ライフサイエンスコーパス: inflammatory reaction

1 lial clearance of misfolded proteins and the inflammatory reaction.

2 titis with apoptotic hepatocytes and minimal inflammatory reaction.

3 and stimulates an innate and adaptive immune inflammatory reaction.

4 interplay of gut microbiota and host immune/inflammatory reaction.

5 synthesis is known to optimize local tissue inflammatory reaction.

6 e complement system plays a key role in this inflammatory reaction.

7 d, to decipher how syndecan-1 impacts on the inflammatory reaction.

8 nse by microglia while triggering a moderate inflammatory reaction.

9 an important role in the coordination of an inflammatory reaction.

10 entrations of bacteria yet only some show an inflammatory reaction.

11 ng at 48 hours and delayed resolution of the inflammatory reaction.

12 and mesenteric lymph nodes, causing an acute inflammatory reaction.

13 cells that are recruited and involved in the inflammatory reaction.

14 of proper neuro-vascular associations and an inflammatory reaction.

15 y in pulmonary sensory neurons during airway inflammatory reaction.

16 ional knockout mouse brain invoked a massive inflammatory reaction.

17 betes within a week, characterized by strong inflammatory reaction.

18 ng to the pathogenesis of this novel type of inflammatory reaction.

19 le response to vaccination versus that of an inflammatory reaction.

20 se plays a crucial role in host defenses and inflammatory reaction.

21 cosal healing, such as the persistence of an inflammatory reaction.

22 , and dendritic cells, in the local allergic inflammatory reaction.

23 ase pattern that is associated with an acute inflammatory reaction.

24 layed oral mucosa healing in the presence of inflammatory reaction.

25 phase of sepsis is characterized by a strong inflammatory reaction.

26 ferent antigens that trigger distinct immune-inflammatory reactions.

27 es to control pathologies with overexuberant inflammatory reactions.

28 lations in the steady-state and during acute inflammatory reactions.

29 function in pathogen- and autoimmune-induced inflammatory reactions.

30 d the types of T cells involved in different inflammatory reactions.

31 icant and influenced by diet, metabolic, and inflammatory reactions.

32 dictating the magnitude of subsequent acute inflammatory reactions.

33 itical role in myeloid cell-mediated in vivo inflammatory reactions.

34 amage inferred from attenuation of glial and inflammatory reactions.

35 T cells (Tregs) are thought to downmodulate inflammatory reactions.

36 by IL-33 probably play an important role in inflammatory reactions.

37 of mitochondria or in induction of untoward inflammatory reactions.

38 ons for bone disorders but may cause serious inflammatory reactions.

39 ocytic, phagocytic, proinflammatory, or anti-inflammatory reactions.

40 role of mast cells and IL-1 in NBP-mediated inflammatory reactions.

41 ns and for preventing excessive or prolonged inflammatory reactions.

42 to facilitate the resolution of pathological inflammatory reactions.

43 ling pathway to protect against exacerbating inflammatory reactions.

44 ide production is believed to participate to inflammatory reactions.

45 betaR) activation controls and downregulates inflammatory reactions.

46 opment and progression of local and systemic inflammatory reactions.

47 and prevents the development of exacerbated inflammatory reactions.

48 g homeostasis and preventing excessive acute inflammatory reactions.

49 dating the molecular mechanisms that lead to inflammatory reactions.

50 re prominent contributor to the pathology of inflammatory reactions.

51 genetically engineered mice induced similar inflammatory reactions.

52 conditions associated with retinal vascular inflammatory reactions.

53 onist could be useful to treat skin-mediated inflammatory reactions.

54 leukocyte recruitment in the development of inflammatory reactions.

55 they simultaneously support tolerogenic and inflammatory reactions.

56 Lactococcus lactis G121 to prevent allergic inflammatory reactions.

57 ly titanium dioxide) leading to peri-implant inflammatory reactions.

58 ced gene 6 (TSG-6), which is up-regulated in inflammatory reactions.

59 municable diseases and are driven by chronic inflammatory reactions.

60 responders" to tissue stress and drivers of inflammatory reactions.

61 n and from reduced allergic, autoimmune, and inflammatory reactions.

62 regulation, orchestrating both pro- and anti-inflammatory reactions.

