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1 es where the immune response to pathogens is immunopathological.
2                       We performed extensive immunopathological analyses of airway tissue inflammatio
3                                              Immunopathological analysis of heart tissues obtained at
4                                              Immunopathological analysis revealed that the cells in t
5                               Therefore, the immunopathological and antiviral effects of CTLs can be
6  normally have a positive effect by limiting immunopathological and autoreactive immune responses, bu
7 e combination of the clinical, histological, immunopathological and immunogenetic features indicate t
8             This disease is considered to be immunopathological and mediated primarily by CD4+ T cell
9                  We aimed at identifying the immunopathological and microbial signatures associated w
10 one marrow transplantation (BMT) rescued the immunopathological and microbial signatures.
11 s review summarizes current knowledge on the immunopathological and MRI aspects of ageing in the CNS
12  provide a characterization of the clinical, immunopathological, and transcriptional signature of fat
13 milarities with lesions in humans suggest an immunopathological basis for HGE.
14 irst to demonstrate cellular immunity as the immunopathological basis of periodontal diseases, recurr
15  action of antiviral therapy in ameliorating immunopathological brain injury and ADC.
16       This may protect lymphoid tissues from immunopathological changes but compromise immune defense
17                  To investigate time-related immunopathological changes in the lacrimal glands (LGs)
18 y failure and death, how they relate to lung immunopathological changes remains unclear.
19 g a self-regulatory mechanism that minimizes immunopathological changes.
20           We describe for the first time the immunopathological characteristics of a novel inflammato
21  acid metabolism regulators and examined the immunopathological characteristics of bile ducts.
22 eases, but it remains unknown whether lethal immunopathological characteristics, caused by Schistosom
23 ent strategy for coronavirus infections with immunopathological complications such as SARS-CoV-2, MER
24  shown to ameliorate and reverse an existing immunopathological condition by inducing tolerance.
25 target to modulate unconventional T cells in immunopathological conditions and cancer.
26 derlie the emerging role of NKG2D in various immunopathological conditions and define new therapeutic
27 rge amounts of IL-10, and suppress excessive immunopathological conditions elicited by recall expansi
28 eful therapeutic option for the treatment of immunopathological conditions including graft-versus-hos
29 iciencies (PIDs and SIDs) who presented with immunopathological conditions related to immunodysregula
30           NKT cells also regulate a range of immunopathological conditions, but the mechanisms and th
31 the development of HLH and potentially other immunopathological conditions.
32 r follow-up of 21 patients with clinical and immunopathological confirmed PV, treated with IVIg as mo
33 nses, and the balance between protective and immunopathological consequences during the chronic stage
34 lize a mouse model system to investigate the immunopathological consequences following ZIKV infection
35  focusing which evidence suggests could have immunopathological consequences from cross-reactive reco
36              Our findings thus reveal unique immunopathological consequences of MyD88 signaling in B
37 play a more prominent role in moderating the immunopathological consequences of parasitism, and dampe
38 ion in microbial burden while minimizing the immunopathological consequences of the host inflammatory
39 -MHC restriction and contribute to undesired immunopathological consequences.
40                                              Immunopathological damage in schistosomiasis consist of
41 ital tract to control infection and minimize immunopathological damage to vital reproductive structur
42 ation of a balanced T-cell response limiting immunopathological damage.
43 T cell responses, which sometimes results in immunopathological damage.
44                  Clinical, radiological, and immunopathological data suggest that NMO-IgG might be pa
45 important but poorly understood roles in the immunopathological demyelinating disease multiple sclero
46 urther details on epidemiological, clinical, immunopathological, diagnostic, and therapeutic aspects
47          Infectious mononucleosis (IM) is an immunopathological disease caused by EBV that occurs in
48 st immunity and how control of parasitic and immunopathological disease might be achieved.
49 control lesion severity in a virally induced immunopathological disease.
50 control lesion severity in a virally induced immunopathological disease.
51 illustrate how aberrant immune responses and immunopathological diseases could be generated by EBV in
52 unctions as a protective factor against many immunopathological diseases, such as infection, inflamma
53 unopathogenesis of Sezary syndrome and other immunopathological diseases.
54  implications for understanding and treating immunopathological diseases.
55 various pathological processes, from chronic immunopathological disorders to cognitive and psychiatri
56 in the clinic for the treatment of different immunopathological disorders.
