ライフサイエンスコーパス: inflammatory lesion

1 50.3%) of benign lesions (including 11 of 12 inflammatory lesions).

2 primary or metastatic tumors, infections, or inflammatory lesions).

3 block migration of leukocytes to the site of inflammatory lesion.

4 ic stress manifest within monocytes in acute inflammatory lesions.

5 ction, with fewer neutrophils present in the inflammatory lesions.

6 in younger patients, particularly those with inflammatory lesions.

7 vel, noncytolytic infection, causing ongoing inflammatory lesions.

8 scular disease characterized by formation of inflammatory lesions.

9 essing cells as a dominant population within inflammatory lesions.

10 y a massive infiltration of neutrophils into inflammatory lesions.

11 neurons and later in infiltrating T cells in inflammatory lesions.

12 ce that received RCA I alone did not develop inflammatory lesions.

13 a shift in tissue trafficking and accrual in inflammatory lesions.

14 ss and can contribute to the pathogenesis of inflammatory lesions.

15 remitting spastic paraplegia and hemorrhagic inflammatory lesions.

16 nt objective was to control the mucogingival inflammatory lesions.

17 an outcome expected to result in more severe inflammatory lesions.

18 interaction to regulate virus-induced immuno-inflammatory lesions.

19 g of arginase I, predominantly in submucosal inflammatory lesions.

20 loping gradually following the appearance of inflammatory lesions.

21 -specific T cells in the initiation of these inflammatory lesions.

22 nced by the increased glycolytic activity of inflammatory lesions.

23 an effective means to control virus-induced inflammatory lesions.

24 recruitment and the migration of cells into inflammatory lesions.

25 nal to EAE effector T cells found within the inflammatory lesions.

26 otype resembled that of T cells infiltrating inflammatory lesions.

27 ins during myeloid cell-cell interactions in inflammatory lesions.

28 destruction of connective tissue in chronic inflammatory lesions.

29 therapeutic transgene factors to autoimmune inflammatory lesions.

30 at lower doses, with development of chronic inflammatory lesions.

31 tissue localization, and their prominence in inflammatory lesions.

32 NFkappaB-mediated cytokine production in the inflammatory lesions.

33 or TNFRp55 in resolution of parasite-induced inflammatory lesions.

34 dy production as well as in the induction of inflammatory lesions.

35 e as a neuroimaging method for assessment of inflammatory lesions.

36 expression of this gene within cells in the inflammatory lesions.

37 g CD11b+ monocyte/macrophage diapedesis into inflammatory lesions.

38 ant in development or maintenance of chronic inflammatory lesions.

39 igodendrocytes and remyelinated axons in the inflammatory lesions.

40 showed many FR-beta-positive macrophages in inflammatory lesions.

41 considered a white matter disease with focal inflammatory lesions.

42 nstituents of the tissue microenvironment of inflammatory lesions.

43 gical environments, such as solid tumors and inflammatory lesions.

44 was performed to look for active and chronic inflammatory lesions.

45 the CNS leading to a selective enrichment in inflammatory lesions.

46 er of cluster of differentiation 45-positive inflammatory lesions.

47 ulation of CD207(+) dendritic cells (DCs) in inflammatory lesions.

48 confirmed uptake of (18)F-FOL in myocardial inflammatory lesions.

49 ided effector and memory T cell migration to inflammatory lesions.

50 acaques that demonstrated moderate to severe inflammatory lesions.

51 enchymal origin in human cancers and chronic inflammatory lesions.

52 suitable marker for the in vivo detection of inflammatory lesions.

53 Elevated PGE(2) is a hallmark of most inflammatory lesions.

54 cantly more when compared to histiocyte-rich inflammatory lesions (0/11, 0%, P<.001) and xanthelasma

55 tion revealed 13 distinct lesion types, with inflammatory lesions (25%) and melanocytic tumors (23%)

56 Of the 18 inflammatory lesions, 3 (17%) showed an increase and 15

57 Here, we report that in inflammatory lesions, a second barrier composed of react

58 dystrophy suffer from early-onset cutaneous inflammatory lesions accompanied by an infiltration of T

59 cy in progressive multiple sclerosis without inflammatory lesion activity.

