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1 ingle nucleotide polymorphisms may be folded improperly.
2  sporulation when coat assembly is initiated improperly.
3 eral but not all mRNA export mutants produce improperly 3' processed transcripts at the nonpermissive
4 1%, 33% to 49%), and 59 assessed overfitting improperly (39%, 31% to 47%).
5 bsurface are discussed, including sealing of improperly abandoned wells and remediation of hydraulic
6 y regions in cap rocks, such as fractures or improperly abandoned wells, are important considerations
7     In a stam-1 mutant, both LOV-1 and PKD-2 improperly accumulate at the ciliary base.
8 d treatment of malignancies where EGFR-TK is improperly activated.
9 evere side effects in animal and humans when improperly added to food and feed due to its toxicity.
10 phase initiation in mitotic cells containing improperly aligned chromosomes and increases the probabi
11 1 resulted in Golgi fragmentation, short and improperly aligned cisternae, and delayed Golgi reassemb
12 HPC and M-HPC due to steric clashes with the improperly aligned methyl group on the C2 carbon, result
13  strands and cells within those strands were improperly aligned.
14  both KvLQT1 and HERG, and L51H is processed improperly and interacts with neither channel.
15                          Terminology is used improperly, and new terms are created in the process wit
16 enorhabditis elegans has been elusive due to improperly annotated transcription start sites (TSSs).
17          However, the errors associated with improperly applying SSA to estimate C turnover time and
18 family, because if this concept is presented improperly (as implied above), it can suggest that it is
19 database creation that the data they omit or improperly assemble may well contain important biologica
20  in rpn5Delta cells, proteasome subunits are improperly assembled and/or localized.
21 ubules and outer dense fibers and accumulate improperly assembled fibrous sheath proteins.
22 required for the formation of the storage of improperly assembled NPCs (SINC) compartment.
23 d, malformed NPCs accumulate in a storage of improperly assembled nuclear pore complexes compartment,
24 elicits premature germination of spores with improperly assembled protective layers resulting in the
25 the endoplasmic reticulum (ER), misfolded or improperly assembled proteins are exported to the cytopl
26 use individual GABA(B) receptor subunits and improperly assembled receptor complexes are not function
27 he protein that may allow the mutant form to improperly associate with other monomer subunits.
28 venting chromosome segregation in cells with improperly attached chromosomes [1, 2 and 3].
29 w studies show that Aurora B kinase corrects improperly attached chromosomes by recruiting molecules
30 rization and error-correction mechanisms for improperly attached chromosomes.
31                                              Improperly attached kinetochores activate the spindle as
32 nt must detect the presence of unattached or improperly attached kinetochores and must then inhibit p
33  The spindle checkpoint senses unattached or improperly attached kinetochores during mitosis, inhibit
34 ical tension signaling pathway that detaches improperly attached kinetochores from their microtubules
35 ult in recruitment of Mad1:Mad2 complexes to improperly attached kinetochores.
36 ces between SAC activation by unattached and improperly attached kinetochores.
37          When a kinetochore is unattached or improperly attached, the protein kinase Mps1 phosphoryla
38 dant high-curvature OS membranes, which were improperly but specifically organized as ectopic incisur
39       C. dubliniensis isolates may have been improperly characterized as atypical Candida albicans du
40                                              Improperly chosen piecewise models might ignore meaningf
41 g to a substantial amount of unattributed or improperly cited text overlap with other sources.
42 apitulate canonical cellular hierarchies and improperly classify major neuronal cell types.
43 ate high levels of cleaved caspase-3 and are improperly cleared due to an essential caspase-3/caspase
44                      Mitotic chromosomes are improperly condensed in mutants, and polytene chromosome
45 ditions for this critical step that is often improperly conducted.
46 marily Taxiphyllin, posing toxicity risks if improperly consumed.
47 f root hair cells in Arabidopsis thaliana is improperly controlled in root hair-defective rhd4-1 muta
48 t analytically and show that the practice of improperly controlling for covariates is the norm in the
49 nting GAPDH endogenous loading controls were improperly cropped, leading to four lanes of GAPDH endog
50       When cells do not properly interact or improperly decode molecular messages, disease ensues.
51 break in China outside Hubei province; using improperly defined serial intervals in this context bias
52  of causality, and the ecological groups are improperly delineated with regard to the hypothesis.
53 how significant, interpretable deviations in improperly developed brains.
54 efects in the head or tail region of larvae, improperly developed gonad in adult worms and embryonic
55 -function mutations in MET1 and CMT3 develop improperly, display altered planes and numbers of cell d
56 jacent environments if Fluv from CWs will be improperly disposed after usage.
57                                              Improperly distanced reference strains generate CGMs tha
58 hich the peptidyl-transfer RNA 3'-CCA end is improperly docked in the peptidyl transferase center.
59  The proportion of spermatocyst bundles with improperly elongated spermatid nuclei explains 71% of th
60                                           If improperly encapsidated, these sequences have the potent
61  target for microbial immune evasion and, if improperly engaged, a trigger point for various clinical
62  the true FDR well, or at least, it does not improperly favor or disfavor any particular method.
