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1 imulate availability of reducible substrates in vivo).
2 ll egress from the spleen and bone marrow by in vivo (19)F-HDL magnetic resonance imaging.
3                                              In vivo, 31a showed significant target coverage in an ic
4 ity in different incubation media as well as in vivo (57-79% intact radiopeptide in blood of BALB/c m
5                                              In vivo, 5D flow demonstrated moderate agreement with co
6 mical cargoes to chloroplasts in plant cells in vivo (74.6 +/- 10.8%) and more specific tunable chang
7 o and potently elicited lower lip retraction in vivo, a component of "serotonergic syndrome".
8 ssible to determine histamine concentrations in vivo, a nasointestinal catheter with histamine-sensin
9 ruses in cell culture leading to attenuation in vivo, a strategy for making vaccines.
10 erentiation of cortical progenitor behaviors in vivo, a variable we have termed the expansion coeffic
11                                              In vivo, ABM300 did not elicit anxiogenic-like or cannab
12 l hyperplasia response upon carotid ligation in vivo, accompanied by decreased MMP14 activation and i
13 st-ever specific genetic manipulation in PCs in vivo, across immunoglobulin isotypes.
14                                              In vivo, activation of MC(1) leads to anti-arthritic eff
15                                              In vivo, AdA treatment significantly alleviated arthriti
16 o oxidative stress in vitro and during aging in vivo, after which, surprisingly, they undergo G2 arre
17 estingly, however, in transplantation assays in vivo, aged HFSCs regenerated HFs when supported with
18 e microscopical methods for parasite staging in vivo, aiding patient management.
19                                              In vivo, all tracers revealed uptake in activated hPBMCs
20 gel screening layer can therefore be applied in vivo, allowing for the fabrication of highly specific
21 s improve the solubility of food ingredients in vivo, along with enhancement in their bioavailability
22 een shown to detect ALS-associated pathology in vivo, although anatomical patterns of disease spread
23 endent desuccinylation activity in vitro and in vivo, among which the desuccinylation activity of SIR
24                            We addressed this in vivo, analyzing RNAPI in S. cerevisiae.
25 s were highly setting specific (in vitro vs. in vivo) and compartment specific (BAL vs. blood) and lo
26  and structural heterogeneity of mural cells in vivo, and allow detailed cellular studies of the norm
27  mutations on the kinetics of TET2 catalysis in vivo, and allows time-resolved monitoring of target g
28 uppressed macrophage phagocytosis of zymosan in vivo, and antibody blockade of IFN-gamma after endoto
29 observed in animal models of BDNF deficiency in vivo, and BDNF is a common downstream intermediary fo
30 but requires lysobisphosphatidic acid (LBPA) in vivo, and can be reconstituted on supported bilayers
31 strain RN6390 promotes bacterial replication in vivo, and deletion of tspA leads to increased bacteri
32 athogen Aspergillus fumigatus forms biofilms in vivo, and during biofilm growth it has reduced suscep
33 nity GR binding in vitro, high tissue uptake in vivo, and efficient passage across the blood-brain ba
34 nt the evidence suggests durotaxis may occur in vivo, and emphasize the urgent need for in vivo demon
35 We also discuss the use of various in vitro, in vivo, and ex vivo models to elucidate the contributio
36 p, determining cis- and trans-acting lncRNAs in vivo, and generating new developments in high-through
37 orted across cancer types, both in vitro and in vivo, and implicated in multiple processes associated
38                         Preclinical studies, in vivo, and in vitro studies, in combination with mathe
39 , causing regression of the malignant clones in vivo, and inducing molecular remission.
40 harmacokinetic properties, displays activity in vivo, and is projected to have a low human efficaciou
41 nce for the antiviral efficacy of remdesivir in vivo, and its potential benefit in selected patients.
