ライフサイエンスコーパス: indoor

1 individuals tend to spend most of their time indoors.

2 from the outdoor environment than that from indoors.

3 the main modulator of ammonia concentration indoors.

4 the amount of ambient NO(x) that infiltrates indoors.

5 toes' biting people when they are in bed and indoors.

6 ogen oxides (NO(x)) of ambient origin occurs indoors.

7 t effects via the consequences on time spent indoors.

8 ad to reduced exposure to hazardous microbes indoors.

9 fate and exposure of organic chemicals used indoors.

10 ts support the hypotheses that (1) transient indoor activities can give rise to intraindividual varia

11 for 12 h varied from 7 mug on a day with no indoor activities to 68 mug during a simulated day (incl

12 Additional considerations are needed for indoor activities with large numbers of persons (places

13 In fact, merely eclosing indoors after an otherwise complete lifecycle outdoors w

14 environment sensor for indoor agriculture system).

15 Vertical farming is a type of indoor agriculture where plants are cultivated in stacke

16 -68%, which encompasses the preferred RH for indoor air (40-60%).

17 ted room using a detailed chemical model for indoor air (the INDCM).

18 ypically present at higher concentrations in indoor air (~10-70 ppb) than in outdoor air (~50 ppt to

19 abromocyclododecane (HBCDD) were measured in indoor air and dust collected from Irish homes, cars, of

20 Settled dust from indoor air and outdoor air and direct samples from build

21 In addition to indoor air chemistry, these results have implications to

22 Indoor air concentrations were estimated for commonly en

23 loss rate muted the impact of ventilation on indoor air concentrations.

24 Indoor air contaminants may act as endocrine-disrupting

25 The indoor air differences between moldy versus no mold home

26 cient K(OW) < 10(11)), whereas inhalation of indoor air dominates human exposure to volatile chemical

27 and the tests should last for at least nine indoor air exchanges for negative pressure difference te

28 egative pressure difference testing and four indoor air exchanges for positive pressure difference te

29 r phthalates found in high concentrations in indoor air have never been examined in allergic individu

30 dict the PM(2.5) concentration in well-mixed indoor air in a commercial office building.

31 This study examined residential indoor air in homes (n = 99) in southeast Louisiana usin

32 determined the levels of these chemicals in indoor air in homes in rural Central Appalachia using pa

33 tant approach to maintain the cleanliness of indoor air in hospitals.

34 Further study of residential indoor air in low-income women's homes in this area is n

35 The indoor air in urban homes of developed countries is usua

36 of chemicals, many of which can be found in indoor air including residential indoor air.

37 Despite the vast time humans spend indoors, indoor air is far less well chemically characterized tha

38 Although a common indoor air pollutant, guidelines for control of incense

39 rts of household stove improvement to reduce indoor air pollution have resulted in higher outdoor air

40 ated homes are a major source of exposure to indoor air pollution in developing countries.

41 Indoor air pollution threats anticipated from climate ch

42 cardiovascular disease side effect profiles, indoor air pollution), and encourage the inclusion of re

43 cyclic aromatic hydrocarbons (from opium and indoor air pollution), and nutrient-deficient diets.

44 usive breastfeeding, crowding, malnutrition, indoor air pollution, incomplete immunisation, and paedi

45 This review summarizes the effects of indoor air pollution, outdoor air pollution, and subsequ

46 made us all think critically about hospital indoor air quality and the approaches to remove, dilute,

47 ize the evidence on the effects of e-cigs on indoor air quality, chemical compositions of mainstream

48 ative pressure testing, adding room-specific indoor air sampling to both negative and positive pressu

49 alkanes were observed to be correlated with indoor air temperature.

50 are exposed to organic chemicals released to indoor air through near-field exposure routes such as ai

51 ion between PFOS in hand wipes and EtFOSE in indoor air was found.

52 Indoor air was sampled using bioaerosol impactors with n

53 ic chlorinated and nitrogenated compounds in indoor air while cleaning with a commercial bleach solut

54 nd nBFRs were 210, 25 000, and 69 pg/m(3) in indoor air, and 49, 670, and 110 ng/g in wristbands, res

55 ng validated external exposure tools such as indoor air, dust, and hand wipes.