63 tertiary structure, causing the enzyme to be improperly folded and retained within the cell.
64  indicating that these mutant receptors were improperly folded and/or processed and thus retained int
65 only three or five transmembrane domains are improperly folded and/or processed.
66 on of the aberrant tubulins, indicating that improperly folded beta-tubulin is degraded by the protea
67 r quality control by preventing transport of improperly folded glycoproteins out of the ER.
68                           Furthermore, these improperly folded K(+) channels are translocated out of
69                                              Improperly folded membrane proteins are retained in the
70 ction in the periplasm to degrade damaged or improperly folded membrane proteins.
71                                              Improperly folded or denatured proteins tend to aggregat
72                              Accumulation of improperly folded polypeptides in the endoplasmic reticu
73 nerally function as molecular chaperones for improperly folded protein substrates.
74    Many mutations are structural and produce improperly folded protein, explaining their effect in pa
75 level of heme loading is probably not due to improperly folded protein.
76                                              Improperly folded proteins are prevented from exiting th
77 biogenesis and the resulting accumulation of improperly folded proteins in the endoplasmic reticulum
78                                              Improperly folded proteins in the endoplasmic reticulum
79                          The accumulation of improperly folded proteins within the endoplasmic reticu
80 uble forms as well as in inclusion bodies as improperly folded proteins.
81 inal half of this domain was consistent with improperly folded receptor sequences.
82  cellular compartments indicated that GFP is improperly folded when localized outside of the cytoplas
83 eing used to study structure/function may be improperly folded.
84  during stress when kinases are denatured or improperly folded.
85 sion of the estimate of the effect) are used improperly, for example reporting only significant findi
86 dal axon membrane, typically associated with improperly formed paranodes, and in unmyelinated segment
87 isrupting even minor pauses can give rise to improperly formed proteins and human disease.
88 chanistic divergence of EndoS and EndoS2, or improperly-formed domain interfaces in the chimeric enzy
89 be targeted for proteolytic degradation when improperly functioning.
90 r transmit to humans, when their viscera are improperly handled.
91                                    Thus, the improperly held exon cannot access the correct AG, but c
92                    Moreover, LacY also folds improperly in proteoliposomes prepared without YidC.
93 CR6 on the Vgamma3(+) gammadeltaT cells also improperly increased their distribution in dermis of the
94 ing the ability to synthesize ppGpp or polyP improperly initiate chromosome replication, proteolyze t
95      The outer hair cells of the cochlea are improperly innervated and express neuronal markers that
96 duplication defects: The newly formed SPB is improperly inserted into the nuclear envelope (NE), prev
97   Connexin43 (Cx43) and zona occludens-1 are improperly localized in Akap9 mutant testes, and Cx43 fa
98 tructure of the fusome and periodically show improperly localized oocytes, stl mutants do not alter o
99 ective root hair morphology, EYFP-RabA4b was improperly localized or was absent from the tips of root
100 structural and functional characteristics of improperly localized poles, we investigated the connecti
101 heir podosome arrays were loosely packed and improperly localized within the cell.
102 COS1 and Sf9 cells appeared to be misfolded, improperly localized, and proteolytically degraded.
103 t with the cortex upon shrinking and Kar3 is improperly localized, suggesting that Gpa1 is a cortical
104 channels Kv1.1 and Kv1.2 channels were often improperly located in the paranodal axon membrane, typic
105 rate that a nonfunctional circuit containing improperly matched components can evolve rapidly into a
106            However, how immunity elicited by improperly matched vaccines affects the degree of IAV ge
107 more likely to occur in pigs vaccinated with improperly matched vaccines.IMPORTANCEUnderstanding how
108 (Yy1) conditional knockout mice that contain improperly matured dysfunctional astrocytes.
109                     Cytochrome rC(557) is an improperly matured, dimeric cytochrome c obtained from e
110 , intrinsically labile metabolites are often improperly measured or omitted from studies due to unwan
111 MT), likely due to degradation of mRNAs with improperly methylated caps.
112 ucleus and preventing NPAT condensation from improperly occurring in the cytoplasm.
113      N. parisii spores in aaim-1 animals are improperly oriented in the intestinal lumen, leading to
114 biorientation by unleashing chromosomes from improperly oriented microtubule attachments.
115        In addition, we occasionally observed improperly oriented mitotic spindles, residing entirely
116 okinesis defects, prolonged cell cycles, and improperly oriented mitotic spindles.
117         Invagination of these rings produced improperly oriented septa, which in turn gave rise to as
118 a result of undergoing nuclear division with improperly oriented spindles, a large percentage of cdp1
119 inary data showed that naive mice exposed to improperly pasteurized milk containing live H5N1 virus d
120  and the myocardial tissue that does form is improperly patterned and fails to maintain tbx5 expressi
121                    Strikingly, we found that improperly patterned cells in these mice converted to th
122 brain region (r7/r8) had been deleted and/or improperly patterned.
123 al skeleton and skeletal muscle form but are improperly patterned.
124 allele, spo14-S251P, the product of which is improperly phosphorylated and fails to relocalize and re
125  are not consistent with the maps as well as improperly placed water molecules.
