ライフサイエンスコーパス: in vitro,

1 Of these, 26 were tested in vitro; 2 were not due to lack of recombinant virus re

2 nous expression of TBL1XR1 was combined with in vitro (2D and 3D cultures) and in vivo (mouse lung an

3 0.1 muM] and inhibits tubulin polymerization in vitro; 4) had no effect upon the polymerization of th

4 In vitro, 44 showed biased agonism for ERK1/2 phosphoryl

5 nd intermediate phenomena have been observed in vitro [9-12], but it is unclear how this relates to m

6 In vitro, a 1.4-fold (p < .01) increase in the depositio

7 d MWF stimulated murine B-cell proliferation in vitro, a hallmark cell subtype found in the pathology

8 To reproduce these responses in vitro, a model system using synthetic lipid vesicles

9 e brain permeability and multiple activities in vitro (acetylcholinesterase, butyrylcholinesterase, b

10 In vitro, adhesion depended on fibrinogen and coagulatio

11 ophages to sense and clear opsonized targets in vitro, allowing for potent and targeted suppression o

12 membranes to study lipid-lipid interactions in vitro, alongside optical microscopy techniques aimed

13 te that ArlS has kinase activity toward ArlR in vitro, although it has slower kinetics than other sim

14 In vitro, an apelin receptor agonist analog elicited end

15 d to coaggregate with F nucleatum subspecies in vitro; an effect that was inhibited by blocking or mu

16 y and profibrogenic responses in fibroblast (in vitro) and cardiac fibrosis in mice.

17 ly neutralized SARS-CoV-2 infection of cells in vitro, and a single intranasal prophylactic dose of d

18 arin can bind independently to spike protein in vitro, and a ternary complex can be generated using h

19 hybrid cells, capable of long-term expansion in vitro, and able to reconstitute an anatomic phenocopy

20 cells suppressed proliferation of B6 T cells in vitro, and adoptive transfer into B6 recipients 2 wee

21 cancer (TNBC) and patient-derived TNBC cells in vitro, and attenuates chemotherapy-induced secretion

22 P) are recruited by the CPSF30-hFip1 complex in vitro, and both hFip1 binding sites in CPSF30 can sup

23 an be recapitulated using iPSC-derived cells in vitro, and co-culture platforms are beginning to yiel

24 onstrated this mechanism in human adipocytes in vitro, and correlations from our flux studies in huma

25 1 reduced proliferation of human C-PVR cells in vitro, and curbed growth of freshly isolated human PV

26 mulation and increased lipid droplet numbers in vitro, and decreased ketogenesis and hepatic mitochon

27 ne platform for advanced testing of implants in vitro, and demonstrate the scientific validity and pr

28 SCLC, leads to hypersensitivity to Hh ligand in vitro, and during neural tube development in vivo.

29 , both destabilize microtubules in cells and in vitro, and expression of a rationally designed tubuli

30 tly promotes GBM cell migration and invasion in vitro, and GBP2 silencing by RNA interference exhibit

31 tively kill various malignant HCC cell lines in vitro, and HCC tumors in xenograft and patient-derive

32 es C1q induced hNSC signaling and chemotaxis in vitro, and hNSC migration and functional repair in vi

33 ells (myofibroblast precursors in the liver) in vitro, and IL-1alpha is elevated in the sera and live

34 Here, we investigate M305L actin in vivo, in vitro, and in silico to resolve emergent pathological

35 143)) and VP11/12(483-497), using in silico, in vitro, and in vivo approaches based on the following:

36 In this article, we describe the synthesis, in vitro, and in vivo characterization of a series of ce

37 piratory syndrome coronavirus 2 (SARS-CoV-2) in vitro, and inhibits Middle East respiratory syndrome

38 eus, exhibits decreased rates of acetylation in vitro, and is effective at lowering bacterial load in

39 RSPO2-RA) enhanced Wnt signaling effectively in vitro, and its activity was limited to ASGR1 expressi

40 independent colony formation, cell migration in vitro, and lung metastasis in vivo.

