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1 diator of cardiac hypertrophy in response to increased blood pressure.
2 on, 40 x 10(8) CFU of the Agg+ strain caused increased blood pressure.
3 hromosomes 2 and 7 in which the Lewis allele increased blood pressure.
4 ation of the DBB is selectively activated by increased blood pressure.
5 Excess body weight correlates closely with increased blood pressure.
6 sed hemoglobin levels, thrombocytopenia, and increased blood pressure.
7 Lactobacillus spp., increased TH17 cells and increased blood pressure.
8 drug treatments fail to regulate a sustained increased blood pressure.
9 the most frequent were anxiety, nausea, and increased blood pressure.
10 /6 mice decreased renal sodium excretion and increased blood pressure.
11 ioventricular node conduction in response to increased blood pressure.
12 eceptors would cause clinically unacceptable increased blood pressure.
13 cumulation of which has been associated with increased blood pressure.
14 where inhibition causes fluid retention and increased blood pressure.
15 A mice have increased plasma aldosterone and increased blood pressure.
16 closures include many known risk factors for increased blood pressure.
17 at -6, and the -6A allele is associated with increased blood pressure.
18 ioavailability, endothelial dysfunction, and increased blood pressure.
19 CETP inhibition by itself might not lead to increased blood pressure.
20 ition of the baroreceptor reflex, leading to increased blood pressure.
22 ) PK levels are elevated in association with increased blood pressure; 2) PK levels are independently
24 co (11.8% [10.5-13.3] of DALYs), followed by increased blood pressure (9.0 % [7.5-10.5]), and high bo
25 try studies showed that while angiotensin II increased blood pressure acutely in all animals, VEGFCc1
26 ventricular function cannot be explained by increased blood pressure alone, because eNOS-KO animals
27 ype mice induced endothelial dysfunction and increased blood pressure, an effect absent in P-selectin
28 rbonate have been reported to associate with increased blood pressure and a higher risk of cardiovasc
29 ctivation and oxidation were associated with increased blood pressure and altered cardiac function, a
30 NX1 in C57BL/6J and BALB/cJ mice resulted in increased blood pressure and blunted natriuretic respons
32 ted in NO synthase inhibition as detected by increased blood pressure and by significantly reduced ph
33 sease has been increasingly identified, with increased blood pressure and decreased cerebral blood fl
34 ed during childhood and young adult life and increased blood pressure and decreased HDL cholesterol l
40 -cigarette aerosols in human subjects led to increased blood pressure and heart rate, similar to trad
44 hat chronic mouthwash use is associated with increased blood pressure and increased risk for prediabe
45 er menopause and probably contributes to the increased blood pressure and increased risk of hypertens
46 of warmth, increased feelings of anger, and increased blood pressure and left-ventricular contractib
47 ass-related off-target effects (particularly increased blood pressure and low serum potassium) relate
48 ntracerebroventricular infusion of PF4800567 increased blood pressure and lumbar sympathetic nerve ac
52 ater Horizon disaster may be associated with increased blood pressure and newly detected hypertension
53 ulation of the renin angiotensin system, and increased blood pressure and oxidative stress in Hap -6A
57 osures have been shown to be associated with increased blood pressure and risk of hypertension in old
58 investigated the mechanisms responsible for increased blood pressure and sympathetic nerve activity
60 cited reports of sedation, dissociation, and increased blood pressure), and serious adverse events fo
61 riglyceridemia, 69% low HDL cholesterol, 31% increased blood pressure, and 25% either increased fasti
62 including dyslipidemia, insulin resistance, increased blood pressure, and abdominal obesity, which t
63 IN in mice leads to endothelial dysfunction, increased blood pressure, and diastolic heart failure.
64 rolongation of late repolarization duration, increased blood pressure, and systemic inflammation.
65 aplotype have increased plasma AGT level and increased blood pressure as compared with -6G haplotype.
67 nfused with angiotensin II had significantly increased blood pressure, as determined using telemetry,
68 nic exposure has been related to the risk of increased blood pressure based largely on cross-sectiona
69 s) improved survival with LPS (p = 0.04) and increased blood pressure before the onset of lethality w
72 h numerous adverse health effects, including increased blood pressure (BP) and vascular dysfunction.
