ライフサイエンスコーパス: influenzae

1 to nontypeable Haemophilus influenzae (NT H. influenzae).

2 lococcus aureus, and potentially Haemophilus influenzae).

3 vaccine responses to tetanus and Haemophilus influenzae.

4 disease caused by H. haemolyticus and NT H. influenzae.

5 iae, Neisseria meningitidis, and Haemophilus influenzae.

6 ccus pneumoniae and non-typeable Haemophilus influenzae.

7 helial cells, facilitating persistence of H. influenzae.

8 ent meningitis, or with disease caused by H. influenzae.

9 egions, for all pathogens except Haemophilus influenzae.

10 causative pathogens are S. pneumoniae and H. influenzae.

11 was designed to detect all serogroups of H. influenzae.

12 included known pathogens such as Haemophilus influenzae.

13 nd Gram-negative bacteria and to Haemophilus influenzae.

14 fense against the human pathogen Haemophilus influenzae.

15 (123/273) S. pneumoniae, and 7% (19/273) H. influenzae.

16 gnificantly reduced cytokine responses to H. influenzae.

17 sence of pneumococcus, meningococcus, and H. influenzae.

18 s associated with qPCR levels of Haemophilus influenzae.

19 sseria meningitidis (13.7%), and Haemophilus influenzae (12.3%) were the predominant isolates from CS

20 ococcus pneumoniae (93 [73.8%]), Haemophilus influenzae (18 [14.3%]), and Neisseria meningitidis (15

21 pneumoniae (93 of 143, 65%) and Haemophilus influenzae (19 of 143, 13%).

22 owed by meningococcus (34.6%: 53/153) and H. influenzae (19.0%: 29/153).

23 st infections (67.3%: 66/98), followed by H. influenzae (23.5%: 23/98) and meningococcus (9.2%: 9/98)

24 Haemophilus influenzae (232/1670 [13.9%]) was the least prevalent or

25 a reduced relative abundance of Haemophilus influenzae (35.3% [5.5-91.6] vs 6.7% [0.8-74.8]; median

26 Of 9 cases of H. influenzae, 8 were type b (Hib) and 1 was type f.

27 Aspergillus species, 11%, 3.2 years; and H. influenzae, 9%, 3.1 years.

28 Pathogenic bacteria such as Haemophilus influenzae, a major cause of lower respiratory tract dis

29 discrimination of H. haemolyticus and NT H. influenzae, a testing scheme combining two targets (H. h

30 Pneumococcus, meningococcus, and H. influenzae accounted for 52.2%, 31.9%, and 16.0% of conf

31 Haemophilus influenzae also uses an enzyme, GlpQ, to hydrolyze ChoP

32 by Streptococcus pneumoniae and Haemophilus influenzae among children has been noted in numerous stu

33 (95% CI, 2.13-3.88) for all serotypes of H. influenzae and 2.90 (95% CI, 2.11-3.89) for unencapsulat

34 NT H. influenzae and H. haemolyticus are often misidentified b

35 h Gram-negative bacteria such as Haemophilus influenzae and Moraxella catarrhalis was found to be ass

36 H. influenzae and N. meningitidis accounted for 6.8% (5 of

37 s of the Gram-negative pathogens Haemophilus influenzae and Neisseria meningitidis We hypothesized th

38 rotype replacement may prevent changes in H. influenzae and S. aureus carriage among PCV7 recipients.

39 ratory burst and killing activity against H. influenzae and S. aureus compared to those transmigrated

40 nical interventions, including changes in H. influenzae and S. aureus disease incidence following pne

41 re high inoculum and pH 5.5 (no growth of H. influenzae and S. pneumoniae by BMD).

