戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (1語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1                             These parameters incompletely account for substantial interindividual sus
2 us amount of MS data generated often remains incompletely analyzed due to a lack of sophisticated bio
3 gressive fatal cardiac and skeletal myopathy incompletely attenuated by synthetic GAA intravenous inf
4 adigms for primary cardiovascular prevention incompletely capture a polygenic susceptibility to CAD.
5 is harbours secondary green plastids, but an incompletely characterised proteome precludes accurate u
6 sorders, but their molecular aetiologies are incompletely characterised.
7 d comprise the microvessel structure, remain incompletely characterized, particularly in humans.
8      Factors mediating the residual risk are incompletely characterized.
9 ancer (CRC), but the underlying mechanism is incompletely characterized.
10 main unknown, and the long-term outcomes are incompletely characterized.
11 lopment, but their lineage potential remains incompletely characterized.
12 en receiving antiretroviral therapy (ART) is incompletely characterized.
13 ediators of islet cellular cross talk remain incompletely characterized.
14 cts of proton pump inhibitor (PPI) drugs are incompletely characterized.
15 relative rarity, the natural history remains incompletely characterized.
16 tic autoimmunity to inflammatory disease are incompletely characterized.
17 ARC and the role this plays in hemolysis are incompletely characterized.
18 ns poorly understood and their phenomenology incompletely characterized.
19 aches, but their antiviral mechanisms remain incompletely characterized.
20 eneity and differentiation landscape remains incompletely characterized.
21 s expression during ageing and in AD remains incompletely clear.
22 d indicates persistent inflammation which is incompletely controlled.
23 ge III/IV at diagnosis (48%), are frequently incompletely debulked (44%) and demonstrate inferior sur
24 ion of an arrhythmic MVP (AMVP) form remains incompletely defined and uncertain.
25                                      Through incompletely defined mechanisms, CovR/S and RocA repress
26 or (EF), respectively, play an important yet incompletely defined role in the pulmonary pathophysiolo
27 V infection and the changes over time remain incompletely defined, we examined CMV diversity and chan
28  to induce acute synaptic dysfunction remain incompletely defined, we recently reported that acutely
29 ribed, although indications and outcomes are incompletely defined.
30 ty; however, its genetic determinants remain incompletely defined.
31 ne homeostasis, though the exact origins are incompletely defined.
32 its activity as an extracellular molecule is incompletely defined.
33 that contribute to the fibrotic response are incompletely defined.
34 f these molecules on immune responses remain incompletely defined.
35 ctors that control Th17 pathogenicity remain incompletely defined.
36 s, which, in almost all populations, remains incompletely defined.
37 he mechanisms and tissue sites of action are incompletely defined.
38 transplant recipients in the current era are incompletely defined.
39 specificities of most RBPs remain unknown or incompletely defined.
40 nduced by fungal cell walls in CGD are still incompletely defined.
41 tacatenin nucleocytoplasmic shuttling remain incompletely defined.
42 antigen specificities of CD8-MP cells remain incompletely defined.
43  important in regulating WNV replication are incompletely defined.
44 on between area surgery rates and CSS remain incompletely defined.
45 ance contributes to its pathogenesis through incompletely-defined mechanisms.
46                       The pathophysiology is incompletely delineated.
47 tients free of significant complications are incompletely described.
48 loci vary in effect across space and time is incompletely described.
49 rs and resistors of the PD-1 response remain incompletely elucidated.
50 eptibility to atrial alternans in HF remains incompletely elucidated.
51      Histopathological images are a rich but incompletely explored data type for studying cancer.
52 rocytes contribute to brain disorders remain incompletely explored.
53 witterionic and cationic PFASs, which may be incompletely extracted from subsurface solids by analyti
54 plication in which structural instability of incompletely glycosylated PrP contributes to the conform
55 t, but the necessary brain components remain incompletely identified.
56 helial neoplasias in KPC/Cdh11(+/+) mice and incompletely in KPC/Cdh11(+/-) and KPC/Cdh11(-/-) mice,
57 in an mRNA transcript because 1,N (6)-erA is incompletely incised by RNase H2.