126                      Maize tan1 mutant cells improperly position the preprophase band (PPB), which pr
127  along the tubule, whereas nondividing cells improperly position their centrosomes.
128                                              Improperly positioned nuclei are a hallmark of numerous
129                   Loss of kinesin-1 leads to improperly positioned nuclei in culture and in vivo.
130 ntaneously revert to structurally normal but improperly positioned nucleosomes, suggesting a novel me
131 nd to the enzyme with its nicotinamide group improperly positioned to allow turnover, while the affin
132                                We noted many improperly positioned transcription start sites that wer
133 etic evidence indicates that C-13 of DHLA is improperly positioned with respect to Tyr-385 in the V34
134 er uterine segment, caesarean scars may heal improperly predisposing it to be a site of improper impl
135 osis in Idaho occurring after consumption of improperly prepared cougar jerky is described.
136                                              Improperly prepared machacado also may have been served
137 rad50 mutants is the result of unrepaired or improperly processed DSBs in the genome and that Rad50 a
138  of structural RNA and in the degradation of improperly processed pre-mRNA.
139 rveillance pathway that retains and degrades improperly processed precursor mRNAs within the nucleus.
140 d complex carbohydrates, an indication of an improperly processed protein.
141  to a quality-control mechanism that retains improperly processed transcripts at or near sites of tra
142 y, retain the ability to polyadenylate these improperly processed transcripts at the nonpermissive te
143 egradation of certain aberrant RNAs, such as improperly processed transcripts, which are often produc
144 nt determined that the mutant molecules were improperly processed, having been retained in the endopl
145 where Rrp3 is depleted, 35S precursor RNA is improperly processed.
146 oteins were elevated and their synthesis was improperly regulated during mGluR-LTD in FMRP-deficient
147 e pathogen clearance, suggesting an immature/improperly regulated immune response in SMA.
148 unction, structure, and molecular motions in improperly regulated Rad50, which reveal the underlying
149  the control of normal cell growth, and when improperly regulated, in cellular transformation and inv
150 d growth but is a potent oncogenic factor if improperly regulated.
151 epair mechanism, is dangerous to the cell if improperly regulated.
152 or cell homeostasis and growth but lethal if improperly regulated.
153 nd breaks (DSBs) are toxic lesions, which if improperly repaired can result in cell death or genomic
154                                              Improperly repaired chromosomal damage can result in the
155                                     However, improperly repaired DSBs can cause meiotic arrest or mut
156 e cell lethality if unrepaired or cancers if improperly repaired.
157  accurately distinguish bona fide iPSCs from improperly reprogrammed cells.
158 te mitochondrial pool through fusion between improperly retained dysfunctional/senescent and normal m
159 gy termed "mesothermic." However, rates were improperly scaled and phylogenetic, physiological, and t
160  claim that the data used in Sabo et al were improperly scaled to account for fishing effort, thereby
161                                      If left improperly sealed, legacy oil and gas wells colocated wi
162 to a developmental mechanism that eliminates improperly specified cell types.
163 in culture, with a rate of approximately one improperly spliced 23S rRNA per 1.3 million copies.
164 key mediator of Purkinje cell pacemaking, is improperly spliced in RbFox2 and Rbfox1 mutant brains, l
165                                              Improperly spliced mRNA (SV1-CYP3A5) was found only in t
166 oforms (<5% of normal) and the appearance of improperly spliced, chimeric transcripts.
167  activity, as mutation of T181 results in an improperly stabilized protein that accumulates at target
168                              Unsupervised or improperly supervised insertions of the central catheter
169  whereby investors, including professionals, improperly take their personal trading history into acco
170                        Constructs generating improperly terminated transgene messenger RNAs (mRNAs) w
171 rn4 than regular constructs, suggesting that improperly terminated transgene mRNAs not only are degra
172                              We propose that improperly terminated, unpolyadenylated mRNA from transg
173 lls that respond excessively and desensitize improperly to the chemokines CXCL12 and CXCL13.
174 e binding partner Rab11a, P-glycoprotein was improperly trafficked and had decreased presence in the
175 echanism for minimizing the use of poorly or improperly translated mRNAs as templates for the product
176 he prion protein (PrP), are inefficiently or improperly translocated using only these components.
177 gression calibration but with many variables improperly treated as surrogates.
178 gression calibration but with many variables improperly treated as surrogates.
179 lating chromatin structure but are absent or improperly treated in models with rigid histone tails.
180 ients (56.9%) when a balloon was not used or improperly used (P=0.0002).
181 e with different atmospheric components, the improperly used atmospheric component should be responsi
182 mproperly used data, but also the effects of improperly used data on any component of an ML model.
183 the elimination or reduction of not only any improperly used data, but also the effects of improperly
184                                    We also = improperly used their figure without attribution.
185               However, some additives may be improperly used to disguise spoilage, thus resulting in
186 in checkpoint pathways respond to DNA damage improperly, which in turn may enhance the rate of cancer
187 , the C-terminal phenylalanine residue packs improperly with the rest of the hydrophobic core.

 
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