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1 as induced in the presence of 2,4-D and IAA (indole-3-acetic acid).
2 ed for polar transport of the hormone auxin (indole-3-acetic acid).
3 e gut microbiome (e.g., CMPF, phenylsulfate, indole-3-acetic acid).
4 play an important role in converting IPA to indole-3-acetic acid.
5 hydrogen peroxide, abscisic acid (ABA), and indole-3-acetic acid.
6 pression in response to exogenously supplied indole-3-acetic acid.
7 e-3-butyric acid (IBA) to the active hormone indole-3-acetic acid.
8 ecific for the carboxyl-bearing phytohormone indole-3-acetic acid.
9 duced conversion of indole-3-butyric acid to indole-3-acetic acid.
10 ty was enhanced by ethylene and inhibited by indole-3-acetic acid.
11 s was increased by treatment with 0.1 microM indole-3-acetic acid.
12 ction of the microbial tryptophan metabolite indole-3-acetic acid.
13 ven by the bacterially-produced phytohormone indole-3-acetic acid.
14 d response of the apx6 mutants to ABA and to indole-3-acetic acid.
18 ntrations of isolated uremic solutes such as indole-3-acetic acid (3.5 mug/mL), indoxyl sulfate (25 m
20 seedlings to 2,3-dihydro-7-hydroxy-2-oxo-1H indole-3-acetic acid-7'-O-beta-D-glycopyranoside with th
21 e intake resulted in reduced serum levels of indole-3-acetic acid, a microbial tryptophan metabolite,
22 d seedlings treated with different hormones (indole-3-acetic acid, abscisic acid, gibberellin, methyl
23 alicylic acid, cinnamic acid, jasmonic acid, indole-3-acetic acid, abscisic acid, unsaturated C(18) f
24 indole-3-aspartic acid (an indicator of high indole-3-acetic acid accumulation, which inhibits lettuc
25 n and that these mutants show reduced auxin (indole-3-acetic acid) accumulation and auxin responses c
26 ce that OsbZIP49 activates the expression of indole-3-acetic acid-amido synthetases OsGH3-2 and OsGH3
29 endogenous hormones measured in leaves, both indole-3-acetic acid and abscisic acid contents were dec
30 nutrient acquisition and the accumulation of indole-3-acetic acid and antioxidants in tissues, are al
32 etics and thermodynamics of the oxidation of indole-3-acetic acid and derivatives and of phenols by h
33 bolic branch point between the primary auxin indole-3-acetic acid and indole glucosinolate biosynthes
35 that neither the naturally occurring auxins indole-3-acetic acid and indole-3-butyric acid, nor the
38 fate, while for weakly bound toxins, namely, indole-3-acetic acid and p-cresyl glucuronide, an increa
39 second compound was found to be an ester of indole-3-acetic acid and the disaccharide rutinose (gluc
41 s (sirtinol, 2,4-dichlorophenoxyacetic acid, indole-3-acetic acid) and a host of pleiotropic phenotyp
42 ation of COR, salicylic acid, jasmonic acid, indole-3-acetic acid, and abscisic acid illustrate the p
43 l analysis of salicylic acid, jasmonic acid, indole-3-acetic acid, and abscisic acid is typically ach
44 osine, kynurenic acid, indole-3-lactic acid, indole-3-acetic acid, and betaine were observed than in
45 cussed to be involved in the biosynthesis of indole-3-acetic acid, and Cytochrome P450 (CYP) 71B6 wer
