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1 ggesting that there is no healthy pattern of increased weight.
2 ine also increased hypoglycemia and modestly increased weight.
3 e with increased expenditure associated with increased weight.
4 d from malalignment and the excess load from increased weight.
5 the vehicle or MS1 conditions significantly increased weight (9-20 g) and produced smaller reduction
6 1-null mice grow longer than wild type, with increased weight and adiposity, when restricted in vitam
8 vided to women living with HIV significantly increased weight and CD4+ T cells and such interventions
9 vided to women living with HIV significantly increased weight and CD4+ T cells, and such intervention
11 may increase dieting, a risk factor for both increased weight and eating disorders in adolescents.
12 and pediatric isolates were associated with increased weight and gestational age of newborns (P </=
13 osure to reduced rations was associated with increased weight and greater indexes of fat deposition a
15 tica pathogenesis, mice lacking Gal-1 showed increased weight and survival, lower bacterial load, and
16 f protein from animal-source foods generally increased weight and weight-for-length in children, but
17 hort-term adverse events-acne, night sweats, increased weight, and altered mood and libido-are recogn
18 e, which results in flies with larger cells, increased weight, and decreased life span compared to wi
21 cerebellar Purkinje neurons reduced ataxia, increased weight, and prolonged life, but it did not pre
22 ies also support the notion that fatigue and increased weight are linked to higher osteoarthritis pai
23 Hand OA was significantly associated with increased weight at ages 26 years, 43 years, and 53 year
27 dia [e.g., redox flow batteries (RFBs)], the increased weight can be better distributed for improved
28 he single mutants had higher oil content and increased weight compared to those of the wild type, wit
29 ce between regular and highly palatable food increased weight compared with access to regular food on
30 , and/or lifestyle behaviors account for the increased weight, fat mass, and central adiposity experi
31 exposed to GDM in utero, infants exposed had increased weight, FM, and percent body fat at birth.
33 pothalamic satiation signaling, hyperphagia, increased weight gain and adiposity, and enhanced lipoge
34 leptin signaling (Leprdb/db mice), including increased weight gain and adiposity, hyperphagia, cold i
37 ictive feeding practices are associated with increased weight gain and higher weight status, and pres
39 ition of GDF-15 with ponsegromab resulted in increased weight gain and overall activity level and red
40 hat these Npc1(+/-) mice were susceptible to increased weight gain characterized by increased whole b
43 glucose intolerance, insulin resistance, and increased weight gain during, but not prior to, pregnanc
44 imuli, with MC3R knockout mice demonstrating increased weight gain following anabolic challenges and
45 Lastly, we showed that while PD consumption increased weight gain in both young and aged rats, this
47 icate that maternal ACEs are associated with increased weight gain in male infants during the first t
50 ght gain (diet-induced obese [DIO] rats) had increased weight gain in response to consuming saccharin
51 that daily insulin injections significantly increased weight gain in the transgenic lines expressing
54 intermediate metabolizers could explain the increased weight gain on dolutegravir compared with efav
55 ng, whereas group-housed female mice display increased weight gain on high-fat diet, reduced behavior
57 ted protein 2 antibodies was associated with increased weight gain with either intensive or conventio
59 , we report that reduced BRD7 levels lead to increased weight gain with little effect on glucose meta
60 hort day hamsters were also characterized by increased weight gain, and heavier adrenal glands (p < 0
61 nd cardiovascular disease-related mortality, increased weight gain, and high risk for severe hypoglyc
63 rdination and cognitive function but exhibit increased weight gain, elevated white adipose tissue dep
66 Chronic HFD/SW feeding led to significantly increased weight gain, serum and liver lipid levels, liv
67 g insulin concentrations are associated with increased weight gain, whereas insulin resistance seems
70 e of deceased donor LT from pediatric donors increased (weighted HR, 1.201.311.42; P < 0.001), and ac
71 essive gestational weight gain may result in increased weight in children; however, studies have not
74 order of magnitude variation in body weight; increased weight is supported solely through disproporti
76 ggest that, despite a known association with increased weight, long-term sulfonylurea therapy may red
78 MC4R (I251L), a rare variant associated with increased weight loss after RYGB and increased basal act
79 ulted in more severe disease, as measured by increased weight loss and airway resistance, as compared
81 istering DSS to IL-17R(-/-) mice resulted in increased weight loss and more severe intestinal inflamm
82 en in mice infected with M. tuberculosis and increased weight loss and mortality in mice infected wit
83 tudy, we found that STING-deficient mice had increased weight loss and roughly 10-fold-increased syst
84 ore susceptible to endotoxemia, evidenced by increased weight loss and serum TNF-alpha, IL-6, and IL-
85 and intestinal-secreted IgA correlated with increased weight loss at the end of DSS administration.
86 za infection, iron deficient mice experience increased weight loss but mount antigen specific T cells
87 es, fully blocked CR-induced hypothermia and increased weight loss during CR independent of calorie i
88 eight gain following anabolic challenges and increased weight loss following anorexic challenges (i.e
89 a significantly reduced survival percentage, increased weight loss, and a more-rapid increase in bact
91 monstrated a reduced UPR in colonic tissues, increased weight loss, and less effective clearance of b
93 ctivated RSV-vaccinated children, as well as increased weight loss, clinical illness, and enhanced pa
94 zed with vacvG results in the development of increased weight loss, clinical illness, and Penh simila
96 sodium (DSS)-induced colitis, manifested by increased weight loss, macrophage infiltration, and infl
97 s in exacerbated disease severity, including increased weight loss, morbidity, and enhanced airway re
100 vation 2 weeks following surgery experienced increased weight loss.CONCLUSIONThe anatomical and/or me
102 on in lateral central beta power reflects an increased weighting of peripheral sensory information im
104 frequencies, below 1 Hz, was associated with increased weighting over parietal channels, which was no
106 effectively decreased soil bulk density and increased weight percentage of water-stable aggregate an
107 stature, black race, age 55 years or older, increased weight, rapid pulse, and smoking history (< or
109 Female TDP-43(Q331K) knock-in mice displayed increased weight relative to wild-type and increased foo
110 , arthralgia, arthritis, increased appetite, increased weight, restlessness, tendon disorder, and pot
111 rs, greater maternal BMI was associated with increased weight SDS (p < 0.001), BMI SDS (p = 0.005), a
113 verse events in both safety populations were increased weight (seven [10%] of 68 patients in the NTRK
114 -fast blood glucose normally associated with increased weight, suggesting a role for resistin in medi
116 ethoxy-4-iodophenyl-2-aminopropane), further increased weight-supported stepping in transplant rats.
117 rease of energy intake needed to sustain the increased weight (the maintenance energy gap) has amount
118 were present in the aged subjects, including increased weight, triglycerides, lactate dehydrogenase (
120 ce estimates using Gray's k-sample P values, increased weight was ascribed to the earlier data becaus
121 mr1(-/y)), but not female Fmr1(-/-), exhibit increased weight when compared to wild-type controls, si