63 Inflammatory reactions activated by pattern recognition

64 om repeated injections, a potential toxic or inflammatory reaction after exposure to the biologic age

65 tin-like domain of TM resulted in a stronger inflammatory reaction after Stx2/LPS with more neutrophi

66 chistosomes results in a CD4 T cell-mediated inflammatory reaction against parasite eggs that varies

67 gainst pathogens while avoiding harmful auto-inflammatory reactions against the host.

68 VCAM-1 in regulating the immune response and inflammatory reactions against viral infections.

69 the poorly understood instant blood-mediated inflammatory reaction and a thorough understanding of ea

70 factors and antigens that stimulate a local inflammatory reaction and activation of the innate immun

71 In pulmonary fibrosis, an inflammatory reaction and differentiation of myofibrobla

72 ommitment of cholangiocytes and enhances the inflammatory reaction and liver fibrosis.

73 ating that TLR2 activation limited the toxic-inflammatory reaction and maintained assembly of the dru

74 fective means of controlling a virus-induced inflammatory reaction and may act mainly by the effects

75 is critically involved in the postinfarction inflammatory reaction and mediates adverse dilative remo

76 We examined neuronal death, inflammatory reaction and neurogenesis in hippocampus 72

77 f sepsis for cytokine storm, and deregulated inflammatory reaction and proapoptotic environment, as w

78 f MRSA pneumonia by attenuating an excessive inflammatory reaction and protecting the lung from patho

79 portant for resolution of the postinfarction inflammatory reaction and regulates fibroblast function.

80 lls (MCs) contribute to the control of local inflammatory reactions and become hyporesponsive after p

81 ntiviral immunity but can exacerbate harmful inflammatory reactions and cause hematopoietic stem cell

82 EX1, NOTCH3, and Col4A1 are known to provoke inflammatory reactions and damage the brain in a wide va

83 teine proteases, have been shown to increase inflammatory reactions and lymphocyte apoptosis.

84 Thus, Foxd1 modulates inflammatory reactions and prevents autoimmunity by dire

85 Inhalation of agricultural dusts causes inflammatory reactions and symptoms such as headache, fe

86 ortant in bacterial killing, but also drives inflammatory reactions and tissue oxidation.

87 eoporosis but have also been found to induce inflammatory reactions and to delay the progression of b

88 shown to cause foreign-body sensation, local inflammatory reaction, and conjunctival hyperemia.

89 insufficient re-epithelialization, decreased inflammatory reaction, and reduced collagen remodeling.

90 ing immunomodulatory effects in allergic and inflammatory reactions, and also plays a key regulatory

91 ns, including IgE-dependent allergies, other inflammatory reactions, and mastocytosis.

92 f regulatory T cells, which help to suppress inflammatory reactions, and plays a significant role in

93 tors, FPR1 and FPR2, play critical roles for inflammatory reactions, and receptor-specific antagonist

94 tions, such as inadequate bone regeneration, inflammatory reactions, and wound infection.

95 receptors, rod and cone opsins; inhibit the inflammatory reactions; and induce the expression of ant

96 Although inflammatory reactions are beneficial and necessary for

97 complement system, known to produce a local inflammatory reaction, are associated with the plaques a

98 d liver dysfunctions and the severity of the inflammatory reaction as evaluated by biomarkers kinetic

99 ar TA injection markedly decreased the acute inflammatory reaction associated with intravitreous Ad-L

100 Controlling the overwhelming inflammatory reaction associated with polymicrobial seps

101 o bone (entheses), the key territory for the inflammatory reaction associated with PsA, being subject

102 nvolved in the generation of the more severe inflammatory reaction associated with the late central n

103 ole for VCAM-1 in promoting successful local inflammatory reactions associated with efficient viral c

104 This results in an inflammatory reaction at several sites, including the sy

105 We also assessed oxidative stress and the inflammatory reaction at the injury site, neuronal and o

106 ipants (40%) experienced a localized sterile inflammatory reaction at the site of the injection, whic

107 ies show the presence of a lymphohistiocytic inflammatory reaction at the sites of delivery of alloge

108 involvement in immediate as well as chronic inflammatory reactions at both local and distal sites po

109 s suggest that vitamin D modulates cutaneous inflammatory reactions, at least in part, by increasing

