w
33 e function and development, such as allergy, immunoglobulin E, and Varicella infection status.
34                                         Anti-immunoglobulin E (anti-IgE) (omalizumab), a humanized mo
35  at year 1 and repeated assessments of serum immunoglobulin E antibodies (year 1, 4.5, 6), atopic der
36 xpectedly, FcgammaRIV 'preferentially' bound immunoglobulin E antibodies of the 'b' allotype (IgE(b))
37 allergens in home dust samples, and specific immunoglobulin E antibodies were measured at outset, and
38 ex sensitization (measured by latex-specific immunoglobulin E antibodies) by using data from 5,512 ad
39 intradermal injections of anti-dinitrophenyl immunoglobulin E antibodies.
40 rotocol including measurement of HE-specific immunoglobulin E-antibodies in serum, skin prick tests,
41                                    The human immunoglobulin E antibody responses to the group 1 aller
42 test positivity (SPT), and allergen-specific immunoglobulin E (asIgE) after 3 years of intervention.
43 e poor correlation between allergen-specific immunoglobulin E (asIgE) and clinical signs of allergy i
44 fector cells in allergic disorders and other immunoglobulin E-associated acquired immune responses th
45 on as effector and immunoregulatory cells in immunoglobulin E-associated allergic disorders, as well
46 -6, CXCL8), IL-12, CCL11, thromboxane B2 and immunoglobulin E at 24 h after local allergen challenge.
47 RI subunits for the formation of functional, immunoglobulin E-binding FcepsilonRI complexes during en
48 n-specific T-cell responses by internalizing immunoglobulin E-bound antigens for presentation to anti
49                             Cross-linking of immunoglobulin E-bound FcepsilonRI triggers multiple cel
50 erin ectodomains fused to the Fc fragment of immunoglobulin (E-cad/Fc, N-cad/Fc, and P-cad/Fc) and im
51 llergen specificity, allergen-specific serum immunoglobulin E concentration, or individual laboratory
52                      Allergen-specific serum immunoglobulin E concentrations ranged from 0.1 to 100 k
53 +)) influx; however, unlike that mediated by immunoglobulin-E crosslinking, it did not require the sp
54 49B1 is expressed on mast cells and inhibits immunoglobulin E-dependent activation and inflammation i
55 ved in the recruitment of neutrophils during immunoglobulin E-dependent gastric inflammation in the m
56                                              Immunoglobulin E-dependent gastric inflammation is chara
57 s can exacerbate ADHD symptoms and cause non-immunoglobulin E-dependent histamine release from circul
58 were increased in gastric tissues undergoing immunoglobulin E-dependent inflammation.
59 ond important action of ISS is inhibition of immunoglobulin E-dependent release of Th2 cytokines, esp
60               The high affinity receptor for immunoglobulin E (designated Fc epsilon RI) is the membe
61                      Allergen-specific serum immunoglobulin E detection and quantification have becom
62 andard was carried out for allergen-specific immunoglobulin E determination using the fluoroimmunoenz
63                      Allergen-specific serum immunoglobulin E determination with the fluoroimmunoenzy
64 kin tests and measurement of serum levels of immunoglobulin E do not accurately identify foods for el
65                                 In addition, immunoglobulin-E enhanced delta-toxin-induced mast cell
66 otal immunoglobulin E (TIgE), serum-specific immunoglobulin E, eosinophil count in peripheral blood,
67 osslinking of the high-affinity receptor for immunoglobulin E, F(c)epsilonRI.
68   Aggregation of high-affinity receptors for immunoglobulin E (Fc epsilon RI) on the surface of mast
69 aling through the high affinity receptor for immunoglobulin E (Fc epsilon RI) results in the coordina
70 ss-linking of the high-affinity receptor for immunoglobulin E (FcepsilonRI) results in release of inf
71 ss-linking of the high-affinity receptor for immunoglobulin E (FcepsilonRI).
72 rough their receptors with high affinity for immunoglobulin E (FcepsilonRI).
73 n mediated by the high-affinity receptor for immunoglobulin E, FcepsilonRI, but the molecular basis i
74 he A-B loop of the Cepsilon3 domain of human immunoglobulin E has been carried out.
75 llergens by skin prick and allergen-specific immunoglobulin E (IgE) (ImmunoCAP((R))) tests.