42 dy responses against nontypeable Haemophilus influenzae and S. pneumoniae, engendering protection aga

43 alization were positively associated with H. influenzae and Streptococcus and negatively associated w

44 with conjugate vaccines against Haemophilus influenzae and Streptococcus pneumoniae has virtually el

45 Haemophilus influenzae and Streptococcus pneumoniae were the commone

46 Haemophilus influenzae and Streptococcus pneumoniae were the main ag

47 Haemophilus influenzae and Streptococcus pneumoniae were the prevale

48 d and characterized IgA protease genes in H. influenzae and studied their expression and proteolytic

49 In the Gram-negative bacteria Haemophilus influenzae and Vibrio cholerae, the master regulator Sxy

50 machinery from the opportunistic pathogen H. influenzae (and the homologous enzymes from A. pleuropne

51 S aureus, 992 CoNS, 330 S pneumoniae, 357 H influenzae, and 389 P aeruginosa) were collected from 72

52 10 Streptococcus pneumoniae, 10 Haemophilus influenzae, and 5 Escherichia coli isolates by MIC and 3

53 CR for Streptococcus pneumoniae, Haemophilus influenzae, and Moraxella catarrhalis were performed on

54 teria (Streptococcus pneumoniae, Haemophilus influenzae, and Moraxella catarrhalis) were identified i

55 occus pneumoniae (pneumococcus), Haemophilus influenzae, and Neisseria meningitidis (meningococcus) w

56 occus pneumoniae (pneumococcus), Haemophilus influenzae, and Neisseria meningitidis (meningococcus).

57 ion of Streptococcus pneumoniae, Haemophilus influenzae, and Neisseria meningitidis, the 3 most commo

58 phosphocholine-modified LPS from Haemophilus influenzae, and phosphocholine-modified protein efficien

59 CoNS), Streptococcus pneumoniae, Haemophilus influenzae, and Pseudomonas aeruginosa.

60 aphylococcus aureus, Nontypeable Haemophilus influenzae, and Pseudomonas aeruginosa.

61 The fact that S. pneumoniae, H. influenzae, and S. aureus polymicrobial carriage pattern

62 udomonas aeruginosa, nontypeable Haemophilus influenzae, and Salmonella enterica serovar Typhi/Typhim

63 eumoniae, Moraxella catarrhalis, Haemophilus influenzae, and Staphylococcus aureus.

64 opathogenic E. coli, nontypeable Haemophilus influenzae, and Staphylococcus epidermidis Importantly,

65 Corynebacterium, Streptococcus, Haemophilus influenzae, and Staphylococcus.

66 pathogens Moraxella catarrhalis, Haemophilus influenzae, and Streptococcus pneumoniae, but not other

67 ainly by Neisseria meningitidis, Haemophilus influenzae, and Streptococcus pneumoniae, inflicts a sub

68 aused by Neisseria meningitidis, Haemophilus influenzae, and Streptococcus pneumoniae.

69 ofiles of children with RSV infection and H. influenzae- and Streptococcus-dominated microbiota were

70 mmunoglobulin (Ig)A proteases of Haemophilus influenzae are highly specific endopeptidases that cleav

71 eningitidis (meningococcus), and Haemophilus influenzae are major causes of this invasive disease.

72 us, Streptococcus pneumoniae and Haemophilus influenzae are the major causes of conjunctivitis.

73 H. parainfluenzae during coinfection with H. influenzae are topics for future work.

74 ruginosa, Staphylococcus aureus, Haemophilus influenzae, Aspergillus species, Streptococcus pneumonia

75 eningitidis (meningococcus), and Haemophilus influenzae, at a sentinel hospital within the capital ci

76 ATCC 49619 (disk and broth), and Haemophilus influenzae ATCC 49247 (disk and broth).

77 23, Escherichia coli ATCC 25922, Haemophilus influenzae ATCC 49247, and Streptococcus pneumoniae ATCC

78 neumoniae ATCC 49619, and 16 to 20 mm for H. influenzae ATCC 49247.

79 umoniae ATCC 49619, and 2 to 8 mug/ml for H. influenzae ATCC 49247.

80 49619, and 0.12 to 1 mug/ml for Haemophilus influenzae ATCC 49247.

81 England immunized with DTaP5/IPV/Haemophilus influenzae b (Hib-TT) vaccine at 2-3-4 months, 13-valent

82 healthy and presented with unencapsulated H. influenzae bacteremia.

83 lla catarrhalis and non-typeable Haemophilus influenzae, bacterial colonizers and pathogens in the re

84 irus, parainfluenza viruses, and Haemophilus influenzae being the most common.

