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1 is isolate with the clinical outcome for the infected patient.
2  is an important complication in the HIV/HBV infected patient.
3 bodies (mAbs) that target IBV NA from an IBV-infected patient.
4 ion was associated with lower viral loads in infected patients.
5 ymphocytes from antiretroviral treated HIV-1 infected patients.
6 an understanding of the antibody response in infected patients.
7 OVID-19 and in choosing proper management of infected patients.
8 d thromboembolism are frequent in SARS-CoV-2-infected patients.
9 against HDV in 12 treatment-naive HBV/HDV co-infected patients.
10 d cells derived from hepatitis C virus (HCV)-infected patients.
11 t cause lethal infections, especially in HIV-infected patients.
12 s, handling of infected livestock or care of infected patients.
13  ccf-mtDNA levels that were higher among HIV-infected patients.
14 expressed in highly permissive cells and HBV-infected patients.
15 unodeficiency virus (HIV) progression in HIV-infected patients.
16  the role of histone PTMs in chronically HBV-infected patients.
17 y facilitate the fast growth rate of UPEC in infected patients.
18 sseminated histoplasmosis, especially in HIV infected patients.
19 places an emphasis on therapeutic aspects of infected patients.
20 zid alone for preventing tuberculosis in HIV-infected patients.
21 may be a surrogate marker of GC in H. pylori-infected patients.
22 vels were significantly elevated in COVID-19-infected patients.
23 hysicians to quickly make their decision for infected patients.
24 milar rates of HCV eradication than HCV mono-infected patients.
25                   ALF occurs in 0.1%-0.5% of infected patients.
26 rs for early identification and isolation of infected patients.
27 f the world, due to migration of chronically infected patients.
28 mbled what is typically observed in Brucella-infected patients.
29 t-associated adverse reactions in loiasis co-infected patients.
30 vaccines or antiviral drugs for treatment of infected patients.
31 eminated disease is especially common in HIV-infected patients.
32 cially in human immunodeficiency virus (HIV)-infected patients.
33 eported following measles vaccination in HIV-infected patients.
34 future therapeutic approaches to treat SFTSV-infected patients.
35  in AFB microscopy smear negative PTB-HIV co-infected patients.
36 d CD69 upregulation on stimulation among HIV-infected patients.
37 , however, continue to be a major problem in infected patients.
38 halitis (MIBE) was identified in several HIV-infected patients.
39 gy-oncology, solid organ transplant, and HIV-infected patients.
40 he development of liver cancer in S. mansoni-infected patients.
41  order to adequately treat and diagnose such infected patients.
42  This approach has not been validated in HIV-infected patients.
43 ses robust innate immune activation in virus-infected patients.
44 e valuable information to more rapidly treat infected patients.
45 dy (nAb) response in clinical cohorts of HCV-infected patients.
46 dicated to investigate treatment response in infected patients.
47 e recognition between ZIKV-infected and DENV-infected patients.
48 xacerbated neurocognitive dysfunction in HIV-infected patients.
49  fibrosis/cirrhosis in a cohort of Greek HIV-infected patients.
50 ECH clears HIV-1 in blood samples from HIV-1-infected patients.
51 t cancer, cholangiocarcinoma, in chronically infected patients.
52 iated with increased incidence of NHL in HIV-infected patients.
53 mily Filoviridae and cause high lethality in infected patients.
54 iagnosis and management of these chronically infected patients.
55 major cause of disease severity and death in infected patients.
56 flammation and neurological disorders in HIV-infected patients.
57 levated production of HERV-K proteins in HIV-infected patients.
58 cytokine profiles in two distinct cohorts of infected patients.
59 V-1 Rev-mediated expression of HERV-K in HIV-infected patients.
60  increased HERV-K expression observed in HIV-infected patients.
61 ARS-CoV-2 infection in vitro and in COVID-19-infected patients.
62  yellow fever viruses in plasma samples from infected patients.
63  of host responses in acute and persistently infected patients.
64 eparation of infected or symptomatic and non-infected patients.
65 eased in bronchoalveolar lavage fluid of HIV-infected patients.