42  local invasion of orthotopic mammary tumors in vivo, and joint up-regulation of Cx43 and ADORA1 in b
43  Decreased Tet2 activity is neuroprotective, in vivo, and may be a new therapeutic target for PD.
44 n Bcl9 affect the expression of TBX3 targets in vivo, and modulation of TBX3 abundance impacts on Wnt
45 VDR knockdown (KD) on mature skeletal muscle in vivo, and myogenic regulation in vitro in C2C12 cells
46 t provide quantification of these components in vivo, and none that can isolate and quantify lipids i
47 andard-of-care antibiotics both in vitro and in vivo, and potentiated the activity of different class
48 ls in vitro, compromises anti-tumor immunity in vivo, and reduces anti-tumor efficacy in an immune-co
49 ence, improves efficacy against DCIS lesions in vivo, and requires 5-fold less CPX to achieve equival
50 tion factors to uniquely spaced DNA elements in vivo, and suggest that differential binding affinitie
51     We demonstrate that PcG targets coalesce in vivo, and that developmentally induced expression of
52 al carbon metabolism in response to exertion in vivo, and that immune cells from trained mice are mor
53 apses are pH sensitive, actuate in vitro and in vivo, and that the electrical signaling is bidirectio
54 lutamate and GABA levels are reduced by FTLD in vivo, and that their deficit is associated with impai
55     BioCer transformed to carbonated apatite in vivo, and the regenerated bone displayed a molecular
56 in vitro and liquid-like nuclear condensates in vivo, and this ability is negatively regulated by Hip
57  lymphocyte (CTL) activity both in vitro and in vivo, and thus promote tumor growth.
58  the pharmacodynamics of cocaine are derived in vivo, and thus this work has widespread implications
59 ctive against C. albicans, both in vitro and in vivo, and to act together with antifungal drugs, sugg
60                CDT proved safe to administer in vivo, and when incorporated into standard frontline c
61 PENDENS (LD) and SET DOMAIN GROUP 26 (SDG26) in vivo, and, together, they prevent accumulation of mon
62 be individually efficacious in RA (in vitro, in vivo, and/or in humans) and provide a strong rational
63 erlie psychosis, compared to SAL rats, using in vivo, anesthetized, electrophysiological recordings.
64 ation within tumours, suboptimal persistence in vivo, antigen escape and heterogeneity, and manufactu
65  although free antitoxin is readily degraded in vivo, antitoxin bound to toxin is protected from prot
66 ERK1/2 phosphorylation, cAMP inhibition) and in vivo (anxiety-like behaviors, cannabimimetic effects,
67 that N6-methyl-dATP is incorporated into DNA in vivo, as indicated by increased N6-methyl-dA DNA leve
68 e show here that CP33B is bound to psbA mRNA in vivo, as was shown previously for CP33C and SRRP1.
69 ATSM signal and levels of reducing molecules in vivo, as well as to evaluate the change in (64)Cu-ATS
70  a critical role in Mn-induced neurotoxicity in vivo, at least in part, by reducing astrocytic GLAST/
71         TrkB.T1 enhances PDGF-driven gliomas in vivo, augments PDGF-induced Akt and STAT3 signaling i
72                     Furthermore, when tested in vivo, azlocillin has shown good efficacy against B. b
73 ntly used as a catalytically inactive mutant in vivo (based on in vitro peptide studies) actually ret
74 s aureus lacking this protease is attenuated in vivo, being rapidly cleared from the airway and leadi
75                                              In vivo, biofilm eDNA can also support rapid electron tr
76                                              In vivo, both MIA-690 and MR-409 induced anxiolytic and
77 ent TNBC in vitro and inhibited tumor growth in vivo, but had no effect on the proliferation of lumin
78 synaptic connectivity in cultured neurons or in vivo, but impaired NMDA-receptor-mediated responses.
79  dermal DCs respond to allergens encountered in vivo, but not in vitro.
80 d-interneuron precursors could differentiate in vivo, but required a prolonged time of four to seven
81 s inhibit cytomegalovirus (CMV) in vitro and in vivo, but their target(s) has been elusive.