76  associated with increased allergen-specific immunoglobulin E (IgE) against a range of environmental
77                         We measured specific Immunoglobulin E (IgE) against prevalent allergens and g
78 etecting human immunoglobulin G (IgG), human immunoglobulin E (IgE) and Aspergillus fumigatus antibod
79 ory-like', increased serum concentrations of immunoglobulin E (IgE) and caused dendritic cells to pro
80 d candidate genes for association with total immunoglobulin E (IgE) and effect modification by 25-hyd
81 -5 and IL-13, which induce the production of immunoglobulin E (IgE) and eosinophils [1,2].
82 cells express the high-affinity receptor for immunoglobulin E (IgE) and have been linked to host defe
83  mastocytosis and the production of both the immunoglobulin E (IgE) and IgG1 isotypes.
84   About 20 years after the identification of immunoglobulin E (IgE) and its key role in allergic hype
85 , this treatment increases parasite-specific immunoglobulin E (IgE) and other Th2 responses in the mo
86 s, characterized by increases in total serum immunoglobulin E (IgE) and specific serum IgG1 levels an
87                                              Immunoglobulin E (IgE) antibodies and mast cells have be
88                                              Immunoglobulin E (IgE) antibodies are a characteristic f
89                                              Immunoglobulin E (IgE) antibodies are known for triggeri
90                                              Immunoglobulin E (IgE) antibodies are pathogenic in asth
91 isdirected type 2 immune responses, in which immunoglobulin E (IgE) antibodies are produced against a
92                                              Immunoglobulin E (IgE) antibodies play a fundamental rol
93             Exposure to allergens crosslinks immunoglobulin E (IgE) antibodies that are bound to mast
94                           In particular, the immunoglobulin E (IgE) antibody response to the carbohyd
95                          The monoclonal anti-immunoglobulin E (IgE) antibody, omalizumab, was the fir
96 nsor, featuring a highly specific anti-human immunoglobulin E (IgE) aptamer as a capture probe, for h
97 isks) and to assess the oxaliplatin-specific immunoglobulin E (IgE) as a novel diagnostic tool.
98 he first to report high levels of functional immunoglobulin E (IgE) autoantibodies recognizing brain
99                             To elucidate the immunoglobulin E (IgE) binding epitopes in the linear se
100 ne if D-amino acids (D-aas) bind and inhibit immunoglobulin E (IgE) binding to peanut allergens.
101 ), stably cross-links anti-2,4-dinitrophenyl-immunoglobulin E (IgE) bound to high affinity receptors
102 n: FM by plethysmography, total and specific immunoglobulin E (IgE) by automated immunosorbent analys
103 everal extracellular cues, including antigen-immunoglobulin E (IgE) complexes, bacteria, viruses, cyt
104                                  While serum immunoglobulin E (IgE) concentration has been shown to b
105  Framingham Heart Study data on total plasma immunoglobulin E (IgE) concentrations and found a number
106 nts stimulated by the mast cell receptor for immunoglobulin E (IgE) correlated with the affinity of a
107                                              Immunoglobulin E (IgE) exhibits a uniquely high affinity
108       Adoptive transfer of allergen-specific immunoglobulin E (IgE) from atopic donors to nonatopic r
109                                              Immunoglobulin E (IgE) has a major role in the pathogene
110 gths (I) on allergen extractability from and immunoglobulin E (IgE) immunoreactivity of peanut, almon
111 ctivated B lymphocyte increases the level of immunoglobulin E (IgE) in a protein kinase A (PKA)- and
112                 Despite the critical role of immunoglobulin E (IgE) in allergy, circulating IgE+ B ce
113 n of low concentrations of allergen-specific Immunoglobulin E (IgE) in human sera using a Photonic Cr
114 itative immunoassay for the determination of immunoglobulin E (IgE) in human serum using gold nanoclu
115 activation of the high-affinity receptor for immunoglobulin E (IgE) in mast cells.
116  high-affinity Fc receptor (FcepsilonRI) for immunoglobulin E (IgE) in MC/9 mouse mast cells stimulat
117                     Capture and detection of immunoglobulin E (IgE) in simple solution and in human s
118 lper (Th) 2 cells, the physiological role of immunoglobulin E (IgE) in the airway remains largely und
119                                              Immunoglobulin E (IgE) is a central mediator of allergic
120                                  Serum total immunoglobulin E (IgE) is a critical intermediate phenot
121                                              Immunoglobulin E (IgE) is believed to be one of the majo
122       The authors investigated if cord blood immunoglobulin E (IgE) is dependent upon birth order and
123 r-based biosensor for the detection of human immunoglobulin E (IgE) is developed using the electroche
124                                              Immunoglobulin E (IgE) is important in mediating human a
125                                              Immunoglobulin E (IgE) is pathogenic in allergic disease
126 mucosal tissues of allergic reactions, where immunoglobulin E (IgE) is produced locally.