47 itination at baseline and that uremic serum, indole-3-acetic acid, and indoxyl sulfate significantly
48 ibution of total radioactivity, radiolabeled indole-3-acetic acid, and radiolabeled ester conjugated
49 wards small substrates including the natural indole-3-acetic acid, and the synthetic auxin 2,4-dichlo
50 t one route to produce another phytohormone, indole-3-acetic acid, and thus, AOs play important roles
51 ulation of FQR1 mRNA begins within 10 min of indole-3-acetic acid application and reaches a maximum o
54 Overexpression of BADC1 in wri1-1 decreased indole-3-acetic acid-Asp content and partially rescued i
55 th reduction in root length and elevation of indole-3-acetic acid-Asp levels relative to the wild typ
56 oxIAA) and increases in the conjugated forms indole-3-acetic acid aspartic acid (IAA-Asp) and indole-
57 ifically, elevated levels of auxin conjugate indole-3-acetic acid-aspartic acid (IAA-Asp) were observ
59 auxin-transcription activators, while auxin/indole-3-acetic acid (Aux/IAA) are auxin-transcription r
64 xin-responsive degradation of multiple auxin/indole-3-acetic acid (Aux/IAA) proteins is essential for
66 pivots on the interaction between the AUXIN/INDOLE-3-ACETIC ACID (Aux/IAA) repressor proteins and th
67 tical model revealed the centrality of auxin/indole-3-acetic acid (Aux/IAA) transcriptional corepress
70 )/AUXIN SIGNALING F-BOX protein and an AUXIN/INDOLE-3-ACETIC ACID (Aux/IAA) transcriptional repressor
71 y two counteracted teams including (1) auxin/indole-3-acetic acid (AUX/IAA)-histone deacetylase (HDA)
72 y two counteracted teams including (1) auxin/indole-3-acetic acid (AUX/IAA)-histone deacetylase (HDA)
73 by facilitating proteolytic removal of auxin/indole-3-acetic acid (AUX/IAA)-inducible repressors, whi
74 n auxin response is the degradation of Auxin/Indole-3-Acetic Acid (Aux/IAA, referred to hereafter as
76 es encode enzymes that convert tryptophan to indole-3-acetic acid (auxin): iaaM (tryptophan mono-oxyg
77 using alpha-(2,4-dimethylphenylethyl-2-oxo)-indole-3-acetic acid (auxinole), alpha-(phenylethyl-2-ox
78 conversion of indole-3-butyric acid (IBA) to indole-3-acetic acid, because ech2 seedlings have altere
79 d metabolism and transport, and key steps in indole-3-acetic acid biosynthesis, catabolism, and trans
80 afb5-5, that responds to conventional auxin (indole-3-acetic acid) but has a strongly diminished resp
81 laced these CCMTs into a clade that includes indole-3-acetic acid carboxyl methyltransferases and a l
83 o lead to a sustained elevation of leaf free indole-3-acetic acid content relative to untreated contr
84 reover, cu-3 mutants retained sensitivity to indole-3-acetic acid, cytokinins, gibberellin, and absci
85 acteria raise the question as to whether the indole-3-acetic acid degradation pathway is present in h
86 , gravity affects the steady state amount of indole-3-acetic acid derived from indole-3-acetyl-myo-in
89 auxin antagonist alpha-(phenyl ethyl-2-one)-indole-3-acetic acid enhanced ABA-regulated stomatal mov
91 it is the same as IAA7, a member of the IAA (indole-3-acetic acid) family of auxin-inducible genes.