110 transcription factors has a central role in inflammatory reactions, being stimulated by multiple cyt

111 sue-resident macrophages during infection or inflammatory reactions, besides self-replication of tiss

112 Loss of Bmal1 aggravated the inflammatory reaction both in vivo and in vitro.

113 Panx3 expression coincided with inflammatory reactions both in vivo and in vitro.

114 ltalpxP32::P(lpxL) lpxL) stimulated a strong inflammatory reaction but sickened mice before recovery

115 In contrast, CD39-null mice had inflammatory reactions but developed only minor pancreat

116 n is important because TNF not only furthers inflammatory reactions but might also be neuroprotective

117 onse to chemical or physical stimuli repress inflammatory reactions, but cells that undergo nonapopto

118 MKP-1 serves to limit the inflammatory reaction by inactivating JNK and p38, thus

119 ther propose that dynamic modulation of this inflammatory reaction by interrupting the vicious cycle

120 CX(3)CR1(+) lining macrophages restrict the inflammatory reaction by providing a tight-junction-medi

121 This inflammatory reaction can spread throughout the systemic

122 Isolated intraocular inflammatory reactions can be an initial manifestation o

123 ces between PPC and CCC in corneal edema, AC inflammatory reaction, capsular fibrosis, ACO, and PCO.

124 ncreatic islet transplantation (PITx), early inflammatory reactions cause an immediate loss of more t

125 occurs during isolation and from the severe inflammatory reactions caused by the transplantation pro

126 9 transgenic mice mounted an enhanced innate inflammatory reaction characterized by increased express

127 egimen-dependent induction of an acute local inflammatory reaction, characterized in part by rapid ne

128 Inflammatory reaction clears dead cells and matrix debri

129 hases of atherogenesis, including well-known inflammatory reactions consequent to intimal trapping an

130 oil' to the stem cells' seed), such as local inflammatory reactions, contribute to successful tissue

131 ith ridge height; thereafter, a more intense inflammatory reaction corresponded to reduction in alveo

132 vironment extended immobility, injuries, and inflammatory reactions demand local microcirculatory ada

133 m day 4 to day 14 (P < 0.001), with distinct inflammatory reaction demonstrated by lymphocyte and neu

134 Although frequently accompanied by local inflammatory reactions detectable in cerebrospinal fluid

135 An inflammatory reaction developed in all nine of the survi

136 A mild inflammatory reaction disappeared after 12 weeks.

137 Loss of T and B cell inflammatory reactions does not protect from impaired ly

138 syndrome is characterized by an overwhelming inflammatory reaction driven by excessive expansion of T

139 itations apply, most notably a chronic local inflammatory reaction due to permanent implantation of a

140 izing autoantibodies, who developed a severe inflammatory reaction during anti-tuberculosis treatment

141 zing bacteria, which is likely to impact the inflammatory reaction during mucosal diseases.

142 n regulates immune cell infiltration and the inflammatory reaction during sepsis, suggesting that act

143 t bear therapeutic potential by reducing the inflammatory reaction during the early phase of AILI.

144 y, focusing on muscle satellite cells (SCs), inflammatory reaction, fibrosis, and formation of new bl

145 gists when there is a need to review diverse inflammatory reactions; for identification of fungi, par

146 lected: VA, degree of anterior and posterior inflammatory reaction, foveal thickness measured by opti

147 hase plasma protein, is a major component of inflammatory reactions functioning as a mediator of inna

148 vs 45 years, P = .05), presence of vitreous inflammatory reactions >2+ (35.9% vs 6.2%, P = .04), vas

149 The remaining inflammatory reactions had a change in the ratio of CD4

150 Systemic inflammatory reactions have been postulated to exacerbat

151 re main effector cells of allergic and other inflammatory reactions; however, only a few anti-MC agen

152 ticipation in allergic responses and related inflammatory reactions; however, the glycan-containing l

153 poor survival of donor cells in response to inflammatory reactions, hypoxia, oxidative stress, or nu

154 This instant blood-mediated inflammatory reaction (IBMIR) activates the coagulation

155 Instant blood-mediated inflammatory reaction (IBMIR) causes significant destruc

156 lets can result in an instant blood-mediated inflammatory reaction (IBMIR) that contributes to early

157 iveness because of an instant blood-mediated inflammatory reaction (IBMIR) that occurs immediately af

158 re is hampered by the instant blood-mediated inflammatory reaction (IBMIR), a thromboinflammatory res

159 adversely trigger the instant blood-mediated inflammatory reaction (IBMIR).