41 amatically increased TNFalpha levels in MDSC in vitro, and neutralizing TNFalpha significantly increa

42 PlsX and membrane phospholipids in vivo and in vitro, and observed that membrane association is nece

43 NA enzymatic activity, neutralize the virus in vitro, and protect against lethal IBV infection in mi

44 determinants for Rab conversion in vivo and in vitro, and reconstitute Rab7 activation with yeast an

45 ited MERS coronavirus (MERS-CoV) replication in vitro, and showed efficacy against Severe Acute Respi

46 sSORLA coprecipitates with EGFR in vitro, and sSORLA treatment increases EGFR Y1173 phos

47 ls or PV(+) cells can trigger SWRs, as shown in vitro, and suggests that PV(+) cell-mediated short-te

48 s cellular susceptibility to viral infection in vitro, and that Snx5 knockout in mice enhances lethal

49 ession of the proteases SspAB and aureolysin in vitro, and that the DeltafakA mutant causes larger le

50 rhangs and directly blocks MRE11 degradation in vitro, and the DNA-binding ability of CST is required

51 analysing synthetic multi-species biofilms, in vitro, and the importance of multi-scale approaches.

52 ures of effector types that can be generated in vitro, and the polarized yet updateable group effecto

53 Newly synthesized compounds were examined in vitro, and their mechanism of action was preliminaril

54 in (CARD) to form filamentous homo-multimers in vitro, and this assembly mediates foci formation and

55 ically relevant, p53-activating chemotherapy in vitro, and this translated into enhanced in vivo effi

56 C-lines might enable us to model the disease in vitro, and to deeper study both the molecular and cel

57 ctivity of polysulfide-inactivated caspase-3 in vitro, and TrxR inhibition potentiated polysulfide-me

58 Importantly, co-cultures studied in vitro, and upon implantation in mice, revealed simila

59 showed markedly reduced growth and invasion in vitro, and were unable to form xenografted tumors.

60 llular domain will rescue the null phenotype in vitro, and will also enhance commitment to differenti

61 ings may be of interest clinically, and this in vitro- and in silico approach could also be applied t

62 tion of (D)CDX-modified liposomes in vivo or in vitro, as assessed with alpha7-knockout mice and by p

63 ls of SOX10 protein in patient-derived cells in vitro, as well as in melanoma patients in vivo.

64 is show p53 dependent enhanced proliferation in vitro, as well as increased susceptibility to inducti

65 complex to regulate its biochemical activity in vitro, as well as overall function in vivo.

66 uding cellular invasion and colony formation in vitro, as well as tumor growth and metastasis in vivo

67 hibits aragonite precipitation from seawater in vitro, at the pH, saturation state and approximate as

68 tes full-length and N-terminal HTT fragments in vitro (at S13/S16), in cells (at S13) and in vivo.

69 In vitro, AZM198 led to a significant reduction in neutr

70 In vitro, beige adipocytes exhibit uncoupled mitochondri

71 In vitro, BLT1(-/-) neutrophils were defective in their

72 cells, which require latency III for growth in vitro, both BHLF1 (-) viruses exhibited variably redu

73 In vitro, both cardiomyocytes and cardiac fibroblasts ex

74 In vitro, both overexpression of mutant COPA and silenci

75 In vitro, both PRC1 and PRC2 can recognize R-loops and o

76 In vitro, both proteins hydrolyzed 2-imino acids (IA) to

77 In germ-like cells in vitro, both serum and PPAR agonist induce PPAR activa

78 In vitro, both tracers displayed preferential binding to

79 Here, we found that during RV infection in vitro, both virus-infected and uninfected bystander c

80 onstrate that vitreous promotes angiogenesis in vitro, but also reveal that Axl is one of receptor ty

81 OMP assembly has been reconstituted in vitro, but assembly reactions have involved the use o

82 michiganensis generally outcompeted E. coli in vitro, but in vivo administration of galactitol-a nut

83 P130CAS mediates VEGF-A and PDGF signalling in vitro, but its cardiovascular function in vivo remain

84 34+ cells differentiated into immature cells in vitro, but less efficiently in vivo.