58 itions, consistent with joint molecules with incompletely invaded 3' ends.
59  the association to mortality largely remain incompletely investigated.
60 h wood-decay and fruiting body formation are incompletely known.
61 sk factors develop across the life course is incompletely known.
62 ons were categorized as completely (100%) or incompletely (<=99%) necrotic based on transplant histol
63 ivocal by consensus, with most (five of six) incompletely necrotic at histopathology.
64 s rated as treatment response equivocal were incompletely necrotic at histopathology.
65                 Most real-world networks are incompletely observed.
66 zygotes showed a palate elevation delay with incompletely penetrant cleft palate.
67  transcript missing exon 3, suggestive of an incompletely penetrant splice defect.
68 al, double heterozygous embryos exhibited an incompletely penetrant syndrome of CHDs with heterotaxy.
69 ification among individuals with highly, but incompletely, penetrant genetic variants.
70 ings highlight that aPL testing may often be incompletely performed.
71  metabolic interactions that were falsely or incompletely predicted by existing methods, including pr
72 neuroinflammation were significantly, though incompletely, prevented.
73 est a critical function of FANCJ to suppress incompletely processed and toxic joint DNA molecules dur
74  E. coli tol-pal mutants are connected by an incompletely processed peptidoglycan (PG) layer.
75 efits they provide are poorly understood and incompletely quantified.
76 the next event.Objectives: To assess whether incompletely recovered COPD exacerbations benefit from a
77                      Missing data were often incompletely reported, and continuous predictors were co
78 il to capture such transitions and therefore incompletely represent care outcomes over time.
79  higher frequency of DNA breaks in PSCs with incompletely reprogrammed DNA replication were found.
80 fferentiation potential, DNA replication was incompletely reprogrammed, and genomic instability incre
81  colorectal polyps 1 to 20 mm are frequently incompletely resected, and that risk increases for polyp
82  organized thrombotic material stemming from incompletely resolved acute pulmonary embolism.
83 ether further therapy with ciprofloxacin for incompletely resolved COPD exacerbations prolonged the t
84 re formed and regulated within cells remains incompletely resolved.
85 ation of hypothalamic hunger neurons that is incompletely reversed by weight loss and may contribute
86 is exported from the nucleus in unspliced or incompletely spliced forms to serve as templates for tra
87 a flexneri or Salmonella Typhimurium remains incompletely understood [5, 6].
88 thelial regeneration and vascular repair are incompletely understood and constitute an emerging area
89              However, the pathophysiology is incompletely understood and specific therapy is lacking.
90                              The reasons are incompletely understood as potential underlying pathomec
91 that govern fetomaternal tolerance are still incompletely understood but may provide critical insight
92 ain unclear due to complex, fluctuating, and incompletely understood developmental conditions.
93 the factors that govern host tropism remains incompletely understood for most pathogens.
94                                        It is incompletely understood how cancer cells escape NK cell
95                                   It remains incompletely understood how TNF can lead to different di
96 ved around the time of fertilization through incompletely understood mechanisms.
97 risk factor for many human disorders through incompletely understood mechanisms.
98 ients in the recent DAPAHF clinical trial by incompletely understood mechanisms.
99 iomyopathies, which promote heart failure by incompletely understood mechanisms.
100 olution of experimental lung inflammation by incompletely understood mechanisms.
101 scular endothelial cell (EC) interactions by incompletely understood mechanisms.
102 ugh different pathways and have distinct but incompletely understood morphogenetic functions during c
103 is a devastating gastrointestinal disease of incompletely understood pathophysiology predominantly af
104 nt.IMPORTANCE Nonenveloped virus entry is an incompletely understood process.
105 nderstanding better both nucleases and their incompletely understood roles in vitro and in vivo.
106 tional yet structurally and biosynthetically incompletely understood varieties of melanin: eumelanin,
107  pneumoniae to cause infection, but it is an incompletely understood virulence trait.