82 nd differentiated into ASC in response to Ag in vivo, but this was inhibited in the presence of NP-sp
83 deal technology to view biological processes in-vivo, but current microendoscopic approaches are limi
84 h in vitro, using a synthetic RNA probe, and in vivo, by quantifying endogenous levels of adenylated
85                                              In vivo, cardiomyocytes undergo numerous adaptive struct
86                                     However, in vivo, cartilage-specific knockout of Yap/Taz does not
87                                              In vivo, CAV1+ Hu-MuSCs demonstrated increased engraftme
88                                              In vivo, CD11b(I332G) animals showed a reduction in recr
89                                              In vivo, CEK1, CEK2, and CEK3 exhibited kinase activity
90 uentially linked receptors shows selectivity in vivo, clearing three-antigen tumors while ignoring re
91                                              In vivo, CO(2) -RWE induced stronger allergic lung infla
92                                              In vivo, colchicine blocked MSU-induced recruitment of n
93                                              In vivo, colocalization of actin filaments and divalent
94                                              In vivo, combination treatment decreased tumor vasculatu
95 ries as to how these receptors are activated in vivo, complicating pharmacological advances.
96 and significantly prolonged leukemia control in vivo, confirmed by a second in vivo model using the l
97  dopaminergic signaling by A(1)R-G279S(7.44) in vivo, consistent with a pathogenic role in Parkinson'
98                                              In vivo, daily administration of marketed long-acting in
99                                              In vivo, DECR1 deletion impairs lipid metabolism and red
100                                              In vivo, deletion of all LAR-RPTPs in the hippocampus at
101             Designs were tested in vitro and in vivo, demonstrating alteration of the E2 antigenic pr
102   Strikingly, this methylation is asymmetric in vivo, detected almost exclusively on one DNA strand,
103  bypassing than monosomes, both in vitro and in vivo, due to their preventing formation of a stem-loo
104 ADAR proteins bind dsRNA substrates tandemly in vivo, each with a 50-bp footprint.
105 ar macrophages) are important HIV reservoirs in vivo, especially in the central nervous system (CNS).
106 ssociated with markers of insulin resistance in vivo (euglycemic clamps and HOMA of insulin resistanc
107 Most importantly, NTZ reduces viral shedding in vivo, exhibiting its potential as a future clinical t
108 losteric activator of RodA both in vitro and in vivo, explaining how a SEDS-bPBP complex can coordina
109                                              In vivo, Fas-4-1BB ACT eradicated leukemia and significa
110   These residues covary across HIV-1 viruses in vivo, favouring depletion of PPP2R5A-E.
111    Overall, this work highlights how various in-vivo 'features' such as tumor penetration, cell inter
112                                              In vivo, femoral hematomas resolved completely between d
113                                              In vivo, flecainide effectively suppressed catecholamine
114  analysis of apparent diffusion coefficients in vivo, for example, enables researchers to determine w
115 memory CD8 T cells to undertake gene editing in vivo, for the first time, to our knowledge.
116 wn to be a promising tool, even transplanted in vivo, for transducing light stimuli to non-functionin
117                                              In vivo, functional protein-based condensates are often
118 emain to be fully characterized or confirmed in vivo, giving the field a direction to grow and furthe
119                                              In vivo, global deletion of miR-155 significantly decrea
120                                              In vivo, global PDE10A deficiency significantly attenuat
121                                              In vivo, glycolysis inhibition led to a reduction in kid
122 on are understood to have impacts on utility in vivo, greater granularity with respect to the impacts
123        Despite being successful in vitro and in vivo, HAPs are yet to achieve successful results in c
124 inetics, and effects on endothelial function in vivo, have been reported in humans.
125 mechanisms underlying the binding affinities in vivo, have remained elusive.
