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1 is isolate with the clinical outcome for the infected patient.
2 is an important complication in the HIV/HBV infected patient.
3 bodies (mAbs) that target IBV NA from an IBV-infected patient.
4 ion was associated with lower viral loads in infected patients.
5 ymphocytes from antiretroviral treated HIV-1 infected patients.
6 an understanding of the antibody response in infected patients.
7 OVID-19 and in choosing proper management of infected patients.
8 d thromboembolism are frequent in SARS-CoV-2-infected patients.
9 against HDV in 12 treatment-naive HBV/HDV co-infected patients.
10 d cells derived from hepatitis C virus (HCV)-infected patients.
11 t cause lethal infections, especially in HIV-infected patients.
12 s, handling of infected livestock or care of infected patients.
13 ccf-mtDNA levels that were higher among HIV-infected patients.
14 expressed in highly permissive cells and HBV-infected patients.
15 unodeficiency virus (HIV) progression in HIV-infected patients.
16 the role of histone PTMs in chronically HBV-infected patients.
17 y facilitate the fast growth rate of UPEC in infected patients.
18 sseminated histoplasmosis, especially in HIV infected patients.
19 places an emphasis on therapeutic aspects of infected patients.
20 zid alone for preventing tuberculosis in HIV-infected patients.
21 may be a surrogate marker of GC in H. pylori-infected patients.
22 vels were significantly elevated in COVID-19-infected patients.
23 hysicians to quickly make their decision for infected patients.
24 milar rates of HCV eradication than HCV mono-infected patients.
25 ALF occurs in 0.1%-0.5% of infected patients.
26 rs for early identification and isolation of infected patients.
27 f the world, due to migration of chronically infected patients.
28 mbled what is typically observed in Brucella-infected patients.
29 t-associated adverse reactions in loiasis co-infected patients.
30 vaccines or antiviral drugs for treatment of infected patients.
31 eminated disease is especially common in HIV-infected patients.
32 cially in human immunodeficiency virus (HIV)-infected patients.
33 eported following measles vaccination in HIV-infected patients.
34 future therapeutic approaches to treat SFTSV-infected patients.
35 in AFB microscopy smear negative PTB-HIV co-infected patients.
36 d CD69 upregulation on stimulation among HIV-infected patients.
37 , however, continue to be a major problem in infected patients.
38 halitis (MIBE) was identified in several HIV-infected patients.
39 gy-oncology, solid organ transplant, and HIV-infected patients.
40 he development of liver cancer in S. mansoni-infected patients.
41 order to adequately treat and diagnose such infected patients.
42 This approach has not been validated in HIV-infected patients.
43 ses robust innate immune activation in virus-infected patients.
44 e valuable information to more rapidly treat infected patients.
45 dy (nAb) response in clinical cohorts of HCV-infected patients.
46 dicated to investigate treatment response in infected patients.
47 e recognition between ZIKV-infected and DENV-infected patients.
48 xacerbated neurocognitive dysfunction in HIV-infected patients.
49 fibrosis/cirrhosis in a cohort of Greek HIV-infected patients.
50 ECH clears HIV-1 in blood samples from HIV-1-infected patients.
51 t cancer, cholangiocarcinoma, in chronically infected patients.
52 iated with increased incidence of NHL in HIV-infected patients.
53 mily Filoviridae and cause high lethality in infected patients.
54 iagnosis and management of these chronically infected patients.
55 major cause of disease severity and death in infected patients.
56 flammation and neurological disorders in HIV-infected patients.
57 levated production of HERV-K proteins in HIV-infected patients.
58 cytokine profiles in two distinct cohorts of infected patients.
59 V-1 Rev-mediated expression of HERV-K in HIV-infected patients.
60 increased HERV-K expression observed in HIV-infected patients.
61 ARS-CoV-2 infection in vitro and in COVID-19-infected patients.
62 yellow fever viruses in plasma samples from infected patients.
63 of host responses in acute and persistently infected patients.
64 eparation of infected or symptomatic and non-infected patients.
65 eased in bronchoalveolar lavage fluid of HIV-infected patients.
