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1  using in vivo and chicken embryonic models (in ovo).
2 hibited the replication of avian RNA viruses in ovo.
3 t implementation of this program until ED 14 in ovo.
4 nd chick in generating mouse-chick chimaeras in ovo.
5 nt to 12:12 hr light-dark cycles in vitro or in ovo.
6 ogenous NT-3, during early heart development in ovo.
7 gonists, Noggin, to the developing inner ear in ovo.
8  avian replication-incompetent viral vectors in ovo.
9 n cells and cell processes from around day 7 in ovo.
10 stinguishable from neurons only after day 16 in ovo.
11 ations of chick gut from as early as day 4.5 in ovo.
12 ich resulted in attenuation both in vivo and in ovo.
13  attenuated IBV strain M41, both in vivo and in ovo.
14 bly maintained during passaging in vitro and in ovo.
15 dendritic arborization of spinal motoneurons in ovo.
16  in vitro, and reveals proangiogenic effects in ovo.
17 ons using a heterochronic grafting strategy, in ovo.
18 s and contributed to multiple NC derivatives in ovo.
19 iogenesis in the chorioallantoic (CAM) assay in ovo.
20 ng and/or maintaining optic cup invagination in ovo.
21 cimol, agents that reduce activation of LMNs in ovo.
22 ity at day 5 while inhibiting them at day 14 in ovo.
23  metastatic melanoma cells were transplanted in ovo adjacent to host chick premigratory neural crest
24                          Conversely, chronic in ovo administration of d-tubocurarine, which causes an
25 e presence of axons for survival because the in ovo administration of NMDA, which excitotoxically eli
26 vulina (3-1E) was used to vaccinate chickens in ovo against coccidiosis both alone and in combination
27 hybridization until embryonic day (ED) 14-16 in ovo, analysis of VP expression by RT-PCR showed that
28 eas of the posterior tectum of chick embryos in ovo and analyzed the resulting changes of retinal pro
29 jected at specific sites in the dermomyotome in ovo and the fates of dye-labeled cells monitored by c
30 rowth, development, and metabolic costs both in ovo and upon hatch.
31 mulating muscarinic receptors with carbachol in ovo and was inhibited by blocking muscarinic choliner
32 tically removed at embryonic day (E)5 and E7 in ovo, and GCs were counted over the following 7 days o
33 ptors, its expression in KS lesions, and its in ovo angiogenic properties.
34 he frequency of bursting activity, caused by in ovo application of the GABA(A) receptor blocker, picr
35  vaccine development, with the potential for in ovo application.
36 es against IBV, which have the potential for in ovo application.
37  in this manner are therefore unsuitable for in ovo application.
38                        Gypsy insertion sites in ovo are clustered within a region in the promoter of
39  EF5, administered by intravascular infusion in ovo, as an indicator of relative tissue oxygen concen
40 enes during chick embryo retinal development in ovo, as well as in retinal cell cultures treated with
41 t sources of MN trophic support in vitro and in ovo, but only ACM can rescue MNs after unilateral lim
42 xperiments were carried out in chick embryos in ovo, by intraocular overexpression of noggin, a prote
43                        Depletion of versican in ovo, by RNA interference, results in retinal arbors w
44                            Knockdown of JNK1 in ovo caused defects in spinal cord commissural axon pr
45            SiRNA-mediated knockdown of DSCAM in ovo causes defects in commissural axon projection and
46 ain-like cell arrangements; however, average in ovo cell speeds are as much as 70% faster.
47    When recombinant Del1 was evaluated in an in ovo chick chorioallantoic membrane assay, it was foun
48                           Furthermore, in an in ovo chick chorioallantoic membrane model, overexpress
49 e cell formation and cell delamination after in ovo chicken endoderm electroporation.
50 nd give rise to germ cells both in vitro and in ovo, confirming their germline competence.
51 mbryos, which is consistent with the data of in ovo culture as well as previous studies.
52                                              In ovo, cyclopamine treatment before the secondary heart
53 the brainstem auditory nuclei was studied by in ovo drug administration to chick embryos.
54 cking or slowing this bursting activity, via in ovo drug applications at precise developmental period
55 fied antisense oligonucleotides (antagomiRs) in ovo during chick development.