126 ng and address how C4A shapes brain circuits in vivo, here, we generated a mouse model with primate-l
127 nesis, but this has not been tested directly in vivo. Here, we investigated synaptic vesicle glycopro
128 served when cells were treated with H(2)O(2) In vivo, high CCL-2 production was detected on hypoxic z
129                                              In vivo, high dietary protein resulted in lower rates of
130 ong-term suppression of neuroblastoma growth in vivo, highlighting the clinical potential of CDK9/2 i
131                                              In vivo, HMGN1 overexpression is linked to decreased qui
132     PT2385 as a single-agent was efficacious in vivo, however, an increase in animal survival was not
133 protein to be long-lived in striated muscles in vivo; however, more rigorous quantitative analysis of
134                                              In vivo, HSCs were activated by repeated CCl(4) administ
135                   Similarly, in tumor models in vivo, hyperpolarized [1-(13)C]pyruvate-to-[1-(13)C]la
136                                              In vivo, hypomethylation of an alternative exon specific
137                                              In vivo, IFNgamma affected neither PD-L1 tumor expressio
138                                              In vivo, IL-24 treatment ameliorated Th17-induced EAU, w
139 nus of Set2p affect H3K36 methylation levels in vivo, illustrating the functional importance of such
140 in primary cultures of mouse glial cells and in vivo, in a mouse model of EcoHIV-associated brain inf
141 tion, superior to glass-ionomer cement alone in vivo, in a rat molar pulpotomy model after six weeks.
142                          This also resulted, in vivo, in a suppression of tumour growth and a decreas
143 e properties and drove CNS axon regeneration in vivo, in part via secretion of a cocktail of growth f
144 ificial bone and other implanted structures (in vivo, in situ, etc).
145                                              In vivo, in the rat parietal cortex, these electrodes co
146 ic understanding of PC biology and pathology in vivo, in their microenvironment.
147 ate exploration of this mechanism in studies in vivo, in wound healing or angiogenesis, in which fibr
148 eptides ex vivo (i.e., to excised tissue) or in vivo (in animals), using antagonists of opioid recept
149 noma cells to radiation therapy in vitro and in vivo (in immunocompetent syngeneic hosts).
150 tro (in bone marrow-derived macrophages) and in vivo (in mice) strategies for activating the inflamma
151 ral dynamic properties of the Reissner fiber in vivo, including embryonic fiber assembly, the continu
152 nhibitors were highly effective in vitro and in vivo, including in imatinib-resistant models.
153 een BRG1 and ARID1A mutant endometrial cells in vivo, including loss of epithelial cell adhesion and
154 stem cell generation, both in time and space in vivo, including the ligand-receptor couple ADM-RAMP2
155 the polarization of macrophages in vitro and in vivo, including the up-regulation of interleukin 6 (I
156 tly prime tumor antigen-specific CD8 T cells in vivo, induce CD8 T cell migration to the tumor site,
157 or antigens sourced from AML cells recruited in vivo) induces local immune-cell infiltration and acti
158                                              In vivo, inhibition of IRE1alpha diminished the intraocu
159                                              In vivo, intraperitoneal injection of an antimiR to miR-
160 ased agonism for ERK1/2 phosphorylation and, in vivo, it preferentially exerted an antidepressant-lik
161 tent decrease in the C. difficile life cycle in vivo, it was able to attenuate an overly robust infla
162 ive capability of the crypt progenitor cells in vivo, lack of crypt base columnar stem cell markers,
163 ive antifibrotic molecules both in vitro and in vivo, leading to improvement in diastolic function in
164                                              In vivo, liver cancer cells but not hepatocytes display
165                       CAR-NKT cells expanded in vivo, localized to tumors and, in one patient, induce
166                                              In vivo, lung metastases developing from orthotopic MDA-
167                                              In vivo, m-RCT was evaluated in mouse models of hypercho