56 total loss of bleach-related compounds from indoor air, respectively; we conclude that uptake to ind

57 be found in indoor air including residential indoor air.

58 predicting the PM(2.5) levels in well-mixed indoor air.

59 organic compounds on indoor surfaces and in indoor air.

60 FAPs) can influence the biological burden of indoor air.

61 (PM < 2.5 mum) and CO levels in ambient and indoor air.

62 g filters are not ideal for the treatment of indoor air.

63 These sequelae implicate a high exposure to indoor allergens (house dust mites, pets, molds, etc), t

64 oach is one of the most important sources of indoor allergens and can lead to IgE sensitization and d

65 re to stress, depression, tobacco smoke, and indoor allergens and microbes differentially associate w

66 nd perennial allergic asthma (PAA) caused by indoor allergens in adults with msAD.

67 found at outdoor concentrations of 1-5 ppb, indoor ammonia concentrations can be much higher.

68 sure, and thus a comprehensive evaluation of indoor ammonia concentrations remains an understudied to

69 The observed distribution of angles in indoor and field settings is opposite to the ideotype.

70 s study can be used to more accurately model indoor and inner-city outdoor NH(3) concentrations and a

71 ood exposures (cross-sectionally), including indoor and outdoor air pollutants, built environment, gr

72 Microplastics have been observed in indoor and outdoor air.

73 racterizing complex mixtures of chemicals in indoor and outdoor environments and biological matrices.

74 chemical transformations of BaP by ozone in indoor and outdoor environments are still not fully eluc

75 xposure risk, may vary significantly between indoor and outdoor environments.

76 We focus on features of the indoor and outdoor human-made environment, their microbi

77 iated development of obesity, and changes in indoor and outdoor lifestyle and activity patterns.

78 different host availabilities and from both indoor and outdoor locations.

79 model) and different types of environments (indoor and outdoor scenes).

80 namic scenes of air bubbles in water in both indoor and outdoor settings are studied using an imaging

81 We previously assessed indoor and personal air pollution exposures in this area

82 total, 68% reported strict social distancing indoors and 53% strict masking indoors; indoor social di

83 Environmental surveillance was undertaken indoors and outdoors at 8 locations with automatic agar

84 he type of coal used for cooking and heating indoors and the presence of stove ventilation.

85 ile organic compounds (VOCs) in atmospheric, indoor, and breath air is essential to understand the fu

86 erized by increased urbanization, time spent indoors, and antibiotic usage.

87 havioral experiments in a tightly controlled indoor arena.

88 Early-life indoor bacterial exposure is associated with the risk of

89 We have now thoroughly examined in indoor behavioral assays (4-way olfactometer) and field

90 useholds, however, continue to regularly use indoor biomass-fueled mud stoves (chulhas) alongside LPG

91 We investigated the relationship between indoor burning of biomass, kerosene, and natural gas wit

92 Air pollution from indoor burning of mosquito repellants is a potential ant

93 Entomological monitoring was conducted by indoor CDC light trap, pyrethrum spray catches (PSC) and

94 exposure model was developed (Activity-Based Indoor Chemical Assessment Model (ABICAM)).

95 125 postnatal exposures relating to outdoor, indoor, chemical, and lifestyle factors were assessed, a

96 Air-cleaning systems suppressed indoor chemistry by reducing indoor levels of oxidants (

97 MEChem campaign was conducted to investigate indoor chemistry in a manufactured test house during pre

98 ts of air-cleaning on indoor TRAP levels and indoor chemistry in a renovated school adjacent an inter

99 s, and surfaces is enabling a renaissance in indoor chemistry; this Feature highlights the challenges

100 hemistry plays a central role in controlling indoor chlorine and reactive nitrogen chemistry during t

101 Indoor chulha use decreased by 90% (95% CI = 80% to 101%

102 ble commitment requiring initially disabling indoor chulhas.