85 telets have an enhanced response to agonists in vitro, but platelet count and survival are normal.

86 patients gave rise to DCs and LC-like cells in vitro, but the driver mutation was not easily detecta

87 li-DFHBI complexes exhibit high fluorescence in vitro, but they exhibit lower fluorescence in mammali

88 sitive HCC cells and cells infected with HBV in vitro, causing a reduction of hepatitis B e antigen a

89 In vitro, CAV1+ Hu-MuSCs are morphologically distinct, a

90 In vitro, CD4+ T cells from all donors respond to nickel

91 In vitro, cell death was induced in more than 80% of the

92 In vitro, cell lines of murine cholangiocytes and human

93 In vitro, cell proliferation was not affected by GM2/GD2

94 hibitors, respectively) demonstrated synergy in vitro (CI(50) = 0.43) while AZD2014 and afatanib also

95 In vitro, CMP-001 induced cytokine production, including

96 In vitro, COL6 promoted steady-state phosphorylated paxi

97 levels, which inactivates cocultured T cells in vitro, compromises anti-tumor immunity in vivo, and r

98 ncy of mtDNA replication was also reproduced in vitro, confirming the pathogenic mechanism.

99 ance alternative pathway convertase activity in vitro, confirming their pathogenic effect.

100 disrupt G4 structures and remove G4 ligands in vitro, consistent with its role in countering cellula

101 CFAP45 binds AMP in vitro, consistent with structural modelling that iden

102 In vitro, costimulation of PBMC with DR3-specific mAb in

103 In vitro, dACE2, which lacks 356 amino-terminal amino ac

104 In vitro, Dgkzeta deficiency results in reduced producti

105 In vitro, different phenazines can exchange electrons in

106 onceptual advances in the study of durotaxis in vitro, discuss to what extent the evidence suggests d

107 In vitro, division of primary fibroblasts occurs without

108 In vitro, DMOG had no proliferative effect on HC, but co

109 In vitro, dolutegravir and, to a lesser extent, raltegra

110 In vitro, EBV can transform primary human B lymphocytes

111 h was below toxic levels determined for MSCs in vitro (EC(50), 33 uM).

112 In vitro, eCRT strongly induces collagen I, fibronectin,

113 In vitro, ectopic HCLCs emerged in regions medial to inn

114 In vitro, eIF2(alphaP) and ISRIB reciprocally opposed ea

115 In vitro, endothelin-1- and, in vivo, pressure overload-

116 tuted human RNAPII promoter-proximal pausing in vitro, entirely with purified factors (no extracts).

117 th I38T/F/M and examined drug susceptibility in vitro, enzymatic properties, replication efficiency,

118 l peptides to neutralize lethal snake toxins in vitro, establishing a potential route to simple, synt

119 Lys(63)-, and Met(1)-linked ubiquitin chains in vitro, establishing UBA(Cez) as a functional ubiquiti

120 peritoneal microenvironment using a panel of in vitro, ex vivo and in vivo assays.

121 te this model, and functionally validated by in vitro, ex vivo and in vivo techniques.

122 mportant antiangiogenic properties, detected in vitro, ex vivo, and in ovum, most likely contributing

123 inhibition induces activation of T cells in in vitro, ex vivo, and in vivo conditions, irrespective

124 he safety potential of Bald's eyesalve using in vitro, ex vivo, and in vivo models representative of

125 ype 1 (SR-B1) and CD15 on brain SHH MB cells in vitro, ex vivo, and in vivo.