160 t has been termed the instant blood-mediated inflammatory reaction (IBMIR).

161 nts without HIV infection caused intraocular inflammatory reaction in all cases and demonstrated more

162 autoimmune disease characterized by a local inflammatory reaction in and around islets followed by s

163 ation of active B burgdorferi infection, the inflammatory reaction in mice with persistent joint infl

164 t cytosolic RLR-RNA-sensing pathway mediated inflammatory reaction in response to acute liver IR.

165 PE-PLD-deficient mice fail to mount a normal inflammatory reaction in response to carrageenan adminis

166 nterica serotype Typhimurium causes an acute inflammatory reaction in the ceca of streptomycin-pretre

167 nterica serotype Typhimurium causes an acute inflammatory reaction in the ceca of streptomycin-pretre

168 SV-1 infection has been shown to initiate an inflammatory reaction in the cornea that leads to tissue

169 h herpes simplex virus 1 (HSV-1) sets off an inflammatory reaction in the cornea which leads to both

170 This infection usually results in a blinding inflammatory reaction in the cornea, orchestrated mainly

171 ve colitis are caused by an excessive immune-inflammatory reaction in the intestinal wall.

172 eficit in short term memory (STM) and strong inflammatory reaction in the ipsilateral cortex and hipp

173 Poly(I:C) injection induced a sustained inflammatory reaction in the SN and in the dorsolateral

174 wild-type or CXCR3-/- mice, they elicited an inflammatory reaction in wild-type but not in CXCR3-/- m

175 epidermis of adult mice also led to stronger inflammatory reactions in a tape-stripping test and in a

176 Inflammatory reactions in anterior segments developed in

177 ection against disease onset, and attenuated inflammatory reactions in arthritic mice.

178 Interleukin 6 plays a key role in mediating inflammatory reactions in autoimmune diseases and cancer

179 Inflammatory reactions in both IRI and infections share

180 invading microorganisms but also potentiate inflammatory reactions in cases of excessive or misdirec

181 R knocked down cholangiocytes fail to induce inflammatory reactions in control large cholangiocytes.

182 lving histamine-induced and allergen-induced inflammatory reactions in forearm skin.

183 sual sphingolipids that are known to promote inflammatory reactions in gingival fibroblasts and Toll-

184 e receptors (TLR) activate multiple steps in inflammatory reactions in innate immune responses.

185 FI is promising for noninvasive detection of inflammatory reactions in IR myocardium because of its m

186 that establish chronic infections and elicit inflammatory reactions in mammals.

187 dampen, rather than provoke, T cell-mediated inflammatory reactions in many clinical conditions where

188 ifs" were recently shown to block pathologic inflammatory reactions in murine models of autoimmune di

189 and IL-1beta was associated with comparable inflammatory reactions in patients and controls.

190 tenance of immune homeostasis or breakout of inflammatory reactions in patients with chronic inflamma

191 3CL1 mRNA, providing fine-tuning of cellular inflammatory reactions in response to IFN-gamma stimulat

192 fied as important factors amplifying the pro-inflammatory reactions in these pathologies.

193 -1 in the negative control of MAPK-regulated inflammatory reactions in vivo.

194 vation and has been used to study immune and inflammatory reactions in which nitric oxide (NO) is a v

195 evalence of ophthalmic findings (intraocular inflammatory reactions [including transient, isolated re

196 gulating tissue homeostasis and coordinating inflammatory reactions, including those involved in chro

197 and that suppressing Pcsk9 can decrease the inflammatory reaction induced by imiquimod treatment and

198 There were no visible inflammatory reactions, infections or extrusions around

199 gates" for transmigrating neutrophils in all inflammatory reactions investigated (elicited by leukotr

200 ils are minimized, which suppress subsequent inflammatory reaction involved in the liver.