74 estricted silencing of Drd1 in C57BL/6J mice increased blood pressure (BP) that was normalized by ren
75 chamber dilatation disproportionate to their increased blood pressure (BP), suggesting, in support of
79 After baroreflex blockade with atropine, PE increased blood pressure but did not change heart rate.
80 ebroventricular treatment with neuronostatin increased blood pressure but suppressed food intake and
81 mm Hg (SE 0.8), whereas subtherapeutic nCPAP increased blood pressure by 0.8 mm Hg (0.7) (difference
82 ted into the arterial supply of the hindlimb increased blood pressure by 39% (baseline, 93.9+/-9.5 mm
83 triuretic peptide receptor A (NPRA)) exhibit increased blood pressure, cardiac hypertrophy, and conge
84 altered mechanical stress, originating from increased blood pressure, changes in blood flow velocity
85 er insight into the mechanism underlying the increased blood pressure characteristic of this model.
88 ed hCYP11B2 mRNA and protein levels, and (c) increased blood pressure compared with TG mice containin
89 hat transgenic animals containing Hap-I have increased blood pressure compared with those containing
90 d volume expansion, except for PEG-Hb, which increased blood pressure due to blood volume expansion a
91 he mediators of abnormal kidney function and increased blood pressure during development of obesity h
92 this CYP in female mice was associated with increased blood pressure, enhanced proximal tubular tran
95 ouse model, including new-onset proteinuria, increased blood pressure, glomerular endotheliosis, and
98 % CI .41-.84; Nhigh-risk = 1509), or without increased blood pressure (iHR .54, 95% CI .36-.81; Nhigh
101 restricted restoration of BKbeta1 expression increased blood pressure in BKbeta1(-/-) strain C mice,
103 ) cardiovascular deaths were attributable to increased blood pressure in China: 2.11 million (2.03-2.
104 as a high-fat diet caused HPA activation and increased blood pressure in control mice, these adaption
107 r N(y)-nitro-l-arginine methyl ester further increased blood pressure in RBC eNOS KOs, demonstrating
108 a cardiopulmonary disorder characterized by increased blood pressure in the small arterioles supplyi
110 t exposure to ambient Ni was associated with increased blood pressure independent of PM2.5 mass in ou
111 in animal lard programmes the development of increased blood pressure, insulin resistance, dyslipidae
113 ether homocysteine is a cause or a marker of increased blood pressure is relevant because blood homoc
116 by body mass index (BMI) was associated with increased blood pressure, LDL-C, triglycerides, and fast
117 tension, younger age, lower body weight, and increased blood pressure load using 24-hour ambulatory b
118 tered ventromedial prefrontal activation and increased blood pressure may represent useful CNS and pe
119 18 [59%] vs 547 of 1468 [37%]; P < .001) and increased blood pressure medication use (21 of 218 [10%]
120 d aspartate aminotransferase (one [2%]), and increased blood pressure (one [2%]), and in the placebo
121 of the DV to close may be the consequence of increased blood pressure or a failure in vasoconstrictio
129 ministration to vehicle-treated SHRs further increased blood pressure, provoked drinking, increased t
130 ients with acute heart failure and normal-to-increased blood pressure, relaxin was associated with fa
132 ectrolytic lesion of the MnPO attenuated the increased blood pressure response to chronic intravenous
133 all reduction is likely a consequence of the increased blood pressure secondary to the blunting of hy
134 AD (OR 1.12; per-allele P = 1 x 10(-5)), and increased blood pressure (systolic and diastolic blood p
135 e sleepiness (Epworth <11 points) or without increased blood pressure (systolic/diastolic <140/90 mmH
136 g versus G6PD(mut) 95+/-4 mm Hg), but Ang II increased blood pressure to a lower level in G6PD(mut) m
139 , 18.56 [95% CI, 2.94-117.05]; P = .002) and increased blood pressure variability (OR, 3.49 [95% CI,
141 n ankle brachial index greater than 1.30 and increased blood pressure variability, are associated wit
143 In contrast, the opposite trend, toward increased blood pressure, was observed in mice with gene
145 + diet, KO mice exhibited hypercalciuria and increased blood pressure, which were reversed by thiazid