66 re receptor blocking of sera from SARS-CoV-2-infected patients.
67 ibuting to elevated HERV-K expression in HIV-infected patients.
68 erapy or vaccination for other causes in HIV-infected patients.
69  target for boosting immune responses in HIV-infected patients.
70 chanically involved molecule in INS, in PUUV-infected patients.
71 ting to the development of dementia in HIV-1 infected patients.
72 flammation and neurological disorders in HIV-infected patients.
73 ytokine responses in two distinct cohorts of infected patients.
74  of ICU beds from 724 to 1381 to accommodate infected patients.
75  ex vivo study of circulating Tfh from HIV-2-infected patients.
76 protein in metabolic changes observed in HCV-infected patients.
77 ent and manage the appropriate treatment for infected patients.
78 - to 6-fold lower than in samples from CHIKV-infected patients.
79                               Two cohorts of infected patients: 1) patients in the ICU with septic sh
80                       Of the 301 chronically infected patients, 158 had follow-up arranged (52%), of
81 uninfected patients (3-19 years), and 25 HIV-infected patients (3-19 years) using multiparametric flo
82                          A total of 1092 HIV-infected patients (51% coinfected with hepatitis C virus
83                           Conclusion: In HCV-infected patients, 6 and 8 weeks of treatment with JNJ-4
84                           Of 885 chronically infected patients, 92.2% received their RNA-positive res
85 genetic evolution of M. avium in chronically infected patients, accompanied by changes in the virulen
86 to COVID-19 has resulted in large numbers of infected patients across the globe and uncertainty regar
87                   Six hundred eighty-two HIV-infected patients admitted to Khayelitsha Hospital, Sout
88 NA in liver biopsies from 56 chronically HBV infected patients after 3 to 5 years of telbivudine trea
89 patitis B (HBV)/hepatitis C virus (HCV) dual-infected patients after anti-HCV therapy.
90  JNJ-53718678 (JNJ-8678) in hospitalized RSV-infected patients aged > 1 to <=24 months.
91 nical Oncology advocate the inclusion of HIV-infected patients alongside HIV- patients in cancer clin
92 antly among IgM(+) memory B cells of all HCV-infected patients analyzed.
93 ripheral blood B cells of 30 MC-negative HCV-infected patients and 15 healthy controls revealed that
94                      Overall, 1,253 HCV mono-infected patients and 175 HIV/HCV co-infected patients w
95                               Twenty-two HIV-infected patients and 20 non-HIV-infected patients were
96 V-1 acquisition to-date, consisting of 6,334 infected patients and 7,247 population controls, to adva
97 rted PC from biopsy specimens of chronically infected patients and analyzed them with respect to thei
98 ms, including circulating rings from malaria-infected patients and artemisinin-induced quiescent para
99 analyses identified a diverse set strains in infected patients and asymptomatic carriers.
100 rgan failure, are now affecting thousands of infected patients and causing widespread mortality.
101 ll-free mitochondrial DNA (ccf-mtDNA) in HIV-infected patients and controls.
102                                 In contrast, infected patients and ferrets exhibit large changes in b
103 se of cancer morbidity and mortality for HIV-infected patients and have increased incidence even in p
104 he heterogeneity of viral dynamics in dengue-infected patients and its precise treatment are still un
105  level was higher in the liver tissue of HBV-infected patients and mice.
106 onfirmed coronavirus disease 2019 (COVID-19)-infected patients and specificity using 1,204 samples su
107 treatment of lymphoma is now similar for HIV-infected patients and the general population: patients w
108 his study, humoral immune responses in CHIKV-infected patients and vaccinees were analyzed.
109 d visitors, actively monitor for potentially infected patients, and implement appropriate infection p
110 oach to study parasite dynamics in naturally-infected patients, and is a significant improvement on t
111             Tobacco smoking is common in HIV-infected patients, and is prevalent among intravenous op
112 nce of venous thromboembolism is 1.5-3.4% of infected patients, and it occurs due to a hypercoagulabl
113 olonized and/or infected) and the outcome of infected patients, and monitored the environmental occur
114  All candidates were recognized in sera from infected patients, and readily induced antibody response
115                    Laboratory diagnostics of infected patients, and the assessment of immunity agains
116  and 2 from NS1) strongly recognized by ZIKV-infected patients' antibodies were identified and were n
117  not cross-recognized by dengue virus (DENV)-infected patients' antibodies.