92 ate flux near neuronal cells; and endogenous indole-3-acetic acid flux near the surface of Zea mays r
93 nphysiological concentrations of ethylene or indole-3-acetic acid for protracted periods (more than 2
95 a photooxidation product of the plant auxin indole-3-acetic acid, functions as an affinity label of
97 le-3-acetic acid aspartic acid (IAA-Asp) and indole-3-acetic acid glutamic acid (IAA-Glu) of 438- and
98 y for indole-3-acetic acid in Zea seedlings: Indole-3-acetic acid --> Oxindole-3-acetic acid --> 7-Hy
99 preference for the naturally occurring auxin indole 3-acetic acid (IAA) and is important for coordina
100 epicotyls were loaded symmetrically with 3H-indole 3-acetic acid (IAA) or 45Ca2+, then subjected to
101 We investigated whether the uremic solute indole-3 acetic acid (IAA) predicts clinical outcomes in
103 years of evidence showing a pivotal role for indole-3-acetic acid (IAA or auxin) in plant development
104 m segments have implicated the plant hormone indole-3-acetic acid (IAA or auxin) in the regulation of
109 ompared with the control group and decreased indole-3-acetic acid (IAA) and abscisic acid (ABA) conce
110 than the wild type at low concentrations of indole-3-acetic acid (IAA) and also under low nutrient c
111 tophan (Trp), including the growth regulator indole-3-acetic acid (IAA) and defense compounds against
112 binding properties of indoxyl sulfate (IS), indole-3-acetic acid (IAA) and hippuric acid (HIPA) and
113 y metabolites including the growth regulator indole-3-acetic acid (IAA) and indole glucosinolate defe
116 auxin biology include synthesis of the auxin indole-3-acetic acid (IAA) and production of virulence f
117 Gretchen Hagen 3.5 (AtGH3.5) conjugates both indole-3-acetic acid (IAA) and salicylic acid (SA) to mo
118 s have suggested that AtGSTF2 interacts with indole-3-acetic acid (IAA) and the auxin transport inhib
119 the auxin precursor IBA and the active auxin indole-3-acetic acid (IAA) and those with restored respo
120 s of indole-3-acetyl-1-O-beta-D-glucose from indole-3-acetic acid (IAA) and uridine diphosphoglucose
122 h plants regulate levels of the phytohormone indole-3-acetic acid (IAA) are complex and not fully und
124 owever, the expression of genes encoding the indole-3-acetic acid (IAA) biosynthesis enzyme TRYPTOPHA
125 Plants can regulate levels of the auxin indole-3-acetic acid (IAA) by conjugation to amino acids
126 -butyric acid (IBA) is converted into active indole-3-acetic acid (IAA) by peroxisomal beta-oxidation
127 acid (IBA) is converted to the active auxin indole-3-acetic acid (IAA) by removal of two side-chain
131 IBA treatments locally increased endogenous indole-3-acetic acid (IAA) content, whereas the combinat
133 s thaliana, cotyledons and leaves synthesize indole-3-acetic acid (IAA) from tryptophan through indol
134 ence indicates that amino acid conjugates of indole-3-acetic acid (IAA) function in auxin homeostasis
135 Furthermore, homologous members of the AUX/indole-3-acetic acid (IAA) gene family mediate the actio
136 tural auxins indole-3-butyric acid (IBA) and indole-3-acetic acid (IAA) has been described in Arabido
138 BA), which is apparently shortened to active indole-3-acetic acid (IAA) in peroxisomes by a process s
140 be the coupling between the concentration of indole-3-acetic acid (IAA) in the cambial region of a tr
141 n induces an asymmetric distribution of free indole-3-acetic acid (IAA) in the cortex-epidermis of th
142 iments show that afb4-2 is hypersensitive to indole-3-acetic acid (IAA) in the hypocotyl, indicating
145 PLC-ESI-MS/MS analysis showed that levels of indole-3-acetic acid (IAA) increased and levels of absci
147 olated pea (Pisum sativum L.) seedlings with indole-3-acetic acid (IAA) induced within 15 min an incr
152 .F.W. Meyer ecotype had significantly higher indole-3-acetic acid (IAA) levels than a UV-sensitive ec
153 nel mass and a recently reported decrease in indole-3-acetic acid (IAA) levels throughout kernel deve
155 tant phenotypes were rescued when endogenous indole-3-acetic acid (IAA) levels were increased by grow