103 are consistent with the hypothesis that the indoor climate of metal-roof houses, with higher tempera

104 Here we review evidence of how outdoor and indoor climates are linked to the seasonality of viral r

105 ures in this area; however, the influence of indoor coal combustion and household ventilation on outd

106 onths, data included: week-long average home indoor concentration of PM <=2.5 mum in aerodynamic diam

107 While indoor conditions may differ from ambient outdoor condit

108 table on surfaces for extended periods under indoor conditions.

109 ng and outdoor commuting compared to ambient indoor conditions.

110 global ubiquity of coal use domestically for indoor cooking and heating and commercially for electric

111 )) reached part-per-billion by volume levels indoors during bleach cleaning-several orders of magnitu

112 Toll-like receptor ligands in indoor dust act as environmental adjuvants to promote se

113 rosol partitioning, chemical accumulation in indoor dust and surfaces, air-vegetation partitioning, i

114 estigated the absorption kinetics of Pb from indoor dust following a single dose instillation into C5

115 chemical (SVOC) partitioning between air and indoor dust is investigated by calculating partition rat

116 The indoor dust microbiota composition appears to be a defin

117 These 24 PIs were also detected in indoor dust samples (GM of EPIs = 1483 ng/g).

118 in various environmental matrices including indoor dust, outdoor air particulates, sea sediment, and

119 vary between the packaging materials and the indoor dust, which is attributed in part to the differen

120 ay, we conducted a study to evaluate PFAS in indoor dust.

121 oducts (GM: 34.8 ng/g) were also detected in indoor dust.

122 r newly identified OPE oxidation products in indoor dust.

123 M) concentrations from 2.46 to 70.4 ng/g, in indoor dust.

124 he concentrations of the parent chemicals in indoor dust.

125 may provide a better exposure estimate than indoor dust.

126 uilding materials are important for reducing indoor energy consumption by enabling better thermal ins

127 anic compounds (SVOCs) are ubiquitous in the indoor environment and a priority for exposure assessmen

128 use and waste disposal outlast those to the indoor environment from the indoor use phase, leading to

129 and relevant transformation products in the indoor environment in North America.

130 timated the human exposure to PFASs from the indoor environment through hand-to-mouth and dermal cont

131 rest in deciphering the interactions between indoor environment, fungi, and host in asthma.

132 exposure analysis and risk assessment in the indoor environment.

133 vior of 14 SVOCs with high relevance for the indoor environment.

134 ewer studies have focused on exposure in the indoor environment.

135 ar to those present in ground-based confined indoor environments and are subject to fluctuations, alt

136 However, the evolution of THS in indoor environments is not well understood.

137 The use of e-cigs in indoor environments leads to high levels of fine and ult

138 jor component sodium hypochlorite, NaOCl) in indoor environments leads to the emission of gaseous hyp

139 ed as a proxy for the exposure to PFASs from indoor environments, but further studies are needed to c

140 terials to biodeterioration phenomena in the indoor environments, offers niches for specialized fungi

141 he dominant neutralizer of acidic species in indoor environments, strongly influencing the partitioni

142 tudies have characterized human emissions in indoor environments, the question remains whether VOCs r

143 can greatly alter the composition of air in indoor environments.

144 various volatile organic compounds (VOCs) in indoor environments.

145 t microbes, and by contributing to unhealthy indoor environments.

146 of PAHs in the atmosphere and their fates in indoor environments.

147 n important indirect source of novel OPEs in indoor environments.

148 Despite the substantial role indoor exposure has played in heat wave-related mortalit

149 Separate models were fitted for three indoor exposure metrics, for individual lag days 0-6, an

150 Compared with indoor exposure pathways, the consumption of contaminate

151 Showers were the driving indoor exposure risk compared to sinks and toilets.

152 e temperature difference between outdoor and indoor exterior surface significantly increases during t

153 intained, and we demonstrated cultivation in indoor farming systems.

