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1 familial transmission (eg, mood disorder and impulsive aggression).
2 n and impulsivity and a composite measure of impulsive aggression.
3 centration were accounted for by measures of impulsive aggression.
4 ygdala circuits have long been implicated in impulsive aggression.
5 t model for gene-environment interactions in impulsive aggression.
6 antisuicidal properties through reduction in impulsive aggression.
7  be mediated by the familial transmission of impulsive aggression.
8 smission of suicidal behavior is mediated by impulsive aggression.
9 s the categorical expression of pathological impulsive aggression.
10 odel for the modulatory role of serotonin in impulsive aggression.
11 irm, refute, or modify our proposed model of impulsive aggression.
12 ne (m-CPP), was examined in 13 subjects with impulsive aggression and 13 normal controls.
13                                              Impulsive aggression and borderline personality disorder
14 ng findings demonstrate associations between impulsive aggression and high serotonin 1B and serotonin
15  To examine further the relationship between impulsive aggression and orbital/medial prefrontal dysfu
16 understudied psychiatric condition marked by impulsive aggression and poorly regulated emotional cont
17 ly treatment of mood disorders and targeting impulsive aggression and sexual trauma may be helpful in
18 order and is also related to the offspring's impulsive aggression and the familial transmission of se
19                                We posit that impulsive aggression and violence arise as a consequence
20 isorder, substance abuse disorder, increased impulsive aggression, and a history of sexual abuse.
21 to psychopathology, suicide attempt history, impulsive aggression, and exposure to familial adversity
22 athologic findings, suicide attempt history, impulsive aggression, and exposure to familial environme
23 bused, such steroids can cause irritability, impulsive aggression, and signs of major depression, but
24 on measures of depressed mood, hopelessness, impulsive aggression, and suicidal behavior, including l
25 uld ensure that clinical features unique for impulsive aggression are appropriately assessed, and we
26 ior, studies of white matter connectivity in impulsive aggression are warranted.
27 spring mood disorder at each time point, and impulsive aggression as a precursor of mood disorder.
28  suicidal behavior include mood disorder and impulsive aggression as well as parental history of suic
29 nd their offspring reported greater lifetime impulsive aggression compared with each of the other two
30        Unlike normal subjects, patients with impulsive aggression did not show activation specificall
31 nin levels as pathophysiologically linked to impulsive aggression has been around for several decades
32                                A disorder of impulsive aggression has been included in DSM since the
33 tanding of the neural mechanisms involved in impulsive aggression has progressed with recent advances
34                                              Impulsive aggression (IA) in adults is associated with b
35 orts the hypothesis that lithium use reduces impulsive aggression in addition to stabilizing mood.
36 se A (MAOA) gene, previously associated with impulsive aggression in animals and humans.
37  Interventions that target mood disorder and impulsive aggression in high-risk offspring may attenuat
38 cture of the central neuromodulatory role of impulsive aggression in human subjects.
39 cture of the central neuromodulatory role of impulsive aggression in human subjects.
40 munoreactivity concentration and measures of impulsive aggression in human subjects.
41              Mood disorder and self-reported impulsive aggression in offspring and a history of sexua
42 ation of inhibitory regions in patients with impulsive aggression in response to a serotonergic stimu
43                             Hopelessness and impulsive aggression independently increase the risk of
44 ic patients with a disorder characterized by impulsive aggression, Intermittent Explosive Disorder (I
45                                              Impulsive aggression is a prevalent problem and yet litt
46 bsequently, renewed interest in disorders of impulsive aggression led to a recent series of community
47 in activity within brain regions involved in impulsive aggression, notably the amygdala, dorsal stria
48                        An increase in either impulsive aggression or hopelessness or a diagnosis of b
49 rs, such as depression, childhood abuse, and impulsive aggression, report inconsistent results.
50  be involved in the association of MAOA with impulsive aggression, suggest neural systems-level effec
51                            In the offspring, impulsive aggression was the most powerful predictor of
52 ation of brain lesions that produce acquired impulsive aggression, we hypothesized that IED subjects
53 idase A genotype specifically predisposes to impulsive aggression, which may be mediated by effects o