60 nitis suppurativa (defined as a total of >=5 inflammatory lesions affecting >=2 distinct anatomical a

61 Homozygotes display chronic inflammatory lesions affecting the feet, salivary glands

62 ims of this study were to assess periodontal inflammatory lesions after P. gingivalis-induced periodo

63 n of dinitrofluorobenzene, and show enhanced inflammatory lesions after topical applications of phorb

64 These inflammatory lesions also exhibited evidence of axonal i

65 Active inflammatory lesions also exhibited staining for decorin

66 le of delivering high doses to periodontitis inflammatory lesion and cells) on experimental periodont

67 whether a few T cells arrive to initiate the inflammatory lesion and subsequently expand locally, or

68 Eleven histiocyte-rich inflammatory lesions and 10 xanthelasma served as contro

69 in oligodendrocyte lineage cells within the inflammatory lesions and a reduction in motor disability

70 nating disorder of the CNS, characterized by inflammatory lesions and an underlying neurodegenerative

71 '-(18)F-fluoromaltotriose is not taken up by inflammatory lesions and appears to be specific to bacte

72 RL/lpr) spontaneously develop lacrimal gland inflammatory lesions and are a model for the human disea

73 ormalities that indicate the distribution of inflammatory lesions and axonal loss (MRI); interference

74 14 weeks, tissue samples were evaluated for inflammatory lesions and cytokine messenger RNA by real-

75 ions revealed no significant differences for inflammatory lesions and demyelination, cytokine product

76 al nervous system (CNS), with characteristic inflammatory lesions and demyelination.

77 utoimmunity as exemplified by the multifocal inflammatory lesions and early demise of the TGF-beta1 n

78 MRI is capable of detecting more inflammatory lesions and erosions than US, X-ray, or CT.

79 NKG2D are highly expressed in carcinomas and inflammatory lesions and have been linked to immunosurve

80 the strains of parasites resulted in severe inflammatory lesions and high numbers of parasites in ey

81 tial restriction is imposed on the spread of inflammatory lesions and highlight systemic ACKR2 induct

82 t from cells, but is found at high levels in inflammatory lesions and in many premalignant and malign

83 TSG-6 protein, up-regulated in inflammatory lesions and in the ovary during ovulation,

84 alpha mice paralleled the development of the inflammatory lesions and included primary and secondary

85 lf may increase the influx of neutrophils in inflammatory lesions and influence disease susceptibilit

86 proach to limit the severity of viral immuno-inflammatory lesions and may be worth exploring as a mea

87 gnancies, whereas the uptake of (18)F-FDG in inflammatory lesions and normal breast tissues decreases

88 r patients (25.0%) had severe acne with deep inflammatory lesions and pitted scars.

89 developed a chronic sustained disease, with inflammatory lesions and primary demyelination in the sp

90 nervous system (CNS) characterized by focal inflammatory lesions and prominent demyelination.

91 ecificities of lymph node cells, and reduced inflammatory lesions and secreted IL-17 and IL-2 from pe

92 that Candida colonization delays healing of inflammatory lesions and that inflammation promotes colo

93 ifferentiate demyelinated lesions from other inflammatory lesions and therefore cannot predict diseas

94 to leukocytes and possibly microglia within inflammatory lesions and TIMP-1 expression that was obse

95 e autotoxic if overproduced, as evidenced in inflammatory lesions and tissue destruction in experimen

96 how that CD56(bright) NK cells accumulate in inflammatory lesions and, in the appropriate cytokine en

97 , increased overt loss of stratum corneum in inflammatory lesions, and impaired stratum corneum thick

98 that RRV- and CHIKV-induced musculoskeletal inflammatory lesions, and macrophages present in these l

99 ay predominate over Th2 lymphocytes in these inflammatory lesions, and that prevention of diabetes by

100 Granulomatous inflammatory lesions are a major histopathological featu

101 findings suggest that fibroproliferative and inflammatory lesions are independent entities, both of w

102 ane inflammatory lesions, but that placental inflammatory lesions are not associated with increased p

103 Because the inflammatory lesions are primarily orchestrated by Th1 c

104 The inflammatory lesions are primarily perpetuated by neutro

105 ating the poorly defined mechanisms by which inflammatory lesions are spatially restricted in vivo is

106 pts regarding the etiology and management of inflammatory lesions around dental implants continue to

107 mice, but not Fas-deficient mice, maintained inflammatory lesions associated with either Leishmania m

108 was helpful in controlling the mucogingival inflammatory lesions associated with this condition.