126 In vitro, exogenous miR-370-3p inhibits HCN4 mRNA and ca

127 In vitro, exposure of LDH to HPS with or without TMAO di

128 In vitro, FAAH exhibits a more restricted substrate scop

129 f tECM-driven hASC tenogenic differentiation in vitro, focusing on the integrin and TGF-beta/SMAD pat

130 ko mice produced similar eicosanoid profiles in vitro: for example, formation of TxA(2) , prostagland

131 ar has been shown to bind both AggR and H-NS in vitro, functional significance of these interactions

132 In vitro, GCBCs had a very low glycolytic extracellular

133 In vitro, GNF2133 is able to proliferate both rodent and

134 In vitro, H1 promotes PRC2-mediated H3K27 methylation an

135 15 was a partial agonist in vitro (hA(3)AR, cAMP inhibition, 31% E(max); mA(3)AR,

136 In vitro, HCMV entry and viral progeny are reduced in vi

137 In human macrophages in vitro, heme activates an AMPK (AMP-activated protein

138 08 is essential for high-affinity GR binding in vitro, high tissue uptake in vivo, and efficient pass

139 s were shown to block angiotensin I cleavage in vitro, highlighting their potential for further devel

140 This new Cal/09 LAIV is ts in vitro, highly attenuated (att) in mice, and protects

141 We demonstrate that in vitro, hMGL-4.0 causes tumor cell death, associated w

142 cytes readily produce proplatelet structures in vitro; however, visualization of platelet release fro

143 cers and has been linked to stem cell traits in vitro; however, whether and how TET2 regulates mammar

144 In vitro, HPG is capable of infecting bat and human cell

145 e demonstrate massive expansion of hiPSC-CMs in vitro (i.e., 100- to 250-fold) by glycogen synthase k

146 t inhibits p53 sequence-specific DNA binding in vitro (IC(50) = 180 nM) and in vivo.

147 In vitro, IL11 activated ERK signaling, but inhibited ST

148 In vitro, IL6 treatment of MDSC-like cells activated STA

149 In vitro, imaging-based, cellular studies have defined k

150 o Abeta peptide 1-42 (Abeta(42)) stimulation in vitro, in aging-associated microgliosis in vivo and i

151 erate selective signal retention for imaging in vitro, in cells, and in mice.

152 species, or model conditions such as growth in vitro, in macrophages and in the mouse.

153 n the context of EcoHIV-induced inflammation in vitro, in primary cultures of mouse glial cells and i

154 bitors that demonstrate telomere restoration in vitro, in stem cell models, and in vivo.

155 We also discuss the use of various in vitro, in vivo, and ex vivo models to elucidate the c

156 shown to be individually efficacious in RA (in vitro, in vivo, and/or in humans) and provide a stron

157 measurements made by an eyemate(R)-IO sensor in-vitro, in an artificial and controlled environment.

158 comprehensive protocol that details several in vitro (in bone marrow-derived macrophages) and in viv

159 e conditions that govern HttEx1 polymorphism in vitro, including concentration and the role of the no

160 ntial to differentiate into various lineages in vitro, including osteogenic, chondrogenic, and adipog

161 AV antibody levels from human plasma samples in vitro, including plasma from prospective gene therapy

162 e that they perform key astrocytic functions in vitro, including trophic support of neurons, glutamat

163 GA has shown pleiotropic effects in vitro, including: antitumor effects through inhibitio

164 vo and can dissociate purified 70S ribosomes in vitro, independent of GTP hydrolysis.