118 eptides were not strongly recognized by ZIKV-infected patients' antibodies.
119 nosis of ZIKV is very difficult because most infected patients are asymptomatic or display nonspecifi
120           Human immunodeficiency virus (HIV)-infected patients are at increased risk of liver-related
121 , which suggests that most deaths in ICUs in infected patients are produced by viral sepsis.
122 screening venues where 2%-10% of chronically infected patients are treated.
123  plasma and in induced skin blisters of DENV-infected patients, as well as concomitant signaling down
124 nocyte/macrophage activation) in 39 COVID-19-infected patients at hospital admission and 2 additional
125 ens were obtained from seropositive HTLV-1/2-infected patients attending the National Centre for Huma
126 to opt out of the care Covid-19 suspected or infected patients before an anticipated surge.
127  regression analyses showed that in COVID-19-infected patients both CD3(+) CD4(+) and CD3(+) CD8(+) e
128 atment initiation for newly diagnosed, HIV-1-infected patients but prospective US data are limited.
129 s are observed only in a small proportion of infected patients but the cellular mechanisms underlying
130  often detectable in VCs than in chronically infected patients, but not in healthy seronegative donor
131 dings suggest that hepatic fibrosis in HIV-1 infected patients can be modulated by the mutation of CC
132     In this prospective cohort study, 30 HIV-infected patients (CD4+ count <100 cells/uL) underwent F
133 at distinct body temperature trajectories of infected patients correlated with survival.
134                       Nevertheless, few long-infected patients develop antibodies, called broadly neu
135 higher titers to both ZIKV and DENV2 in ZIKV-infected patients diagnosed with GBS compared with non-G
136                       CD4 T cells from HIV-1 infected patients die at excessive rates compared to tho
137                                       In HCV-infected patients, disruption of the BMP6/hepcidin axis
138 in 29 HCV-infected LTx-recipients and 17 HCV-infected patients during DAA-treatment.
139 igated cellular immune responses in COVID-19-infected patients during disease but little is known reg
140                                    Among CMV-infected patients, each log2 increase in nAb titer was a
141 entral nervous system and cause paralysis in infected patients, especially young children and immunoc
142 ere acute respiratory syndrome coronavirus 2 infected patients, even in the absence of hypoxia.
143 pecific binding towards the blood samples of infected patients, even in the presence of 'matrix' and
144                                     One GT1a-infected patient experienced late viral relapse after ac
145 s (12/14), which was carried in the blood of infected patients for 10-59 days until treatment with do
146                                          HIV-infected patients from 2 Canadian cohorts underwent tran
147                              ART-naive HIV-1-infected patients from Cameroon were subjected to a mult
148 arch databases were used to select adult HIV-infected patients from each payer.
149                                          HIV-infected patients from eight prospective cohorts were in
150 ate analysis was employed to distinguish HIV-infected patients from healthy uninfected controls in a
151 V genomes sampled from nonhuman primates and infected patients from the southeastern region.
152 9 isolates, primarily C. neoformans from HIV-infected patients, from 5 countries in Asia and Africa.
153 Indonesian patients and 20% of reported H5N1-infected patients globally.
154  DTG regimen in virologically suppressed HIV-infected patients &gt;=50 years old or with a Framingham sc
155  probability weighting method on the PS, HIV-infected patients had a lower risk of HCC [powered HR=0.
156                                          HIV-infected patients had a significantly higher rate of SST
157                                              Infected patients had higher risk for death (18 vs. 8 de
158              Moreover, we found that >43% of infected patients had no serological evidence of prior i
159  that more than 80% of CD4+ T cells from HIV-infected patients have morphological abnormalities.
160 mprovements over time for both, but with HIV-infected patients having greater improvements (P-trends
161 mprovements over time for both, but with HIV-infected patients having greater improvements (P-trends=
162 826-like sequences can be identified in H7N9-infected patients, healthy adults, and newborn babies.