109 erythematosus characterized by cold-induced inflammatory lesions at acral locations presenting in ea

110 ystemic and tissue-specific autoimmunity and inflammatory lesions at mucosal interfaces.

111 ased infiltration of CD11b(+) cells into the inflammatory lesions at the acute phase.

112 outcome that was based on assessment of new inflammatory lesions, best corrected visual acuity, ante

113 treatment is not solely derived from stromal inflammatory lesions but likely also originates from the

114 are at increased risk for placental membrane inflammatory lesions, but that placental inflammatory le

115 e infected for 365 d have very few organized inflammatory lesions, but the bacteria continue to persi

116 FTY720 has been used to control inflammatory lesions, but the mechanisms by which the dr

117 e disease activity index, decreasing colonic inflammatory lesions by 4-fold, and suppressing inflamma

118 rials, COC was associated with reductions in inflammatory lesions by 62%, placebo was associated with

119 ates specifically in tuberculosis-associated inflammatory lesions by selective retention within macro

120 te that some of the neutrophils recruited to inflammatory lesions can differentiate into neutrophil-D

121 tantial demyelination results from the focal inflammatory lesion caused by the injection of lipopolys

122 The SUV of inflammatory lesions caused by radiation therapy (+1.16%

123 objective was to compare the distribution of inflammatory lesions centrally and laterally within the

124 tient with metastatic melanoma who developed inflammatory lesions circumscribing pigmented areas of s

125 and more (3.1 vs 0.3, P = .008) subclinical inflammatory lesions compared with DTPA-Gd, including in

126 2-weighted images was seen in 12 of 14 (86%) inflammatory lesions compared with four of 21 (19%) HNF-

127 Atherosclerotic plaques are chronic inflammatory lesions composed of dysfunctional endotheli

128 IL-26 expressed in inflammatory lesions confers proinflammatory properties

129 tained numerous perivascular and parenchymal inflammatory lesions consisting of predominantly CD4+ an

130 In biopsies with active inflammatory lesions containing Langerhans' cells, hyper

131 cell histiocytosis (LCH) is characterized by inflammatory lesions containing pathologic CD207(+) dend

132 success with DFD-29 vs placebo and (2) total inflammatory lesion count reductions with DFD-29 vs plac

133 nd 9% (-16 to 38) in controls (p=0.023), and inflammatory lesion counts reduced by 49% (30 to 75) in

134 (+18% +/- 8%; P < 0.01), whereas the SUVs of inflammatory lesions decreased over time (-17% +/- 13% o

135 1c and MHC II expression when recovered from inflammatory lesions, demonstrating neutrophil --> hybri

136 tem (CNS) tuberculosis (TB) worsening due to inflammatory lesions, despite optimal antitubercular the

137 ochemical demonstration of HHV-6 antigens in inflammatory lesions, detection of increased HHV-6 speci

138 Significant inflammatory lesions develop initially in the periductal

139 lved in the mechanism by which these chronic inflammatory lesions develop.

140 ture, but secreted TNF is needed for optimal inflammatory lesion development.

141 contrast in liver, spleen, lymph nodes, and inflammatory lesions (eg, atherosclerotic plaques).

142 he switch from a pro-inflammatory to an anti-inflammatory lesion environment.

143 l validated models that mimic aspects of the inflammatory lesion evident in diseases such as COPD.

144 ultinucleated giant cells from patients with inflammatory lesions expressed CD44V6- and CD44V9-contai

145 Multifocal inflammatory lesions featuring destruction of lipid-rich

146 cells increased disease activity and colonic inflammatory lesions following C. difficile infection.

147 late BBB permeability changes and autoimmune inflammatory lesion formation within the brain elicited

148 Rapamycin treatment reduced inflammatory lesions formed in the ears of Leishmania-in

149 ly sensitive and specific in differentiating inflammatory lesions from malignant lesions.

150 ypical severe combined immunodeficiency with inflammatory lesions, granulomas, and autoimmunity was t

151 e and attract T cells and eosinophils to the inflammatory lesion has not been determined.

152 d spatial relationship between HSV-1 DNA and inflammatory lesions has been previously reported for th

153 Gingivitis is a non-specific inflammatory lesion in response to the accumulation of o

154 Gingivitis is a nonspecific inflammatory lesion in response to the accumulation of o

155 After induction of a focal inflammatory lesion in the brain, there is rapid hepatic

156 in the serum following generation of a focal inflammatory lesion in the brain.

