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1 cid also resulted in the attenuation of MDMA-induced hyperthermia.
2              Lactulose had no effect on METH-induced hyperthermia.
3  +/- 3%CVCmax) sites in response to exercise-induced hyperthermia.
4 n-linear scattering is optimal for NIR laser-induced hyperthermia.
5 ehavior suggests a role for movement in METH-induced hyperthermia.
6 anandamide metabolism, did not alter cocaine-induced hyperthermia.
7 ic-adrenal-pituitary axis activity or stress-induced hyperthermia.
8 he potential morbidity and mortality of drug-induced hyperthermia.
9 ) significantly attenuated the deltorphin-II-induced hyperthermia.
10 DA receptor activation maximizes mu receptor-induced hyperthermia.
11 njection) with a time course similar to PGE2-induced hyperthermia.
12  physiological antagonist to reduce morphine-induced hyperthermia.
13 ranslationally, inhibits IL-1beta and PGE(2)-induced hyperthermia.
14 ere studied under conditions conducive to MA-induced hyperthermia.
15 on, highlighting their role in female stress-induced hyperthermia.
16 ced BAs might play an important role in MDMA-induced hyperthermia.
17 s agonist-induced hypothermia and antagonist-induced hyperthermia.
18 single alpha-subtype had no effect on stress-induced hyperthermia.
19  (9 of 21 died) vs. asphyxiated rats without induced hyperthermia (3 of 21) or with hyperthermia indu
20 =+30 s after morphine) can decrease morphine-induced hyperthermia; (3) neither the opioid receptor an
21 4 hrs vs. rats subjected to asphyxia without induced hyperthermia (33 +/- 13 vs. 67 +/- 36; p=.002).
22 ed to: no temperature manipulation (n = 21), induced hyperthermia (40 +/- 0.5 degrees C) for 3 hrs be
23 differ from that in rats asphyxiated without induced hyperthermia (59 +/- 21 vs. 67 +/- 36; p=.885).
24 iffer from those in rats asphyxiated without induced hyperthermia (6.4 +/- 3.0 vs. 6.2 +/- 2.6; p=.90
25 ores than rats subjected to asphyxia without induced hyperthermia (9.3 +/- 1.5 vs. 6.2 +/- 2.6; p=.00
26 ided significant protection against the METH-induced hyperthermia and depletion of DA, DOPAC and HVA.
27 bogaine can completely block methamphetamine-induced hyperthermia and HSP-72 expression in the striat
28 erapies for the treatment of psychostimulant-induced hyperthermia and its sequelae.
29 eview the current literature describing drug-induced hyperthermia and its treatment.
30 ngs indicate that UCP-3 is important in MDMA-induced hyperthermia and point to a new therapeutic dire
31 dicate that NMDA receptors modulate morphine-induced hyperthermia and suggest that increases in gluta
32 tors may contribute to the mediation of MDMA-induced hyperthermia and that drugs targeting these rece
33                 As previously reported, METH-induced hyperthermia and the subsequent loss of striatal
34  reduced acoustic startle, attenuated stress induced hyperthermia, and a blunted increase in startle
35  was assessed using acoustic startle, stress-induced hyperthermia, and a challenge with the anxiogeni
36 1 and 3 (UCPDK) fail to show methamphetamine-induced hyperthermia, and have a markedly accelerated lo
37 nnabinoid CB1 receptors to attenuate cocaine-induced hyperthermia, and that dopamine D2 receptor acti
38  METH showed a temporal relationship to METH-induced hyperthermia; and 2.) describe the temporal patt
39 tality rate in asphyxiated rats treated with induced hyperthermia at 24 hrs (9 of 21 died) vs. asphyx
40 cal procedures except asphyxia) treated with induced hyperthermia at 24 hrs (n = 4) or 48 hrs (n = 4)
41 ) for 3 hrs beginning at 24 hrs (n = 21), or induced hyperthermia at 48 hrs (n = 10).
42 investigated which EP receptors mediate PGE2-induced hyperthermia by injecting selective EP receptor
43 l was modestly effective in attenuating MDMA-induced hyperthermia by moderately inhibiting skin vasoc
44 ontrast to the powerful potentiation of MDMA-induced hyperthermia by social interaction and warm ambi
45 ioral phenotypes, including increased stress-induced hyperthermia, defective spatial learning, impair
46                                        D-Fen-induced hyperthermia does not appear to cause generalize
47                              Methamphetamine-induced hyperthermia during the 6 h immediately after tr
48 ring core body temperature and preventing MA-induced hyperthermia from exceeding 41.5 degrees C.
49                                        D-Fen-induced hyperthermia has been shown to coincide with an
50 urrent strategies to treat pathological MDMA-induced hyperthermia in humans are palliative and margin
51 ter maternal separation, to attenuate stress-induced hyperthermia in mice, to increase punished drink
52 circadian rise in Tc or psychological stress-induced hyperthermia in mice.
53 alterations in MDMA pharmacokinetics or MDMA-induced hyperthermia in rats previously exposed to MDMA
54                                         MDMA-induced hyperthermia is highly variable, unpredictable,
55                     However, because cocaine-induced hyperthermia occurs primarily in hot weather, it
56 nd 2.) describe the temporal pattern of METH-induced hyperthermia over an extended dose range.
57 ed to be the key mediators of phenethylamine-induced hyperthermia (PIH).
58 ore temperature increases; the onset of METH-induced hyperthermia ranged from 45 min post-treatment t
59  agents is increasing, the treatment of drug-induced hyperthermia remains unchanged and continues to
60                          Acute noradrenaline-induced hyperthermia requires UCP1 but not UCP3.
61 Galectin-1 (Gal-1) promotes thermal-ablation-induced hyperthermia resistance in HCC and demonstrate t
62 administration, thus indicating that AMG0347-induced hyperthermia results from blockade of tonic TRPV
63 econstruction assistance following microwave-induced hyperthermia to remove the bone tumor.
64 gh fluence rate gave a contribution of laser-induced hyperthermia to the photodamage.
65 uced a marked and immediate reversal of MDMA-induced hyperthermia via inhibition of brain metabolic a
66                                       Stress-induced hyperthermia, via brown adipose tissue (BAT) the
67                           The CCK-8 (300 ng)-induced hyperthermia was blocked by pretreatment of rats
68                                      AMG0347-induced hyperthermia was brought about by one or both of
69                 In contrast, methamphetamine-induced hyperthermia was essentially identical in the tw
70                                     Morphine-induced hyperthermia was first detected at P60 and peake
71                                              Induced hyperthermia when administered at 24 hrs, but no