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1 exposed bees to a second ecological stressor infecting individuals with 10,000 spores of the fungal g
2 d the kinetics of SIE in individual cells by infecting them sequentially with 2 isogenic IAVs, each e
3       From this, we identified 102 mammalian-infecting viruses, with 65 described for the first time.
4 s infecting Trioza urticae and at some sites infecting Urtica dioica with 77.55% and 24.37% average i
5 h sets of antagomir effects were mimicked by infecting cells with a p220 cDNA-encoding adenoviral vec
6 MAPK pathway was constitutively activated by infecting cells with a v-mos retrovirus.
7 tidylinositol 3-kinase/protein kinase Akt or infecting endothelial cells with a dominant-negative Akt
8 terial can be generated using this system by infecting insect cells with a baculovirus expressing the
9           We investigated this hypothesis by infecting MDCK cells with a transfectant influenza virus
10 f Pseudomonas aeruginosa but dispensable for infecting mutants with a truncated lipopolysaccharide de
11           Influenza A virus (IAV) is a human-infecting pathogen with a history of causing seasonal ep
12 s an opportunistic human pathogen capable of infecting patients with a deficient immune system.
13 PI anti-PnPs antibody levels to the presumed infecting serotypes, with a geometric mean level of 0.83
14 d purified lipooligosaccharide of homologous infecting strains and with a series of mutants deficient
15  vivo depleted these cells from RMs prior to infecting the primates with a pathogenic strain of SIV.
16  complementing the cells with human CD81, or infecting them with a strain of HCV with less restricted
17 -phase induction in quiescent human cells by infecting them with Ad N-terminal E1A mutants with mutat
18 B-/- double knockout mouse lens epithelia by infecting primary cells with Ad12-SV40 hybrid virus.
19                                              Infecting cells with adenovirus encoding constitutively
20      In addition, stabilizing HIF1/2alpha by infecting mammalian cells with adenoviruses containing t
21 ring rate in response to MPO and showed that infecting flies with African trypanosomes alters the fli
22 n bieneusi is the most common microsporidian infecting patients with AIDS.
23 als could be generated quickly and easily by infecting susceptible blastocysts with ALV-based retrovi
24                This hypothesis was tested by infecting cells with an M protein mutant virus defective
25 ocked the binding of AP-1 proteins to DNA by infecting keratinocytes with an adenovirus encoding a do
26  of the highest pre-infection titre than the infecting serotype, consistent with antigenic seniority
27                                         Upon infecting macrophages separately with antimony drug-sens
28 irus (IDV) has emerged as a multiple-species-infecting pathogen with bovines as a primary reservoir.
29                                              Infecting glial progenitors with Cre-recombinant retrovi
30              G10P[11] strains were also seen infecting older children with dehydrating gastroenteriti
31  study, we use a sequential infection model, infecting AG129 mice with DENV-1, followed by DENV-2 6-8
32                                              Infecting human MKs with DENV selectively increased type
33 the control of a pathogen that is capable of infecting multiple hosts with different rates of transmi
34 e cells to the JNK inhibitor SP600125 and by infecting cells with dominant-negative JNK or AMPK adeno
35 en validated these analytical predictions by infecting mice with doses below or above the predicted t
36                 These cells were produced by infecting fibroblasts with dox-inducible lentiviruses, r
37                                              Infecting these mice with EGFP-expressing murine herpes
38 y chemically inhibiting actin assembly or by infecting mammalian cells with EHEC mutants that translo
39 ally maintaining T cells was investigated by infecting mice with either a demyelinating, paralytic (V
40 her Tir modification could not be induced by infecting cells with EPEC, suggesting that Tir must be c
41 broad range, with laboratory-adapted viruses infecting all clones with equal efficiencies and primary
42                                Specifically, infecting mice with H3N2 influenza before challenging wi
43 ta-lipoproteins may reduce the efficiency of infecting hepatocytes with HCV by competitively inhibiti
44 ng the brain via HBCE cells and subsequently infecting microglial cells with high efficiency, leading
45 d and primary T-cell targets was examined by infecting cells with HIV-1 reporter viruses containing k
46 o targets the immunocompromised, chronically infecting people living with HIV and primary immunodefic
47 00 in infected patients, regardless of their infecting strain, correlated with increased production o
48 g the plasmid into recipient cells, and then infecting those cells with influenza virus.