154 Indoor farms often use soil-free techniques such as hydr

155 ilation will not be as effective at reducing indoor formaldehyde concentrations as it is for other VO

156 gatherings, particularly those taking place indoors, have been linked to multitransmission events th

157 alyses suggest stronger associations between indoor heat and emergency hospital admissions among Afri

158 e associations between short-term changes in indoor heat exposure and cause-specific mortality and mo

159 deling approaches may adequately account for indoor heat exposure in vulnerable neighborhoods.

160 Accounting for indoor heat exposure may improve the estimation of the t

161 We evaluated the role of indoor heat exposure on mortality and morbidity among th

162 We developed a novel methodology to estimate indoor heat exposure that can be adapted to other U.S.

163 Summer indoor heat exposure was modeled at the U.S. Census bloc

164 Indoor heat-health associations were examined using time

165 As a result, knowledge gaps regarding indoor heat-health thresholds, vulnerability, and adapti

166 e examined the health effects of exposure to indoor heat.

167 ns in Houston who are likely exposed to high indoor heat.

168 and Cl(2) tend to be low (10s-100s of ppt), indoor HOCl and Cl(2) can reach high levels during clean

169 Indoor HOCl and nitrogen trichloride (NCl(3)) mixing rat

170 Indoor HONO varied from 0.2 to 84.0 ppb (mean: 5.5 ppb;

171 d compositions may improve the estimation of indoor human exposure to SVOCs present in air and dust a

172 for further characterization of outdoor and indoor impacts of aviation emissions at the neighborhood

173 commonly used as plasticizers, can be found indoors in the gas phase, in airborne particulate matter

174 work (DMN); and (3) the interactions between indoor incense burning and vascular disease markers upon

175 Indoor incense burning is (1) associated with poorer cog

176 To investigate (1) the effects of indoor incense burning upon cognition over 3 years; (2)

177 Indoor incense burning was associated with reduced perfo

178 n over 3 years; (2) the associations between indoor incense burning with the brain's structure and fu

179 Indoor incense use was self-reported as having burnt inc

180 Despite the vast time humans spend indoors, indoor air is far less well chemically characte

181 al distancing indoors and 53% strict masking indoors; indoor social distancing was significantly asso

182 The dynamic behavior of phthalates indoors is not fully understood.

183 hibitor DOlutegravir On the viral Reservoir (INDOOR) is a phase 4 open-label clinical trial that rand

184 tems suppressed indoor chemistry by reducing indoor levels of oxidants (NO(2), O(3)) and reactive org

185 [Formula: see text]) dB(A) and indoor LF WTN [Formula: see text] (vs.

186 d with OH and HO(2) under a typical range of indoor lighting and ventilation conditions.

187 tential for particle formation to occur with indoor lighting during the use of common cleaning produc

188 sunlight harvesting and offering comfortable indoor lighting.

189 Whole-house emission rates and indoor loss coefficients of formaldehyde and other volat

190 an other VOCs, consistent with a significant indoor loss rate attributed to surface uptake.

191 tive to changes in the ACH because its large indoor loss rate muted the impact of ventilation on indo

192 ethods to measure the penetration factor and indoor loss rates for ambient NO(x) constituents using t

193 nds in the total reactive flux were remote < indoor < rural < urban < megacity.

194 d phthalate concentrations in hand wipes and indoor media in their residences.

195 s study was to investigate the links between indoor microbial exposures and pulmonary microbial commu

196 e dysregulation, as was previously shown for indoor microbial exposures.

197 hma cohort were enrolled for analyzing their indoor microbial flora through the use of electrostatic

198 decrease in fungal alpha-diversity of their indoor microbial florae, the latter being significantly

199 ch could be used to model a health-promoting indoor microbiome.

200 d deep sequencing results from pulmonary and indoor microbiomes of patients with asthma with spiromet

201 rmine whether individual bacterial genera in indoor microbiota predict the development of asthma.

202 prehensive inhalation exposure assessment of indoor microenvironments at six commercial printing cent

203 i o 2) was purified from Rhizopus oryzae, an indoor mold causing allergic sensitization.