56 were used to overexpress in the chick retina in ovo either follistatin (FS), an activin-binding prote
57                          The introduction of in ovo electroporation a decade ago has helped the chick
58 ssed Gdf11 in chick embryonic spinal cord by in ovo electroporation and found that ectopic expression
59 -function experiments in chick embryos using in ovo electroporation and found that Shh signaling is r
60 g a combination of in vitro cell culture and in ovo electroporation assays, we demonstrate that GAS1
61  loss-of-function and gain-of-function using in ovo electroporation in chick LMC neurons, of either s
62 l overexpression in the developing retina by in ovo electroporation increases the number of RGCs, whe
63                           We demonstrated by in ovo electroporation of a dominant negative retinoid r
64                                     Finally, in ovo electroporation of axonally targeted GAP-43 mRNA
65                                        Using in ovo electroporation of chick embryos, we show that ec
66 n altered photoreceptor phenotype, while the in ovo electroporation of chicken embryos resulted in fa
67                                              In ovo electroporation of Pax2 into the chick ventral op
68                                        Using in ovo electroporation of PSM cells, we demonstrate that
69 muscle precursor migration, we used targeted in ovo electroporation to express ephrin-A5 ectopically
70 anded RNA (dsRNA) interference combined with in ovo electroporation to knock down RPTP expression lev
71 clin-dependent inducible system Tet-Off with in ovo electroporation to monitor mRNA stability in the
72                      Here, we use unilateral in ovo electroporation together with Atoh1 and Neurog1 e
73    To answer this question we combined focal in ovo electroporation with transposon mediated gene tra
74  gain-of-function studies in the chick using in ovo electroporation, and loss-of-function studies in
75                                        Using in ovo electroporation, we show that misexpressing wild-
76 g EphA4 and disrupting EphA4 signaling using in ovo electroporation.
77 es or in the presumptive neural retina using in ovo-electroporation.
78 ription factor, Chx10, was carried out using in ovo electroporations in chick and transgenic mice.
79   In 1976 an incomplete egg clutch including in ovo embryos of this dinosaur, the oldest known exampl
80 ation (hpf); CYP1 activity was determined by in ovo ethoxyresorufin-o-deethylase (EROD) at 96 hpf, an
81 cities in Xenopus laevis as a consequence of in ovo exposure through maternal transfer of dietary Se.
82                                              In ovo exposure to SeMet increased mortality and deformi
83 PAHs from contaminated sediments, leading to in ovo exposure.
84                                        While in ovo exposures to oil alone did not affect survival, o
85 significant mortality that corresponded with in ovo exposures to sediments containing >=1455 mug/kg t
86 locked glutamatergic or GABA(A) transmission in ovo for 2 days while maintaining relatively normal ne
87 tested in vitro using cells windowed-labeled in ovo for 5 hr on ED 5.
88 todermal cells was traced for the first time in ovo from prestreak stages XI-XII through HH stage 3,
89                                   We used an in ovo function blocking assay in chick and a conditiona
90                                              In ovo functional perturbations of the GJ isoform, conne
91 e double mutations E119V I222V) have similar in ovo growth kinetics and infectivity in guinea pigs.
92 ors suggests that no single mechanism guides in ovo hindbrain neural crest cell migration into the br
93                                              In ovo i.v. injection of PE resulted in deletion of VLR(
94                                              In ovo imaging allowed us to visualize neural crest cell
95 ing neural crest cells over time using a new in ovo imaging technique.
96 ration strategy used in utero in rodents and in ovo in poultry, and apply it to posthatch zebra finch
97 hh expression, using experimental embryology in ovo in the chick.
98  in the chick embryo retina was investigated in ovo, in dissociated and explant cultures, and in cDNA
99                Furthermore, blockade of trkC in ovo induced reductions in subpopulations of DRG neuro
100      Disruption of that gradient in vitro or in ovo induces changes in hair cell morphologies consist
101 rations of MARIA to developing chick embryos in ovo induces precocious expression of M(2) mAChRs in t
102 ugh AY-9944 does not interrupt Shh signaling in ovo, it blocks the response to Shh-N in explants cult
103  this, we performed fate mapping experiments in ovo, labelling at indifferent stages the coelomic epi
104                                              In ovo light exposure yielded strongly lateralized brain
105 ominant chemical species of Se in diets) via in ovo maternal transfer and (2) to investigate the pers
106 nsory stimulation to the embryo (in utero or in ovo) may be crucial for brain development is widespre
107                                              In ovo microinjection of PDGF inhibitor causes a similar
108                                    Following in ovo misexpression of NKL, neuroepithelial cells exit
109                  By blocking HH signaling by in ovo misexpression of Ptc1(Delta)(loop2), we show that
110 erimentally generated hypoganglionic hindgut in ovo model.
111 t to specific differentiated fates, using an in ovo modification of the in vitro "window-labeling" te
112 riod, assessing their effects on activity by in ovo motility measurement and muscle nerve recordings
113                    There were aspects of the in ovo neural crest cell migration patterning which were
114   When neuropilin-1 function is knocked down in ovo, neural crest cells fail to fully invade the bran
115                      Thyroxine (T4) injected in ovo on E9 induces precocious transparency by E12.
116                                              In ovo, onset of gene expression was asynchronous, becom
117 viding an initial glimpse into titanosaurian in ovo ontogeny.