49 d when pre-integration complexes are made by infecting cells with integrase-minus virus, demonstratin
50 eld-isolate reovirus strains were capable of infecting cells transfected with JAM-A but not those tra
51 loviruses and nudiviruses, a group of insect-infecting viruses with key roles in biocontrol.
52 velop has been impaired by the difficulty of infecting B cells with KSHV in vitro, and the inability
53 of HIF-1alpha or HIF-2alpha was abrogated by infecting astrocytes with lentiviral particles encoding
54  chronic infection were moderately better at infecting cells with low CCR5 concentrations.
55 -infection V1-V2 sequences showed a trend in infecting cells with low CD4 concentrations more efficie
56 ence in inflamed tissues was demonstrated by infecting transgenic mice with MCMV recombinant virus co
57  of these mutations was further evaluated by infecting naive mice with MHV-JHM variants isolated from
58 could be used to predict the genotype of the infecting virus with moderate success.
59                                              Infecting Ramos cells with MV did not result directly in
60 viral escape from autophagic degradation, as infecting DC with Nef- or Env-deficient HIV strains did
61 ur knowledge has never been described as the infecting organism with oropharyngeal candidiasis (OPC).
62 oglobulin G (IgG) concentrations against the infecting serotype compared with other vaccine serotypes
63 ed with lower IgG concentrations against the infecting serotype compared with other vaccine serotypes
64 f IOMT-overexpressing plants with CuCl(2) or infecting these plants with Phoma medicaginis leads to g
65 den and Zika disease varies depending on the infecting primary flavivirus; with primary Zika virus in
66 icipant characteristics, symptom profile and infecting variant with prospective PCR sampling, and the
67 human cells with recombinant plasmids and by infecting insect cells with recombinant baculoviruses.
68 DS has primarily consisted of experimentally infecting macaques with related simian immunodeficiency
69 gastric cancer cell line was investigated by infecting these cells with retroviral construct (MFG) ex
70 ed or increased p53 activity were created by infecting VSMCs with retroviruses containing a dominant-
71 ing cells with a proteasomal inhibitor or by infecting cells with small interfering (si)RNA against S
72                      Geminiviruses are plant-infecting viruses with small circular single-stranded DN
73                      The model was tested by infecting Galleria mellonella with spores and confirmed
74                                              Infecting BFTE cells with sporulated oocysts provided a
75 RhCMV would be more accurately reproduced by infecting macaques with strains of RhCMV that reflect th
76 acteriophages are a diverse group of viruses infecting Mycobacterium with substantial therapeutic pot
77 le to directly correlate the genotype of the infecting antigen with the antibody response.
78 erminal-repeat circle junctions derived from infecting cells with the mutant viruses indicated that t
79 in the five major commercial turkey lines by infecting each with the parental B. avium strain and thr
80                                              Infecting flies with the knockout, followed by live conf
81                                           By infecting mice with the attaching and effacing bacteria
82                                    Moreover, infecting mice with the LRV1-cured Leishmania guyanensis
83        In these studies, we demonstrate that infecting mice with the Th1-inducing parasite Toxoplasma
84                                          The infecting phages colocalized with the polar protein mark
85 ne response of these animals was explored by infecting them with the Th1-inducing parasite Toxoplasma
86           Mice are primarily responsible for infecting ticks with the Lyme disease agent.
87 nteractions compare with those of vertebrate-infecting viruses and with the Sigma rhabdovirus that in
88 ere we report a high-efficiency protocol for infecting yeast with the [PSI+] prion using amyloids com
89 directly the effects of a human transgene by infecting them with Theiler's murine encephalomyelitis v
90                                     After co-infecting C. elegans with these organisms, we observed t
91                      Given the difficulty of infecting laboratory mice with these diarrhea-causing pa
92                                           By infecting nonpermissive cells with this library and scre
93  specifically destroy the malignant cells by infecting them with toxic cargo.
94                        We determined this by infecting monocytes with two strains of ZIKV: South Amer
95 o induce DNA repair was also demonstrated by infecting NWTb3 cells with UV-irradiated adenovirus.
96 avital microscopy and ex vivo analyses after infecting mice with vaccinia virus (VV), a large DNA vir
97                                           By infecting animals with various doses of Listeria and ana
98                     Baculoviruses are insect-infecting pathogens with wide applications as biological
99 The latter possibility was studied by dually infecting rhesus macaques with X4 and R5 chimeric simian