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1 is in which T and NK cells dominate a severe inflammatory infiltrate.
2  to periodontitis displayed higher levels of inflammatory infiltrate.
3  and complement deposition and a conspicuous inflammatory infiltrate.
4 L-1alpha and attraction of a pro-tumorigenic inflammatory infiltrate.
5  dysregulated angiogenesis and a substantial inflammatory infiltrate.
6 muscle fibers, myophagocytosis, and a sparse inflammatory infiltrate.
7 ized by a spindle-cell proliferation with an inflammatory infiltrate.
8  divide and that they do so by triggering an inflammatory infiltrate.
9  the increased expression of VEGF-C, the SCC inflammatory infiltrate.
10 serum creatine kinase levels and a prominent inflammatory infiltrate.
11 s accompanied by a disproportionately sparse inflammatory infiltrate.
12 mmatory gene synthesis and resolution of the inflammatory infiltrate.
13 mesenchymal cells and was independent of the inflammatory infiltrate.
14  of tissues and are key orchestrators of the inflammatory infiltrate.
15  represented 10% or more of the interstitial inflammatory infiltrate.
16 s characterized by an eosinophil-predominant inflammatory infiltrate.
17 liferation of basal keratinocytes and a skin inflammatory infiltrate.
18 our incidence correlates with wound size and inflammatory infiltrate.
19 rel had a significant effect on reducing the inflammatory infiltrate.
20 ells with characteristic morphology among an inflammatory infiltrate.
21 lls, consistent with Th9 lymphocytes, in the inflammatory infiltrate.
22 athological CD207(+) dendritic cells with an inflammatory infiltrate.
23 acrophages or fibrin as well as intervillous inflammatory infiltrates.
24 ntestinal epithelial cells (IEC) and colonic inflammatory infiltrates.
25  gland function is preserved despite intense inflammatory infiltrates.
26 treatment was indicated through detection of inflammatory infiltrates.
27 e weakness and the presence of intramuscular inflammatory infiltrates.
28 ApoE((-/-)) versus ApoE((-/-)) mice, as were inflammatory infiltrates.
29 ological changes consisting of peribronchial inflammatory infiltrates.
30 3 is highly expressed in postmortem MS brain inflammatory infiltrates.
31 g of cerebral tissue was performed to detect inflammatory infiltrates.
32 hich are characterized by early perivascular inflammatory infiltrates.
33 ere mucosal erosion, villous congestion, and inflammatory infiltrates.
34 nohistochemical analysis for the presence of inflammatory infiltrates.
35 d histopathologic analysis showed no greater inflammatory infiltrates.
36 rved within the islet endocrine cells and in inflammatory infiltrates.
37  nonspecific chronic hepatitis (14.3%), mild inflammatory infiltrates (10.7%), and steatosis (3.5%).
38    Surviving grafts were essentially free of inflammatory infiltrates 15 weeks after transplantation.
39 hoid follicles (32%), a plasma cell-enriched inflammatory infiltrate (63%), and central perivenulitis
40  in PsA by reducing, but not abolishing, the inflammatory infiltrate, adhesion molecule expression, a
41 is, a predominance of polymorphonuclear cell inflammatory infiltrate and a reduced extent of fibrosis
42 suppress airway hyperreactivity, bronchiolar inflammatory infiltrate and allergen-specific IgE produc
43 rogenitors, are a prominent component of the inflammatory infiltrate and are believed to directly con
44 rous connective tissue response with minimal inflammatory infiltrate and bone formation.
45 val immune processes, including an increased inflammatory infiltrate and edema seen with IVCM.
46 e complex cellular composition of the portal inflammatory infiltrate and HPC/DR niche in NAFLD will s
47 iated skin and was associated with decreased inflammatory infiltrate and increased angiogenesis.