157 ection with herpes simplex virus leads to an inflammatory lesion in the cornea orchestrated by CD4+ T

158 on with HSV causes a chronic T cell-mediated inflammatory lesion in the cornea.

159 Early SD OCT revealed an inflammatory lesion in the photoreceptor outer segment l

160 nition of a similar self antigen provokes an inflammatory lesion in the target organ.

161 MRI ruled out the presence of active inflammatory lesions in 60.4% of patients with sacroilii

162 that CD56(bright) NK cells accumulate within inflammatory lesions in a wide variety of clinical disea

163 te to glomerular macrophage infiltration and inflammatory lesions in aging.

164 measure to gauge the intensity of individual inflammatory lesions in axial spondyloarthritis (SpA) ha

165 In contrast, inflammatory lesions in B10.D2 recipients were reminisce

166 FDP is also helpful for inflammatory lesions in BP patients.

167 DR15 MS mice uniquely developed inflammatory lesions in brain and spinal cord gray matte

168 ffects important for the recovery from acute inflammatory lesions in CNS autoimmunity.

169 ce correlate with spontaneous development of inflammatory lesions in different organs of aged Sin(-/-

170 With MRI, we were able to detect inflammatory lesions in diseased rats, which correlated

171 essary step in the development of pathologic inflammatory lesions in experimental autoimmune encephal

172 ocess, but predispose patients to developing inflammatory lesions in extraarticular tissues.

173 venules (high endothelial venules [HEVs]) in inflammatory lesions in Helicobacter species-infected li

174 nd confirm more, smaller, and earlier active inflammatory lesions in living mice by in vivo MRI.

175 on may facilitate the development of chronic inflammatory lesions in Lyme disease.

176 ious inflammatory stimuli and a reduction of inflammatory lesions in models of demyelinating disease.

177 utoimmune renal, vasculitic, and intraocular inflammatory lesions in MRL/lpr mice.

178 d-brain barrier (BBB) is a hallmark of acute inflammatory lesions in multiple sclerosis (MS) and its

179 be a second step during the formation of new inflammatory lesions in multiple sclerosis.

180 ic gastrointestinal inflammation, as well as inflammatory lesions in other tissues.

181 The assembly of inflammatory lesions in rheumatoid arthritis is highly r

182 ltrating mononuclear cells into CNS, minimal inflammatory lesions in spinal cord, and preservation of

183 doscopies and was found out to have multiple inflammatory lesions in the area extending from the cecu

184 is (MS) is characterized by demyelinated and inflammatory lesions in the brain and spinal cord that a

185 e macrophages and microglia were detected in inflammatory lesions in the brain of children with sever

186 drocytes are exposed to various cytokines in inflammatory lesions in the central nervous system.

187 OX-40R+ T cells isolated from inflammatory lesions in the CNS of animals with experime

188 ression is induced in astrocytes surrounding inflammatory lesions in the CNS.

189 ppressed clinical signs of EAE and abolished inflammatory lesions in the CNS.

190 roptosis, Paneth cell loss and focal erosive inflammatory lesions in the colon.

191 istence of HSV-1 DNA and its relationship to inflammatory lesions in the conjunctiva and eyelid skin

192 S from MOG-immunized mice revealed extensive inflammatory lesions in the cPLA2alpha+/- mice, whereas

193 Spinal cord inflammatory lesions in the EAE model can be noninvasive

194 skin patches and alleviated pneumonitis and inflammatory lesions in the extremities of me/me mice co

195 o most mouse models of mucosal inflammation, inflammatory lesions in the gastrointestinal tract of Si

196 aR(-/-)) mice uniformly develop angiocentric inflammatory lesions in the lung.

197 but noninvasively, visualize the lymphocytic/inflammatory lesions in the pancreatic islets.

198 Therefore, in order to avoid missing inflammatory lesions in the paranasal sinuses, it is rea

199 ically predisposed individuals, resulting in inflammatory lesions in the proximal small intestine.

200 ecruitment of circulating cells into chronic inflammatory lesions in the skin and elsewhere.

201 ical subunit of LUBAC, spontaneously develop inflammatory lesions in the skin and other organs.