168  and cut dsDNA targets with high specificity in vivo, making it an ideal candidate for expanding the
169 ty to jointly analyze large-scale TF-binding in vivo, making possible the discovery of the potential
170 the pharmacology and toxicology in vitro and in vivo (mice and dogs), and the biodistribution and cle
171 sses activity in both visual thalamic nuclei in vivo, moderate-frequency (10 Hz) stimulation powerful
172                                              In vivo, NatB was seen to preferentially acetylate N ter
173                                              In vivo, neutrophil-specific deletion of genes encoding
174  the role of this inhibitory phosphorylation in vivo, new phosphorylation-deficient p53-S180A knock-i
175                                              In-vivo, newly recruited cells on the vascular lumen exp
176 course of influenza A virus (IAV) infections in vivo, none have considered the impact of both diffusi
177 iated [1-(13)C]pyruvate transmembrane influx in vivo, not glycolytic flux or LDHA activity, driving a
178  cell death of multiple myeloma in vitro and in vivo, offering a therapeutic strategy for this malign
179                                              In vivo, one single topical deposition of CURC-muPLs out
180                                              In vivo, only gain-of-function cancer-associated mutatio
181 re reversed by dichloroacetate (in RVfib and in vivo) or siRNA targeting PDK 1 and 3 (in RVfib).
182                                              In vivo, oxytocin strongly decreased the frequency and p
183                                              In vivo, PEG hydrogels induce local immune responses com
184                                              In vivo, Pfn1 ablation limited regeneration of growth-co
185                                              In vivo, Pi16 (-/-) mice show reduced endothelial barrie
186     To test the effect of these interactions in vivo, pneumococci were preincubated with human sIgA b
187 led to achieve significant target engagement in vivo, possibly because the protein is present in cell
188 ammation, anemia, dyslipidemia, and fibrosis in vivo, potentially by binding to key metabolic regulat
189 ese Tregs display potent regulatory activity in vivo, promoting long-term skin allograft survival in
190 e responsive to porcupine (PORCN) inhibition in vivo, providing clear evidence of RNF43 impairment.
191                                              In vivo, PX treatment caused substantial downregulation
192                                              In vivo, R-VECs implanted subcutaneously in mice self-or
193                                              In vivo, Rag1 (-/-) mice, which lack functional T cells,
194 valuated ex vivo (IPAH-PAAF, IPAH-PASMC) and in vivo (rat chronic hypoxia-induced PH and zebrafish an
195 ng of dopamine with other circuit components in vivo, RdLight1 opens avenues for understanding many a
196 l cord stimulation system provides the first in vivo, real-time, continuous objective measure of spin
197             Debio-1452-NH3 is well tolerated in vivo, reduces bacterial burden in mice and rescues mi
198 s, including how their numbers are regulated in vivo, remains poor.
199                                              In vivo, reperfusion of carotid artery thrombotic occlus
200                                              In vivo, responses to lipopolysaccharide (LPS) result in
201 ed activity consistent with a threshold dose in vivo, resulting in decreased target cell burden, decr
202                                              In vivo, rtPDT induces cellular damage in tumors, shown
203 ells to clonal density, to mimic lung injury in vivo, selects for rare subsets of HBECs that activate
204                                              In vivo, senescence markers were also increased in the b
205 st the effect of C674 oxidation on apoptosis in vivo, SERCA knock-in mice were subjected to chronic a
206                                              In vivo, SHH interference in colon cancer cell lines dec
207 ity, in controlled laboratory conditions and in vivo, should lead to a better understanding of oral d
208 by disturbed flow required Nck1 in vitro and in vivo, showing endothelial Nck1 and IRAK-1 staining in