201 A well-orchestrated inflammatory reaction involves the induction of effector

202 Resolution of inflammatory reactions involves emigration of monocyte-d

203 s do not express COX-2), intensifies a broad inflammatory reaction involving numerous cytokines and o

204 ins unknown but a timely and well-controlled inflammatory reaction is essential for the integrity and

205 tants of B. cenocepacia, suggesting that the inflammatory reaction is likely induced by an as yet unc

206 The inflammatory reaction is orchestrated by gamma interfero

207 nd an inducible chemokine, whose role in the inflammatory reaction is poorly known.

208 ized to the vessel wall, the etiology of the inflammatory reaction is unknown.

209 A characteristic feature of chronic inflammatory reactions is their persistence and predilec

210 hibit a cell death-type modulatory effect on inflammatory reactions is unknown.

211 (FA) dietary supplementation would modulate inflammatory reactions leading to periodontal disease in

212 though beneficial at early stages, excessive inflammatory reaction leads to cell death and tissue dam

213 The current VPC regime induced local skin inflammatory reaction limited to subcutaneous injection

214 To promote the inflammatory reaction, lipopolysaccharide was administra

215 Failure to terminate such inflammatory reactions may lead to detrimental systemic

216 The evidence for a chronic inflammatory reaction mediated by microglial cells in th

217 During granulomatous inflammatory reactions, myeloid cells can differentiate

218 No intraocular inflammatory reaction occurred after intravitreous injec

219 tachment appeared once more, however without inflammatory reaction of the anterior chamber.

220 ryptamine (5-HT)) is implicated in enhancing inflammatory reactions of skin, lung, and gastrointestin

221 Although signals that trigger acute inflammatory reactions often stimulate dendritic cell ma

222 (i.e. similar to untreated tissues), with no inflammatory reaction or fibrous encapsulation occurring

223 There were no intraocular hemorrhages, inflammatory reactions, or pigment dispersion and the IO

224 We propose that this plaque-independent inflammatory reaction originates from neurons burdened w

225 The inflammatory reaction over several days will drive robus

226 That the human whole blood inflammatory reactions paralleled the host response show

227 c strategies incorporating methods to elicit inflammatory reactions, particularly NK cell-driven lysi

228 2 entities characterized by a granulomatous inflammatory reaction pattern but with unique genetic an

229 s play a crucial role during stroke, and the inflammatory reaction promotes neurodegeneration.

230 (anterior chamber cells and flare, vitreous inflammatory reactions, retinal whitening), without clin

231 -evoked responses are elevated during airway inflammatory reaction, revealing the potential convergen

232 ted disease (IgG4-RD) is characterized by an inflammatory reaction rich in IgG4-positive plasma cells

233 sed impaired life span and intensive cardiac inflammatory reactions, showing that the cardiotoxic IFN

234 E. coli may contribute to the resolution of inflammatory reactions since E. coli can inhibit hydroxy

235 Although the granulocytes contribute to the inflammatory reaction, stem cells may promote tissue rep

236 yelination and persists despite the residual inflammatory reaction subsiding to levels seen in contro

237 ter a treatment that deliberately induces an inflammatory reaction, such as intravesical instillation

238 t defense, but also relevant in pathological inflammatory reactions, such as sepsis.

239 occurs as part of the central nervous system inflammatory reaction suggests a potential mechanism of

240 The type of inflammatory reaction suggests cytotoxic CD8+ T lymphocy

241 r the cause, or a feature of severe systemic inflammatory reaction syndrome, whatever the cause.

242 pates in the molecular events underlying the inflammatory reaction that accompanies cerebral ischemia

243 ayed immune response, followed by an intense inflammatory reaction that causes widespread tissue dama

244 Myocardial necrosis triggers an inflammatory reaction that clears the wound from dead ce

245 The injury causes a rapid inflammatory reaction that includes activation of the ti

246 Granulomas represent a localized inflammatory reaction that is characteristically observe

247 Myocardial infarction triggers an inflammatory reaction that is involved in cardiac remode

248 regeneration are closely associated with an inflammatory reaction that is usually characterized by s

249 ing that this is associated with the intense inflammatory reaction that occurs in this condition.