163 est that the enzymatic reactions involved in indole-3-acetic acid (IAA) production via IPyA are diffe
165 es facilitates ubiquitination of auxin (aux)/indole-3-acetic acid (IAA) repressor proteins in the pre
166 although AgNO(3) dramatically decreased root indole-3-acetic acid (IAA) responsiveness in inhibition
168 first enzyme-catalyzed reaction leading from indole-3-acetic acid (IAA) to the myo-inositol esters of
169 companied by a rapid increase in radioactive indole-3-acetic acid (IAA) transport and its accumulatio
170 mutation significantly enhance radiolabeled indole-3-acetic acid (IAA) transport in both the acropet
172 were to determine whether both polarities of indole-3-acetic acid (IAA) transport occur in roots of A
174 the promotion of lateral root initiation by indole-3-acetic acid (IAA) was reduced as the IAA concen
176 of bacterial cultures and the production of indole-3-acetic acid (IAA), a ubiquitous plant hormone t
177 onversion of indole-3-pyruvic acid (IPyA) to indole-3-acetic acid (IAA), acting downstream of CKRC1/T
179 rabidopsis genes were specifically active on indole-3-acetic acid (IAA), and one was active on both I
180 ant-associated microbes synthesize the auxin indole-3-acetic acid (IAA), and several IAA biosynthetic
182 synthesis and degradation of the main auxin, indole-3-acetic acid (IAA), by sugars requires changes i
183 hey were also screened for the production of indole-3-acetic acid (IAA), hydrogen cyanide (HCN), ammo
184 IBA is converted to the more abundant auxin, indole-3-acetic acid (IAA), in a mechanism that parallel
185 l mechanisms to regulate levels of the auxin indole-3-acetic acid (IAA), including the formation and
187 A plant growth-promoting substance, such as indole-3-acetic acid (IAA), known to be produced by G. d
188 nscripts that were consistently regulated by indole-3-acetic acid (IAA), partitioning into 60 cluster
191 an antagonistic manner to that of the auxin indole-3-acetic acid (IAA), the mechanism by which remai
194 airway lactobacilli associated with reduced indole-3-acetic acid (IAA), which was in turn linked to
195 changes in auxin metabolism, mediated by the indole-3-acetic acid (IAA)-amido synthetase Gretchen Hag
197 ulation of a conjugated form of the hormone, indole-3-acetic acid (IAA)-Asp, to promote disease devel
199 a that are resistant to growth inhibition by indole-3-acetic acid (IAA)-phenylalanine have been isola
200 al root primordia; decreased auxin maxima in indole-3-acetic acid (IAA)-treated root apical meristems
212 nclude camalexin, indole glucosinolates, and indole-3-acetic acid (IAA); however, the steps in their
214 static regulation of the phytohormone auxin [indole-3-acetic acid (IAA)] is essential to plant growth
215 Spatial regulation of the plant hormone indole-3-acetic acid (IAA, or auxin) is essential for pl
217 sion of GRETCHEN HAGEN3.2 (ZmGH3.2, encoding indole-3-acetic acid [IAA] deactivating enzyme), and inc
218 he biosynthesis of the main auxin in plants (indole-3-acetic acid [IAA]) has been elucidated recently
221 rile (IAN; a possible precursor of the auxin indole-3-acetic acid [IAA]) was carried out under mild c
222 (1-NAA); however, traditional auxins (e.g., indole-3-acetic acid [IAA], 2,4-D, 1-NAA) have no effect
223 IAR3 hydrolyzes an inactive form of auxin (indole-3-acetic acid [IAA]-alanine) and releases bioacti
224 er to survey the entire literature on auxin (indole-3-acetic acid, IAA) action in all plants, with sp
226 indole-3-butyric acid (IBA) to active auxin (indole-3-acetic acid; IAA) modulates lateral root format
230 pplied to the endosperm supplies some of the indole-3-acetic acid in the mesocotyl but essentially no
231 induction of primary auxin response genes by indole-3-acetic acid in the presence of growth-inhibitor
232 bution and chemical form of the radiolabeled indole-3-acetic acid in the shoot depending upon whether
234 route as the principal catabolic pathway for indole-3-acetic acid in Zea seedlings: Indole-3-acetic a
235 etic acid, and radiolabeled ester conjugated indole-3-acetic acid, in the shoots was then determined.