85 e majority of serotyped meningococcus and H. influenzae belonged to meningococcus serogroup W (45.5%)

86 gainst PC-expressing nontypeable Haemophilus influenzae, but not PC-negative nontypeable Haemophilus

87 RV significantly impaired phagocytosis of H. influenzae by 23% in MDM (n = 37; P = 0.004) and 18% in

88 016 (an insertion sequence associated with H influenzae capsule genes).

89 resent the Arg160His mutation of Haemophilus influenzae carbonic anhydrase (HICA), which mimics the e

90 showed an apparent transient increase in H. influenzae carriage but no further significant differenc

91 increases in density of other species and H. influenzae carriage prevalence.

92 is, Streptococcus pneumoniae, or Haemophilus influenzae cases were confirmed and N. meningitidis/H. i

93 Pneumococcus, meningococcus, and Haemophilus influenzae cause a similar spectrum of infections in the

94 and in vivo in 169 independent strains of H. influenzae collected longitudinally over 10 years from a

95 re is evidence for an association between H. influenzae colonization density and H. influenzae-confir

96 90 (95% CI, 2.11-3.89) for unencapsulated H. influenzae compared with the background rate for pregnan

97 en H. influenzae colonization density and H. influenzae-confirmed pneumonia in children; the associat

98 Non-typeable Haemophilus influenzae contains an N(6)-adenine DNA-methyltransferas

99 The development of a rapid H. influenzae diagnostic assay that would allow for the imp

100 Invasive H. influenzae disease confirmed by positive culture from a

101 years with laboratory-confirmed invasive H. influenzae disease during 2009-2012, encompassing 45,215

102 ristics, and outcome of neonatal invasive H. influenzae disease in England and Wales over a 5-year pe

103 nhanced national surveillance of invasive H. influenzae disease in England and Wales.

104 responsible for the majority of invasive H. influenzae disease, and its prevalence has been markedly

105 susceptibility to invasive unencapsulated H. influenzae disease.

106 eonates had laboratory-confirmed invasive H. influenzae disease: 115 (97%) were NTHi, 2 were serotype

107 f IgA proteases are variably expressed by H. influenzae during infection of the human airways.

108 Using Haemophilus influenzae Eagan strains expressing well-characterized l

109 In the postvaccine era, nontypeable H. influenzae emerged as the most dominant group causing di

110 Haemophilus influenzae exclusively colonizes the human nasopharynx a

111 ved, because the Fur homolog from Hemophilus influenzae expressed in E. coli cells also reversibly bi

112 atives of a laboratory strain of Haemophilus influenzae expressing either surface-associated Cha1 or

113 our attention to bacteria, i.e., Haemophilus influenzae, expressing cell-surface adhesins including N

114 Unencapsulated Haemophilus influenzae frequently causes noninvasive upper respirato

115 ity to serve as biomarkers to distinguish H. influenzae from H. haemolyticus.

116 rapid in silico serotype prediction from H. influenzae genome sequences.

117 sity and distribution in 691 high-quality H. influenzae genomes from GenBank.

118 To survive and propagate in vivo, H. influenzae has evolved mechanisms for subverting this ho

119 owing interest in genomic epidemiology of H. influenzae Here we present hicap, a software tool for ra

120 onal lasso analysis, we selected Haemophilus influenzae (HI) and Mycoplasma penetrans (MP) for target

121 Haemophilus influenzae (Hi) causes respiratory tract infections and

122 occus pneumoniae ([Sp] 13%), and Haemophilus influenzae ([Hi] 2%).

123 catalytic activity of DapE from Haemophilus influenzae (HiDapE) and ArgE from Escherichia coli (EcAr

124 ing two targets (H. haemolyticus purT and H. influenzae hpd, encoding protein D lipoprotein) was also

125 iae was cultured in 33 episodes (51%) and H. influenzae in 11 episodes (17%).

126 cus aureus in 22% of samples and Haemophilus influenzae in 14%, and both a viral and bacterial target

127 on of S. pneumoniae, N. meningitidis, and H. influenzae in CSF, and that application of molecular dia

128 c acid diagnostics approaches that detect H. influenzae in RTIs have been described in the literature

129 s of the periplasmic domain from Haemophilus influenzae in which N- and C-terminal residues had been

130 of Streptococcus pneumoniae and Haemophilus influenzae in wild-type mice but not CD68.hMcl-1 transge

131 study, we found that nontypeable Haemophilus influenzae induces the association of Itch with Ndfip1.

