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1 ondrial intermembrane protein AIF (apoptosis-inducing factor).
2  of mitochondrial cytochrome c and apoptosis-inducing factor.
3 tress and mitochondrial release of apoptosis-inducing factor.
4  of Toll-like receptor-associated interferon-inducing factor.
5 , to mature caspase-8, which is an apoptosis-inducing factor.
6 the ability to respond to the male soporific-inducing factor.
7 ic apoptosis through caspase-3 and apoptosis-inducing factor.
8 lled and released cytochrome c and apoptosis-inducing factor.
9 n, controls production of a diffusible heart-inducing factor.
10 ic protein with some similarity to apoptosis-inducing factor.
11 hondrial proteins cytochrome c and apoptosis-inducing factor.
12 mac/DIABLO, but not the release of apoptosis-inducing factor.
13 tein was termed S. mansoni-derived apoptosis-inducing factor.
14 riosteal cells release a soluble cholinergic inducing factor.
15 /TRX, originally described as an IL-2R alpha-inducing factor.
16 ulation of the proapoptotic factor apoptosis inducing factor.
17 eceptor-interacting protein 1, and apoptosis-inducing factor.
18 ptotic pathway associated with the apoptosis-inducing factor.
19 e it also coincided with increased apoptosis-inducing factor.
20 fect of HDLs on beta-cells against ER stress-inducing factors.
21 for the production of endoderm-derived heart-inducing factors.
22 rasites and reversed the induction of anergy-inducing factors.
23 y, suggesting that bone may provide dormancy-inducing factors.
24 rounds of stimulation in the presence of Th1-inducing factors.
25 termediates (ROI), and release of cell death-inducing factors.
26 on requires cooperation with differentiation-inducing factors.
27 ntified in studies to reveal cell senescence-inducing factors.
28  expression of miR-200 in the absence of EMT-inducing factors.
29                              Differentiation-inducing factor 1 (DIF-1) is a polyketide-derived morpho
30 to the prestalk cell inducer Differentiation inducing factor 1 (DIF-1), or are subjected to hyper-osm
31 ette-inducing small molecule, dubbed rosette-inducing factor 1 (RIF-1), produced by the Gram-negative
32 re the release of cytochrome C and apoptotic inducing factor-1 (AIF1) by Western blotting.
33                              Differentiation-inducing factor-1 (DIF-1) is a polyketide that induces D
34 DNA arrays were used to identify the hypoxia-inducing factor-1 (HIF-1) as a downstream target of TGF-
35 looxygenase-1 (COX-1) and COX-2, and hypoxia-inducing factor-1 alpha (HIF-1 alpha) and vascular endot
36  of several stress pathways, such as hypoxia-inducing factor-1-alpha, receptor-interacting protein 1,
37 cancer specimens and correlates with hypoxia-inducing factor 1alpha (HIF1alpha) expression.
38                                      Hypoxia-inducing factor 1alpha (HIF1alpha) is known to be hypera
39      Glycolysis enhancement required hypoxia-inducing factor-1alpha to up-regulate glucose transporte
40                         A well-characterized inducing factor, 2, 6-dimethoxy-p-benzoquinone (DMBQ), c
41 rocess involving secretion of the cell death-inducing factor 24p3 by mouse leukemia cells, raising th
42 nsity, release of cytochrome C and apoptosis inducing factor, (4) chromatin condensation, nuclear lam
43 poxia, (3) nanoparticles suppressing hypoxia-inducing factor, (4) nanoparticles that relieve tumor hy
44 derived activator of caspases, and apoptosis-inducing factor, accompanied by a proteolytic cascade wi
45  survival factors or overexpression of death-inducing factors accounted for the phenotypes observed.
46 al transition (EMT) in that a variety of EMT-inducing factors activate signaling pathways that conver
47 sed on our initial finding that the mesoderm-inducing factor activin A is suppressed by DeltaNp63 in
48           ECs released B cell-activating AID-inducing factors after sensing microbial products throug
49                                    Apoptosis-inducing factor (AIF) and AMID (AIF-homologous mitochond
50 ecursor, mature, and apoptotic) of apoptosis-inducing factor (AIF) and co-localizes with apoptotic AI
51 ted Bcl-2, resulting in release of apoptosis-inducing factor (AIF) and cytochrome c from mitochondria
52 e of proapoptotic factors, such as apoptosis-inducing factor (AIF) and cytochrome c.