48  stages of infection, with a more pronounced inflammatory infiltrate and increased specific productio
49 e investigated the composition of the portal inflammatory infiltrate and its relationship to the duct
50 CLs and highlights the interplay between the inflammatory infiltrate and malignant B cells in these t
51 determine the effects of methotrexate on the inflammatory infiltrate and on cytokine and metalloprote
52 cular steatosis, whereas demonstration of an inflammatory infiltrate and pattern of liver fibrosis co
53 stained sections were used to study cellular inflammatory infiltrate and quantitative histomorphometr
54 followed by the characterization of cellular inflammatory infiltrate and quantitative histomorphometr
55 28 significantly reduces the formation of an inflammatory infiltrate and reduces bone loss, measured
56 placental factors, such as the nature of the inflammatory infiltrate and slightly higher expression o
57                    The interplay between the inflammatory infiltrate and tissue resident cell populat
58 aque segments, demonstrating the presence of inflammatory infiltrate and various compositional elemen
59 athological features include T-cell mediated inflammatory infiltrates and aberrant accumulations of p
60 ropathological analysis shows a reduction in inflammatory infiltrates and demyelinated areas in the b
61 fer of myelin-specific T cells, resulting in inflammatory infiltrates and demyelination in the centra
62 ed neurological functional recovery, reduced inflammatory infiltrates and demyelination, and increase
63 ssion of HIF, which initiated an increase in inflammatory infiltrates and edema in the colon.
64                                              Inflammatory infiltrates and enlarged blood vessels were
65 reased atherosclerosis severity and arterial inflammatory infiltrates and increased neoangiogenesis w
66 was mediated, in part, by the suppression of inflammatory infiltrates and inflammatory cytokines/medi
67 , and histopathologists should be aware that inflammatory infiltrates and late presentation are poten
68  greater cellularity and significantly fewer inflammatory infiltrates and lipid pools in the ISR grou
69 -/-) mice had a lower number of perivascular inflammatory infiltrates and mast cells than those found
70                                              Inflammatory infiltrates and neuronal damage were presen
71 nonuclear cells at the junction of the focal inflammatory infiltrates and normal acinar tissue in bot
72 active EAE of C57Bl/6 mice, decreased spinal inflammatory infiltrates and spinal infiltration of Th1
73                 Moreover, the total cellular inflammatory infiltrates and the CD3(+), CD4(+), HLA-DR(
74 h levels of inflammatory cytokines, alveolar inflammatory infiltrates and vascular microthrombi.
75 rized by the proliferation of spindle cells, inflammatory infiltrate, and aberrant angiogenesis cause
76  antagonist IRS954 reduced pancreatic edema, inflammatory infiltrate, and apoptosis.
77  of a thin choroid, a perivascular choroidal inflammatory infiltrate, and atrophic changes within the
78 parietal cell loss, induction of a prominent inflammatory infiltrate, and emergence of SPEM.
79 ss, temperature, virus titres, the pulmonary inflammatory infiltrate, and interferon [IFN]-gamma leve
80 dual viable tumor, necrosis, fibrous tissue, inflammatory infiltrate, and Ki-67 proliferative index w
81 dontal disease, bleeding index, intensity of inflammatory infiltrate, and loss of connective tissue i
82 ac allograft vasculopathy, the nature of the inflammatory infiltrate, and origin of endothelial cells
83 embrane attack complex, the character of the inflammatory infiltrate, and the primacy of capillary ab
84 o acute worsening of EAE symptoms, increased inflammatory infiltrate, and was associated with the dea
85 ged with H. pylori, and the bacterial loads, inflammatory infiltrates, and cytokine responses were ev
86 educing acanthosis (thickening of the skin), inflammatory infiltrates, and expression of Th17 cytokin
87 ice exhibited more extensive tubular injury, inflammatory infiltrates, and fibrosis than wild-type mi
88                The severity of the fibrosis, inflammatory infiltrates, and glomerular and tubular cha
89 ells, interstitial endothelium, interstitial inflammatory infiltrates, and glomerular cells.Primary V
90 c asthma include airway hyperresponsiveness, inflammatory infiltrates, and structural changes in the
91 okine production (cytokine storm), excessive inflammatory infiltrates, and virus-induced tissue destr
92 nd histopathologic presentation and reducing inflammatory infiltrates, antibody treatment during alle
93 s that both products of the bacteria and the inflammatory infiltrate are capable of stimulating gastr
94                                              Inflammatory infiltrates are common but are small and of
95                                              Inflammatory infiltrates are frequently observed in pros
96        Axonal injury was correlated with the inflammatory infiltrate as well as clinical characterist
97 in diseased rats, which correlated well with inflammatory infiltrates as determined by histology.
