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1 Aging, for virtually all life, is inescapable.
2 after sleep deprivation is intense and seems inescapable.
3 s with time spent awake, until sleep becomes inescapable.
4 ain vasculature, areas of low blood flow are inescapable and cannot be removed by functional hyperemi
6 psychological, that are overwhelming (i.e., inescapable and unpredictable), can measurably affect su
18 ting from such neutrophil accumulation is an inescapable consequence of parenchymal cell death has no
20 Thus, intercellular competition creates an inescapable double bind that makes aging inevitable in m
22 -escape deficits caused by prior exposure to inescapable electric shock in rats (learned helplessness
25 etrieval emotional arousal (restraint stress/inescapable foot shock, exposure to the predator odor TM
27 ntral medial striatum throughout training on inescapable footshock and signaled active avoidance task
28 mice, we found that repeated forced swim and inescapable footshock both produced aversive behaviors t
30 y old rats were exposed to unpredictable and inescapable footshocks, and fear memory for the shock co
31 im test and reduced levels of freezing after inescapable footshocks, suggesting that M(1)R(-/-) mice
35 that exposure of rats to randomly presented, inescapable loud sound, referred to as sound stress, inc
36 variations within each age, activities in an inescapable novel environment and novel object explorati
39 mal in their behavioral response to a novel, inescapable open field and in their preference for a nov
40 ; spontaneous locomotor activity in a novel, inescapable open field; and novelty place preference.
46 a new and general method for exploiting the inescapable protein corona to target nanomaterials to sp
47 of serotonergic neuronal function, show that inescapable, randomly presented sound pulses activate se
52 EPM) after exposure to escapable shock (ES), inescapable shock (IS) or fear conditioning (FC), result
56 g network analysis, we find that exposure to inescapable shock (IS), compared to context training, re
58 rain IL-1beta was explored after exposure to inescapable shock (IS; 100 1.6 mA tail shocks for 5 sec
60 ges occur in the dorsal raphe nucleus during inescapable shock and that such changes may contribute t
61 se inhibitor, mimicked the effect of earlier inescapable shock at a dose of 2.5 microM in previously
62 shuttle-escape performance in the manner of inescapable shock in a dose-dependent manner and acted s
63 eceptor impairs escape performance following inescapable shock in the learned helplessness paradigm.
67 their own preshock baseline, rats exposed to inescapable shock showed an increase in extracellular 5-
68 s produced by EHNA or by earlier exposure to inescapable shock were reversed by intraperitoneal injec
73 ynergistically with an ineffective number of inescapable shocks to maximally impair test performance.
74 spects of fear (i.e., fear conditioning) and inescapable stress (i.e., struggling and helplessness).
75 disorder wherein a subset of mice exposed to inescapable stress (IS) develop a deficit in escape beha
80 l raphe nucleus increases active coping with inescapable stress in rats and mice in a time-locked man
85 e (HC), during passive coping in response to inescapable stress under tail suspension (TS), and durin
87 swim is usually considered a consequence of inescapable stress, and is used to screen antidepressant
88 uisition, either with or without exposure to inescapable stress, consistent with the hypothesis that
97 task in which mice were exposed to repeated inescapable stressors followed by an active avoidance ta
98 at ACh levels in the mPFC during exposure to inescapable stressors were positively correlated with la
100 entricular (i.c.v.) IL-1beta and exposure to inescapable tail shock (IS) activate acute phase respons
101 al responses assessed 24 h after exposure to inescapable tail shock stress (IS) in adult male rats.
102 In this study, male rats were exposed to inescapable tail shock, loud noise or restraint, and the
107 the caudal dorsal raphe nucleus (DRN) during inescapable tailshock (IS) has been shown to be critical
108 le Sprague-Dawley rats to escapable or yoked inescapable tailshock and assessed LC activity by measur
109 ve been observed following stressors such as inescapable tailshock and social isolation, while no cha
112 A prior report indicated that exposure to inescapable tailshocks (IS) raised levels of brain IL-1b
113 he paradigm established by these data, it is inescapable that going forward, investigators will opera
114 the breadth of topics covered reflected how inescapable the influence of noncoding RNAs is in develo
119 ochemical and behavioral changes that follow inescapable, uncontrollable tail shocks (ISs) in Sprague
120 eviously reported that rats that experienced inescapable-unpredictable stress subsequently exhibited
121 wed how a similar condition, if perceived as inescapable, would result in a downregulation of interoc