202 ve distribution of meningeal and parenchymal inflammatory lesions in the spinal cord and optic nerves

203 n of mice develops discontinuous, transmural inflammatory lesions in the terminal ileum with 100% pen

204 YitFc mice develop discontinuous, transmural inflammatory lesions in the terminal ileum, similar to w

205 at induce histologically distinct autoimmune inflammatory lesions in the thyroid.

206 load caused elevated levels of apoptosis and inflammatory lesions in these mice and produced a smalle

207 Thus, it appears plausible that the inflammatory lesions in top3beta(-/-) mice are caused by

208 Inflammatory lesions in VEGF-A transgenic mice closely r

209 regulate chemokine secretion by monocytes in inflammatory lesions in vivo and provide a model for the

210 permitting noninvasive visualization of the inflammatory lesions in vivo in real time.

211 ylactic treatment in a hamster model of oral inflammatory lesions in vivo.

212 lymph node T cells from Tg mice, and caused inflammatory lesions in white matter of the CNS and symp

213 Radiographs do not allow early inflammatory lesions indicating sacroiliitis to be diagn

214 resulting in localized tissue hypoxia within inflammatory lesions ("inflammatory hypoxia").

215 Inflammatory lesions involving the brainstem are associa

216 The elimination of lymphocytes within inflammatory lesions is a critical component in the reso

217 Although development of multifocal inflammatory lesions is the underlying pathological proc

218 channel 1 expression in neurons and glia in inflammatory lesions is unknown and, crucially, acid-sen

219 5 in periapical tissues in bacterium-induced inflammatory lesions largely reduced bone destruction, i

220 rosis (MS) is an autoimmune disease in which inflammatory lesions lead to tissue injury in the brain

221 al roles in the development of virus-induced inflammatory lesions, leading to eye disease and causing

222 e detected in histologically normal skin and inflammatory lesions (lichen sclerosus), chromosomal abn

223 Resection of the inflammatory lesion may be curative.

224 uch as foreign body reactions and peripheral inflammatory lesions, monocytes fuse to form large, mult

225 At the LM level, the inflammatory lesion of peri-implantitis was in most case

226 ic factors do not permit the evolution of an inflammatory lesion of sufficient magnitude to induce ov

227 tis of unknown etiology (VUE), a destructive inflammatory lesion of villous placenta, is characterize

228 ctin function to recruit leukocytes into the inflammatory lesions of diseases such as rheumatoid arth

229 ion of interleukin 10 by glial cells in both inflammatory lesions of EAE and normal-appearing CNS whi

230 The features of acute inflammatory lesions of EG with limited tissue destructi

231 Fewer apoptotic cells were detected in inflammatory lesions of lpr mice than in wild-type lesio

232 y astrocytes and infiltrating macrophages in inflammatory lesions of MS patients.

233 polymorphisms on periodontal parameters and inflammatory lesions of oral mucosa as a characteristic

234 Inflammatory lesions of periodontal disease contain all

235 TSP2 has regulatory function in the chronic inflammatory lesions of rheumatoid arthritis.

236 noninflammatory lesions, and 14 (87.5%) had inflammatory lesions of seborrheic regions.

237 Inflammatory lesions of target organs are characterized

238 ASIC1 contributes to axonal degeneration in inflammatory lesions of the central nervous system (CNS)

239 t extracellular matrix proteins found within inflammatory lesions of the gastrointestinal tract throu

240 isite cell population for the genesis of the inflammatory lesions of the islets of Langerhans.

241 thologies pancreatic ductal adenocarcinomas, inflammatory lesions of the pancreas, postpancreatectomy

242 Because telomerase activity can be found in inflammatory lesions of the skin, this indicates that te

243 quired to induce apoptosis of T cells within inflammatory lesions once pathogens are eliminated, and

244 nder the acidotic tissue conditions found in inflammatory lesions opens to allow influx of sodium and

245 ometimes not differentiate malignancies from inflammatory lesions or detect malignant transformation

246 coxibs are ideal candidates for detection of inflammatory lesions or early-stage COX-2-expressing hum

247 of cancer, and to facilitate the analysis of inflammatory lesions or gastrointestinal bleeding during

248 may develop within newly formed acute focal inflammatory lesions or in chronic pre-existing lesions

249 47 +/- 0.09, compared with 0.73 +/- 0.18 for inflammatory lesions (P = .0004), 0.82 for the beta-cate

250 of lesional cells in all 11 histiocyte-rich inflammatory lesions (P<.001) and all 10 xanthelasma les