98 ons of the epididymides showed a focal acute inflammatory infiltrate at 2 weeks p.i.
99        DC and T cells form aggregates in the inflammatory infiltrates at active disease sites in huma
100                            In humans, a rich inflammatory infiltrate bearing macrophage markers has a
101                                  Analysis of inflammatory infiltrates by fluorescence-activated cell
102 -field microscopy, (2) histology to quantify inflammatory infiltrates by immunostaining for macrophag
103 ive correlations could be observed among the inflammatory infiltrate, c-kit, and tryptase in individu
104 gic examination, a layer of collagen tissue, inflammatory infiltrate cells, perivascular infiltrate,
105 f clinical manifestations, the nature of the inflammatory infiltrating cells, the cytokine response p
106  is characterized by a myocardial macrophage inflammatory infiltrate, changes in the distribution of
107 per stromal remodeling and perdurance of the inflammatory infiltrate characteristic of pancreatic tum
108 t and tryptase mast cells and the density of inflammatory infiltrate, cluster of differentiation 4 (C
109                     Despite these equivalent inflammatory infiltrates, CNS tissues from CXCR3(-/-) mi
110 logy of C3(-/-) mice revealed a reduction in inflammatory infiltrate compared with C3(+/+) mice.
111  of regenerative fibers, with a reduction in inflammatory infiltrates, compared with those in vehicle
112 QTS/CPVT patients revealed mild but distinct inflammatory infiltrates composed of T lymphocytes and m
113 prominent spleen, liver, and skeletal muscle inflammatory infiltrates comprised of NK, dendritic, B a
114 marked edema of the papillary dermis with an inflammatory infiltrate consisting mainly of mature neut
115                                     An acute inflammatory infiltrate consisting of polymorphonuclear
116 perproliferation, elongated rete ridges, and inflammatory infiltrates consisting of T cells, macropha
117  skin transplanted onto NSG mice develops an inflammatory infiltrate, consisting predominately of hos
118 tis), i.e. replacememt of bone marrow fat by inflammatory infiltrates containing macrophages, T lymph
119 of the human pathology, are characterized by inflammatory infiltrates containing T helper 1 (T(H)1) a
120 es in dermal fibroblasts that, like the skin inflammatory infiltrate, contribute to tumour formation.
121 to determine the extent of connective tissue inflammatory infiltrate (CTII) using the following marke
122                                              Inflammatory infiltrate, DCs, chemokines, classification
123 gy scores, vascular densities, and levels of inflammatory infiltrates decreased significantly compare
124 own efficacy in the neoadjuvant setting with inflammatory infiltrates demonstrated in tumors.