66 re receptor blocking of sera from SARS-CoV-2-infected patients.
67 ibuting to elevated HERV-K expression in HIV-infected patients.
68 erapy or vaccination for other causes in HIV-infected patients.
69 target for boosting immune responses in HIV-infected patients.
70 chanically involved molecule in INS, in PUUV-infected patients.
71 ting to the development of dementia in HIV-1 infected patients.
72 flammation and neurological disorders in HIV-infected patients.
73 ytokine responses in two distinct cohorts of infected patients.
74 of ICU beds from 724 to 1381 to accommodate infected patients.
75 ex vivo study of circulating Tfh from HIV-2-infected patients.
76 protein in metabolic changes observed in HCV-infected patients.
77 ent and manage the appropriate treatment for infected patients.
78 - to 6-fold lower than in samples from CHIKV-infected patients.
81 uninfected patients (3-19 years), and 25 HIV-infected patients (3-19 years) using multiparametric flo
85 genetic evolution of M. avium in chronically infected patients, accompanied by changes in the virulen
86 to COVID-19 has resulted in large numbers of infected patients across the globe and uncertainty regar
88 NA in liver biopsies from 56 chronically HBV infected patients after 3 to 5 years of telbivudine trea
91 nical Oncology advocate the inclusion of HIV-infected patients alongside HIV- patients in cancer clin
93 ripheral blood B cells of 30 MC-negative HCV-infected patients and 15 healthy controls revealed that
96 V-1 acquisition to-date, consisting of 6,334 infected patients and 7,247 population controls, to adva
97 rted PC from biopsy specimens of chronically infected patients and analyzed them with respect to thei
98 ms, including circulating rings from malaria-infected patients and artemisinin-induced quiescent para
100 rgan failure, are now affecting thousands of infected patients and causing widespread mortality.
103 se of cancer morbidity and mortality for HIV-infected patients and have increased incidence even in p
104 he heterogeneity of viral dynamics in dengue-infected patients and its precise treatment are still un
106 onfirmed coronavirus disease 2019 (COVID-19)-infected patients and specificity using 1,204 samples su
107 treatment of lymphoma is now similar for HIV-infected patients and the general population: patients w
109 d visitors, actively monitor for potentially infected patients, and implement appropriate infection p
110 oach to study parasite dynamics in naturally-infected patients, and is a significant improvement on t
112 nce of venous thromboembolism is 1.5-3.4% of infected patients, and it occurs due to a hypercoagulabl
113 olonized and/or infected) and the outcome of infected patients, and monitored the environmental occur
114 All candidates were recognized in sera from infected patients, and readily induced antibody response
116 and 2 from NS1) strongly recognized by ZIKV-infected patients' antibodies were identified and were n
119 nosis of ZIKV is very difficult because most infected patients are asymptomatic or display nonspecifi
123 plasma and in induced skin blisters of DENV-infected patients, as well as concomitant signaling down
124 nocyte/macrophage activation) in 39 COVID-19-infected patients at hospital admission and 2 additional
125 ens were obtained from seropositive HTLV-1/2-infected patients attending the National Centre for Huma
127 regression analyses showed that in COVID-19-infected patients both CD3(+) CD4(+) and CD3(+) CD8(+) e
128 atment initiation for newly diagnosed, HIV-1-infected patients but prospective US data are limited.
129 s are observed only in a small proportion of infected patients but the cellular mechanisms underlying
130 often detectable in VCs than in chronically infected patients, but not in healthy seronegative donor
131 dings suggest that hepatic fibrosis in HIV-1 infected patients can be modulated by the mutation of CC
132 In this prospective cohort study, 30 HIV-infected patients (CD4+ count <100 cells/uL) underwent F
135 higher titers to both ZIKV and DENV2 in ZIKV-infected patients diagnosed with GBS compared with non-G
139 igated cellular immune responses in COVID-19-infected patients during disease but little is known reg
141 entral nervous system and cause paralysis in infected patients, especially young children and immunoc
143 pecific binding towards the blood samples of infected patients, even in the presence of 'matrix' and
145 s (12/14), which was carried in the blood of infected patients for 10-59 days until treatment with do
150 ate analysis was employed to distinguish HIV-infected patients from healthy uninfected controls in a
152 9 isolates, primarily C. neoformans from HIV-infected patients, from 5 countries in Asia and Africa.
