ライフサイエンスコーパス: induced hypothermia

1 rt rate with a remarkable resistance to cold-induced hypothermia.

2 survival and neurological benefit with mild induced hypothermia.

3 ndent signals do not contribute to adenosine-induced hypothermia.

4 ce, resistant to tumor necrosis factor (TNF)-induced hypothermia.

5 gly, this change was secondary to anesthesia-induced hypothermia.

6 antibody Ba103 significantly reduced peanut-induced hypothermia.

7 analogous to the protective effects of drug-induced hypothermia.

8 ury through the use of hypertonic saline and induced hypothermia.

9 were found in all phenotypes except ethanol-induced hypothermia.

10 ine treatment could be ascribed to adenosine-induced hypothermia.

11 asphyxial cardiac arrest known to occur with induced hypothermia.

12 a CB(2) antagonist, did not affect capsaicin-induced hypothermia.

13 They failed to antagonize nicotine-induced hypothermia.

14 es in the hypothalamus may be related to 2DG-induced hypothermia.

15 ocked the higher dose (10 micrograms) of SST-induced hypothermia.

16 ardiac arrest was decreased with neurotensin-induced hypothermia (14+/-11 secs) and prolonged externa

17 ss ischemic neuronal damage with neurotensin-induced hypothermia (28+/-24%) and prolonged external co

18 For more than a decade, mild induced hypothermia (32 degrees C-34 degrees C) has been

19 ings included perioperative settings without induced hypothermia (60 of 77 comparisons), perioperativ

20 urvival duration was significantly higher in induced hypothermia (7 hrs 22 mins +/- 0 hr 12 mins at 3

21 77 comparisons), perioperative settings with induced hypothermia (8 of 77), and induced hypothermia w

22 In this trial, induced hypothermia added to standard care was not assoc

23 rolonged external cooling) or by neurotensin-induced hypothermia administration 30 mins after asphyxi

24 Mild-to-moderate induced hypothermia after cardiac arrest is feasible and

25 In muscle, induced hypothermia also reversed low oxygen consumption

26 ribe the interaction between the use of mild induced hypothermia and acute diffuse coronary spasm.

27 show that only the former abrogates agonist-induced hypothermia and antagonist-induced hyperthermia.

28 contrast, a TRPA1 antagonist inhibited APAP induced hypothermia and APAP was without effect on body

29 Induced hypothermia and brain death determination.

30 rin rats were hypersensitive to oxotremorine-induced hypothermia and demonstrated an increased sensit

31 ility (AVH) and passive systemic anaphylaxis-induced hypothermia and edema.

32 response, but also blunted 5-HT(1A) agonist-induced hypothermia and increased 5-HT(2A) receptor leve

33 this study was to highlight the link between induced hypothermia and increased survival duration as o

34 terial titres in blood and exudates, E. coli-induced hypothermia and increased survival, demonstratin

35 u opioid receptor subtypes, fully blocked CR-induced hypothermia and increased weight loss during CR

36 background action in protecting against drug-induced hypothermia and speeds recovery from hypothermia

37 For 20 years, induced hypothermia and targeted temperature management

38 s for the understanding of pharmacologically induced hypothermia and thermoregulatory mechanisms.

39 tioned taste aversion, low tolerance to EtOH-induced hypothermia, and high stimulated activity after

40 se of paralytic agents, seizure medications, induced hypothermia, and intracranial pressure monitorin

41 m experimental models and clinical trials of induced hypothermia as treatment after cerebral hypoxia-

42 ccharides significantly attenuated infection-induced hypothermia, augmented leukocyte recruitment and

43 als, practice guidelines recommend that mild induced hypothermia be considered for comatose survivors

44 mized to a normothermic control, neurotensin-induced hypothermia, brief external cooling, or prolonge

45 al was designed to evaluate mild-to-moderate induced hypothermia by use of external cooling blankets

46 sed severe disease in mice, characterized by induced hypothermia, clinical signs, rapid weight loss,

47 with anti-CXCR3 significantly attenuated CLP-induced hypothermia, decreased systemic cytokine product

48 ose tissue, and are more susceptible to cold-induced hypothermia despite having no defect in adrenerg

49 vin receptor ALK7 in BAT resulted in fasting-induced hypothermia due to exaggerated catabolic activit

50 NT77L induced hypothermia (ED(50)=6.5 mg/kg, i.p.) but induced

51 During the induced hypothermia, EEG amplitude and heart rate were m

52 LPS induced hypothermia followed by fever in Wistar rats, wh

53 ombolytic therapy for acute ischemic stroke, induced hypothermia for comatose survivors of cardiac ar

54 essed little, although limited examples (eg, induced hypothermia for out-of-hospital ventricular fibr

55 ates such as near drowning in cold water and induced hypothermia for surgery.