125 nted into C57Bl/6 recipients exhibited mixed inflammatory infiltrates, destruction of the mucosa, and
126      At 3 days postinfection, well-organized inflammatory infiltrates developed in the spleen and liv
127   Later in the course of this illness, focal inflammatory infiltrates disappeared from the spinal cor
128   These results highlight differences in the inflammatory infiltrates during Lyme arthritis and cardi
129 idermis caused epidermal hyperplasia, dermal inflammatory infiltrate, dysmorphic hair follicles, and
130                 AD shows a Th2 cell-dominant inflammatory infiltrate, elevated serum IgE levels, a pe
131 alomyelitis (EAE), characterized by lesions, inflammatory infiltrates, elevated STAT activation, and
132                          A rapid pericentral inflammatory infiltrate enriched in F4/80(+) monocytes c
133 mistry of the pancreatic remnant revealed an inflammatory infiltrate expressing cathepsin B, independ
134 y of the muscle showed a mainly perivascular inflammatory infiltrate, fibrotic degeneration, and neov
135 d with the induction of an intense cutaneous inflammatory infiltrate following topical application of
136                       On day 3, however, the inflammatory infiltrate had subsided in the control grou
137                Synovial tissues with diffuse inflammatory infiltrates had high levels of TSP2, wherea
138 ntinued axon loss for months after acute CNS inflammatory infiltrates had subsided, and massive astro
139                            The subepithelial inflammatory infiltrate has a larger amount of CD20 posi
140                                  We detected inflammatory infiltrates, hepatocellular necrosis, and a
141                                          The inflammatory infiltrates identified were of chronic infl
142                  T cells are the predominant inflammatory infiltrate in affected tissue and are thoug
143                                          The inflammatory infiltrate in atherosclerotic plaque is com
144                            Data on placental inflammatory infiltrate in biopsies from 68 cases and 65
145 AC expression is elevated selectively in the inflammatory infiltrate in human and murine colitis.
146  positively correlated with the intensity of inflammatory infiltrate in lesions from pregnant women.
147 hilic infiltrate compared with a mononuclear inflammatory infiltrate in normal mice.
148     Macrophages account for 5% to 30% of the inflammatory infiltrate in periodontitis and are activat
149 ameters, periodontal bacterial profiles, and inflammatory infiltrate in placental chorionic villi are
150 egulating the pattern and persistence of the inflammatory infiltrate in RA and suggest a new and impo
151                      Characterization of the inflammatory infiltrate in selected cases used a novel s
152                  This resulted in a profound inflammatory infiltrate in the adventitia and necrotic c
153  abscesses within the epidermis, and a mixed inflammatory infiltrate in the dermis.
154 , we analyzed the monocytic component of the inflammatory infiltrate in the draining popliteal lymph
155 e congestion, alveolar hemorrhage, and acute inflammatory infiltrate in the interstitial compartment
156    Neutrophils were the predominant cellular inflammatory infiltrate in the lungs of superinfected mi
157 haracteristic stricturing by day 16, with an inflammatory infiltrate in the neuromuscular layers incl
158 in serum IL-1beta and IL-18, reduced myeloid inflammatory infiltrate in the skin and spleen, and subs
159 cells and plasma cells are components of the inflammatory infiltrates in both active and chronic MS l
160 lls of normal colonic lamina propria, and in inflammatory infiltrates in colon tissues from Crohn's d
161         The majority of cells comprising the inflammatory infiltrates in kidney allografts undergoing
162  that IFNgamma influences the composition of inflammatory infiltrates in Lyme carditis by promoting t
163             Macrophages constitute the major inflammatory infiltrates in musculoskeletal tissues duri
164 n was localized mainly in cardiomyocytes and inflammatory infiltrates in peri-infarct areas, but not
165  or growth restriction were characterized by inflammatory infiltrates in placentas, functional defici
166 s, and in neutrophil survival, which affects inflammatory infiltrates in RA.
167 trol study, in which we immunophenotyped the inflammatory infiltrates in renal biopsy specimens with
168 rity and incidence of EAE, the appearance of inflammatory infiltrates in spinal cord, and the subsequ
169 an skin scores and significantly fewer focal inflammatory infiltrates in submandibular salivary gland
170 nical course of EAE and showed markedly less inflammatory infiltrates in the CNS than wild-type mice.
171 anthosis, parakeratosis, hyperkeratosis, and inflammatory infiltrates in the dermis.