53 dependent mitochondrial release of apoptosis-inducing factor (AIF) and cytochrome complex (Cyt c) is
54 ax and Bak were enhanced, and both apoptosis-inducing factor (AIF) and endonuclease G (Endo G) were r
55 ation of the proapoptotic factors, apoptosis-inducing factor (AIF) and endonuclease G (EndoG), throug
56 bserved a nuclear translocation of apoptosis-inducing factor (AIF) and endonuclease G in CNGA3(-/-)/N
57 h involve release of mitochondrial apoptosis-inducing factor (AIF) and its translocation to the nucle
58 cellular activities, we identified apoptosis-inducing factor (AIF) as an XIAP binding protein.
59               We further show that apoptosis-inducing factor (AIF) cooperated with Bnip3 to promote l
60 nylate kinase-2, cytochrome c, and apoptosis-inducing factor (AIF) during apoptosis and compared the
61 lease and nuclear translocation of apoptotis-inducing factor (AIF) followed by irreversible caspase-i
62               The translocation of apoptosis-inducing factor (AIF) from mitochondria to the nucleus h
63 sociated with the translocation of apoptosis-inducing factor (AIF) from the cytoplasm to nuclei.
64 ctivation and the translocation of apoptosis-inducing factor (AIF) from the mitochondria to the nucle
65 n is required for translocation of apoptosis-inducing factor (AIF) from the mitochondria to the nucle
66 ion as a proviral insertion in the apoptosis-inducing factor (Aif) gene, causing about an 80% reducti
67 duced the nuclear translocation of apoptosis-inducing factor (AIF) in A2058 and SKMEL5 cells, and the
68 on in the mitochondrial release of apoptosis-inducing factor (AIF) in cisplatin-treated renal tubular
69 ized by activation of calpains and apoptosis-inducing factor (Aif) in dying photoreceptors.
70                        The role of apoptosis inducing factor (AIF) in promoting cell death versus sur
71 ndrial release of cytochrome c and apoptosis-inducing factor (AIF) in the penumbra region were reduce
72                                    Apoptosis-inducing factor (AIF) is a bifunctional mitochondrial fl
73                      Mitochondrial apoptosis-inducing factor (AIF) is a central player in the caspase
74                                    Apoptosis-inducing factor (AIF) is a mitochondrial flavoprotein th
75                                    Apoptosis-inducing factor (Aif) is a mitochondrial flavoprotein th
76                                    Apoptosis-inducing factor (AIF) is a mitochondrial flavoprotein wi
77                                    Apoptosis-inducing factor (AIF) is an evolutionarily conserved, ub
78                We report here that apoptosis-inducing factor (AIF) mediates PARP-1-dependent glutamat
79 93Val) in AIFM1, the gene encoding apoptosis-inducing factor (AIF) mitochondrion-associated 1.
80 tivation, and cleavage, as well as apoptosis-inducing factor (AIF) nuclear translocation and executio
81                                    Apoptosis-inducing factor (AIF) promotes cell death yet also contr
82 haracterize the mechanism by which apoptosis-inducing factor (AIF) regulates CI biogenesis by trackin
83 dependent cell death, triggered by apoptosis-inducing factor (AIF) release from mitochondria and its
84 nction suppressed cell killing and apoptosis-inducing factor (AIF) release from mitochondria.
85 sm of cisplatin-induced apoptosis, apoptosis-inducing factor (AIF) release into the cytosol was obser
86 tor Bax in mitochondria, while the apoptosis-inducing factor (AIF) remains unchanged.
87 nsity and nuclear translocation of apoptosis-inducing factor (AIF) suggesting caspase-independent cel
88 release of cytochrome c (CytC) and apoptosis-inducing factor (AIF) through upregulation of Bax expres
89 hat the intramitochondrial protein apoptosis-inducing factor (AIF) translocates to the nucleus and pr
90 ndent neuronal death that involves apoptosis-inducing factor (AIF) translocation from mitochondria to
91 through a unique pathway involving apoptosis-inducing factor (AIF) translocation into the nucleus.