56 Specifically, Oxo-M-induced hypothermia, hypolocomotion, and salivation were

57 Neurotensin-induced hypothermia improved neurologic outcome after as

58 antigen inhibited the development of antigen-induced hypothermia in sensitized mice in vivo and inhib

59 rotein-coupled receptor in vitro (TAAR1) and induced hypothermia in vivo on a rapid time scale.

60 s C (induced normothermia) and 34 degrees C (induced hypothermia)-in septic rats.

61 Induced hypothermia is a promising modality of treatment

62 Induced hypothermia is beneficial in other conditions wi

63 Moreover, 5-HT(1A) receptor agonist-induced hypothermia is blunted and frontal cortex 5-HT(2

64 The proposed mechanism for uridine-induced hypothermia is inhibition of adenosine transport

65 sesses hypothermic activity and mediates LPS-induced hypothermia is known.

66 Taken together, the results show that 5-HT-induced hypothermia is mediated by the 5-HT(7) receptor,

67 hypothesized that 5-hydroxytryptamine (5-HT)-induced hypothermia is mediated by the 5-HT(7) receptor,

68 mice lacking the 5-HT(7) receptor, that 5-HT-induced hypothermia is mediated by the 5-HT(7) receptor.

69 de proof-of-principle showing that capsinoid-induced hypothermia is neuroprotective in an experimenta

70 Induced hypothermia is recommended to improve neurologic

71 ion of brain death after cardiac arrest when induced hypothermia is used.

72 Overall, our results suggest that anesthesia-induced hypothermia leads to ERK inhibition, which in tu

73 Mild-to-moderate induced hypothermia (MIH) is effective in animal models.

74 siological response in vivo in the capsaicin-induced hypothermia model in rats; however, it was ineff

75 Extracorporeal cardiopulmonary resuscitation induced hypothermia more rapidly than surface cooling (p

76 Furthermore, without these LC neurons, DEX induced hypothermia more strongly.

77 We found that after anesthesia-induced hypothermia, MT-free tau was hyperphosphorylated

78 We evaluated the effect of induced hypothermia on neurologic outcomes in patients w

79 ough examination of the effect of anesthesia-induced hypothermia on the risk and progression of AD is

80 L-NAME and 7-NI attenuated hypoxia-induced hypothermia or hypometabolism in lean rats, but

81 to evaluate 3 key questions: (1) Should mild induced hypothermia (or some form of targeted temperatur

82 monstrate the potential of pharmacologically induced hypothermia (PIH) by the novel neurotensin recep

83 e shown to be efficient in pharmacologically induced hypothermia (PIH).

84 scription of successful implementation of an induced hypothermia protocol by one of the authors is pr

85 proved postischemic brain blood flow and ADO-induced hypothermia, rather than adenylate supplementati

86 The anesthesia-induced hypothermia resolved within 10 hrs, and perioper

87 Induced hypothermia restored the fall in oxygen consumpt

88 al cardiac arrest was lower with neurotensin-induced hypothermia (score, 0) and prolonged external co

89 vering intervention in the perioperative and induced hypothermia settings.

90 rdiac care unit admission (baseline), during induced hypothermia (T1), directly after rewarming (T2),

91 Parents were more supportive of a trial of induced hypothermia than of a trial of a new medication

92 Induced hypothermia, the standard treatment for hypoxic-

93 ed level of consciousness, who received mild induced hypothermia to reduce brain damage as suggested

94 he safety, feasibility, and outcomes of mild induced hypothermia treatment following in-hospital card

95 seizures impaired CO(2) chemoreception, and induced hypothermia, two effects reported with serotoner

96 8-OH-DPAT-induced hypothermia was absent in female 5-HTT -/- and m

97 Mild induced hypothermia was apparently the trigger of a seve

98 n contrast, adenosine 5'-monophosphate (AMP)-induced hypothermia was attenuated in QKO mice, demonstr

99 ct of large electrolytic DMH lesions on cold-induced hypothermia was due to suppressed thermogenesis.

100 Capsinoid-induced hypothermia was linked to an activation of heat

101 uced locomotor depression; however, nicotine-induced hypothermia was unaffected.

102 cardiac arrest population treated with mild induced hypothermia, we found a 41% good outcome at hosp

103 type of delta receptor which mediates SNC-80-induced hypothermia, we injected SNC-80 and phenotype se

104 ospital cardiac arrest and treated with mild induced hypothermia were included between January 2004 a

105 less than or equal to 79.5 years and use of induced hypothermia were not.

106 he hypothalamic preoptic area reduced the CR-induced hypothermia, whereas chemogenetic activation of

107 esia per se, but a consequence of anesthesia-induced hypothermia, which led to inhibition of phosphat

108 vival were observed by the pharmacologically induced hypothermia with WIN55, 212-2.

109 d versions 2a and 2b described of a trial of induced hypothermia, with version 2a in paragraph format

110 ings with induced hypothermia (8 of 77), and induced hypothermia without anesthesia (9 of 77).