127                                              Immunoglobulin E (IgE) is well known for its role in all
128         B cells expressing antibodies of the immunoglobulin E (IgE) isotype are rare, yet are heavily
129  antibody levels revealed an increase in the immunoglobulin E (IgE) level in immunized mice.
130 and no significant elevation in the systemic immunoglobulin E (IgE) level.
131                               Elevated serum Immunoglobulin E (IgE) levels and increased airway respo
132 study examined the association between serum immunoglobulin E (IgE) levels for a panel of common indo
133 rhinitis, and itchy rash) and serum-specific immunoglobulin E (IgE) levels from the 2005-2006 Nationa
134                   Comparison of OVA-specific immunoglobulin E (IgE) levels in the serum and numbers o
135             Cytokines, chemokines, and total immunoglobulin E (IgE) levels were measured using immuno
136                                  Total serum immunoglobulin E (IgE) levels were significantly lower i
137 ry, maternal and paternal total and specific Immunoglobulin E (IgE) levels, and cord blood IgE were r
138 erresponsiveness (BHR), elevated total serum immunoglobulin E (IgE) levels, and skin tests positive t
139 ed based on increased IL-5 production, total immunoglobulin E (IgE) levels, antigen-specific IgG1 res
140 f intermediate phenotypes, one each on serum immunoglobulin E (IgE) levels, blood eosinophil counts a
141                                  Total serum immunoglobulin E (IgE) levels, for example, show strong
142                                 Canonically, immunoglobulin E (IgE) mediates allergic immune response
143                                              Immunoglobulin E (IgE) mediates its effector functions v
144                                              Immunoglobulin E (IgE) mediates many of the inflammatory
145 estigated how the high-affinity receptor for immunoglobulin E (IgE) modulates the response of mast ce
146 ceptor for the Fc region of antigen-specific immunoglobulin E (IgE) molecules.
147 acebo-controlled clinical trials of the anti-immunoglobulin E (IgE) monoclonal antibody, rhuMAb-E25 (
148 e current study, we used omalizumab, an anti-immunoglobulin E (IgE) monoclonal antibody, to demonstra
149 ividuals is that parasite-induced polyclonal immunoglobulin E (IgE) out-competes allergen-specific Ig
150 omosome 14q was screened for loci modulating immunoglobulin E (IgE) phenotypes in 15 extended and 45
151 ing the serum of the patient showed specific immunoglobulin E (IgE) positivity for wasps; therefore,
152 cell type 2 (Th2) responses leading to hyper-immunoglobulin E (IgE) production and allergy.
153 as suggested an increase in antigen-specific immunoglobulin E (IgE) production during viral infection
154  draining lymph node (DLN) cell infiltrates, immunoglobulin E (IgE) production, and airway hyper-resp
155 igated the relationship between the specific immunoglobulin E (IgE) profile for 40 allergens using a
156                          This study analysed Immunoglobulin E (IgE) profiles to single allergen compo
157           Cross-linkage of the high-affinity immunoglobulin E (IgE) receptor (FcvarepsilonRI) on mast
158 tiated by cross-linking of the high-affinity immunoglobulin E (IgE) receptor FcepsilonRI.
159 abilities, we analyzed a published model for immunoglobulin E (IgE) receptor signaling using syntheti
160                             The low-affinity immunoglobulin E (IgE) receptor, CD23 (FcepsilonRII), bi
161 and basophils that express the high-affinity immunoglobulin E (IgE) receptor, Fc epsilon receptor 1 (
162 ates upon cross-linking of the high-affinity immunoglobulin E (IgE) receptor.