92 d with the mitochondrial localized apoptosis inducing factor (AIF) under both normal and oxidant stre
93                       Induction in apoptosis inducing factor (AIF) was observed, suggesting a paralle
94                                    Apoptosis-inducing factor (AIF) was originally discovered as a mit
95 Cytosolic cytochrome c and nuclear apoptosis-inducing factor (AIF) were increased 3 h after OGD, and
96 uction increased the activation of apoptosis-inducing factor (AIF), a caspase-independent cell death
97                                    Apoptosis-inducing factor (AIF), a mitochondrial oxidoreductase, i
98                                    Apoptosis-inducing factor (AIF), a mitochondrial oxidoreductase, i
99                                    Apoptosis Inducing Factor (AIF), a mitochondrial proapoptotic prot
100 sitive for nuclear localization of apoptosis-inducing factor (AIF), an early event in apoptosis.
101 d AIFM1 gene encodes mitochondrial apoptosis-inducing factor (AIF), an FAD-containing and NADH-specif
102 rylation, as well as cytochrome c, apoptosis-inducing factor (AIF), and endonuclease G (EndoG) releas
103 tivation, nuclear translocation of apoptosis-inducing factor (AIF), and induction of p53, and prevent
104 nd modified caspase-3, Bcl-2, Bad, apoptosis-inducing factor (AIF), and PARP were quantified by immun
105  apoptogenic factors cytochrome c, apoptosis-inducing factor (AIF), and proinflammatory high-mobility
106  of cytochrome c, Smac/DIABLO, and apoptosis-inducing factor (AIF), but not endonuclease G.
107 etion and mitochondrial release of apoptosis-inducing factor (AIF), but the causal relationships betw
108 ction in the mitochondrial protein apoptosis-inducing factor (AIF), exhibited signs of oxidative stre
109  of caspase-independent apoptosis, apoptosis-inducing factor (AIF), from mitochondria is induced by H
110 ear translocation of mitochondrial apoptosis-inducing factor (AIF), known to trigger both apoptotic m
111  of mitochondrial cytochrome C and apoptosis inducing factor (AIF), LC3B-positive neurons, and expres
112 ial release of cytochrome c, Smac, apoptosis-inducing factor (AIF), or loss of mitochondrial membrane
113 iators, including cytochrome c and apoptosis-inducing factor (AIF), was studied in the absence and pr
114 zed a human gene homologous to the apoptosis-inducing factor (AIF), which is named AIF-like (AIFL).
115 X, and release of cytochrome c and apoptosis-inducing factor (AIF), which was translocated to the nuc
116                         AMID is an apoptosis-inducing factor (AIF)-homologous and mitochondria-associ
117 ential, reduced levels of ATP, and apoptosis-inducing factor (AIF)-induced apoptosis.
118         Although hsp70 antagonizes apoptosis-inducing factor (AIF)-mediated cell death, the relative
119 DP-ribose) polymerase (PARP-1) and apoptosis-inducing factor (AIF).
120 type of neuronal PCD involving the apoptosis-inducing factor (AIF).
121 e-independent mechanism, involving apoptosis-inducing factor (AIF).
122 defective nuclear translocation of apoptosis-inducing factor (AIF).
123 rmined by nuclear translocation of apoptosis-inducing factor (AIF).
124 ding cytochrome c, Smac/DIABLO and apoptosis-inducing factor (AIF).
125 but not endonuclease G (EndoG) and apoptosis-inducing factor (AIF).
126 ates in the nucleus, together with apoptosis-inducing factor (AIF).
127 in the release of cytochrome c and apoptosis-inducing factor (AIF).
128 ition and nuclear translocation of apoptosis-inducing factor (AIF).
129    Liberibacter solanacearum": the apoptosis-inducing factor AIF3 was downregulated in LsoA-infected
130 dentified mitochondrion-associated apoptosis inducing factor (AIFM1) have roles in the induction of a
131 loss was linked to upregulation of apoptosis-inducing factor, although only a minute fraction of cell
132 pathway involving translocation of apoptosis-inducing factor and caspase 12 to the nucleus.