154 DTG regimen in virologically suppressed HIV-infected patients >=50 years old or with a Framingham sc
155 probability weighting method on the PS, HIV-infected patients had a lower risk of HCC [powered HR=0.
160 mprovements over time for both, but with HIV-infected patients having greater improvements (P-trends
161 mprovements over time for both, but with HIV-infected patients having greater improvements (P-trends=
162 826-like sequences can be identified in H7N9-infected patients, healthy adults, and newborn babies.
163 with only 50% case detection among HIV/TB co-infected patients, hence the need to determine the diagn
166 ence of antibody sequences across SARS-CoV-2-infected patients, highlighting stereotyped naive respon
167 ecognized as an important comorbidity of HIV-infected patients, however, the exact molecular mechanis
168 ity among Human Immunodeficiency virus (HIV) infected patients; however no consensus exists on HIV-re
169 (HBsAg) from the circulation of chronically infected patients (i.e., seroconversion) is usually asso
170 tion and protracted illness observed in H5N1-infected patients.IMPORTANCE Influenza viruses cause upp
173 lymorphisms that were overrepresented in HIV-infected patients in Japan sharing the same HLA genotype
174 enerate 649 SARS-CoV-2 genome sequences from infected patients in New Zealand with samples collected
180 d between 20 February and 15 March 2020 from infected patients in Washington state in the United Stat
182 direct-acting antiviral treatment-naive, gt3-infected patients, including those with rarer subtypes,
187 leading to profound immunodeficiency of HIV-infected patients is still only partially understood.
188 ikelihood of drug-drug interactions in HIV-1-infected patients is through the development of long-act
189 has led to better long-term outcomes in HIV infected patients, it has not been as effective at rever
190 ph nodes, where filarial nematodes reside in infected patients, leading to an effective therapy for l
193 type analyses in a cohort of HIV-1 subtype C-infected patients (n = 168), together with site-directed
196 e prospectively followed a cohort of 84 DENV-infected patients of whom 29 were coinfected with HIV un
197 ross-sectional study was conducted among HIV-infected patients on antiretroviral therapy at 20 AIDS c
199 a potential target for immunotherapy in HIV-infected patients on ART with insufficient immune recons
200 udy (1:1) we reviewed medical records of HIV-infected patients on cART in a referral AIDS center in S
203 ls accumulate in the blood of aviremic HIV-1-infected patients on long-term antiretroviral therapy, a
204 ately 1 in 10(6) cells and in cells from HIV-infected patients on suppressive antiretroviral therapy