163 with only 50% case detection among HIV/TB co-infected patients, hence the need to determine the diagn
164                          For 33 pairs of HIV-infected patients (hereafter, "index patients") and thei
165                                       In HIV-infected patients, higher plasma sodium was uniformly as
166 ence of antibody sequences across SARS-CoV-2-infected patients, highlighting stereotyped naive respon
167 ecognized as an important comorbidity of HIV-infected patients, however, the exact molecular mechanis
168 ity among Human Immunodeficiency virus (HIV) infected patients; however no consensus exists on HIV-re
169  (HBsAg) from the circulation of chronically infected patients (i.e., seroconversion) is usually asso
170 tion and protracted illness observed in H5N1-infected patients.IMPORTANCE Influenza viruses cause upp
171 (representative of Italian hepatitis C virus-infected patients in care).
172                This result suggests that HIV-infected patients in Guangxi maintaining a BMI of 19-28
173 lymorphisms that were overrepresented in HIV-infected patients in Japan sharing the same HLA genotype
174 enerate 649 SARS-CoV-2 genome sequences from infected patients in New Zealand with samples collected
175 n of different leukocyte subsets in COVID-19-infected patients in relation to disease severity.
176 imal second-line treatment options for HIV-1-infected patients in resource-limited settings.
177 ctions in human immunodeficiency virus (HIV)-infected patients in resource-limited settings.
178 STI incidence among HIV-infected and non-HIV-infected patients in the United States after 2010.
179                         From a cohort of HBV-infected patients in Vietnam, we assessed the proportion
180 d between 20 February and 15 March 2020 from infected patients in Washington state in the United Stat
181 ancers in human immunodeficiency virus (HIV)-infected patients in Zambia.
182 direct-acting antiviral treatment-naive, gt3-infected patients, including those with rarer subtypes,
183                                       In HIV-infected patients initiating ART at advanced disease, th
184 contributes to mortality in hospitalized HIV-infected patients investigated for TB.
185    The diagnosis of tuberculosis (TB) in HIV-infected patients is challenging.
186 ntification of effective antivirals to treat infected patients is of the highest importance.
187  leading to profound immunodeficiency of HIV-infected patients is still only partially understood.
188 ikelihood of drug-drug interactions in HIV-1-infected patients is through the development of long-act
189  has led to better long-term outcomes in HIV infected patients, it has not been as effective at rever
190 ph nodes, where filarial nematodes reside in infected patients, leading to an effective therapy for l
191                                      In Zika-infected patients, levels of IL-1beta showed a significa
192                A static rate of LT among HIV-infected patients may reflect improvements in cirrhosis
193 type analyses in a cohort of HIV-1 subtype C-infected patients (n = 168), together with site-directed
194 ized trial of behavioral weight loss for HIV-infected patients (n = 40).
195            Gastric biopsies of 814 H. pylori infected patients naive for treatment were analyzed befo
196 e prospectively followed a cohort of 84 DENV-infected patients of whom 29 were coinfected with HIV un
197 ross-sectional study was conducted among HIV-infected patients on antiretroviral therapy at 20 AIDS c
198  for monitoring human immunodeficiency virus-infected patients on antiretroviral therapy.
199  a potential target for immunotherapy in HIV-infected patients on ART with insufficient immune recons
200 udy (1:1) we reviewed medical records of HIV-infected patients on cART in a referral AIDS center in S
201 interfere with their reconstitution in HIV-1-infected patients on cART.
202 ohort of HIV-HTLV-1 co-infected and HIV mono-infected patients on cART.
203 ls accumulate in the blood of aviremic HIV-1-infected patients on long-term antiretroviral therapy, a
204 ately 1 in 10(6) cells and in cells from HIV-infected patients on suppressive antiretroviral therapy
205                   In 44.6% of HCV genotype 1-infected patients, only the variant rs12785878 in the DH
206 rigger immunopathogenic events in SARS-CoV-2-infected patients or enhance infection.