165 support the constant development of B cells in vitro, indicating a possible low frequency or exhaust

166 disruption and inflammatory gene expression in vitro, indicating enhanced pathogenic potential of th

167 Its denaturation, either in vivo or in vitro, involves autoxidation to methemoglobin, follow

168 In vitro, irisin (2-10 ng/mL) increased osteoclast diffe

169 h-promoting activity, which is also observed in vitro, is Rab11a-dependent, involves ERK-MAPK-signall

170 tial for the parasite growth and replication in vitro, it contributes to the virulence and growth of

171 st cell shutoff activity of the PA-X protein in vitro, it could not be overridden by frameshift site

172 pha alone less effectively activates Rubisco in vitro, it is not known how CO(2) assimilation and pla

173 on of IL6 expression exerted limited effects in vitro, its attenuation significantly impaired tumor g

174 of an embryo development after fertilization in vitro (IVF), but the available embryo culture medium

175 In vitro, KB105 efficiently infected TGM1-deficient huma

176 In vitro, KLAK-MCP-1 micelles were observed to bind and

177 In vitro, knockdown of GPRC5B in human aortic SMCs resul

178 In vitro, LDH with or without TMAO was exposed to HPS an

179 th the ATR-inhibitor AZD6738 (ceralasertib), in vitro, leading to selective cell death in ATM-deficie

180 PLA2G1B also decreased CD4+ T cell survival in vitro, likely playing a role in CD4 lymphopenia in co

181 In vitro, LPS-stimulated migratory TNFRp55(-/-) DCs of M

182 In vitro, many S. epidermidis isolates stimulated nasal

183 When cocultured with breast cancer cells in vitro, MCs hindered activation of cMET, a master regu

184 is a Modular Platform for Epilepsy Modelling In Vitro (MEMO), a lab-on-chip device, in which three hP

185 In vitro, microglia innately responded to NA-containing

186 In vitro, MinC and MinD are known to coassemble into lin

187 nti-inflammatory/immunosuppressive phenotype in vitro, mirn23a (-/-) mice inoculated with syngeneic t

188 been shown to catalyze reversible reactions in vitro, most of them function unidirectionally in vivo

189 In vitro, murine T cells were stimulated in the presence

190 ce axon outgrowth in a dose-dependent manner in vitro [neurite length PACAP 1065.0 um (285.5), vehicl

191 Finally, in vitro, newborn Tregs exhibited an increased requireme

192 s and provided more sustained release of CsA in vitro, nFIB enhanced cellular uptake and promoted loc

193 Even in vitro, NRF2 activation or prostaglandin E2 supplement

194 In vitro, NUCKS1 stimulates the ATPase activity of RAD54

195 Tested against a panel of GBM cell lines in vitro, paclitaxel was found to be effective at nanomo

196 d so that it behaves as a "super-repressor." In vitro, ParA(R351A) binds and hydrolyzes ATP, and unde

197 In vitro, patient-derived cerebral organoids are smaller

198 In vitro, PDE10A deficiency or inhibiting PDE10A with se

199 th measured drug combination efficacies both in vitro (Pearson's correlation = 0.93 when comparing pr

200 At high concentrations in vitro, PLA2G1B acted alone, independently of gp41, an

201 In vitro, PLK-1 and SPD-2 directly protected centrosome

202 In vitro, podocyte NPY signaling occurred via the NPY2 r

203 e becomes insensitive to warfarin inhibition in vitro, preventing the characterization of inhibition

204 In vitro, protease-activated receptor-2-dependent vascul

205 ffects of mutations on contractile behaviour in vitro, providing new insights into genotype-phenotype

206 In vitro, R406W-desmin formed unusually thick filaments

207 at HIS4 in Sui- cells (Ssu- phenotype); and in vitro, R55G-R57E accelerated dissociation of the eIF2

208 In vitro, RAD52 displays an increased affinity for DNA:R

209 ls do not produce infectious cell-free virus in vitro, raising the question about the mechanism invol

210 740, Olfr741, and Olfr743-which, when tested in vitro, recapitulated OSN responses.