163  ROSA(KIT WT), expressing a fully functional immunoglobulin E (IgE) receptor.
164                                 Targeting of immunoglobulin E (IgE) represents an interesting approac
165                                     Specific immunoglobulin E (IgE) responses are upregulated during
166  by which allergic patients develop specific immunoglobulin E (IgE) responses to environmental allerg
167               Cross-linking of high-affinity immunoglobulin E (IgE) results in the life-threatening a
168 he basis of immune deviation that results in immunoglobulin E (IgE) sensitization and allergic reacti
169                                    Assessing immunoglobulin E (IgE) sensitization to allergens is an
170 ociated with concomitant atopic diseases and immunoglobulin E (IgE) sensitization to food allergens i
171 gher baseline levels of schistosome-specific immunoglobulin E (IgE) than did children with > or =2 re
172                               The binding of immunoglobulin E (IgE) to high affinity IgE receptors (F
173 s is reported for the sensitive detection of immunoglobulin E (IgE) using fluorescence polarization (
174 isease and asthma, and more than 30 yr since immunoglobulin E (IgE) was identified as the key molecul
175 nce and binding properties of an aptamer for immunoglobulin E (IgE) was investigated using custom DNA
176 ns at the site of infection and infection of immunoglobulin E (IgE)(-/-) mice, lead us to suggest tha
177 gnificant reduction in STH prevalence, total immunoglobulin E (IgE), and eosinophil count.
178  CD4-CD8- peripheral T cells, elevated serum immunoglobulin E (IgE), and possible pulmonary inflammat
179  markers of Th2 inflammation, reducing serum immunoglobulin E (IgE), chemokine ligands 13 and 17 by a
180 r, the level of a systemic Th2 marker, serum immunoglobulin E (IgE), correlated significantly with SR
181 cells by phosphorylation of the receptor for immunoglobulin E (IgE), FcepsilonRI, by Lyn kinase after
182 g by antigen, the high affinity receptor for immunoglobulin E (IgE), FcepsilonRI, is phosphorylated b
183 y fever, allergic sensitization, serum total immunoglobulin E (IgE), forced expiratory volume in one-
184 of allergens, and geometric mean serum total immunoglobulin E (IgE), in 3451 participants aged 18-75
185 lpha), IL-6, IL-12/23, IL-17, IL-4/13, IL-5, immunoglobulin E (IgE), integrins and B cells.
186              We know now that this factor is immunoglobulin E (IgE), sensitizing mast cells and basop
187     The distinguishing structural feature of immunoglobulin E (IgE), the antibody responsible for all
188      We identified unique signatures for AD (Immunoglobulin E (IgE), thymus- and activation-regulated
189                         Their activation via immunoglobulin E (IgE)-antigen interactions is promoted
190 orm of treatment for patients suffering from immunoglobulin E (IgE)-associated allergy; the most comm
191 parvalbumin, which was shown to have reduced immunoglobulin E (IgE)-binding capacity upon calcium dep
192 ophil IL-4 production after stimulation with immunoglobulin E (IgE)-containing immune complexes.
193 f LPA were functional, as evidenced by their immunoglobulin E (IgE)-dependent histamine release and b
194                             The structure of immunoglobulin E (IgE)-Fc(3-4) has been solved in three
195 an emphasis on novel FcepsilonRI regulators, immunoglobulin E (IgE)-independent pathways [e.g., Mas-r
196 during pregnancy may reduce the incidence of immunoglobulin E (IgE)-mediated allergic disease.
197                                              Immunoglobulin E (IgE)-mediated anaphylaxis is a potenti
198  of intracellular calcium concentration upon immunoglobulin E (IgE)-mediated crosslinking of the high
199                                    Atopic or immunoglobulin E (IgE)-mediated diseases include the com
200          Atopy is characterized by immediate immunoglobulin E (IgE)-mediated hypersensitivity to agen
201 psilonRI beta chain, a molecule important in immunoglobulin E (IgE)-mediated immunity.
202       Classical itch studies have focused on immunoglobulin E (IgE)-mediated mast cell activation and
203          Allergic rhinitis (AR) is caused by immunoglobulin E (IgE)-mediated reactions to inhaled all
204  a consequence of antigen-aggregation of the immunoglobulin E (IgE)-occupied high affinity receptor f
205                                              Immunoglobulin E (IgE)-sensitization to peanut is common
206 ndent on the acquisition of antigen-specific immunoglobulin E (IgE).
207 bited increased blood levels of Ova-specific immunoglobulin E (IgE).
208 nificant reduction in peanut specific plasma Immunoglobulin E (IgE).
209 y allergen in complex with allergen-specific immunoglobulin E (IgE).
210 erized by substantial increases in levels of immunoglobulin E (IgE).
211 immunoglobulin class switch-recombination to immunoglobulin E (IgE).