133  expression of apoptotic proteins (apoptosis-inducing factor and cleaved caspase-3) and autophagy pro
134 rleukin (IL)-18 is an interferon (IFN)-gamma-inducing factor and contributes to the Th1 immune respon
135 rial depolarization and release of apoptosis-inducing factor and cytochrome c Furthermore, this prote
136 in both cell types, but release of apoptosis-inducing factor and endonuclease G was detected only in
137 ed from the mitochondrion, such as apoptosis-inducing factor and endonuclease G, may induce caspase-9
138               CIMD also depends on apoptosis-inducing factor and endonuclease G, which are effectors
139 ribofuranosylbenzimidazole (DRB) sensitivity-inducing factor and found that the IC(50) determined was
140 ented intranuclear localization of apoptosis-inducing factor and protected neurons from excitotoxic i
141 ssing the mitochondrial release of apoptosis-inducing factor and Smac/DIABLO.
142  further examined the association of Th1/Th2 inducing factors and allergic disease and intestinal inf
143  under inflammatory conditions; however, the inducing factors and underlying mechanisms remain unknow
144 mitochondrial caspase-independent (apoptosis-inducing factor) and caspase-dependent (Smac/Diablo and
145 in (IL)-1beta, IL-18 (interferon (IFN)-gamma inducing factor) and IFN-gamma, but not tumour-necrosis
146 g., cytochrome c, Smac/DIABLO, and apoptosis-inducing factor), and caspase activation.
147 irectly the release of Cyt c, AIF (apoptosis-inducing factor), and Smac (second mitochondria-derived
148 elongation factor) and DSIF (DRB sensitivity-inducing factor)--and P-TEFb (positive elongation factor
149 ndria, release of cytochrome c and apoptosis-inducing factor, and activation of caspase-9 and caspase
150  translocation of cytochrome c and apoptosis inducing factor, and active caspases 3 and 7, consistent
151 release of mitochondrial proteins, apoptosis-inducing factor, and cytochrome c.
152 itochondrial proapoptotic factors, apoptosis inducing factor, and endonuclease G.
153 osolic release of cytochrome c and apoptosis-inducing factor, and mitochondrial membrane potential ch
154 t stress, mitochondrial release of apoptosis inducing factor, and nuclear DNA fragmentation resulting
155  respiration, prevented release of apoptosis-inducing factor, and reduced neuronal cell death trigger
156 e leakage of both cytochrome c and apoptosis-inducing factor, and significantly improved cell surviva
157 ded MHC class II molecules, interferon-gamma inducing factor, apolipoprotein E, and cathepsin S.
158         In this study, we show that the CCR8-inducing factors are heat stable and protease resistant
159  illustrates the complex regulation to which inducing factors are subject.
160 and the pausing factor DSIF (DRB sensitivity-inducing factor) are still present at the hsp70 loci in
161  CCK-induced caspase 3 activation, apoptosis-inducing factor, as well as X-linked inhibitor of apopto
162 d that in this nondividing oocyte, the nerve-inducing factor Ascl1 can remain bound to a specific chr
163  one copy of the binding site for the neural-inducing factor Ascl1 is injected directly into a Xenopu
164 ation and mitochondrial release of apoptosis-inducing factor at low microM concentrations.
165                 Total and modified apoptosis-inducing factor, Bcl-2 family proteins, phospho-PKC-alph
166 otic cells and expression of total apoptosis-inducing factor, Bcl-2, Bak, and Bax in the pre-CP/Rep a
167        One model for NMD suggests that decay-inducing factors bound to mRNAs during early processing
168          NGF was found to be a proliferation-inducing factor but not a survival factor.
169 varian cancer, we show that "helper" NK cell-inducing factors can be used to enhance local production
170 d key apoptotic mediators, such as apoptosis-inducing factor, caspase 3, caspase 8, caspase 9, poly(a
171  Bcl-2 and increased expression of apoptosis-inducing factor, caspase-3, and cleavage of BID, c-IAP-1
172 mediated by the enhanced induction of anergy-inducing factors cbl-b, c-cbl (cbl is abbreviation for C
173  FLP represses the expression of the mitosis-inducing factor CDKB1;1, which, along with CDKB1;2, is s
174 ial meningitis and induces a novel apoptosis-inducing factor-dependent (AIF-dependent) form of brain
175 ath proceeded predominately via an apoptosis-inducing factor-dependent pathway in XY neurons versus a
176 sion is induced by the stalk differentiation-inducing factor DIF-1 and is restricted to a subset of p
177 osed to the prestalk inducer differentiation inducing factor (DIF-1), a chlorinated hexaphenone.