118 ation on OP body compartment composition and in ovo or in utero transfer for any given wildlife speci
119 nt to 12-hour light/12-hour dark (LD) cycles in ovo or in vitro.
120 tive vasculogenic cells in the proepicardium in ovo or tagging dissected proepicardial cells in vitro
121 -function Msx2 expression within rhombomeres in ovo prior to the emigration of cranial neural crest c
122 the M1 layer in mature virions and inhibited in ovo propagation of multiple IAV strains including H1N
123 we show that ectopic expression of ephrin-A5 in ovo reduced arborization of cutaneous axons in skin o
124 fold increase in the efficiency of infection in ovo relative to infection with virus particles bearin
125 al canal of the developing chick spinal cord in ovo resulted in guidance errors for commissural axons
126 in the AV canal and overexpression of Wnt-9a in ovo results in enlarged endocardial cushions and AV i
127 he chicken (Gallus domesticus) and performed in ovo retroviral transduction and plasmid electroporati
128 n axon guidance, we downregulated them using in ovo RNAi and analyzed the trajectory of commissural a
129 ssessing the developmental stage of extinct, in-ovo saurians.
130  this amphibian species is less sensitive to in ovo Se exposure than most of the fish species studied
131  that Raman spectroscopy enables contactless in ovo sex determination of the domestic chicken (Gallus
132            Existing experimental methods for in ovo sexing require taking samples and are applicable
133 iets, and are important in understanding how in ovo stressors (representing maternal stress), affect
134                                              In ovo studies involving shifts from the male- or female
135             In contrast, marking experiments in ovo suggest that the atrioventricular canal, atria an
136                  Here in atrial cells 5 days in ovo, TGFbeta increased carbamylcholine inhibition of
137   In manipulated embryos cultured for 3 days in ovo, the mesonephros as well as the pronephros failed
138 recently in neuronal cell death in vitro and in ovo, the role of specific genes belonging to this fam
139  appear in the pia on embryonic day (E)13-14 in ovo, then along blood vessels extending from the pia
140  (MDCK) cells and grew robustly in vitro and in ovo They had a lower pH of fusion and increased therm
141                           During development in ovo, TIM4 was present on the large majority of macrop
142  observed in F1-generation zebrafish exposed in ovo to 6.8 and 12.7 mug Se/g d.m and raised to adulth
143 rotations, and translocations were performed in ovo to identify the tissues that control inner ear mo
144 current study, 100 ng nicotine applied daily in ovo to yolk during embryonic days (E) 1-7 mimicked ma
145 whole egg (1.2 mug), although depuration via in ovo transfer was substantial.
146 in atrial cells cultured from chicks 14 days in ovo, transforming growth factor beta (TGFbeta) decrea
147                     Through a combination of in ovo transplantation, co-culture and electroporation t
148                                              In ovo treatment significantly affected latency to retur
149 in E11 K(Ca) density was found after chronic in ovo treatment with the neuronal nicotinic antagonist
150 est cells were labeled with DiI and followed in ovo using a new approach for long-term time-lapse con
151  We provide evidence that targeted infection in ovo using adenovirus containing Msx2 and a reporter m
152                 Shh overexpression, achieved in ovo using Shh-encoding retrovirus and in organ cultur
153 hese results provide the first evidence that in ovo vaccination with the recombinant 3-1E Eimeria pro
154 eptor to limited numbers of progenitor cells in ovo, we examined the effects of disrupted trk C signa
155 myocytes cultured from chick embryos 14 days in ovo were used as an in vitro model for ischemic preco
156 also promoted tumor formation and metastasis in ovo, where depleting ILK significantly abrogated the
157 t pathway was identified and tested using an in ovo whole chick embryo culture assay.
158               Chick epicardial cells labeled in ovo with DiI invaded the subepicardial extracellular
159 ival, we chronically treated chicken embryos in ovo with either d-tubocurarine (dTC) or muscimol duri
160              Similarly, treatment of embryos in ovo with exogenous HGF/SF rescues lumbar but not othe
161 oporated channelrhodopsin-2 can be activated in ovo with light flashes to drive waves at precise inte
162  report here that treatment of chick embryos in ovo with muscarinic agonist causes decreases in mRNA
163 ling, chick embryos were chronically treated in ovo with picrotoxin to block GABA(A) receptors, while
164 D, we transduced the embryonic chicken heart in ovo with recombinant adenovirus expressing a death (F
165                   A single-dose immunization in ovo with the att H5N1 vaccine virus in 18-day-old chi
166  in the presomitic mesoderm of chick embryos in ovo, Wnt-1 differentially affects the expression of d

 
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