204 of environmental variables on the density of indoor mosquitoes, sporozoite infected mosquitoes, and m

205 st time, a potential association between the indoor mycobiome and clinical features of patients with

206 For indoor nighttime LF WTN, the HRs (95% CIs) among persons

207 We found no overall association with indoor nighttime LF WTN.

208 relative importance of common activities on indoor nitrous acid (HONO) mixing ratios was explored du

209 sites for directly driving PEC oxidation of indoor NO gas, even in the absence of water vapor.

210 Indoor NO loss rates were strongly correlated with the e

211 The mean (+/-s.d.) indoor NO(2) loss rate was 0.27 +/- 0.12 h(-1), ranging

212 ere tested, including natural and artificial indoor NO(x) elevation with and without accounting for i

213 ted the HONO-photolysis-induced formation of indoor OH, the transformation of OH to hydroperoxy radic

214 dants (NO(2), O(3)) and reactive organics of indoor origin.

215 ide DNAm with allergic sensitization against indoor, outdoor and food allergens, using linear mixed m

216 ted with both negative and positive pressure indoor-outdoor differentials of about 10-15 Pa, and the

217 dance on test design parameters, such as the indoor-outdoor pressure difference (or exhaust fan flow

218 licate meals (0-96 h), feces (24-120 h), and indoor/outdoor dust (<250 mum) were collected.

219 factors (namely, local host availability and indoor/outdoor trapping location) than intrinsic factors

220 x) elevation with and without accounting for indoor oxidation reactions.

221 ted ~3.4 mg/h of indoor SOA formation due to indoor ozone-monoterpene chemistry.

222 ings, rooftops, transparent decorations, and indoor panels.

223 The indoor particles acquired a higher DEHP fraction than la

224 We sampled ammonium sulfate particles and indoor particles of outdoor origin through a small chamb

225 absorption by organic matter present in the indoor particles.

226 Indoor photolysis of nitrous acid (HONO) generates hydro

227 ive tests after being exposed to low ambient indoor PM concentrations and elevated ambient outdoor PM

228 Regulations on the indoor PM(2.5) level and measurement schemes are not wel

229 to PM(2.5) has serious health implications, indoor PM(2.5) monitoring is not a widely applied practi

230 ity and may modify the asthmatic response to indoor PM.

231 on between fatty acid intake and strength of indoor, PM-related asthma symptoms, albuterol use, and s

232 ion of aviation-origin emissions resulted in indoor PNC that were comparable to ambient concentration

233 ent PEC air-purification filter for treating indoor polluted air under ambient conditions.

234 Heated tobacco products are a weaker indoor pollution source than conventional cigarettes, bu

235 everyday living, and control of ambient and indoor pollution.

236 al hygiene measures (including wearing masks indoors); principles on ventilation and sanitization; re

237 06-0.47 h(-1), with strong correlations with indoor relative and absolute humidity.

238 ons, by modeling aggregate human exposure to indoor-released chemicals with diverse partitioning beha

239 le organic compounds (SVOCs) partition among indoor reservoirs in (1) a manufactured test house under

240 mpared outcomes in areas receiving different indoor residual spray (IRS) strategies in Eastern Provin

241 al Malaria Control Centre conducted targeted indoor residual spraying (IRS) in Nchelenge District, Lu

242 Indoor residual spraying (IRS) is widely used as a vecto

243 Indoor residual spraying (IRS) of insecticides is a majo

244 er 2017-June 2018) were deployed with annual indoor residual spraying (IRS), programmatically distrib

245 s-long-lasting insecticidal nets (LLINs) and indoor residual spraying (IRS)-primarily protect against

246 intervention models are informed by targeted indoor residual spraying (TIRS) experiments; trial outco

247 Targeted indoor residual spraying (TIRS) has the potential to mor

248 th birth outcomes in populations residing in indoor residual spraying areas.

249 modeling suggests that the use of form II in indoor residual spraying in place of form I would signif

250 Indoor residual spraying of insecticide (IRS) has been a

251 Although effective in controlling malaria, indoor residual spraying results in elevated exposure to

252 h artemether-lumefantrine, and RAVC involved indoor residual spraying with pirimiphos-methyl.