132 N-Glc, to establish a connection between H. influenzae infection and MS.

133 The primary outcome was H. influenzae infection and the secondary outcomes were pre

134 Unencapsulated H. influenzae infection during the second half of pregnancy

135 rate after a lethal non-typeable Haemophilus influenzae infection in wild-type mice, but not in IRAK-

136 nta, the incidence of invasive nontypeable H influenzae infection increased significantly from 2017-2

137 1 women had laboratory-confirmed invasive H. influenzae infection, which included 144 (84.2%; 95% CI,

138 of IgA proteases in clinical settings of H. influenzae infection.

139 ssociated with a greater risk of invasive H. influenzae infection.

140 Novel mouse models of Chlamydia, Haemophilus influenzae, influenza, and respiratory syncytial virus r

141 Haemophilus influenzae is a Gram-negative human pathogen that reside

142 Haemophilus influenzae is a significant causative agent of respirato

143 Non-typeable Haemophilus influenzae is an opportunistic pathogen of the human upp

144 Nontypeable H influenzae is genetically diverse and clusters of infect

145 ammation induced by non-typeable Haemophilus influenzae is significantly attenuated in IRAK-M-deficie

146 A key virulence determinant of H. influenzae is the polysaccharide capsule, of which six s

147 onal S. aureus isolates and 25/92 (27.2%) H. influenzae isolates, which were more frequently discorda

148 In this study, we identified an H. influenzae lipoprotein having the ability to bind factor

149 specimens: Escherichia coli K1, Haemophilus influenzae, Listeria monocytogenes, Neisseria meningitid

150 enetic diversity (P = 0.026) and Haemophilus influenzae load (P < 0.0001).

151 olled asthma, azithromycin reduced airway H. influenzae load compared with placebo but did not change

152 l (n = 20), high gammaP:F ratio, increased H influenzae, low diversity measures and increased pro-inf

153 wn that the C-terminal domain of Haemophilus influenzae LpoA (HiLpoA) has a highly conserved, putativ

154 without protein D of nontypeable Haemophilus influenzae, M. catarrhalis has become a high-priority pa

155 Haemophilus influenzae meningitis fluctuated over the surveillance p

156 Hib caused 67% (2/3) of the H. influenzae meningitis isolates serotyped.

157 Annual incidence of H influenzae meningitis per 100,000 children decreased fro

158 pn), Neisseria meningitidis, and Haemophilus influenzae meningitis within the WHO African Region.

159 with bacterial coinfection with Haemophilus influenzae, Moraxella catarrhalis, or Streptococcus pneu

160 We assessed this association for Haemophilus influenzae, Moraxella catarrhalis, Staphylococcus aureus

161 ; 56%), S. pneumoniae (n = 1758; 40%), or H. influenzae (n = 180; 4%).

162 rus [n = 5], adenovirus [n = 5], Haemophilus influenzae [n = 5], and Streptococcus pneumoniae [n = 5]

163 ogens (Streptococcus pneumoniae, Haemophilus influenzae, Neisseria meningitidis, Mycoplasma pneumonia

164 ems in the major human pathogens Haemophilus influenzae, Neisseria meningitidis, Neisseria gonorrhoea

165 Emergence of H. influenzae nontypeable meningitis was observed after H.

166 s closely related to nontypeable Haemophilus influenzae (NT H. influenzae).

167 s a major adhesin of nontypeable Haemophilus influenzae (NTHi) and has long been investigated as a va

168 Biofilms formed by nontypeable Haemophilus influenzae (NTHI) are central to the chronicity, recurre

169 lus haemolyticus and nontypeable Haemophilus influenzae (NTHi) are closely related upper airway comme

170 occus pneumoniae and nontypeable Haemophilus influenzae (NTHi) are frequently implicated in complex o

171 Nontypeable Haemophilus influenzae (NTHI) are Gram-negative bacteria that coloni

172 commensal bacterium nontypeable Haemophilus influenzae (NTHI) can cause respiratory tract diseases t

173 and pneumococcal and nontypeable Haemophilus influenzae (NTHi) carriage were assessed prevaccination

174 h H. influenzae strain Rd and nontypeable H. influenzae (NTHi) clinical isolate NT127.