207  CD4(+) T-cells among influenza A/H3N2 and B-infected patients (p = 0.006 and 0.004 respectively).
208 le and SVR12 rates of 96.7% among HIV/HCV co-infected patients participating in an Italian compassion
209 CWs) have elevated risk of contact with EBOV-infected patients, particularly if safety precautions ar
210 of morbidity and mortality in the HBV/HIV co-infected patient population.
211                           The B. burgdorferi-infected patient presented with fever, whereas the remai
212 itor, in naive and paired HIV-1/O vs HIV-1/M infected (+) patients (ratio 1:2), matched on several cr
213                                              Infected patients received significantly more saline flu
214                                              Infected patients received significantly more saline flu
215 OV) proteome was characterized in an acutely infected patient receiving supportive care alone to eluc
216                                       In HIV-infected patients receiving ART, chronic co-infection wi
217                                          HCV-infected patients receiving cancer treatment at our inst
218  (HBV)-human immunodeficiency virus (HIV) co-infected patients receiving combined antiretroviral ther
219 pplication to the number of CD4 cells of HIV-Infected patients recruited in the CAPRISA 002 Acute Inf
220 ctions in human immunodeficiency virus (HIV)-infected patients remains increased despite antiretrovir
221 ere acute respiratory syndrome coronavirus 2 infected patients, resource levels, concerns about being
222     In addition, lung metagenomic studies on infected patients revealed overrepresented Prevotella sp
223 inical Rif(R) RNAP substitution found in Mtb infected patients (S456>L of the beta subunit).
224 ne can take months up to years, detection of infected patients seems like one of the best ideas for c
225 mbrane/envelope and NS1 antibodies from ZIKV-infected patient sera, although lower cross-reactivity t
226 particles, but are the major antigens in HEV-infected patient sera.
227 should not contraindicate cancer therapy and infected patients should have access to multiple cancer
228 riological status at diagnosis among 127 HIV-infected patients starting TB treatment.
229 ty adjusted for age was higher in HIV/HCV co-infected patients (subhazard ratio [SHR] = 1.88; 95% con
230           Previous studies investigating HIV-infected patients suggested a direct link between underw
231 2C expression and disease progression in HBV-infected patients suggests a role in HBV-induced liver d
232                                    Of the 80 infected patients, test results for 74 (93%) would have
233 ut CDI cases are more likely linked to other infected patients than colonized patients in this cohort
234 sed by a higher frequency of NK cells in HIV-infected patients than in controls.
235 ovide potential insight for treatment of HIV-infected patients that are at risk of developing cerebro
236        In conclusion, Mpro is a potent Ag in infected patients that can be used in serological tests,
237                     In avian influenza virus-infected patients, the host immune system is activated i
238 es can be detected in the lungs and blood of infected patients, the role of extra-respiratory organs
239 a robust decrease in viral RNA levels in HCV-infected patients, thereby validating our hypothesis tha
240  certain conditions may increase the risk of infected patients to develop severe COVID-19.
241 s HLA genotypes and spVL data from 6,311 HIV-infected patients to interrogate the entire HIV-1 proteo
242  cancer and elevated cancer incidence in HIV-infected patients together increase the need for detaile
243 of CCL18 levels was detected among the HIV-1-infected patients treated with combined antiretroviral t
244 CV spontaneous resolution and 21 chronically infected patients treated with directly acting antiviral
245                             Among genotype 1-infected patients treated with ledipasvir/sofosbuvir mon
246 cluded studies that reported on AEs in HIV-1-infected patients undergoing TIs.
247                             In P. falciparum-infected patients, Vgamma9Vdelta2 T cells presented incr
248             In contrast to chronically HIV-1-infected patients, we found that at the earliest phases
249 s bearing HCV envelope proteins from acutely infected patients, we show that HCV variants isolated pr
250 d infection, yet we observed that 82% of the infected patients were adults.
251 fic CD4+ and CD8+ T-cell responses of 32 HDV-infected patients were analyzed by enzyme-linked immunos
252                                      507 HIV-infected patients were divided into a training (n=318) a
253 nty-two HIV-infected patients and 20 non-HIV-infected patients were evaluated.
