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1 vely on the C/EBPalpha promoter prior to the induction of differentiation.
2 ement of cell viability may be linked to the induction of differentiation.
3 /Waf1 displayed decreased sensitivity to the induction of differentiation.
4 expressed at lower levels in cells after the induction of differentiation.
5 tions permitting continuous cell division or induction of differentiation.
6 iated with an apoptotic response elicited by induction of differentiation.
7 AU565, a decreased rate of cell growth, and induction of differentiation.
8 with activating PPARgamma ligands, efficient induction of differentiation.
9 e Ac-mut-expressing cells prior to and after induction of differentiation.
10 NTERA-2 cells, even several weeks after the induction of differentiation.
11 ese molecules is insufficient to mediate the induction of differentiation.
12 to make rapid changes in those profiles upon induction of differentiation.
13 unlike the Dnmt1 null ES cells that die upon induction of differentiation.
14 sitive to the stage of the cell cycle and to induction of differentiation.
15 ase are necessary to block pluripotency upon induction of differentiation.
16 e switch in the mode of DNA replication upon induction of differentiation.
17 tissues and in two cell lines following the induction of differentiation.
18 nduction of cell death as well as during the induction of differentiation.
19 sis undergo the strongest upregulation after induction of differentiation.
20 the consequence of Cox6b2 knockdown was the induction of differentiation.
21 This metabolic setup did not change after induction of differentiation.
22 pressed of the synthases and increased after induction of differentiation.
23 gulated expression of pluripotency genes and induction of differentiation.
24 he stem state and rapidly declined following induction of differentiation.
25 l gene expression signatures consistent with induction of differentiation.
26 nificantly attenuated NSC proliferation upon induction of differentiation.
27 ic regulatory factors MyoD and myogenin upon induction of differentiation.
28 cytes show adipogenesis defects 1 week after induction of differentiation.
29 from Th1 to Th2 cytokine production upon the induction of differentiation.
30 l self-renewal that normally occurs with the induction of differentiation.
31 nt Eed, and were preferentially activated on induction of differentiation.
32 ere significantly up-regulated subsequent to induction of differentiation.
33 nhibition of tumor growth associated with an induction of differentiation.
34 dipocytes and their expression declines upon induction of differentiation.
35 d severely delayed adipogenesis 1 week after induction of differentiation.
36 stem cells can reform spheres even after the induction of differentiation.
37 tor interferes with C/EBPbeta function after induction of differentiation.
38 -transduced wild type bone marrow cells upon induction of differentiation.
39 n expression, diminished growth capacity and induction of differentiation.
40 ous role in the suppression of apoptosis and induction of differentiation.
41 inhibition of cellular proliferation and the induction of differentiation.
42 nly when added between 12 and 24 h after the induction of differentiation.
43 exhibits negative regulatory effects on the induction of differentiation.
45 he downregulation of pluripotency markers on induction of differentiation after withdrawal of the ES
46 preventing retinoic-acid-receptor-dependent induction of differentiation along a neuronal pathway in
48 d by all-trans-retinoic acid (RA) to mediate induction of differentiation and apoptosis of malignant
49 render hematopoietic cells refractory to the induction of differentiation and are, thereby, likely to
50 EVI-1-positive AML cases respond to ATRA by induction of differentiation and decreased clonogenic ca
51 d induction of p21WAF1 may be crucial to the induction of differentiation and G1 arrest in Daudi cell
52 was active in clonal inhibition, as well as induction of differentiation and WAF1 expression of HL-6
53 creased expression of the c-fos target mRNA, induction of differentiation, and inhibition of s.c. tum
54 sults in the inhibition of proliferation and induction of differentiation, and that U2OS cells transd
55 -grown keratinocyte cultures, decreases upon induction of differentiation, and then rises to levels a
56 morpholinosydnonimine-induced neurotoxicity, induction of differentiation, and up-regulation of hypox
58 erexpressing the G-CSFR to G-CSF resulted in induction of differentiation as measured by (1) the abil
59 ll adhesion is an essential prerequisite for induction of differentiation, as observed at each step o
60 on, retention of pluripotency, and premature induction of differentiation, associate with teratoma su
63 siRNA treatment had no effects on C/EBPalpha induction of differentiation but inhibited proliferation
65 played a 2-3-fold increase in sensitivity to induction of differentiation by 2 mM sodium butyrate.
66 eratin 10, and loricrin, with or without the induction of differentiation by an elevated extracellula
70 In contrast, inhibition of proliferation and induction of differentiation by TNF were still observed
71 Growth inhibition by RA may be exerted by induction of differentiation, cell cycle arrest, or apop
78 ified as a gene that was up-regulated by the induction of differentiation in a colon carcinoma cell l
80 inuous telomere instability triggered by the induction of differentiation in ATRX-deficient stem cell
81 estigated for a role in growth inhibition or induction of differentiation in breast cancer cells.