178                              DRB sensitivity-inducing factor (DSIF or Spt4/5) is a conserved transcri
179 eta-d-ribofuranosylbenzimidazole sensitivity-inducing factor (DSIF) and negative elongation factor (N
180 eta-D-ribofuranosylbenzimidazole sensitivity-inducing factor (DSIF) are involved in pausing RNA Polym
181 elongation factor (NELF) and DRB sensitivity inducing factor (DSIF) contribute in the establishment o
182 ribofuranosylbenzimidazole (DRB) sensitivity-inducing factor (DSIF).
183 hloro-1-beta-D-ribobenzimidazole-sensitivity inducing factor (DSIF).
184  reversed by the presence of DRB sensitivity-inducing factor (DSIF).
185  by the retinoid in cells exposed to the EMT-inducing factors EGF and heregulin-beta1.
186 her mitochondrial related factors (apoptosis-inducing factor, endonuclease G, and HtrA2/Omi).
187 itochondria, and cytochrome c, and apoptosis-inducing factor escaped from mitochondria to the cytopla
188  or Toll-like receptor-associated interferon-inducing factor expression does not affect the LPS-trigg
189 rental cells, whereas knockdown of apoptosis-inducing factor expression suppressed lapatinib toxicity
190 pase inhibitor and by knockdown of apoptosis-inducing factor expression.
191 itioned medium (MG63CM) contains a migration-inducing factor for MCF-7 cells.
192 brane permeability, and release of apoptosis-inducing factor from mitochondria are partially blocked
193 AD(+) blocked translocation of the apoptosis-inducing factor from mitochondria to nuclei, a step prev
194 phate levels, and translocation of apoptosis-inducing factor from mitochondria to the nucleus, result
195 ing cytochrome c and translocating apoptosis inducing factor from mitochondria to the nucleus.
196  of cytochrome c, Smac/DIABLO, and apoptosis inducing factor from mitochondria, and reduced mitochond
197 nd the release of cytochrome c and apoptosis inducing factor from mitochondria.
198 before release of cytochrome c and apoptosis-inducing factor from mitochondria.
199 hondrial mu-calpain and release of apoptosis-inducing factor from the mitochondrial intermembrane spa
200 sary for the complete discharge of apoptosis-inducing factor from the mitochondrial intermembrane spa
201                             Isolation of CD1-inducing factors from M. tuberculosis using normal phase
202 sion of caspase 4, cathepsin B, or apoptosis-inducing factor function significantly suppressed cell k
203 ced immunotherapeutic efficacy; (c) the MDSC-inducing factors G-CSF and GM-CSF facilitated IRF-8 down
204     No new classes of extracellular mesoderm-inducing factors have been identified in more than two d
205 e feedback loop with Dronc and the apoptosis-inducing factors Hid and Reaper.
206               Loss of the C. elegans hypoxia-inducing factor HIF-1, which regulates iron homeostasis,
207 inase (PDK1) and EGFR along with the hypoxia-inducing factor (HIF)-1alpha in human glioblastoma multi
208 are also induced by the exudate (hormogonium-inducing factor [HIF]) of a symbiotic plant partner.
209 es of T. versicolor roots to these haustoria inducing factors (HIFs) included localized swelling and
210                              AMID (apoptosis-inducing factor-homologous mitochondrion-associated indu
211 The equine herpesvirus 1 (EHV-1) alpha-trans-inducing factor homologue (ETIF; VP16-E) is a 60-kDa vir
212 tes recruitment of Smac/Diablo and apoptosis-inducing factor in chronic neurodegeneration.
213 dependent nuclear translocation of apoptosis-inducing factor in NMDA-treated neurons and reduced tPA-
214 hanism independent of caspase- and apoptosis-inducing factor in nonproliferating U937 cells.