253 ted an increased rate of outdoor relative to indoor resting; with An. arabiensis reducing the proport

254 and lower humidity, reduces survivorship of indoor-resting mosquitoes and may have contributed to th

255 Environmental interventions targeting indoor school NO(2) levels may improve asthma health for

256 Compared to other indoor settings, PM(2.5) prediction models for large off

257 Many explosive SSEs have occurred in indoor settings, stoking the pandemic and shaping its sp

258 lower numbers of Gram-negative organisms at indoor sites (p < 0.0001).

259 , exposure to environmental tobacco smoke (1 indoor smoker, aRR, 1.19 [95% CI, 1.04-1.35]; >=2 indoor

260 r smoker, aRR, 1.19 [95% CI, 1.04-1.35]; >=2 indoor smokers, aRR, 1.30 [95% CI, 1.02-1.64]; P for tre

261 ons show air-cleaning prevented ~3.4 mg/h of indoor SOA formation due to indoor ozone-monoterpene che

262 ning system was inactive, our data show that indoor SOA formation within the school was elevated.

263 cing indoors and 53% strict masking indoors; indoor social distancing was significantly associated wi

264 ovide insight into vapor entry locations and indoor source contributions.

265 and BTEX compounds likely come from the same indoor source(s).

266 Particles from indoor sources, typically dominated by organic matter, w

267 easured outdoor values, indicating important indoor sources.

268 The INDOOR study is the first evaluation of changes in HIV r

269 g for a few days due to slow desorption from indoor surface reservoirs.

270 react with unsaturated organic compounds on indoor surfaces and in indoor air.

271 oxygenates will form via ozonolysis of oily indoor surfaces and skin oil.

272 Organic films on indoor surfaces are ubiquitous, but details about their

273 ir, respectively; we conclude that uptake to indoor surfaces is an important additional loss process.

274 Experimental observations indicate that indoor surfaces of the residence are large reservoirs of

275 urred via heterogeneous reactions of HOCl on indoor surfaces.

276 Using the GOC, it has been observed that the indoor temperature (at the substrate) of the prototype c

277 he prototype chamber is to maintain the high indoor temperature while the outdoor temperature is low.

278 a concentrations were strongly influenced by indoor temperatures and heating, ventilation, and air co

279 igher particle mass concentrations and lower indoor temperatures in correspondence with theory.

280 rical evidence suggests that human occupants indoors, through their presence and activities, can infl

281 tions compensated low oxidant concentrations indoors to result in an appreciable reactive flux.

282 We investigate impacts of air-cleaning on indoor TRAP levels and indoor chemistry in a renovated s

283 Air-cleaning reduced indoor TRAP to below or near urban background.

284 ansmitted viruses is vector control, such as indoor, ultra-low volume (ULV) spraying.

285 should consider that this allergy can occur indoors, unlike allergies to other types of venom.

286 ast those to the indoor environment from the indoor use phase, leading to slower decline rates in the

287 ern societies, there is increased time spent indoors, use of antibiotics, and consumption of processe

288 mixed with new scenes and classified them as indoor versus outdoor (encoding task) or old versus new

289 wn on a single hectare of land in a 10-layer indoor vertical facility could produce from 700 +/- 40 t

290 , respectively, corresponding to over 88% of indoor VOC oxidant reactivity being consumed outdoors af

291 may be confined within the HONO-photolyzing indoor volume of light.

292 Results suggest that, during indoor walking, increased cognitive load impacted a rang

293 of caught malaria vectors: mosquitoes caught indoors were ten-fold more likely to have sourced their

294 Independent of climate, season, and region, indoor wheat farming could be environmentally superior,

295 Although it is unlikely that indoor wheat farming will be economically competitive wi

296 ved substantial variations in 23 residential indoor WSOG concentrations measured in real time in a No

297 In contrast to nonpolar organic gases, indoor WSOG loss rate coefficients were substantial for

298 that the AC system is an important sink for indoor WSOGs.

299 imated hourly outdoor and low frequency (LF) indoor WTN for each dwelling and derived 1-y and 5-y run

300 ted nighttime outdoor and low-frequency (LF) indoor WTN, using information on WT type and simulated h