175 ons is important for nontypeable Haemophilus influenzae (NTHi) colonization in the airway mucosa.

176 Non-typeable Haemophilus influenzae (NTHi) contains the phase-variable methyltran

177 Nontypeable Haemophilus influenzae (NTHi) efficiently colonizes the human nasoph

178 Nontypeable Haemophilus influenzae (NTHI) forms biofilms in the middle ear durin

179 Nontypeable Haemophilus influenzae (NTHi) frequently causes noninvasive upper re

180 pneumoniae (Spn) and nontypeable Haemophilus influenzae (NTHi) in stringently defined otitis-prone (s

181 Invasive nontypeable Haemophilus influenzae (NTHi) infection among adults is typically as

182 Nontypeable Haemophilus influenzae (NTHi) initiates infection by colonizing the

183 Nontypeable Haemophilus influenzae (NTHI) is a commensal bacterial species of th

184 Nontypeable Haemophilus influenzae (NTHI) is a common commensal and opportunisti

185 Nontypeable Haemophilus influenzae (NTHi) is a Gram-negative, opportunistic path

186 Nontypeable Haemophilus influenzae (NTHI) is a leading cause of opportunistic in

187 Nontypeable Haemophilus influenzae (NTHi) is a major bacterial pathogen for OM.

188 Nontypeable Haemophilus influenzae (NTHi) is a major pathogen causing acute otit

189 Nontypeable Haemophilus influenzae (NTHi) is a pathogen known for being a freque

190 al colonization with nontypeable Haemophilus influenzae (NTHi) is a prerequisite for developing NTHi-

191 Nontypeable Haemophilus influenzae (NTHI) is the causative agent of multiple res

192 Nontypeable Haemophilus influenzae (NTHi) is the leading bacterial pathogen duri

193 Nontypeable Haemophilus influenzae (NTHi) is the primary cause of bacterially in

194 pe IV pilus (Tfp) of nontypeable Haemophilus influenzae (NTHI) mediates adherence, colonization, moti

195 ne the impact of the nontypeable Haemophilus influenzae (NTHI) ModA2 phasevarion on pathogenesis and

196 Pneumococci and nontypeable Haemophilus influenzae (NTHi) often cocolonize children.

197 Nontypeable Haemophilus influenzae (NTHi) persists in the airways in chronic obs

198 Nontypeable Haemophilus influenzae (NTHi) was selected as a model pathogenic spe

199 ted for pneumococcal, nontypable Haemophilus influenzae (NTHi), Moraxella catarrhalis, Streptococcus

200 In nontypeable Haemophilus influenzae (NTHi), the oligopeptide-binding protein (Opp

201 f biofilms formed by nontypeable Haemophilus influenzae (NTHI), those directed against a recombinant

202 pathogens, including nontypeable Haemophilus influenzae (NTHI), yet the reasons for this increased su

203 e commonly caused by nontypeable Haemophilus influenzae (NTHi).

204 g microbiota such as nontypeable Haemophilus influenzae (NTHi).

205 of ccl3(-/-)mice to nontypeable Haemophilus influenzae (NTHi).

206 ry pathogens such as nontypeable Haemophilus influenzae (NTHi).

207 rategies employed by nontypeable Haemophilus influenzae (NTHi).

208 nd colonization with nontypeable Haemophilus influenzae (NTHi).

209 wo major antigens of nontypeable Haemophilus influenzae (NTHi).

210 d with bacteria [eg, nontypeable Haemophilus influenzae (NTHi)] that cause pulmonary inflammation and

211 e for S. pneumoniae, N. meningitidis, and H. influenzae, only 10 were culture positive.

212 cytosis of fluorescently labeled Haemophilus influenzae or Streptococcus pneumoniae was assessed by f

213 nfluence total carriage of S. pneumoniae, H. influenzae, or S. aureus.

214 low concentrations (S. aureus, P < 0.001; H. influenzae, P < 0.0001) and in sputum-type specimens (S.