236 ns of auxin response factors (ARF) and auxin/indole 3-acetic acid inducible proteins regulate transcr
237 ly of transcription factors as well as AUXIN/INDOLE-3-ACETIC ACID INDUCIBLE (AUX/IAA) proteins that r
238 analyses revealed that expression of several indole-3-acetic acid-inducible genes, including Aux/IAA,
240 /SL signaling, D14-LIKE2, KAR-UP F-BOX1, and INDOLE-3-ACETIC ACID INDUCIBLE1, is rescued in smax1 max
241 t increases in the expression of some genes (INDOLE-3-ACETIC ACID-INDUCIBLE1 and PHYTOCHROME B ACTIVA
242 ated tissues and others through signaling of INDOLE-3-ACETIC ACID INDUCIBLE28 (IAA28), CRANE (IAA18),
245 According to the classic model, the auxin indole-3-acetic acid is produced in the shoot tip and tr
247 We also show that the widespread auxin, indole-3-acetic acid, is synthesized by a parallel pathw
250 of this enzyme in the homeostatic control of indole-3-acetic acid levels in Zea mays is discussed.
252 hypocotyls contained considerably lower free indole-3-acetic acid levels when compared with wild-type
253 nhibitor response1/auxin-related f-box-auxin/indole-3-acetic acid-mediated auxin-signaling machinery
254 ft1-2, and 35S:PFT1 seedlings in response to indole-3-acetic acid, naphthaleneacetic acid, and the po
255 uding a number of transcripts encoding Auxin/Indole-3-Acetic Acids, negative regulators of auxin sign
256 ere fully assigned as tetrahydropentoxyline, indole-3-acetic-acid-O-glucuronide, p-cresol glucuronide
258 id in the shoot depending upon whether 5-[3H]indole-3-acetic acid or 5-[3H]indole-3-acetyl-myo-inosit
259 d an auxin-inhibitor (a-(phenyl ethyl-2-one)-indole-3-acetic acid (PEO-IAA)), together with the MIR17
260 c acid (auxinole), alpha-(phenylethyl-2-oxo)-indole-3-acetic acid (PEO-IAA), and 5-fluoroindole-3-ace
262 atranscriptome analyses that show widespread indole-3-acetic acid production by Sulfitobacter-related
265 ene family products, together with the AUXIN/INDOLE-3-ACETIC ACID proteins, regulate auxin-mediated t
267 intermediate indole-3-pyruvic acid (IPA) and indole-3-acetic acid rescues the tir2 short hypocotyl ph
268 sitivity to reactive oxygen species, reduced indole-3-acetic acid secretion, reduced biofilm and pell
269 This compound was also formed from labeled indole-3-acetic acid supplied to intact seedlings and ro
270 m cell division via secretion of the hormone indole-3-acetic acid, synthesized by the bacterium using
271 se mutant, but it also is less responsive to indole-3-acetic acid, synthetic auxins, auxin transport
275 unds related to the biosynthetic pathways of indole-3-acetic acid, the primary growth regulator in pl
278 tration of the in vitro enzymic oxidation of indole-3-acetic acid to oxindole-3-acetic acid in higher
279 tic acid in the mesocotyl but essentially no indole-3-acetic acid to the coleoptile or primary leaves
280 perm provides both free and ester conjugated indole-3-acetic acid to the mesocotyl and coleoptile.
281 nositol and 1 picomole per plant per hour of indole-3-acetic acid to the shoot and thus is comparable
282 with auxin-regulated proteolysis of an auxin/indole-3-acetic acid transcription factor, and two impar
286 avitropic response, a reduction in basipetal indole-3-acetic acid transport, and a delay in the asymm
287 Y6) mutant makes no flavonoids, has elevated indole-3-acetic acid transport, and exhibits a delayed g
292 m was applied unilaterally to the cap and 3H-indole-3-acetic acid was applied to the basal cut surfac
294 noyl-Ile, salicylic acid, abscisic acid, and indole-3-acetic acid were compromised due to PAE9 loss o
298 homolog RAMOSA1 ENHANCER LOCUS2, maize auxin/indole-3-acetic acids were able to repress AUXIN RESPONS
299 nd 3-mercaptopropionate, and the plant auxin indole 3-acetic acid, were released by S. elongatus at m
300 eld [3H]indole-3-acetyl-myo-inositol and [3H]indole-3-acetic acid which were then transported to the