215  is the major cyclooxygenase-2 mRNA and PGE2-inducing factor in pulmonary edema fluid and accounts fo
216  failed to release cytochrome c or apoptosis-inducing factor in response to recombinant Bax or trunca
217 t an additional function of EHD1 as a tubule-inducing factor in the Arf6 pathway for recycling of pla
218 ble protein 3alpha was the most active mucin-inducing factor in the RSV-infected human small airway e
219     One candidate for an endogenous mesoderm-inducing factor in Xenopus is derriere, a member of the
220 growth by down-regulating other angiogenesis-inducing factors in addition to VEGF and that the centra
221                                        Heart-inducing factors in amphibians are not known, and althou
222  management and enable identification of NET-inducing factors in individual patients for targeted tre
223 omous manner, indicating a role for secreted inducing factors in the response to FoxD3.
224      Exposure of hAd-PSCs to differentiation-inducing factors in vitro upregulated steroidogenic gene
225 ce and reveal a role for Id4 as a quiescence-inducing factor, in contrast with its role of promoting
226       Both types of endoderm express cardiac-inducing factors, including WNT antagonists Dkk1 and Sfr
227 1) peptide has been reported as an autophagy-inducing factor inhibiting the replication of pathogens
228 resulting in release of the interferon gamma inducing factor interleukin-18 (IL-18).
229 transfer experiments reveal the PS30-derived inducing factor is soluble and promotes mitogenic ERK an
230 and Toll-like receptor-associated interferon-inducing factor, is blocked by both FADD and phosphatidy
231 eling and in cytosolic and nuclear apoptosis-inducing factor labeling within 60 min.
232 ormation on whether Smac/Diablo or apoptosis-inducing factor might play a role in chronic neurodegene
233     We found that the flavoprotein apoptosis-inducing factor mitochondria-associated 2 (AIFM2) is a p
234 tein 1 (FSP1) (previously known as apoptosis-inducing factor mitochondrial 2 (AIFM2)) as a potent fer
235 ndrial release of cytochrome c and apoptosis-inducing factor, mitochondrial membrane depolarization,
236 ) for the phosphorylation of DRB sensitivity-inducing factor, negative elongation factor, and C-termi
237   Accordingly, ablation of the primary pause-inducing factor NELF does not increase expression of lin
238 ss of pausing through knockdown of the pause-inducing factor NELF leads to broadly attenuated immune
239 e earliest identified BMP antagonists/neural-inducing factors, noggin and chordin, were expressed in
240 ds to a POU motif in the promoter of the rod-inducing factor Nrl to repress its expression.
241 se) polymerase) cleavage, and AIF (apoptosis-inducing factor) nuclear translocation.
242  and efflux rate modulation as the strongest inducing factor of chronic inflammation for a wide range
243 e the activity of Nodal, a secreted mesoderm-inducing factor of the transforming growth factor-beta (
244                The existence of an ovulation-inducing factor (OIF) in seminal plasma has broad implic
245 be maintained by continued expression of the inducing factor or by a mechanism that confers a stable
246 enotype after in vitro treatment with neural-inducing factors or after delivery into the brain.
247 induction of autophagy, release of apoptosis-inducing factor, or opening of the mitochondrial permeab
248 the elongation factors DSIF (DRB Sensitivity-Inducing Factor), P-TEFb (Positive Transcription Elongat
249 ng RNA to reduce the expression of apoptosis-inducing factor partially inhibited CDDO-induced apoptos
250 o occur by a caspase 3-independent apoptosis-inducing factor pathway.
251 f biphenyl dioxygenase (BphA4) and apoptosis-inducing factor, Pdr lacks one of the arginine residues
252 ep, the amount of apoptotic cells, apoptosis-inducing factor, phospho-Bad, phospho-PKC-alpha, phospho
253 ukin (IL) -1beta, TNF-alpha, and proteolysis-inducing factor (PIF) by cancer cells used in this study
254                 A novel protein, proteolysis-inducing factor (PIF), has been isolated from the urine
255 d delta-toxin as the mast cell degranulation-inducing factor produced by S. aureus.
256 ollagen XVIII (NC1) functioned as a motility-inducing factor regulating the extracellular matrix-depe
257 y transition, and cytochrome C and apoptosis-inducing factor release from isolated mitochondria.