57 maladaptive to the host, owing to the severe immunopathological effects that are brought about by imp
58 effective strategies to circumvent excessive immunopathological effects.
59 nervous system (CNS), is believed to have an immunopathological etiology arising from gene-environmen
60 ons predispose to sepsis, key aspects of the immunopathological events during sepsis, and the long-te
61 mune cells, leading to higher viral load and immunopathological events that augment disease.
62 terns of oligodendrocyte destruction and the immunopathological evidence of complement activation.
63 erns of oligodendrocyte destruction, and the immunopathological evidence of complement activation.
64 timicrobic susceptibility testing or in vivo immunopathological experiments.
65  is a powerful technique for deciphering the immunopathological factors common to multiple individual
66            The concomitant presence of these immunopathological features in the transgenic mice sugge
67 nism of Brucella-induced T-cell responses to immunopathological features is not known.
68 oter polymorphisms and CD4 decline and other immunopathological features like immune activation and C
69         The clinical, immunoserological, and immunopathological features of eligible patients were ev
70 ne the prevalence, prognostic relevance, and immunopathological features of HIC-derived transcriptomi
71 euroinflammatory disorder, recapitulates key immunopathological features of multiple sclerosis.
72 romosomal regions (NOD.Abd3), reproduces the immunopathological features of NOD ABD.
73 propria eosinophil numbers, but not in other immunopathological features, blood neutrophils, FeNO, FE
74  to support the concept of patient-dependent immunopathological heterogeneity in early MS and suggest
75  Multiple sclerosis (MS) lesions demonstrate immunopathological heterogeneity in patterns of demyelin
76 accination strategies targeting allergens or immunopathological key cytokines, illustrate cross-speci
77               Stromal keratitis is a chronic immunopathological lesion of the eye caused by HSV-1 inf
78          Stromal keratitis (SK) is a chronic immunopathological lesion of the eye caused by HSV-1 inf
79 n corneal neovascularization during blinding immunopathological lesion stromal keratitis (SK).
80 e have evaluated the role of T(reg) in viral immunopathological lesion stromal keratitis.
81 se corneas can lead to the development of an immunopathological lesion, termed herpetic stromal kerat
82 tuents impacts the subsequent development of immunopathological lesions.
83                                We found that immunopathological manifestations (IMs) were strongly as
84 haracterized by severe hyperinflammation and immunopathological manifestations in several tissues.
85 the MHC/CD8 complex is considered a specific immunopathological marker because it distinguishes the a
86 distinct sets of such strains did not elicit immunopathological markers at levels above those observe
87                                              Immunopathological mechanisms appear to contribute to th
88 to be elucidated, the evidence suggests that immunopathological mechanisms are involved.
89 mon skin and synovial antigens led to shared immunopathological mechanisms at these different sites,
90 cal manifestations of AMR, and discusses the immunopathological mechanisms contributing to antibody-m
91                                          The immunopathological mechanisms driving the development of
92 om direct ehrlichia-mediated injury but from immunopathological mechanisms initiated by ehrlichial in
93 knowledge of costimulatory molecule-mediated immunopathological mechanisms of uveitis and suggests a
94 al histopathological changes, the underlying immunopathological mechanisms remain largely unexplored.
95                 Dissection of the underlying immunopathological mechanisms revealed the elaborate Tre
96 ing CVB4-V infection is able to suppress the immunopathological mechanisms that lead to chronic disea
97 dings provide new insights into the distinct immunopathological mechanisms underlying airway remodeli
98 eater understanding of time and place in the immunopathological mechanisms underlying human tuberculo
99                                          The immunopathological mechanisms underlying TB-IRIS are inc
100  the present study we aimed to elucidate the immunopathological mechanisms with which the H(4) recept
101 ecent literature in the understanding of the immunopathological mechanisms, diagnosis, and treatment
102 s disease heterogeneity with unknown diverse immunopathological mechanisms.
103 ytokine storm, and lymphopenia, with unknown immunopathological mechanisms.
104 ronic pancreatitis, in this model, is due to immunopathological mechanisms.
105 bundant ELSs and found that they constituted immunopathological microniches wherein malignant hepatoc
106  and highlight the importance of LPDA1 as an immunopathological molecule.
107 lly, this may leave them more susceptible to immunopathological morbidity when attempting to clear ex
108 cutaneous IL-36 signaling is crucial for the immunopathological outcome of various skin diseases.