108 le of these pathways in different tissues is incompletely understood, an issue particularly relevant
109 erentiation of specific nephron segments are incompletely understood, and the factors that maintain t
110  regulation of mucociliary transport remains incompletely understood, and therapeutic targets for its
111 se mechanisms underlying these processes are incompletely understood, and were proposed to differ bet
112 etabolism, cofactor binding interactions are incompletely understood, as is the molecular basis for p
113                  The pathophysiology remains incompletely understood, but it is probably related to d
114 thophysiology of irritable bowel syndrome is incompletely understood, but it is well established that
115 coronavirus disease 2019 (COVID-19) is still incompletely understood, but it seems to involve immune
116 ms linking age and body weight to cancer are incompletely understood, but recent studies have provide
117    Key aspects of its pathogenesis are still incompletely understood, but severe brain swelling ident
118  polyP, a molecule with prominent, yet often incompletely understood, contributions to cellular funct
119 m cells to neuronal and glial generation are incompletely understood, despite their importance for ne
120 n to downstream physiological systems remain incompletely understood, however.
121        The mechanisms of proton delivery are incompletely understood, in part due to an absence of in
122 ersons with infectious tuberculosis (TB) are incompletely understood, particularly the number of expo
123 and signal coupling of these proteins remain incompletely understood, particularly upon expression of
124  rise to these pronounced differences remain incompletely understood, they are generally attributed t
125               Since underlying mechanics are incompletely understood, we investigated left ventricula
126 roperties of pristine graphene flakes remain incompletely understood, with conflicting reports about
127  pathways promoting excessive activation are incompletely understood, with limited experimental model
128 f anesthetic-induced unconsciousness remains incompletely understood, with some advocating for a quan
129 s adipocyte development and function remains incompletely understood.
130  for regular bone formation, however, remain incompletely understood.
131 allosteric mechanism by which this occurs is incompletely understood.
132 y profile of common anti-VEGF agents remains incompletely understood.
133 ffected with diffuse white matter injury, is incompletely understood.
134 transfer and B cell activation by DCs remain incompletely understood.
135  ligands, but how neurexins are regulated is incompletely understood.
136 chanisms controlling recognition of GSLs are incompletely understood.
137  specificity of these signaling pathways are incompletely understood.
138 re initiated following virus recognition are incompletely understood.
139  influences on intestinal development remain incompletely understood.
140 isms underpinning cancer cell plasticity are incompletely understood.
141 ng multiple NAD(+) biosynthesis pathways are incompletely understood.
142 HIFalpha in developmental myelination remain incompletely understood.
143  syndrome, but the underlying mechanisms are incompletely understood.
144 cal trials, but its clinical significance is incompletely understood.
145 th taking at any particular point in time is incompletely understood.
146 ring events for acute aortic dissections are incompletely understood.
147 g ZIKV infection and pathogenic outcomes are incompletely understood.
148 ts of ER stress on beta-cell function remain incompletely understood.
149 subsets and associated protein expression is incompletely understood.
150 f effector T cell-derived chemokines remains incompletely understood.
151 hanism of BAX and BAK pore formation remains incompletely understood.
152 ions has only recently been addressed and is incompletely understood.
153 ficity of the gammadelta T cell receptors is incompletely understood.
154 chanisms by which mosaic vessels form remain incompletely understood.
155 r mechanisms that govern these processes are incompletely understood.
156 L-1beta-mediated viral RNA reduction remains incompletely understood.
157 ic changes that underlie this malignancy are incompletely understood.
158 ecular basis for mastermind function remains incompletely understood.
159 chanisms underlying this coordination remain incompletely understood.
160 ng factors orchestrating these events remain incompletely understood.
161 A receptors and the induction mechanisms are incompletely understood.
162 ical and pathophysiological responses remain incompletely understood.
163 avior through molecular programs that remain incompletely understood.
164 on and importance in brain physiology remain incompletely understood.
165 re and neural dysfunctions over time remains incompletely understood.
166 olecular targets, and thereby the genes, are incompletely understood.
167 confined migration-induced DNA damage remain incompletely understood.
168 ich epithelial cells sense tissue damage are incompletely understood.
169 euronal circuits mediating these effects are incompletely understood.
170 ollaborates with c-Myc in lymphomagenesis is incompletely understood.
171  but the consequences of 12HBA abundance are incompletely understood.