209                                              In vivo, signal-dependent fluctuations in NS levels are
210                                              In vivo, (SN38 + DACHPt)-loaded micelles displayed super
211     This is due, in large part, to a lack of in vivo, spleen-specific lineage tagging strategies.
212                                              In vivo, studies in the Xenopus system showed that TFG i
213 nd significantly mitigate its immunogenicity in vivo, suggesting an EK peptide cloak as a promising a
214 nalgesic effects in inflammatory pain models in vivo, suggesting potential translational applicabilit
215 gene expression, EMT, and distant metastasis in vivo, suggesting that AR may play a role in distant m
216  in the primary tumor site when reinoculated in vivo, suggesting that these cells are primed to grow
217                                              In vivo, syncytin-1 protein expression was confirmed in
218 comparable anticoagulant effects ex vivo and in vivo (tail-bleeding assay and FeCl(3)-induced thrombo
219 t a series of experiments, both in vitro and in vivo, that reveal previously unrecognized silent pH-s
220 ve anti-inflammatory compound well tolerated in vivo, that shows efficacy in reducing disease in a mo
221 phisticated investigation of neural circuits in vivo, that would otherwise be impossible in completel
222                                              In vivo, the antimicrobial mixture reduced the virus loa
223                                              In vivo, the AtaT2 activity induces ribosome stalling at
224 netics, and photopharmacology, we show that, in vivo, the centrosome's position relative to the nucle
225                                              In vivo, the condensate-forming region of Lge1 is requir
226                                              In vivo, the dictionary method performed over 140-fold f
227                                    Secondly, in vivo, the efficiency of vaccination was improved by p
228                            Both in vitro and in vivo, the expression of pro-inflammatory cytokines (I
229                                              In vivo, the extent of tumor regression induced by combi
230                                              In vivo, the functional form of FtsZ is the polymeric, r
231                                              In vivo, the gel completely degrades after two weeks and
232                                              In vivo, the implants released 348 +/- 107 mug/day (medi
233                       During infusion of ISO in vivo, the incidence of delayed afterdepolarizations (
234                                              In vivo, the knockdown of osa and brahma was shown to en
235  binding able to sustain, for over six weeks in vivo, the localized activity of the clinically licens
236                                              In vivo, the parA (R351A) allele is compromised for part
237 on has been studied extensively in vitro and in vivo, the precise role of cardiac myosin light chain
238      The efficiency of GALC cross-correction in vivo, the role of the GALC substrate galactosylcerami
239        To prevent extraosseous calcification in vivo, the serum protein fetuin-A stabilizes calcium a
240 ow that if fiber formation is at equilibrium in vivo, the vast majority of cells in most tissues woul
241                                              In vivo, the vitamin K postprandial response was higher
242 are collectively effective both in vitro and in vivo, thereby inducing stem cell differentiation.
243                                              In vivo, these groove mutations were found to significan
244                                              In vivo, these memory cells preferentially home to lymph
245                                              In vivo, this can be accomplished with a single dose of
246                                              In vivo, this translated into a tissue-specific vulnerab
247                                              In vivo, this would result in lower ZTL levels at high t
248 bing osteocytes cause the probe to fluoresce in vivo, thus allowing imaging by intravital two-photon
249 h CDKA;1- and CDKB1-containing CDK complexes in vivo, thus promoting endoreplication in developing Ar
250                 Therefore, we recorded BFCNs in vivo, to examine their behavioral functions, and in v
251                                              In vivo, TTK inhibition combined with RT led to a signif
252                                              In vivo, tumour growth of implanted human TNBC cells and
253                                              In vivo, two B mAb-Ds with 77-81% fucosylation cleared r
254 ent portal glucose signaling was identified, in vivo, using a novel (68)Ga-labeled GLP-1r positron-em
255  low-passage patient-derived cell lines, and in vivo, using orthotopic models of glioblastoma.