172 M/WT mice we detected variable expression of inflammatory infiltrates in the lungs and kidneys.
173 ls were co-localized with CD4(+) Th cells in inflammatory infiltrates in the lungs of mycoplasma-infe
174 onchoalveolar lavage fluid, allergen-induced inflammatory infiltrates in the lungs, and Bla g 1-speci
175 nation in the cerebral cortex was related to inflammatory infiltrates in the meninges, which was pron
176 ase of unknown etiology, is characterized by inflammatory infiltrates in the pancreas with conspicuou
177 s, total serum Ig isotypes, lymphadenopathy, inflammatory infiltrates in the salivary gland and kidne
178  acute clinical disease and showed only mild inflammatory infiltrates in the spinal cord white and gr
179 glycolysis associated with paralysis-causing inflammatory infiltrates in the spinal cord; and (iii) t
180 vo clinical examination at 24 hours revealed inflammatory infiltrates in the vitreous and retina, whi
181 treated mice have reduced fibrosis and fewer inflammatory infiltrates in their adipose tissues.
182  with an increased number of cysts and fewer inflammatory infiltrates in their brains.
183                                          The inflammatory infiltrates in various organs predominantly
184 h levels of serum proinflammatory cytokines, inflammatory infiltrates in various organs, and amyloid
185  the development of chronic small-intestinal inflammatory infiltrates in vivo.
186  oxidase and myeloperoxidase activities, and inflammatory infiltrate) in chronically infected hearts.
187 udies of patients have shown the presence of inflammatory infiltrates, in some cases with features of
188 ccompanied by a significant reduction of the inflammatory infiltrate including macrophages, dendritic
189 elitis and decreased the accumulation of CNS inflammatory infiltrates, including dendritic cells, B c
190 NBS-treated mice contained acute and chronic inflammatory infiltrates, increased collagen, fibrogenic
191                              Analysis of the inflammatory infiltrate indicated a reduction of cytotox
192 s have similarities in their desmoplasia and inflammatory infiltrates, indicating overlapping inflamm
193 echanisms underlying the recruitment of this inflammatory infiltrate into the involved lymph nodes ar
194 omicrobial peritonitis, leading to increased inflammatory infiltrate into the peritoneum and target o
195 is abundant EBV replication but the expected inflammatory infiltrate is absent.
196                                         This inflammatory infiltrate is predominately composed of T l
197 r-treated NAFLD livers had notably fewer pro-inflammatory infiltrating macrophages and a significantl
198                                   The portal inflammatory infiltrate may contribute directly to fibro
199 riodontal disease, these cells, found in the inflammatory infiltrate, may drive the progressive perio
200 ing cholangitis is characterized by a portal inflammatory infiltrate mostly composed of memory T cell
201 sis of IOM were splaying of muscle fibers by inflammatory infiltrates (n = 9) and mild fibrosis (n =
202 ng the musculature that are characterized by inflammatory infiltrates, necrosis and connective tissue
203 atures such as eosinophilic and neutrophilic inflammatory infiltrates, necrosis, vascular hyalinizati
204                                          The inflammatory infiltrate, NOS-2-positive macrophages, myo
205                                              Inflammatory infiltrates occurred in the majority of HIV
206 cells were found in increased numbers in the inflammatory infiltrate of CD lesions and induced expres
207 hocytes and plasma cells) predominate in the inflammatory infiltrate of human chronic periodontitis.
208 rophils are the predominant cell in the lung inflammatory infiltrate of infants with respiratory sync
209 ndle cell proliferation with diffuse chronic inflammatory infiltrate of lymphocytes, plasma cells and
210         The ear bud area develops an intense inflammatory infiltrate of OT-I cells.
211  response to kidney injury includes a robust inflammatory infiltrate of PMNs and macrophages.
212 ant, revealed staining in the perifollicular inflammatory infiltrate of rosacea for both proteins.