254                             For decades, HIV-infected patients were excluded from consideration for k
255                                     1035 HCV-infected patients were included, 667 (64%) coinfected wi
256 his US urban setting, over two thirds of HCV-infected patients were linked to care; although treatmen
257                                          HIV-infected patients were significantly less accurate on th
258                                     P. vivax-infected patients were treated radically with chloroquin
259                                P. ovale spp. infected patients were treated with artemether-lumefantr
260 loping hepatocellular carcinoma (HCC) in HCV-infected patients who achieve sustained virological resp
261 e considered in genotype 1 hepatitis C virus-infected patients who are treatment-naive, do not have c
262 ed substitutions identified in NS3 from GT1a-infected patients who failed therapy with grazoprevir (i
263 onducted in 302 human immunodeficiency virus-infected patients who had a CD4 T-cell count <100 cells/
264 tterns between DAA-treated and untreated HCV-infected patients who had achieved a complete response t
265  propensity score-matched cohort of 8064 HCV-infected patients who had at least a 12-month follow-up
266                  A total of 192 HBV/HCV dual-infected patients who had received pegylated interferon
267 HCV treatment was highly effective among HIV-infected patients who received care within an integrated
268 mation about the clinical characteristics of infected patients who require intensive care is limited.
269  of transplanted patients, among which 7 HIV-infected patients who subsequently developed Kaposi sarc
270 of 26 critically ill hospitalized SARS-CoV-2-infected patients who underwent electroencephalography t
271 hs of isoniazid alone (9-month group) in HIV-infected patients who were living in areas of high tuber
272                                 About 30% of infected patients will develop chronic Chagas cardiomyop
273 ffness (LS) predicts clinical outcome in HIV-infected patients with active HCV coinfection, but infor
274                                    Among HCV-infected patients with advanced fibrosis, who achieve SV
275                                    Among HIV-infected patients with advanced immunosuppression, enhan
276  carcinoma (HCC) in 15,059 hepatitis C virus-infected patients with advanced liver disease defined by
277 E), among human immunodeficiency virus (HIV)-infected patients with and without hepatitis B virus (HB
278 ic inflammation and thrombosis in SARS-CoV-2-infected patients with cancer, with the concern that the
279                        We identified 333 HIV-infected patients with CD4 + T-cell-counts >= 500/ul, am
280                           Conclusion: In HCV-infected patients with cirrhosis, HIV co-infection was n
281 CV mono-infected patients and 175 HIV/HCV co-infected patients with cirrhosis, included in two prospe
282 lood total and memory B cells between 27 HIV-infected patients with CM who developed C-IRIS and 63 wh
283 ecommendation of a single dose of BPG in HIV-infected patients with early syphilis.
284  Selected human immunodeficiency virus (HIV)-infected patients with end organ failure can safely rece
285 wn to be safe and effective for selected HIV-infected patients with hematological malignancies.
286 ity for all patients with HF and in COVID-19-infected patients with HF.
287 bactam-fosfomycin has the potential to offer infected patients with high bacterial burdens a therapeu
288 ut susceptibility to the outcome of COVID-19-infected patients with MM.
289 egravir (RAL) on hepatic steatosis among HIV-infected patients with nonalcoholic fatty liver disease
290                                          HIV-infected patients with OcC have a high incidence of live
291 no effective vaccine available: treatment of infected patients with praziquantel is the mainstay of c
292  (PIB) +/- ribavirin (RBV) in HCV genotype 1-infected patients with prior virologic failure to HCV DA
293 ortion of human immunodeficiency virus (HIV)-infected patients with specific comorbidities receiving
294  with elevated KSHV-VL in critically ill HIV-infected patients with suspected but not microbiological
295  evaluating the LAM test in hospitalized HIV-infected patients with suspected TB.
296                                     Some HCV-infected patients with sustained virological response (S
297                                In genotype 1 infected patients with treatment failure and 156-RASs, w
298 ) in some human immunodeficiency virus (HIV)-infected patients with underlying neoplasia or opportuni
299 onic hepatitis C virus (HCV) genotype (GT)-1-infected patients without cirrhosis and has now been eva
300 As remain expensive, so treatment of all HCV-infected patients would substantially affect health care

 
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