109 trated that one lung infection modulates the immunopathological outcome to a subsequent unrelated pat
110 hat infect the intestine but differ in their immunopathological outcomes.
111  and disease progression, but the underlying immunopathological pathways are poorly understood.
112                                          The immunopathological pathways enabling post-coronavirus di
113 ntrinsic PDL1 signals regulate non-canonical immunopathological pathways mediating treatment resistan
114                                Four distinct immunopathological patterns have been described in multi
115 y-one patients (95%) showed a persistence of immunopathological patterns in tissue sampled from diffe
116 a profound, interindividual heterogeneity in immunopathological patterns of active demyelination, whi
117                           The structural and immunopathological patterns of demyelination suggest tha
118  active multiple sclerosis lesions show four immunopathological patterns of demyelination.
119 l nervous system is characterized by several immunopathological patterns that suggest the involvement
120     Additionally, inflammatory responses and immunopathological phenotypes in visceral adipose tissue
121 ilies that influence susceptibility to other immunopathological phenotypes, including survival after
122 ntial role of CTLs as final effectors of the immunopathological process in narcolepsy.
123 tax gene expression provokes and sustains an immunopathological process that progressively destroys m
124 gression, and various cytokines increase the immunopathological process.
125                   We conclude that different immunopathological processes (endotypes) may underlie ty
126 c disorders, with genetic loads that reflect immunopathological processes in response to the intestin
127 oprotective processes while limiting ongoing immunopathological processes that cause ongoing CNS dise
128  have emerged as important in the control of immunopathological processes.
129                                 Two distinct immunopathological reaction patterns of lethal COVID-19
130 tution inflammatory syndrome (TB-IRIS) is an immunopathological reaction to mycobacterial antigens in
131 ous system (CNS) is relatively intolerant of immunopathological reactions.
132  may also initiate negative feedback against immunopathological reactions.
133  applied as a means to control virus-induced immunopathological reactions.
134 g-term sustained, clinical, serological, and immunopathological remission.
135  this protozoan parasite induces an abnormal immunopathological response consisting of pancreatic tis
136 dengue virus infection, is believed to be an immunopathological response to a secondary infection wit
137 odeficiency virus pneumonia may represent an immunopathological response to macrophage-tropic virus.
138 immunoinflammatory lesion that represents an immunopathological response to the infection.
139 hat both T-cell subsets were involved in the immunopathological response.
140  environmental promoting factors provokes an immunopathological response.
141  molecular circuits that prevent or regulate immunopathological responses against one parasite is lik
142 egulatory T (T(R)) cells can inhibit harmful immunopathological responses directed against self or fo
143 e modulation that we might apply to diminish immunopathological responses that cause human diseases.
144  development of novel interventions to limit immunopathological responses to infection.
145 is model should generate novel insights into immunopathological responses to viral ocular infection.
146 onses to sequential infection, and excessive immunopathological responses, co-infections are noted by
147 y individuals exhibit apparently maladaptive immunopathological responses.
148 y to differentiate beneficial versus harmful immunopathological responses.
149 efinitive evidence that T(reg) cells have an immunopathological role in human cancer is lacking.
150                       These data confirm the immunopathological role of C5a/C5aR1 signaling in COVID-
151 ersial, but most studies admit the important immunopathological role of infiltrating CD8+ (cytotoxic)
152 has potent proinflammatory actions and plays immunopathological roles in inflammatory diseases, we in
153 ion between chlamydial hsp60 and destructive immunopathological sequelae in infected patients.
154 sis of TBM to discover strategies to prevent immunopathological sequelae.
155  and suggesting the potential development of immunopathological side effects when interfering with th
156           These findings underscore a unique immunopathological signature in HIC, characterized by pl
157              These results identify distinct immunopathological signatures in pCOVID-19 and MIS-C tha
158                                  Clinically, immunopathological staining showed strong PTGR2 expressi
159                                              Immunopathological studies demonstrate the deposition of
160                                              Immunopathological studies indicated that TCRdelta(-/-)
161                                              Immunopathological studies suggest that the target of im
162                                     Advanced immunopathological techniques hold promise for more prec
163 wever, adverse FRC reprogramming can promote immunopathological tissue damage during infection and au
164  include dysregulated antiviral immunity and immunopathological tissue injury.

 
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