172 nd function of lung EC heterogeneity remains incompletely understood.
173 xactly how beta-catenin inhibits WT1 remains incompletely understood.
174 lar mechanisms underlying this crosstalk are incompletely understood.
175 ng inflammation; however, the mechanisms are incompletely understood.
176  the mechanisms that control persistence are incompletely understood.
177 s underlying allosteric PfPKG inhibition are incompletely understood.
178              Mechanisms and risk factors are incompletely understood.
179 ns traffic to this proximal axonal domain is incompletely understood.
180  impact of these radicals on gut microbes is incompletely understood.
181  causes of multiple congenital anomalies are incompletely understood.
182 cast information to cortical networks remain incompletely understood.
183 2 in directing de novo TE methylation remain incompletely understood.
184 erlying this activation process of plexin is incompletely understood.
185 t underlie heightened pain processing remain incompletely understood.
186  Clostridioides difficile infection (CDI) is incompletely understood.
187  involvement in lung cancer, if any, remains incompletely understood.
188  variety of diseases, but how this occurs is incompletely understood.
189 xes (ICs), but the underlying mechanisms are incompletely understood.
190 ination, though underlying mechanisms remain incompletely understood.
191 troke and dementia, but its genetic basis is incompletely understood.
192 ypertrophic growth and heart failure remains incompletely understood.
193 sus (SLE), but the underlying mechanisms are incompletely understood.
194 and disease severity and adverse outcomes is incompletely understood.
195 y for proper lamination in these tissues are incompletely understood.
196 , the regulatory pathway and outcomes remain incompletely understood.
197 orage and subsequent BDNF quantification are incompletely understood.
198 ted in the differentiation of LPCs are still incompletely understood.
199 programming, but the underlying mechanism is incompletely understood.
200 eservoir for lifelong EBV infection, remains incompletely understood.
201 ts a breach of its epithelial border remains incompletely understood.
202 utoreactive antibody responses in humans are incompletely understood.
203 nutrient transport across their membranes is incompletely understood.
204 ed fusogenicity of different MuV strains are incompletely understood.
205 NPCs, but the chromatin-based mechanisms are incompletely understood.
206  cytokine production, but these pathways are incompletely understood.
207 al mechanisms leading to hypertension remain incompletely understood.
208 specially the transcallosal pathway, remains incompletely understood.
209 sms that underlie this susceptibility remain incompletely understood.
210 port, and transport to integration sites are incompletely understood.
211 ring systemic metabolic perturbations remain incompletely understood.
212 gnition and uptake, several key areas remain incompletely understood.
213 owever, the underlying mechanism(s) remained incompletely understood.
214  mammalian cells form such a nucleus remains incompletely understood.
215 nsplantation, the mechanisms involved remain incompletely understood.
216 ity, limits therapeutic success, and remains incompletely understood.
217 DK4/6 is activated relative to CDK2 remained incompletely understood.
218  programs mediating tumor suppression remain incompletely understood.
219 ver, underlying dentoalveolar defects remain incompletely understood.
220 ism underlying the oncogenic role of WASp is incompletely understood.
221 ver, the proteomics of hematopoiesis remains incompletely understood.
222 genitors into these cell types, however, are incompletely understood.
223 fibroblasts but underlying mechanisms remain incompletely understood.
224  in neurons, the role of PS1 in microglia is incompletely understood.
225 f aging-related disease, such as cancer, are incompletely understood.
226 tively called beige or brite adipocytes, are incompletely understood.
227 eria acquire iron during gut inflammation is incompletely understood.
228 that allow W to modulate innate immunity are incompletely understood.
229 ystem and their underlying mechanisms remain incompletely understood.
230 activity in HF, but these pathways are still incompletely understood.
231 nical study, the mechanism of action remains incompletely understood.
232 mechanisms that determine these outcomes are incompletely understood.
233 sp104 hexamers disaggregate proteins remains incompletely understood.
234 an withstand endogenous hydrogen peroxide is incompletely understood.
235 on within the tumor microenvironment remains incompletely understood.
236 ly recognized and removed from organelles is incompletely understood.
