ライフサイエンスコーパス: impaired consciousness

1 ndirectly to depressed cortical function and impaired consciousness.

2 patients with acute brain injury (ABI) with impaired consciousness.

3 n had severe malaria; 421/2,208 (19.1%) with impaired consciousness, 665/2,240 (29.7%) with inability

4 ren had severe malaria: 421/2208 (19.1%) had impaired consciousness, 665/2240 (29.7%) had an inabilit

5 Absence seizures are 5-10 s episodes of impaired consciousness accompanied by 3-4 Hz generalized

6 data acquired from 17 patients with severely impaired consciousness and 20 healthy volunteers.

7 nships between frontoparietal slow waves and impaired consciousness and between cortical slowing and

8 ) results in heat intolerance that may cause impaired consciousness and death.

9 The neural underpinnings of impaired consciousness and of the variable severity of b

10 However, the brain mechanisms underlying impaired consciousness and the specific network interact

11 ry outcome was a composite of survival time, impaired consciousness, and functional status at 3 month

12 onary edema, significant bleeding, seizures, impaired consciousness, and laboratory abnormalities suc

13 Coma and other states of impaired consciousness are signs of extensive dysfunctio

14 haviour during and following seizures showed impaired consciousness at the time of SPECT tracer injec

15 Absence seizures are brief episodes of impaired consciousness, behavioral arrest, and unrespons

16 Among those with impaired consciousness (Blantyre Coma Score <=4), transf

17 Age, symptoms at baseline (prostration, impaired consciousness, convulsions, coma), and malaria

18 were assessed, out of which 5 (base deficit, impaired consciousness, convulsions, elevated blood urea

19 eatures of haemorrhagic rash, meningism, and impaired consciousness developed late (median onset 13-2

20 Regarding symptoms, impaired consciousness during initial ICD therapy was pr

21 Here, we tested the hypothesis that impaired consciousness during NREM sleep is associated w

22 In patients with impaired consciousness, epileptic seizure, or temporal l

23 peak spontaneous sudden death and profoundly impaired consciousness following seizures during the sub

24 ous complication of septic encephalopathy is impaired consciousness, for which the patient may requir

25 Impaired consciousness has been incorporated in predicti

26 al analog scale (HR: 1.7; 95% CI: 1.1, 2.6), impaired consciousness (HR: 16.7; 95% CI: 3.1, 90.4), an

27 Impaired consciousness in absence seizures is related to

28 To prevent impaired consciousness in epilepsy, it is necessary to u

29 an experimental treatment for restoration of impaired consciousness in patients with severe acquired

30 Impaired consciousness in temporal lobe seizures has a m

31 al frontoparietal slow waves associated with impaired consciousness in temporal lobe seizures.

32 be crucial for motor manifestations and for impaired consciousness in tonic-clonic seizures.

33 Management of impaired consciousness includes prompt stabilization of

34 high proportion of respiratory arrest (68%), impaired consciousness level (82%) and mechanical ventil

35 Impaired consciousness limited understanding of patients

36 ity observed in two independent cohorts with impaired consciousness: one with arousal-impairing strok

37 Among those with impaired consciousness or hyperlactataemia, transfusion

38 with neurological manifestations (ie, coma, impaired consciousness, or repeated seizures).

39 Impaired consciousness requires altered cortical functio

40 mon clinical reasons for CSF collection were impaired consciousness, seizures, and coma (47%, 22%, an

41 ry of whole-brain irradiation presented with impaired consciousness with or without epileptic seizure