256 hoblastic acute leukemia growth in vitro and in vivo, validating OGG1 as a potential anti-cancer targ
257    To test for MeCP2 binding to these motifs in vivo, we analysed human neuronal cells using ChIP-seq
258 sess the performance of the trimeric complex in vivo, we compared the ability of L(1p)M-FH and L(1p)T
259 biologic impact of this second-site mutation in vivo, we created a mouse model with the corresponding
260                    To address their efficacy in vivo, we created and used what we believe to be a nov
261 iling upon acute fusion protein inactivation in vivo, we defined the core set of direct transcription
262 sed microenvironmental TGFbeta concentration in vivo, we developed a conditional transgenic mouse mod
263                             To study T cells in vivo, we developed a new knock-in mouse line, which e
264                            Both in vitro and in vivo, we find that RFX6 specifically labels a subset
265        Using the hypoxia marker pimonidazole in vivo, we found that MCs were largely located in the l
266          For effective lymphoma cell killing in vivo, we further functionalized CD22 ligand-modified
267          To assess the functions of miR-145a in vivo, we generated a pericyte-specific miR-145a-knock
268 le of GRK1 phosphorylation in rods and cones in vivo, we generated mutant mice in which Ser21 is subs
269  affects alpha-syn-induced neurodegeneration in vivo, we generated triple transgenic mice that overex
270 ilitate the investigation of miRNA functions in vivo, we have developed a method based on a genetical
271 ating property attributes to synaptic damage in vivo, we have generated adeno-associated viruses AAV-
272                                    Secondly, in vivo, we have investigated its efficacy against infec
273 m to modulate Pten expression in melanocytes in vivo, we highlighted the utility and advantages of ge
274 o mimic the geometry of Spire and Cappuccino in vivo, we immobilized Spire on beads and added Cappucc
275                               To detect DEK1 in vivo, we inserted the tdTomato fluorophore into PpDEK
276                          To investigate this in vivo, we knocked out WAVE1 and WAVE2 genes, individua
277 cells and dendritic cells (DCs) in vitro and in vivo, we map T cell-DC interaction preferences, and d
278                      To test this hypothesis in vivo, we mutated Nae1, an obligative subunit of the E
279                                              In vivo, we observed decreased canonical WNT target gene
280 o define the mechanism of action of beta-Pix in vivo, we optimize single-cell live-embryo imaging, ce
281                                              In vivo, we show that maternal metformin treatment along
282 ls began to emerge to study chlamydial genes in vivo, we speculated as to what degree Tarp function c
283 on during the genesis of partial obstruction in vivo, we tested whether rapamycin could improve persi
284 ubiquitylation and deubiquitination kinetics in vivo, we used a rapid and reversible optogenetic tool
285 hich can induce non-native conformations, or in vivo, where contributions from homologous antenna com
286 ive in vitro and to maintain yeast telomeres in vivo, whereas the DeltaCEH and 1- and 2-bp alleles do
287 ranslates into meaningful metabolic benefits in vivo, wherein the dynamics of insulin signaling and r
288 low and high net oxygenation in the placenta in vivo, which are consistent with efficient delivery of
289 at is enriched at astrocyte-neuron junctions in vivo, which includes neuronal cell adhesion molecule
290 e antigen-specific CD8(+) T-cell populations in vivo, which may serve prognostic and diagnostic roles
291                                              In vivo, while AZD2014 and dasatinib each inhibit tumor
292 geting alloreactive lymphocytes in vitro and in vivo, while sparing resting lymphocytes.
293 ion, and migration in vitro and tumor growth in vivo, while the depletion or inhibition of PRODH bloc
294  vitro and maintain their antiviral activity in vivo, while the glycooligomers exert their inhibitory
295 feration studies in cell lines (in vitro and in vivo) with genetic manipulation, and the adverse prog
296 ction requires at least eight cell divisions in vivo, with BLIMP-1 being required for differentiation
297 holds and competitive landscapes for B cells in vivo, with implications for vaccine design.
298 localization followed by H3K36me3 deposition in vivo, with total H3K36me3 levels correlating with RNA
299                                              In vivo, Yap is indispensable for Sca-1(+) cell expansio
300   Tumour cells adapt to nutrient deprivation in vivo, yet strategies targeting the nutrient poor micr

 
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