237  However, the molecular mechanisms are still incompletely understood.
238 the immune response to prostate infection is incompletely understood.
239 infection leads to subsequent disease remain incompletely understood.
240 titerritorial subclinical atherosclerosis is incompletely understood.
241 nd conduction of APs at mammalian NRs remain incompletely understood.
242 ell differentiation, though the mechanism is incompletely understood.
243   The cues that promote this confinement are incompletely understood.
244 lar and molecular mechanisms responsible are incompletely understood.
245  intertwined DNA repair and transcription is incompletely understood.
246 ver, the pathogenesis of myocarditis remains incompletely understood.
247 gy that predispose to metabolic syndrome are incompletely understood.
248 idities, and mortality from COVID-19 remains incompletely understood.
249 orticoid action and whole-body metabolism is incompletely understood.
250  of astrocytes within neural circuits remain incompletely understood.
251 eptor signaling that promote axon growth are incompletely understood.
252 ng, with the underlying molecular mechanisms incompletely understood.
253  storage, consolidation and retrieval remain incompletely understood.
254 high mortality, yet the pathogenesis remains incompletely understood.
255 LE) on subcortical arousal structures remain incompletely understood.
256 ng although the underlying mechanism remains incompletely understood.
257 pects of cancer, its role in this disease is incompletely understood.
258 a-dependent cyst progression in ADPKD remain incompletely understood.
259 ability and energy landscape of proteins are incompletely understood.
260 ations lead to the migraine phenotype remain incompletely understood.
261 sual cues on the processing of speech remain incompletely understood.
262 e molecules mediating their crosstalk remain incompletely understood.
263 he signaling mediates AD pathologies remains incompletely understood.
264  the mechanism underlying this phenomenon is incompletely understood.
265  chromatin opening by mechanisms that remain incompletely understood.
266 ysiological role and molecular regulation is incompletely understood.
267 +) (mtCa(2+)) regulates tumorigenesis remain incompletely understood.
268 but the underlying circuit mechanism remains incompletely understood.
269 t their specialised functions in mitosis are incompletely understood.
270 2 engages with native-like chromatin remains incompletely understood.
271 n metastasis but its molecular basis remains incompletely understood.
272 nt to inflammatory micro-environments remain incompletely understood.
273 S-mediated macroautophagy stimulation remain incompletely understood.
274  role of RSV genotypes in human infection is incompletely understood.
275 ociated factors that drive functionality are incompletely understood.
276 clinical problem of which the mechanisms are incompletely understood.
277 tion both centrally and peripherally remains incompletely understood.
278  the mechanisms that govern this process are incompletely understood.
279 nisms responsible for these phenomena remain incompletely understood.
280 ) and amyotrophic lateral sclerosis (ALS) is incompletely understood.
281 hat enforce dependence on co-stimulation are incompletely understood.
282   The role of TCR in tTreg selection remains incompletely understood.
283 velopment, but the underlying mechanisms are incompletely understood.
284 ors, but the nature of these associations is incompletely understood.
285 tion, and repair of germ cell genomes remain incompletely understood.
286 nisms governing their quality control remain incompletely understood.
287 function of the isoforms, MR1B and MR1C, are incompletely understood.
288 owever, the mechanism of its biosynthesis is incompletely understood.
289 in disease, its pathogenic mechanisms remain incompletely understood.
290 ation of gammadelta T cells in the thymus is incompletely understood.
291 ial signaling dynamics in CD8 T cells remain incompletely understood.
292  substrates for top-down sensory control are incompletely understood.
293 chanisms behind this cellular process remain incompletely understood.
294  that govern its spatial organization remain incompletely understood.
295 ogenesis, but the relevant mechanisms remain incompletely understood.
296 s linking obesity with these diseases remain incompletely understood.
297         Its inflammatory characteristics are incompletely understood.Objectives: To define the cytoki
298 lying the development of immunopathology are incompletely understood.Objectives: We aimed to identify
299 ure death and incident complications remains incompletely understood; likewise, the potential reasons
300  of common additives/impurities or unreacted/incompletely utilized reagents or catalysts.

 
Page Top