213                                          The inflammatory infiltrate of SeTA was similar to SeA but c
214 IL-1beta, accompanied by significantly fewer inflammatory infiltrates of macrophages and neutrophils
215        The increase in CD4(+) T cells in the inflammatory infiltrates of the lacrimal gland, and the
216                                              Inflammatory infiltrates, often with granulomatous arran
217 iosis, vascular proliferation, and a chronic inflammatory infiltrate on the subepithelial connective
218   No ratios were correlated with presence of inflammatory infiltrate or foamy macrophages.
219 increased in osteoclast numbers, whereas the inflammatory infiltrate or on gene expression were simil
220  mice; in contrast, there was no evidence of inflammatory infiltrates or responses in any other tissu
221  increased the intensity and duration of the inflammatory infiltrate (P < 0.05).
222 -derived LCs was associated with nonspecific inflammatory infiltrates (P=0.009).
223 cells cause enhanced disease, with increased inflammatory infiltrate, particularly rich in neutrophil
224                         Associations between inflammatory infiltrate patterns and 18F-FDG-PET/CT upta
225      Likewise, at 2 weeks, acute and chronic inflammatory infiltrates persisted in the bladders and k
226 d; P < .001), presence of neutrophils in the inflammatory infiltrate (predominantly mononuclear vs mi
227 gs; fibrous hyperplasia; a low-grade chronic inflammatory infiltrate, predominantly consisting of lym
228                 Adenoviral infection induced inflammatory infiltrates primarily made of T and B cells
229                            The prevalence of inflammatory infiltrate, proinflammatory cytokines, and
230 data suggest that the P2X7 receptor promotes inflammatory infiltrates, proinflammatory cytokines, and
231 ator synthesis and mediate resolution of the inflammatory infiltrate, promoting formation of a scar.
232 d tumours, colorectal cancer exhibits immune/inflammatory infiltrates, referred to as 'tumour-elicite
233 maller, less aggressive lesions with reduced inflammatory infiltrate relative to tumors generated by
234 a-Cell replication stimulated by the initial inflammatory infiltrate results in increased production
235                              Analyses of the inflammatory infiltrate revealed a classic acute inflamm
236 ent bacterial colonization and heterogeneous inflammatory infiltrates rich in CD15+ neutrophils, as h
237                              Despite similar inflammatory infiltrates, RRV-infected C3(-/-) mice exhi
238 gmentation and loss, Wallerian degeneration, inflammatory infiltrates, Schwann cell proliferation, an
239 ght account in part for the heterogeneity of inflammatory infiltrates seen in lungs of asthmatics.
240                                The resulting inflammatory infiltrate serves to aid bacterial clearanc
241                              Analysis of CNS inflammatory infiltrates showed that female but not male
242    Histologic examination revealed increased inflammatory infiltrate, significantly increased immunos
243          Morphologic immunologic parameters (inflammatory infiltrate, stromal content, and budding ac
244                                       In the inflammatory infiltrate subjacent to the pocket epitheli
245 ariable numbers of malignant cells within an inflammatory infiltrate, suggesting that these tumors es
246                  The presence of a CD8+ rich inflammatory infiltrate suggests an ongoing immune respo
247 ivascular interstitial edema associated with inflammatory infiltrates surrounding larger pulmonary ve
248 ys of GTC-treated mice had less fibrosis and inflammatory infiltrate than kidneys of vehicle-treated
249 eed-Sternberg (RS) cells within an extensive inflammatory infiltrate that includes abundant T helper
250      The remaining tissue is comprised of an inflammatory infiltrate that includes myeloid cells.
251 cosal diseases are often characterized by an inflammatory infiltrate that includes polymorphonuclear
252 ancer is frequently characterized by a large inflammatory infiltrate that includes T cells.
253 y to be one of several cytokines produced by inflammatory infiltrates that contribute to this and sub
254 monia (peak days, 4-5) and chronic pulmonary inflammatory infiltrates that lasted up to 154 days afte
255 , associated dysfunction, and development of inflammatory infiltrates that result in abnormal tears.