207 CD4(+) T-cells among influenza A/H3N2 and B-infected patients (p = 0.006 and 0.004 respectively).
208 le and SVR12 rates of 96.7% among HIV/HCV co-infected patients participating in an Italian compassion
209 CWs) have elevated risk of contact with EBOV-infected patients, particularly if safety precautions ar
212 itor, in naive and paired HIV-1/O vs HIV-1/M infected (+) patients (ratio 1:2), matched on several cr
215 OV) proteome was characterized in an acutely infected patient receiving supportive care alone to eluc
218 (HBV)-human immunodeficiency virus (HIV) co-infected patients receiving combined antiretroviral ther
219 pplication to the number of CD4 cells of HIV-Infected patients recruited in the CAPRISA 002 Acute Inf
220 ctions in human immunodeficiency virus (HIV)-infected patients remains increased despite antiretrovir
221 ere acute respiratory syndrome coronavirus 2 infected patients, resource levels, concerns about being
222 In addition, lung metagenomic studies on infected patients revealed overrepresented Prevotella sp
224 ne can take months up to years, detection of infected patients seems like one of the best ideas for c
225 mbrane/envelope and NS1 antibodies from ZIKV-infected patient sera, although lower cross-reactivity t
227 should not contraindicate cancer therapy and infected patients should have access to multiple cancer
229 ty adjusted for age was higher in HIV/HCV co-infected patients (subhazard ratio [SHR] = 1.88; 95% con
231 2C expression and disease progression in HBV-infected patients suggests a role in HBV-induced liver d
233 ut CDI cases are more likely linked to other infected patients than colonized patients in this cohort
235 ovide potential insight for treatment of HIV-infected patients that are at risk of developing cerebro
238 es can be detected in the lungs and blood of infected patients, the role of extra-respiratory organs
239 a robust decrease in viral RNA levels in HCV-infected patients, thereby validating our hypothesis tha
241 s HLA genotypes and spVL data from 6,311 HIV-infected patients to interrogate the entire HIV-1 proteo
242 cancer and elevated cancer incidence in HIV-infected patients together increase the need for detaile
243 of CCL18 levels was detected among the HIV-1-infected patients treated with combined antiretroviral t
244 CV spontaneous resolution and 21 chronically infected patients treated with directly acting antiviral
249 s bearing HCV envelope proteins from acutely infected patients, we show that HCV variants isolated pr
251 fic CD4+ and CD8+ T-cell responses of 32 HDV-infected patients were analyzed by enzyme-linked immunos
256 his US urban setting, over two thirds of HCV-infected patients were linked to care; although treatmen
260 loping hepatocellular carcinoma (HCC) in HCV-infected patients who achieve sustained virological resp
261 e considered in genotype 1 hepatitis C virus-infected patients who are treatment-naive, do not have c
262 ed substitutions identified in NS3 from GT1a-infected patients who failed therapy with grazoprevir (i
263 onducted in 302 human immunodeficiency virus-infected patients who had a CD4 T-cell count <100 cells/
264 tterns between DAA-treated and untreated HCV-infected patients who had achieved a complete response t
265 propensity score-matched cohort of 8064 HCV-infected patients who had at least a 12-month follow-up
267 HCV treatment was highly effective among HIV-infected patients who received care within an integrated
268 mation about the clinical characteristics of infected patients who require intensive care is limited.
269 of transplanted patients, among which 7 HIV-infected patients who subsequently developed Kaposi sarc
270 of 26 critically ill hospitalized SARS-CoV-2-infected patients who underwent electroencephalography t
271 hs of isoniazid alone (9-month group) in HIV-infected patients who were living in areas of high tuber
273 ffness (LS) predicts clinical outcome in HIV-infected patients with active HCV coinfection, but infor
276 carcinoma (HCC) in 15,059 hepatitis C virus-infected patients with advanced liver disease defined by
277 E), among human immunodeficiency virus (HIV)-infected patients with and without hepatitis B virus (HB
278 ic inflammation and thrombosis in SARS-CoV-2-infected patients with cancer, with the concern that the
281 CV mono-infected patients and 175 HIV/HCV co-infected patients with cirrhosis, included in two prospe
282 lood total and memory B cells between 27 HIV-infected patients with CM who developed C-IRIS and 63 wh
284 Selected human immunodeficiency virus (HIV)-infected patients with end organ failure can safely rece
287 bactam-fosfomycin has the potential to offer infected patients with high bacterial burdens a therapeu
289 egravir (RAL) on hepatic steatosis among HIV-infected patients with nonalcoholic fatty liver disease
291 no effective vaccine available: treatment of infected patients with praziquantel is the mainstay of c
292 (PIB) +/- ribavirin (RBV) in HCV genotype 1-infected patients with prior virologic failure to HCV DA
293 ortion of human immunodeficiency virus (HIV)-infected patients with specific comorbidities receiving
294 with elevated KSHV-VL in critically ill HIV-infected patients with suspected but not microbiological
298 ) in some human immunodeficiency virus (HIV)-infected patients with underlying neoplasia or opportuni
299 onic hepatitis C virus (HCV) genotype (GT)-1-infected patients without cirrhosis and has now been eva
300 As remain expensive, so treatment of all HCV-infected patients would substantially affect health care