215 The H. influenzae pangenome has 2 alleles of IgA protease genes

216 The results showed that coinfection with H. influenzae promoted clearance of H. parainfluenzae from

217 ns: the aforementioned KLH and rTTHC; the H. influenzae protein D (HiD); and the cross-reactive mater

218 valent pneumococcal non-typeable Haemophilus influenzae protein D conjugate vaccine (PCV10) was intro

219 Haemophilus influenzae protein F (PF) is an important virulence fact

220 but not PC-negative nontypeable Haemophilus influenzae, relative to wild-type mice.

221 This novel interaction is important for H. influenzae resistance against complement activation and

222 s for N. meningitidis, S. pneumoniae, and H. influenzae, respectively, were 7.5, 2.5, and 0.3.

223 00% specificity for the identification of H. influenzae, respectively.

224 ndent transcription factor that modulates H. influenzae response to formaldehyde, with two cysteine r

225 respiratory pathogen nontypeable Haemophilus influenzae resulted in a marked increase in expression o

226 allenge of Trim29(-/-) mice with Haemophilus influenzae resulted in lethal lung inflammation due to m

227 umoniae, Neisseria meningitidis, Haemophilus influenzae, S suis) and O tsutsugamushi, Rickettsia typh

228 s (GBS), Listeria monocytogenes, Haemophilus influenzae, S. aureus, Klebsiella spp. and non-typhoidal

229 Incidence of invasive disease due to H. influenzae serotype a (Hia) increased an average of 13%

230 in contrast to Hib, infections caused by H. influenzae serotype f (Hif) are emerging.

231 lthy adult patient, secondary to Haemophilus influenzae serotype f infection, and we review literatur

232 s.Measurements and Main Results: Haemophilus influenzae, Staphylococcus aureus, Pseudomonas aeruginos

233 ential factor in serum resistance of both H. influenzae strain Rd and nontypeable H. influenzae (NTHi

234 recent years a resurgence of encapsulated H. influenzae strains has also been observed, most notably

235 The most prevalent meningococcal and H. influenzae strains were serogroup W and serotype b, resp

236 cterized by enrichment of either Haemophilus influenzae, Streptococcus, Corynebacterium, Moraxella, o

237 glucocorticoids and non-typeable Haemophilus influenzae synergistically upregulate IRAK-M expression

238 ned in this work highlight the ability of H. influenzae to utilize a single protein to perform multip

239 H. influenzae TolR(62-133) is a symmetrical dimer with a la

240 detection of 2 cases of invasive Haemophilus influenzae type a (Hia) disease in Italy.

241 to meningococcus serogroup W (45.5%) and H. influenzae type b (54.5%), respectively.

242 f all preterm groups, except for Haemophilus influenzae type b (88.1%).

243 rtussis, hepatitis B, polio, and Haemophilus influenzae type b (DTaP-IPV-Hib) and pneumococcal vaccin

244 A conjugate vaccine containing Haemophilus influenzae type b (Hib) and group C meningococcal polysa

245 Encapsulated H. influenzae type b (Hib) and type f (Hif) are the most co

246 pneumoniae (S. pneumoniae), and Haemophilus influenzae type b (Hib) are three most common pathogens

247 to Streptococcus pneumoniae and Haemophilus influenzae type b (Hib) between 2000 and 2015.

248 Haemophilus influenzae type b (Hib) conjugate vaccine, delivered as

249 The incidence of invasive Haemophilus influenzae type b (Hib) disease has significantly decrea

250 Haemophilus influenzae type b (Hib) vaccine and the 13-valent pneumo

251 H. influenzae type b (Hib) was historically responsible for

252 ) polysaccharides extracted from Haemophilus influenzae type b (Hib), and the corresponding glycoconj

253 00 from influenza, and 7200 from Haemophilus influenzae type b and 24,700 diarrheal deaths from rotav

254 fants in both groups received the combined H influenzae type b and capsular group C Neisseria meningi

255 predominant invasive pathogen as Haemophilus influenzae type b and pneumococcal vaccine use in Mali h

256 Anti-H influenzae type b anti-polyribosylribitol phosphate IgG

257 s varied between 83.0% and 100%, Haemophilus influenzae type b between 34.7% and 46.2% (40.6% among a

258 Nasopharyngeal H influenzae type b carriage was detected in one (0.2%) of

259 llular pertussis-inactived polio-Haemophilus influenzae type b combined vaccine (DTaP-IPV-Hib) at 2,

260 ysaccharide vaccine (PsACWY); or Haemophilus influenzae type b conjugate vaccine (Hib-TT).