212 sthma, atopic dermatitis, and elevated total immunoglobulin E (IgE).
213 llergic reactions that are often mediated by immunoglobulin E (IgE).
214 nsitivity (DTH) to SEA; high levels of serum immunoglobulin E (IgE); a strong T2 cytokine phenotype w
215 ation through the high-affinity receptor for immunoglobulin E (IgE; FcepsilonRI).
216                            The initiation of immunoglobulin-E (IgE)-mediated allergic responses requi
217                                              Immunoglobulins E (IgEs) trigger allergic reactions by s
218 tood, the disease is mediated by an abnormal immunoglobulin-E immune response in the setting of skin
219                Cat- and cockroach-sensitive (immunoglobulin E immunocap [Cap] class > or = 1) women w
220  complete lack of prevailing Sm-p80-specific immunoglobulin E in a high-risk or infected population w
221  maternal parasitemia and levels of PLAP and immunoglobulin E in cord blood.
222 gen-specific serum immunoglobulins and total immunoglobulin E in different groups of animals were mea
223 ecurrent respiratory infections and elevated immunoglobulin E in serum.
224 sis in the jejunum and had reduced ovalbumin-immunoglobulin E in their serum, compared with TSLPR(+/+
225     These studies highlight a mast cell- and immunoglobulin E-independent role for CCR6-bearing T cel
226  essential role in antigen sensitization and immunoglobulin E induction, stimulate dendritic cell acc
227 Also, substantial reductions in plasma total immunoglobulin E, interleukin (IL)-1beta, and tumor necr
228 nificantly correlated with decrease in total immunoglobulin E level (rho = 0.47; P = .04).
229 can data set and incorporate the total serum immunoglobulin E level in the analysis.
230 rd the T helper 2 type, with increased total immunoglobulin E levels (P<.06) and a tendency toward in
231  OVA-specific Th1 to Th2 cells and decreased immunoglobulin E levels after allergen challenge as adul
232 antigen treatments resulted in reduced total immunoglobulin E levels and peripheral blood eosinophil
233 g doxorubicin and trastuzumab, high baseline immunoglobulin E levels are associated with a lower risk
234 tions between multiple VDR polymorphisms and immunoglobulin E levels were also observed (p = 0.006-0.
235 l alkaline phosphatase (PLAP) and polyclonal immunoglobulin E levels were also quantified in paired m
236 h doxorubicin and trastuzumab, high baseline immunoglobulin E levels were associated with a significa
237                                        Serum immunoglobulin E levels were increased in IL-13Ralpha2-/
238 4 production by NKT cells, to increase serum immunoglobulin E levels, and to promote the generation o
239 urrent viral and bacterial infections, hyper-immunoglobulin E levels, eczema, and greater susceptibil
240 allenge with ovalbumin, as measured by total immunoglobulin E levels, ovalbumin-specific immunoglobul
241 iotemporal dynamics of the redistribution of immunoglobulin E-loaded receptors (IgE-FcepsilonRI) on r
242 etion of cytokines involved in regulation of immunoglobulin E, mast cells, basophils and eosinophils,
243 lower immunoglobulin A (median, 1:3,200) and immunoglobulin E (median, 1:128) titers to rAls3p-N by e
244                             Hypersensitivity Immunoglobulin E mediated syndromes are primary immunode
245 ceptor (FcepsilonRI) complex is dedicated to immunoglobulin E-mediated allergic responses.
246 ted with traits underlying asthma and atopy (immunoglobulin E-mediated allergy).
247 d mouse mast cells inhibited antigen-induced immunoglobulin E-mediated cell activation.
248 tive incidence to be 0.34% by 1 year of age; immunoglobulin E-mediated cow's milk allergy was 0.5%.
249                   Rgs13-/- mice had enhanced immunoglobulin E-mediated mast cell degranulation and an
250                     Mast cells contribute to immunoglobulin-E-mediated allergic disorders including a
251 rvoir after antigen capture through specific immunoglobulin E molecules bound to their FcepsilonRI.
252                          Omalizumab, an anti-immunoglobulin E monoclonal antibody, has transformed th
253  FcepsilonRI, the high-affinity receptor for immunoglobulin E, on RBL-2H3 mast cells results in its c
254  was not apparent following plate-bound anti-immunoglobulin E or SEA stimulation.
