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1 ures incompatible with effective delivery of inhalational anesthetics.
2 lar concentration is central to the study of inhalational anesthetics.
3 ose receptors is less certain in the case of inhalational anesthetics.
4 the PDZ domain as a new molecular target for inhalational anesthetics.
5                            Administration of inhalational anesthetics.
6                           We now report that inhalational anesthetics affect gene expression of nitri
7 advantages for any of the commonly available inhalational anesthetic agents and each can be used for
8                                              Inhalational anesthetic agents have also been shown to r
9                Xenon and dichloromethane are inhalational anesthetic agents whose binding to myoglobi
10 ative, analgesics, benzodiazepines, opioids, inhalational anesthetic agents, nitrous oxide, ketamine,
11 ous oxide (N2O, laughing gas), a widely used inhalational anesthetic and drug of abuse.
12 ersing the effects of some anesthetic drugs (inhalational anesthetics and muscle relaxants) are descr
13          This is a novel interaction between inhalational anesthetics and the NO signaling pathway an
14                                              Inhalational anesthetics are bronchodilators with immuno
15                           They also received inhalational anesthetics because of refractory bronchoco
16                                  To identify inhalational anesthetic binding domains in a ligand-gate
17                                 To determine inhalational anesthetic binding domains on a ligand-gate
18                        Postconditioning with inhalational anesthetics can reduce ischemia-reperfusion
19            At surgical depths of anesthesia, inhalational anesthetics cause a loss of motor response
20                     These data indicate that inhalational anesthetics cause activation of RTN neurons
21                Currently, it is thought that inhalational anesthetics cause anesthesia by binding to
22  during surgery (derived from mean end-tidal inhalational anesthetic concentrations).
23 esthetized with a virtually nondefluorinated inhalational anesthetic (desflurane) or with a nonfluori
24                                              Inhalational anesthetic dose increase and reduced risk o
25                                         High inhalational anesthetic dose of 1.20 (1.13-1.30) (median
26         We sought to determine the effect of inhalational anesthetic dose on risk of severe postopera
27                           Additionally, high inhalational anesthetic dose was associated with lower 3
28 e that clinically relevant concentrations of inhalational anesthetics dose-dependently and specifical
29                 Intraoperative use of higher inhalational anesthetic doses is strongly associated wit
30          Median effective dose equivalent of inhalational anesthetics during surgery (derived from me
31  can be utilized to probe the binding of the inhalational anesthetic halothane to an anesthetic-bindi
32               Clinical concentrations of the inhalational anesthetic, halothane (1 rat MAC, 1.2 vol.%
33                                              Inhalational anesthetics have been shown to inhibit the
34                            Increasing use of inhalational anesthetics in the ICU underscores the need
35 sting differences in the binding domains for inhalational anesthetics in the nAChR.
36 amuscular sedative was given, followed by an inhalational anesthetic induction and mechanical ventila
37 sthesia is indicated for procedures in which inhalational anesthetics may not be safely or effectivel
38 w that clinically relevant concentrations of inhalational anesthetics modulate neuronal Ih and the co
39      A better understanding of the effect of inhalational anesthetics on fetal cardiac function and s
40              The potential adverse impact of inhalational anesthetics on the developing brain was hig
41 ulate based on these data that sedation with inhalational anesthetics outside of the operating room m
42             Isoflurane, the most widely used inhalational anesthetic, releases inorganic fluoride dur
43                The molecular pharmacology of inhalational anesthetics remains poorly understood.
44 iments were performed with the commonly used inhalational anesthetic sevoflurane.
45  in experimental traumatic brain injury with inhalational anesthetics, these results indicate that th
46 ectroscopic probe to study the binding of an inhalational anesthetic to a model membrane protein.
47 the native RTN current (i.e., suppression by inhalational anesthetics, weak rectification, inhibition