261 The incidence of invasive H influenzae type b disease in children younger than 5 yea

262 ation (2010-14), only one case of invasive H influenzae type b disease was detected in a child younge

263 ficant and sustained reduction in invasive H influenzae type b disease.

264 We analysed sterile site cultures for H influenzae type b from children (aged </=12 years) admit

265 cines, pneumococcal vaccine serotypes and H. influenzae type b remain associated with bacterial menin

266 in 1994, after the introduction of routine H influenzae type b vaccination.

267 pertussis, measles, rubella, and Haemophilus influenzae type b vaccine antigens were comparable betwe

268 2, meningococcal serogroup A and Haemophilus influenzae type b vaccine each in 1 patient).

269 nontypeable meningitis was observed after H. influenzae type b vaccine introduction.

270 pertussis-inactivated poliovirus/Haemophilus influenzae type b vaccine; age 6/10/ 14 weeks) and 13-va

271 cell pertussis; hepatitis B; and Haemophilus influenzae type b) and pneumococcal vaccine.

272 ertussis, hepatitis B virus, and Haemophilus influenzae type b), yellow fever, measles, and tuberculo

273 (diphtheria, tetanus, pertussis, Haemophilus influenzae type b, and hepatitis B) at 6, 10, and 14 wee

274 mococcus, group B Streptococcus, Haemophilus influenzae type b, and meningococcus vaccines.

275 tetanus, pertussis, hepatitis B, Haemophilus influenzae type b, Streptococcus pneumoniae, rotavirus,

276 y series and booster, except for Haemophilus influenzae type b.

277 he pathogens Vibrio cholerae and Haemophilus influenzae use tripartite ATP-independent periplasmic tr

278 ected, and 36 isolates were identified as H. influenzae using a gold standard methodology that combin

279 P6 lipoprotein from nontypeable Haemophilus influenzae, using 17-HDHA and aspirin-triggered-resolvin

280 solvent to ligand binding in the Haemophilus influenzae virulence protein SiaP.

281 cid-specific SBP, SiaP, from the Haemophilus influenzae virulence-related SiaPQM TRAP transporter.

282 nst an efflux-negative strain of Haemophilus influenzae was 4- to 8-fold higher, the combined improve

283 Phagocytosis of H. influenzae was also impaired by poly I:C but not IFN-bet

284 ction of pneumococcus, meningococcus, and H. influenzae was confirmed through microbiological techniq

285 whereas resistance among P aeruginosa and H influenzae was low against the antibiotics tested.

286 Regardless of rhinovirus status, H influenzae was not associated with respiratory symptoms.

287 eningitidis (meningococcus), and Haemophilus influenzae was performed by microbiological culture and/

288 The IL-10 response to H. influenzae was significantly impaired by poly I:C, IFN-b

289 Haemophilus influenzae was unencapsulated in all 10 episodes with kn

290 Working with the bacterium Haemophilus influenzae, we found that MolBC-A functions as a low aff

291 enomic analysis of H. haemolyticus and NT H. influenzae, we identified genes unique to H. haemolyticu

292 tococcus pneumoniae, N. meningitidis, and H. influenzae were done.

293 ae, Entrobacter species, K. pnemoniae and H. influenzae were each accounted 6.5% isolation rate.

294 Higher densities of H. influenzae were observed in both microbiologically confi

295 cases were confirmed and N. meningitidis/H. influenzae were serogrouped/serotyped by real-time polym

296 d Corynebacterium propinquum and Haemophilus influenzae were significantly more abundant in control s

297 expressed in nontypeable (unencapsulated) H. influenzae, which did not bind FH, an increased FH affin

298 nfluenzae and its close relative Haemophilus influenzae, which is also commonly carried within the sa

299 s with Staphylococcus aureus and Haemophilus influenzae, with later emergence of Pseudomonas aerugino

300 A 5.9 log10 copies/mL density cutoff for H. influenzae yielded 86% sensitivity and 77% specificity f