211 ABM300 was characterized in vitro (receptor binding, beta-arrestin2 recruitment,

212 In vitro, recombinant Crm proteins from different orthop

213 In vitro, recombinant FSTL-1 treatment of macrophages at

214 In vitro, recombinant LCN2 induced COL1A1 expression.

215 ExNef were rapidly taken up by neural cells in vitro, reducing the abundance of ABC transporter A1 (

216 In vitro, remdesivir inhibited replication of SARS-CoV-2

217 on enhances CGI DNA binding by PCLs in pairs in vitro, reminiscent of the widespread phenomenon of tr

218 ugh flecainide inhibits single RyR2 channels in vitro, reports have claimed that RyR2 inhibition by f

219 In vitro, restoration of p190A expression in carcinoma c

220 In vitro, rhizolutin substantially decreased Abeta-induc

221 In vitro, SCFAs modulated inflammation in renal tubular

222 In vitro, selumetinib downregulated cellular and membran

223 Ahr-deficient tumours and CPCs grown in vitro, showed elevated activation of the TGFbeta medi

224 transfer PIP into PA-rich membrane bilayers in vitro, suggesting that CPSFL1 potentially facilitates

225 ated zebularine also did not disrupt editing in vitro, suggesting that PPR65 cannot bind modified bas

226 Although it did not form filaments in vitro, Syc2039 from Synechococcus elongatus PCC 7942

227 In vitro, T cells stimulated in the presence of liraglut

228 In vitro, T21 cells cultured with apigenin had significa

229 In vitro, TGFbeta1-mim downregulated TNF-alpha productio

230 In vitro, the addition of MC(TC)LUVA potentiated fetal h

231 y have been previously generated and studied in vitro, the assembly of this multi-synaptic circuit ha

232 The aim of this study was to investigate, in vitro, the effects of a topical estrogen treatment on

233 Unlike previous measurements carried out in vitro, the loops assume well-defined configurations i

234 ted short transient burst of competent state in vitro, the naturally developed competent state was pr

235 In vitro, the onset of gas production was significantly

236 In vitro, the particulate systems tended to promote cell

237 described the generation of beige adipocytes in vitro, their potential utility in cell therapy and dr

238 D1 variant would share similar perturbations in vitro, thereby explain horses' susceptibility to cert

239 ses to mediate vitreous-induced angiogenesis in vitro, thereby providing a molecular basis for remova

240 In vitro, these antibody drug conjugates (ADCs) exhibite

241 In vitro, these antimicrobials had expressed antiviral a

242 vate, and induce epitope presentation in DCs in vitro; they recruit and activate DCs with Th1-dominan

243 In vitro, this inhibitor blocks TLR signalling and infla

244 lations promoted high association with cells in vitro, those formulations containing the fusogenic li

245 ponse when LN cells were challenged with Ova in vitro, though the number of regulatory T cells (Treg)

246 nd, we now show, xyloglucan polysaccharide - in vitro, thus exhibiting CXE (cellulose:xyloglucan endo

247 nd the flanking QTQTN sequence are not fixed in vitro; thus, there appears to be distinct selection p

248 , to examine their behavioral functions, and in vitro, to study their intrinsic properties.

249 In vitro, UNC-87 bundled actin filaments, whereas CLIK-1

250 ARN can decrease its enzymatic activity both in vitro, using a synthetic RNA probe, and in vivo, by q

251 P and CV2 were detected in endothelial cells in vitro, using quantitative real-time polymerase chain

252 rch focused on arsenite exposure in vivo and in vitro, using relevant end points to elucidate potenti

253 In vitro, V2R antagonists OPC31260 and Tolvaptan, or V2R

254 In vitro, VE-cadDEE mutant cells displayed defects in po

255 In vitro, VEGF-A from infected corneas repressed sensory

256 In vitro, VPA-induced oxidative stress was independent o

257 In vitro, we exposed human primary monocytes to (nor)adr

258 Using these models in vivo and in vitro, we fail to observe a requirement for phosphory

259 g voltammetry in brain preparations isolated in vitro, we found that PT stimulation evoked dopamine r

260 To investigate this phenomenon in vitro, we infected primary CD4+ T cells with an HIV c

261 omal cells are exposed to hypoxic conditions in vitro, we observed a striking enhancement in HIF2alph