256    Although the IL-1R(-/-) mice developed an inflammatory infiltrate, the number of infiltrating neut
257 ions with respect to demyelinating activity, inflammatory infiltrates, the presence of meningeal infl
258 ells and collagen deposition, independent of inflammatory infiltrates; these changes were also observ
259 Western blot were used to evaluate levels of inflammatory infiltrate, TLR2, TLR4, and RAGE expression
260    Fibrosis was present in most with similar inflammatory infiltrate to viremic cases.
261  vivo EP also recruited large mixed cellular inflammatory infiltrates to the site of inoculation.
262  examined for the presence of CNS tissue and inflammatory infiltrates using direct microscopy, enhanc
263             In this study we used MRI to map inflammatory infiltrates using high-field MRI and fluore
264                                        Liver inflammatory infiltrate was characterized by immunohisto
265 bserved in the urinary bladder, although the inflammatory infiltrate was delayed by approximately a w
266                                              Inflammatory infiltrate was evaluated and quantified in
267         However, when the composition of the inflammatory infiltrate was examined by flow cytometric
268                            In hairless mice, inflammatory infiltrate was found around the rudimentary
269                          Further, an intense inflammatory infiltrate was found in the cervix and vulv
270 was observed in IL-6(-/-) mice, in which the inflammatory infiltrate was markedly reduced and synovia
271                          The formation of an inflammatory infiltrate was measured in hematoxylin-and-
272               On day 1, a moderate to severe inflammatory infiltrate was noted in both the diabetic a
273 gnificant histopathological reduction of the inflammatory infiltrate was observed after clobetasol tr
274   At 3 weeks p.i., a mixed acute and chronic inflammatory infiltrate was observed in the urethra that
275 ull were not different from those in WT, the inflammatory infiltrate was reduced in SP/ON-null PDL.
276 tive responses increased, although the total inflammatory infiltrate was reduced.
277                            After 3 days, the inflammatory infiltrate was significantly higher in the
278 lar channels, and adventitial and periaortic inflammatory infiltrates were assessed in an equal numbe
279               Axonal swelling and perineural inflammatory infiltrates were detectable at every time p
280                                              Inflammatory infiltrates were not associated with neurog
281 ubstantial amounts of perivascular edema and inflammatory infiltrates were noted in the lungs of Tg m
282                           Renal interstitium inflammatory infiltrates were observed in 50% of cases.
283                    Villus blunting and heavy inflammatory infiltrates were seen in the 5-FU-challenge
284                                   Persistent inflammatory infiltrates were seen in the lungs and brai
285 gical features, such as inclusion bodies and inflammatory infiltrates, were more severe and prominent
286 en, proinflammatory cytokine production, and inflammatory infiltrate while simultaneously preventing
287  mucosal biopsy showed a slight perivascular inflammatory infiltrate while skin biopsy was normal.
288 ical disease can occur in the presence of an inflammatory infiltrate with eosinophilic, neutrophilic,
289 n muscle biopsy--namely, rimmed vacuoles, an inflammatory infiltrate with invasion of non-necrotic mu
290 acterized by epidermal thickening and dermal inflammatory infiltrates with a dominant Th2 profile dur
291 tic islets from these transgenic mice showed inflammatory infiltrates with corresponding destruction
292                   CRTH-positive Th2-dominant inflammatory infiltrate, with increased IL-4 expression,
293 on, accompanied by a significantly increased inflammatory infiltrate within the central nervous syste
294 ear layers of the retina, and a perivascular inflammatory infiltrate within the choroid.
295 ons are required for the generation of early inflammatory infiltrate within the injured brain, and wh
296                      Characterization of the inflammatory infiltrate within the skeletal muscle tissu
297 ls were present in perifollicular and dermal inflammatory infiltrates within acne lesions, suggesting
298  mice had similar numbers and composition of inflammatory infiltrates within hind limb skeletal muscl
299 o concanavalin A stimulation, and absence of inflammatory infiltrates within the brain.
300 stologic analysis showed absent to low-grade inflammatory infiltrates without cardiomyocyte necrosis.

 
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