251 k4a and p19/Arf in CNT- and asbestos-induced inflammatory lesions precedes mesothelioma; this results

252 However, inflammatory lesions, predominantly lymphocytic, develop

253 l/macrophage activation at the site of focal inflammatory lesions, presumably by preventing leukocyte

254 P) are cleared from sites of inflammation as inflammatory lesions resolve.

255 degeneration specifically at the site of the inflammatory lesions, suggesting that locally produced i

256 are also associated with the pathogenesis of inflammatory lesions, suggesting that the modulation of

257 inducer, and iNOS, before the appearance of inflammatory lesions, suggests that failed regulation of

258 yocardial (18)F-FOL uptake colocalizing with inflammatory lesions (SUV(mean), 2.1 +/- 1.1), whereas u

259 gPL1 are more resistant to TE and have fewer inflammatory lesions than mice infected with the wild ty

260 ue with a CD4(+) T-cell-orchestrated chronic inflammatory lesion that impairs vision.

261 Because immunotherapy may cause new inflammatory lesions that are detectable on (18)F-FDG PE

262 ain viable in microenvironments of immune or inflammatory lesions that are rich in cytotoxic inflamma

263 Disease exacerbation with more extensive inflammatory lesions that contained activated cells of t

264 Chronic inflammatory lesions that developed in the kidney and pa

265 elapsing-remitting clinical course and focal inflammatory lesions that have a predilection for the pe

266 estinal helicobacter species develop hepatic inflammatory lesions that have previously been described

267 ing modality for the assessment intensity of inflammatory lesions.The aim of this study was to measur

268 findings from chronic (NG) versus acute (EG) inflammatory lesions, these data reveal that NG displays

269 It is postulated that IL-13 drives inflammatory lesions through the modulation of both hema

270 te-stage syphilis can lead to development of inflammatory lesions throughout the body, which can lead

271 the tetracycline-based MMP inhibitor in the inflammatory lesion, thus synergistically enhancing the

272 e that proceeds from initial peribronchiolar inflammatory lesions to characteristic, fibrotic cellula

273 ing our ability to diagnose malignancies and inflammatory lesions, to grade severity of pancreatitis,

274 phalomyelitis to reproducibly induce 2 brain inflammatory lesion types, differentiated by their T cel

275 In giant cell arteritis (GCA), inflammatory lesions typically produce interferon-gamma(

276 lows for defining localization and extent of inflammatory lesions, visualizes soft tissue involvement

277 cy in least-squares mean reductions in total inflammatory lesions vs placebo (MVOR-1: TD, -9.2; 95% C

278 Spinal cord inflammatory lesions were also reduced in Panx1 KO EAE m

279 , the prevalence and severity of multisystem inflammatory lesions were comparable in backcross rats b

280 ues 3 days after virus challenge, and severe inflammatory lesions were found in the spleen and liver

281 the two groups of mice, distinct patterns of inflammatory lesions were found in their livers at 7-14

282 Inflammatory lesions were more diffuse and extensive in

283 No inflammatory lesions were observed in the ileum or colon

284 one of the sinuses.Measurements of the same inflammatory lesions were performed sequentially with di

285 otype, infiltrates of MS2-3C8 Tg T cells and inflammatory lesions were predominantly located in the b

286 In Trial 2, inflammatory lesions were significantly reduced at 12 we

287 segment, and, contrary to previous studies, inflammatory lesions were the predominant finding.

288 coccus or group G streptococcus developed an inflammatory lesion with high bacterial counts, marked n

289 oimmune syndrome characterized by multiorgan inflammatory lesions with a marked female predominance,

290 characteristics were compared between active inflammatory lesions with and without CNV activity.

291 At 38 weeks, RFC1(+/-) mice developed local inflammatory lesions with or without epithelial dysplasi

292 The disease is characterized by focal inflammatory lesions with perifollicular T-cell infiltra

293 immunized rats showed focal macrophage-rich inflammatory lesions, with FR-beta expression occurring

294 by insulin-producing pancreatic beta cells, inflammatory lesions within islets (insulitis), and beta

295 monstrated that patients with characteristic inflammatory lesions within the brain on magnetic resona

296 gh little is known about the distribution of inflammatory lesions within the structures of the spine.

297 f therapy, our patient had resolution of his inflammatory lesions without serious infections or adver