255                            Allergen-specific immunoglobulin E (present in allergic sensitization) has
256 y enhanced type 2 responses (IL-4, IL-5, and immunoglobulin E production) upon in vitro TCR stimulati
257 mal allergen-induced airway hyperreactivity, immunoglobulin E production, mucus metaplasia, and airwa
258  reduced T2 cytokine production and no serum immunoglobulin E production.
259                  Furthermore, enhancement of immunoglobulin-E production and dermatitis by delta-toxi
260 inst the protein extracts revealed different Immunoglobulin E reactivity of sera according to the ins
261 sera from soybean-sensitive individuals have immunoglobulin E reactivity to abundant storage proteins
262 d performed serological analyses of specific immunoglobulin E reactivity.
263 these cells, cross-linking the high-affinity immunoglobulin E receptor (FcepsilonR1) leads to activat
264                            The high affinity immunoglobulin E receptor (FcepsilonRI) complex is dedic
265  mediators after antigen crosslinking of the immunoglobulin E receptor FcepsilonRI.
266 f macrophages that express the high-affinity immunoglobulin E receptor FcepsilonRIalpha and are in cl
267 on when RBL-2H3 cells are stimulated via the immunoglobulin E receptor, Fc epsilon RI.
268                       CD23, the low-affinity immunoglobulin E receptor, is an important modulator of
269 lso showed functional defects; high-affinity immunoglobulin E receptor-mediated degranulation was sig
270 tive [Lin-]/CD34hi/CD117int/hi/high-affinity immunoglobulin E receptor-positive [FcepsilonRI+]), with
271          We find that antigen stimulation of immunoglobulin E receptors causes much less Orai1/CRACM1
272  (IL)-4 and IL-5 and posttreatment levels of immunoglobulin E recognizing the parasite's tegument (Te
273   Robert L. Coffman recounts how his work on immunoglobulin E regulation along with data from Tim Mos
274 duce iBALTs, which coincided with subsequent immunoglobulin E responses, and IL-1-receptor-deficient
275                                              Immunoglobulin E sensitization was almost exclusively di
276 d treatments of asthma, rhinitis and eczema; immunoglobulin E sensitization; weight; and height.
277 disease, which is frequently associated with immunoglobulin-E sensitization to ubiquitous fungi, typi
278 rgies were excluded by negative results from immunoglobulin E serology analysis and skin tests for co
279 ences in the number of sensitizations, total immunoglobulin E serum levels and predilection of the sk
280 termined by skin prick tests (SPT); specific immunoglobulin E (sIgE) titers against 12 common allerge
281 story rates and peanut- and Ara h 2-specific immunoglobulin E (sIgE) titers were significantly higher
282  to analyze the diagnostic value of specific immunoglobulin E (sIgE) to Gly m 2S albumin, Gly m 4, 5,
283 e, history, skin prick test, peanut specific immunoglobulin E (sIgE), and total IgE minus peanut sIgE
284 s leads to diseases such as cancer and hyper-immunoglobulin E syndrome (HIES).
285 efit from targeted anti-interleukin and anti-immunoglobulin E therapies, and in monitoring subsequent
286 ts for seasonal allergic rhinitis, and alpha-immunoglobulin E therapy for asthma.
287 om inhaled corticosteroids and targeted anti-immunoglobulin E therapy.
288 hacholine (MTCH)-challenge test, serum total immunoglobulin E (TIgE), serum-specific immunoglobulin E
289  immunoglobulin E levels, ovalbumin-specific immunoglobulin E titers, and eosinophil content of bronc
290 sing allergic lung inflammation and elevated immunoglobulin E titers.
291                                     Specific immunoglobulin E to Gly m 2S albumin had the best accura
292                                     Specific immunoglobulin E to Gly m 2S albumin had the highest AUC
293 re added, with oral corticosteroids and anti-immunoglobulin E treatment with omalizumab for the most
294                                              Immunoglobulin E-triggered anaphylactic responses, inclu
295  in serum antigen-specific immunoglobulin G1/immunoglobulin E using ovalbumin or Aspergillus fumigatu
296 d gel-phase membranes displaying ligands for immunoglobulin E, using total internal reflection fluore
297                                              Immunoglobulin E was lower in children with measles, des
298 kers of oxidative stress, thromboxane B2 and immunoglobulin E were measured in bronchoalveolar lavage
299 alloproteinase-9 total, apolipoprotein E and immunoglobulin E--were used along with co-variates in mu
300 t differences between cases and controls was immunoglobulin E, with higher levels detected at baselin

 
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