82 timulation, growth inhibition, apoptosis and induction of differentiation in cells expressing the HER
83 identified as a gene strongly upregulated by induction of differentiation in colon carcinoma cells in
85 DNAs was strongly regulated in NIH3T3 cells, induction of differentiation in PC-12 cells by FGF-4 (as
87 ype I receptor, dramatically accelerated the induction of differentiation in sparse, proliferating cu
88 ole during cellular differentiation and that induction of differentiation in the absence of Dab2 expr
90 atinocytes in culture, and upregulated after induction of differentiation in vitro, along with upregu
91 the coactivator DRIP205 is a determinant of induction of differentiation, in response to PPARgamma a
92 ess of histone H3-Lys4 methylation following induction of differentiation, indicating that CFP1 restr
94 t the decrease in PDGFR expression following induction of differentiation is a transcriptionally regu
95 ness of 3T3-L1 preadipocytes to RA after the induction of differentiation is initially due to the red
96 e is repressed by AP-2alpha/CUP, which, upon induction of differentiation, is down-regulated, allowin
98 HuR is constitutively expressed and prior to induction of differentiation localized predominantly to
100 n a time-dependent manner that is similar to induction of differentiation markers such as keratin 10
103 te that control of cell proliferation during induction of differentiation may directly involve, at th
104 p21(waf1) provides a novel link between the induction of differentiation, mRNA stability, and the te
106 he C/EBPbeta gene promoter occurs only after induction of differentiation of 3T3-L1 preadipocytes and
110 mulation and phosphorylation associated with induction of differentiation of leukemic cells suggested
111 ses revealed that, during the first hours of induction of differentiation of mammalian embryonic stem
112 Id2 mRNA levels markedly increased with induction of differentiation of myeloid blasts (HL-60, P
113 and a key role for glucagon in the paracrine induction of differentiation of other pancreatic compone
114 e their expression and is downregulated upon induction of differentiation of pluripotent stem cells;
115 ivity were found to be up-regulated upon the induction of differentiation of the human monocyte cell
119 pendent manner, and the order of potency for induction of differentiation parallels the potencies for
121 can regulate cell cycle exit coincident with induction of differentiation programs during development
123 UVB (290-320 nm wavelengths) exposure before induction of differentiation reduced expression of diffe
124 s of this pathway revealed that IL6-mediated induction of differentiation resulted in rapid cell surf
125 either striatal deafferentation nor in vitro induction of differentiation resulted in significant neu
126 ghly expressed in pluripotent cells, and the induction of differentiation results in the down-regulat
127 tochemical, and molecular analysis after the induction of differentiation showed that EMSC maintain a
128 ic inhibitor (U0126 or PD98059) prior to the induction of differentiation significantly attenuated th
129 though MyoD expression is not inhibited, the induction of differentiation-specific genes such as myog
130 shed ability of RA to activate RAR following induction of differentiation stems from down-regulation
131 tured C2C12 myoblasts increased apoptosis on induction of differentiation, suggesting a need for bag3
132 een eliminated, XBP-1 is induced normally on induction of differentiation, suggesting that activation
133 nesis only occurred following a delay in the induction of differentiation that coincided with the del
134 ommitment is known to occur 9-14 h after the induction of differentiation, the factors that regulate
135 ar processes are invoked simultaneously upon induction of differentiation, the regulated progression
137 was observed in Dnmt1(-/-) ESC cultures upon induction of differentiation through the withdrawal of l
138 proliferation by N-CAM was accompanied by an induction of differentiation to the neuronal lineage, as
139 to S6K) is gradually inverted upon in vitro induction of differentiation toward the neutrophilic phe
140 ations provide a mechanistic explanation for induction of differentiation upon treatment with DNA met
141 PPARgamma expression was reduced prior to induction of differentiation using a novel, chemically m
142 ntiation (early, middle, and late) following induction of differentiation using Hes5::GFP, Nurr1::GFP
144 on in six of nine patient samples; of those, induction of differentiation was documented in four pati
145 g growth stimulation, growth inhibition, and induction of differentiation, we systematically examined
146 mor suppressor through growth inhibition and induction of differentiation whereas in advanced cancers
147 a rapid induction of cell death and a slower induction of differentiation, which are likely to be rec
148 ased upon serum addition and decreased after induction of differentiation with either sodium butyrate
149 vel and in individual cells before and after induction of differentiation with hemin or DMSO show tha
151 otch1 and Notch2 biosynthesis is enhanced by induction of differentiation with serum-containing media
152 expression of Wnt10b mRNA is suppressed upon induction of differentiation, with a 50% decline by 6 h
153 tected in 3T3-L1 cells, before and after the induction of differentiation, with the level increasing