250 Surgery launches a systemic inflammatory reaction that reaches the brain and associa

251 Infarct healing is dependent on an inflammatory reaction that results in leukocyte infiltra

252 y preterm labor or preeclampsia, triggers an inflammatory reaction that results in spontaneous preter

253 rystals into the rat joint provoked a marked inflammatory reaction that was significantly inhibited (

254 innate immune response involves a variety of inflammatory reactions that can result in inflammatory d

255 deposits but damaged blood vessels caused by inflammatory reactions that lead to ischemia, amyloid ac

256 Bone loss in periodontitis results from inflammatory reactions that stimulate osteoclastic bone

257 of leprosy is challenging, particularly its inflammatory reactions, the major cause of irreversible

258 ponse to pathological stimuli by suppressing inflammatory reactions through AdipoR1/AMPK-dependent me

259 counteracts scar resolution by perpetuating inflammatory reactions through release of proinflammator

260 osapentaenoic acid (EPA, 20:5n-3)], modulate inflammatory reactions through various mechanisms, inclu

261 Macrophages play vital functions in host inflammatory reaction, tissue repair, homeostasis and im

262 n of CCR7 implies that during a TH2-mediated inflammatory reaction, TLSP-activated CD1c(+) DCs are re

263 The immune-mediated inflammatory reaction to allogeneic and xenogeneic stem

264 Histology found little inflammatory reaction to any hydrogel implant.

265 The inflammatory reaction to coated discs was assessed in th

266 letion of NK cells significantly altered the inflammatory reaction to corneal wounding, as evidenced

267 corneal healing by limiting the innate acute inflammatory reaction to corneal wounding.

268 uneventful bone healing, although the early inflammatory reaction to femur fracture was marginally e

269 Local inflammatory reaction to inoculation with E coli was att

270 may serve as a surrogate marker of unwanted inflammatory reaction to microbicide products topically

271 steady state and after induction of a type 2 inflammatory reaction to ovalbumin (OVA).

272 Periodontal disease is an inflammatory reaction to the bacteria in dental plaque.

273 Modulation of the inflammatory reaction to the wear products represents a

274 ts are dysregulated, leading to an increased inflammatory reaction to unspecific triggers.

275 test subject to assess cytotoxicity and pro-inflammatory reactions to diatom-biosilica.

276 cutaneous sensitization susceptibilities and inflammatory reactions to environmental insults in human

277 ppreciated mechanism that prevents prolonged inflammatory reactions to Gram-negative bacteria and LPS

278 try to draw an arc from initiation of acute inflammatory reactions to microbial pathogens to develop

279 f transplantation is associated with instant inflammatory reactions to the graft and subsequent islet

280 vidence that this concept holds true for the inflammatory reaction underlying AD, where IL-13 is over

281 ate HLA to evade host immune responses, host inflammatory reactions upregulate HLA, and differences a

282 behavioral deficits, neuronal injury and the inflammatory reaction using the kainic acid (KA) seizure

283 nly poised to initiate potentially injurious inflammatory reactions via immune complex formation asso

284 This inflammatory reaction was dependent on the interaction o

285 In 1 case an inflammatory reaction was observed in the anterior chamb

286 events, though a local sterile conjunctival inflammatory reaction was observed.

287 arization were increased, while the extended inflammatory reaction was repressed in the relaxin-treat

288 lymphatic system is a critical regulator of inflammatory reactions, we tested the hypothesis that ob

289 ith virulent pathogens often induce a strong inflammatory reaction, what drives the increased immune

290 culature and towards the core of the ensuing inflammatory reaction where they exert effector function

291 Th1 lymphocytes participate in cell-mediated inflammatory reactions, where the selectins play a role

292 resent the major effector cells driving this inflammatory reaction whereas Foxp3(+) regulatory T cell

293 at RAGE is a critical mediator of the type 2 inflammatory reactions which drive the development of T2

294 Sepsis is associated with a systemic inflammatory reaction, which can result in a life-endang

295 role of Foxp3(+)CD4(+) Tregs during ongoing inflammatory reactions, which is the topic of this artic

296 In contrast, persistent inflammatory reactions, which may be driven by infection

297 of action of neuropeptides and on immune and inflammatory reactions will likely yield new treatment o

298 , and the orchestration of acute and chronic inflammatory reactions within the arterial wall.

299 afts in four recipients with MC developed an inflammatory reaction without skin graft loss.

300 These results affirm that metabolic and inflammatory reactions yield electrophilic products that