262 erize the mechanism of LPS-induced secretion in vitro, we reveal an important role for cellular FLICE

263 Using zebrafish and human endothelial cells in vitro, we show ECs deficient in CDP-diacylglycerol sy

264 Using purified erythroid progenitors in vitro, we show that IL-33 directly inhibited terminal

265 In vitro, we show that these effects are largely driven

266 ere restored after incubation with IRL201104 in vitro, when added alone, or in combination with LPS o

267 enesis in the preBotzinger complex (preBotC) in vitro, where experimental tests remain inconsistent w

268 This interaction has been studied in vitro, where mimicking the membrane curvature of the

269 c capacity was assessed by functional assays in vitro, whereas flow cytometry and transcriptomic anal

270 ular weight HA promoted HPASMC proliferation in vitro, whereas pharmacologic inhibition of hyaluronid

271 nM), showed slightly lower affinity for FAP in vitro, whereas plasma protein binding was higher for

272 We tested, in vivo and in vitro, whether DCS-LTD occurs throughout the cortical

273 e in the ability of pDCs to produce IFNalpha in vitro, which correlated with decreased phosphorylatio

274 s sufficient to evict H-NS from promoter DNA in vitro, which is dependent on LuxR DNA binding activit

275 alciparum, cannot be cultivated continuously in vitro, which limits our understanding of its biology

276 s inhibitor (carboxypeptidase B2) activation in vitro, which may contribute to the observed in vivo e

277 ically, neutrophils were able to kill larvae in vitro, which was enhanced by neutralizing Nb-DNase II

278 (with high-efficiency BBB crossing observed in vitro), while competing for the ER/Golgi/PM pathway.

279 feration, and an inability to form spheroids in vitro, while in vivo they generated highly secretory

280 the most abundant Rca isoform by 5 degrees C in vitro, while maintaining the efficiency of Rubisco ac

281 RGE) was shown to eliminate integrin binding in vitro, while mouse genetics revealed that FNRGE prese

282 ugments PDGF-induced Akt and STAT3 signaling in vitro, while next generation sequencing broadly impli

283 osteoblast differentiation and proliferation in vitro, while simultaneously inhibiting osteoclastogen

284 , a classic model for studying primary cilia in vitro, with a genetic dissection of the protein-prote

285 ARS-CoV-2-specific CD8(+) and CD4(+) T cells in vitro, with CD4(+) T cells being robust.

286 positive and Gram-negative bacteria aerosols in vitro, with CFU reductions observed as early as withi

287 ed PGE(2)-induced sensitization was observed in vitro, with differences identified in non-peptidergic

288 re highly sensitive to sacituzumab govitecan in vitro, with IC(50) values in the range of 0.18 to 0.2

289 R T cells showed improved antitumor capacity in vitro, with increased granzyme B and decreased PD-1 e

290 ed a novel HIF2alpha inhibitor, PT2385, both in vitro, with low-passage patient-derived cell lines, a

291 ic arrest) had synergistic antitumor effects in vitro, with minimal effect on normal prostate epithel

292 y one predicted off-target site is cleavable in vitro, with negligible deletions observed in vivo.

293 er domain was important for Pmp22 expression in vitro, with particular impact on a Schwann cell-speci

294 as primordial lung progenitors differentiate in vitro, with some progeny reaching their AEC2 fate tar

295 ant micelles and was monodisperse and stable in vitro, with sufficient structural definition to suppo

296 l cultures of HeLa cells and ovarian tissues in vitro, with superior outcomes than static conventiona

297 Most variants (> 100) were active in vitro, with the fastest having a turnover number of 2

298 significantly induced B cell IgA production in vitro, with the increased expression of genes related

299 iatal neuron action potential burst duration in vitro, without altering other electrophysiological ch

300 We find that, in vitro, Yap/Taz double knockout impairs murine chondro