258 x translocation, cytochrome c, and apoptosis-inducing factor release) and apoptosis by imatinib mesyl
259 tivation) and caspase-independent (apoptosis-inducing factor release) pathways, and limited neuronal
260 g., cytochrome c, Smac/DIABLO, and apoptosis-inducing factor release), caspase activation, and apopto
261 P binding protein with low pI, and apoptosis-inducing factor release), caspase activation, poly(ADP-r
262 rial dysfunction (cytochrome c and apoptosis-inducing factor release), caspase-3 and -8 activation, a
263 rial dysfunction (cytochrome c and apoptosis-inducing factor release), caspase-3 and -8 activation, a
264 ificantly reduced cytochrome c and apoptosis-inducing factor release, loss of mitochondrial membrane
265 ed by the mitochondrial release of apoptosis-inducing factor, resulting in caspase-independent cell d
266 ng the release of cytochrome c and apoptosis-inducing factor, resulting in DNA fragmentation.
267 in some chicken embryos and named resistance-inducing factor (RIF) because it interferes with RSV.
268                  Here we show that a rosette inducing factor (RIF-1) produced by A. machipongonensis
269 ergizes with activating sulfonolipid rosette-inducing factors (RIFs) to recapitulate the full bioacti
270                The keratinocyte-derived CCR8-inducing factor(s) were soluble, and independent of vita
271     These exosomes increased chemoresistance-inducing factor, Snail, in recipient epithelial cells an
272  cells were hypersensitive to various stress-inducing factors, such as salts, SDS, and H(2)O(2), espe
273            The presence of cleaved apoptosis inducing factor (tAIF) and the absence of activated casp
274                 Furthermore, differentiation-inducing factors that elevate MITF expression in melanom
275        Thus, bone secretes multiple dormancy-inducing factors that employ distinct signaling pathways
276 melanocortin peptide agonists act as satiety-inducing factors that mediate their action through the m
277 ial-derived signal in the intestinal stroma, inducing factors that restrict epithelial proliferation
278          Although IL-12 is also an IFN-gamma-inducing factor, the activity of IL-18 (but not IL-12) i
279 ly delivering a specific combination of Treg inducing factors through degradable polymer microspheres
280 ces BMP4, a well-established HFSC quiescence-inducing factor, thus contributing to a proliferation-in
281                 The first candidate mesoderm-inducing factor to be identified was activin, a member o
282 lease and nuclear translocation of apoptosis-inducing factor to initiate chromatinolysis and cell dea
283 s activation, the translocation of apoptosis-inducing factor to the nucleus, and DNA fragmentation in
284 e permeability or translocation of apoptosis-inducing factor to the nucleus.
285 ted, at least in part, by specific fugetaxis-inducing factors to which only mature cells respond.
286 , and Egf and up-regulation of the apoptosis-inducing factor Trail in the oviduct.
287 lycolysis in response to the differentiation-inducing factor transforming growth factor beta1 (TGF-be
288 ough mitochondrial dysfunction and apoptosis-inducing factor translocation from the mitochondria to t
289 induced mitochondrial dysfunction, apoptosis-inducing factor translocation, and subsequent cell death
290 yD88 adaptor-like protein, Toll receptor IFN-inducing factor (Trif), and Trif-related adaptor molecul
291                       Interferon (IFN)-gamma-inducing factor was previously termed interleukin (IL)-1
292                           However, apoptosis-inducing factor was released from mitochondria into the
293 eta-synuclein can act as a neurodegeneration-inducing factor, we demonstrated that wild-type beta-syn
294 d release from mitochondria of the apoptosis-inducing factor were selectively abrogated in iNOS(-/-)
295                        Glial-derived barrier-inducing factors were characterized using size-exclusion
296 polarization and relocalization of apoptosis-inducing factor, whereas the BRAF-V600E-mutated melanoma
297 lease and nuclear translocation of apoptosis-inducing factor with subsequent cell death.
298 ation of caspases-1, -3, and -8 or apoptosis-inducing factor within MNs, with a blockade of apoptosis
299 ion in ECs infected with the TB40E or fusion-inducing factor X (FIX) HCMV strains.
300 ndothelial cells by BADrUL131 and the fusion-inducing factor X clinical human cytomegalovirus isolate

 
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