ライフサイエンスコーパス: inhale

1 d the volume and duration of cigarette puffs inhaled.

2 rolled, phase 3 study comprising two trials (INHALE 1 and INHALE 2) done in 153 hospital intensive-ca

3 3 study comprising two trials (INHALE 1 and INHALE 2) done in 153 hospital intensive-care units in 2

4 y and 725 were randomly assigned to Amikacin Inhale (362 patients) or aerosolised placebo (363 patien

5 icantly improved by treating recipients with inhaled 50% oxygen, in conjunction with prevascularizati

6 stem that confirmed enrolment eligibility to inhaled 7% hypertonic saline or 0.9% isotonic saline neb

7 of the CO boost on FMD, hookah smokers (n=8) inhaled a 0.1% CO gas mixture to approximate their CO bo

8 ne JAK inhibitors with profiles suitable for inhaled administration.

9 nsidered, and deposition distribution of the inhaled aerosols on the internal airway walls was quanti

10 sitization, resulting in airway responses to inhaled Ags.

11 decreasing the partial pressure of oxygen in inhaled air stepwisely (pO(2); 21.25 kPa (0 k), 16.42 kP

12 ne) inhibited the cardiac reflexes evoked by inhaled AITC but not injected AITC.

13 Conversely, inhaled AITC in conscious SH rats evoked complex brady-t

14 treatment with an oral corticosteroid and 3 inhaled albuterol and ipratropium treatments.

15 cebo-controlled trial testing the effects of inhaled albuterol on resting and exercise hemodynamics i

16 9 with airway hyperresponsiveness) underwent inhaled allergen challenge after 2-hour exposures to DE,

17 DM allergen, particularly when the amount of inhaled allergen is low, by expanding allergen-specific

18 ulates the balance of T(H) cell responses to inhaled allergen.

19 dministered at 30 minutes and 24 hours after inhaled allergen/diluent challenge.

20 immunoglobulin E (IgE)-mediated reactions to inhaled allergens and is one of the most common chronic

21 The upper airways present a barrier to inhaled allergens and microbes, which alter immune respo

22 sease caused by aberrant immune responses to inhaled allergens, which leads to airway hyperresponsive

23 ergic asthma is mediated by Th2 responses to inhaled allergens.

24 Inhaled allyl isothiocyanate (AITC) evoked atropine-sens

25 Our findings do not support use of inhaled amikacin adjunctive to standard-of-care intraven

26 tential influence of the anaesthetic method (inhaled anaesthetics versus total-intravenous anaesthesi

27 ality, decline in FEV1, and response to both inhaled and systemic corticosteroids.

28 ults establish a membrane-mediated target of inhaled anesthesia and suggest PA helps set thresholds o

29 rrent study investigated whether PreCon with inhaled anesthetic sevoflurane (SF-PreCon) remains cardi

30 Here we show that inhaled anesthetics (chloroform and isoflurane) activate

31 Inhaled anesthetics are a chemically diverse collection

32 nuclei, but causal investigations of potent inhaled anesthetics have not been conducted.

33 Bacterial eradication was increased with inhaled antibiotic therapy (odds ratio [OR] 3.36, 1.63 t

34 Rationale: The principal underlying inhaled antibiotic treatment in bronchiectasis is that a

35 lysis of all randomised controlled trials of inhaled-antibiotic use in adult patients with bronchiect

36 xacerbation was significantly prolonged with inhaled antibiotics (hazard ratio 0.83, 0.69 to 0.99; p=

37 Although inhaled antibiotics are conditionally recommended for lo

38 Inhaled antibiotics are well tolerated, reduce bacterial

39 e aimed to assess the efficacy and safety of inhaled antibiotics for the long-term treatment of adult

40 Associations Innovative Medicines Initiative inhaled Antibiotics in Bronchiectasis and Cystic Fibrosi

41 e is insufficient evidence to support use of inhaled antibiotics in patients with bronchiectasis not

42 Although use of inhaled antibiotics is the standard of care in cystic fi

43 Inhaled antibiotics significantly reduced exacerbation f

44 of colony forming units per g of sputum with inhaled antibiotics was -2.32 log units (95% CI -3.20 to

45 Randomised controlled trials of inhaled antibiotics were included if the patients were a

46 me, and test the hypothesis that response to inhaled antibiotics would be predicted by baseline bacte

47 nchiectasis and optimal outcome measures for inhaled antibiotics.

48 cells and impaired immunologic tolerance to inhaled antigens.

49 cacy of the combination drug device Amikacin Inhale as an adjunctive therapy to intravenous standard-

50 rs (95% CI, -0.44 to 0.13), whereas users of inhaled aztreonam lysine and azithromycin experienced a

51 lusions: Improvement of quality of life with inhaled aztreonam was only evident in patients with high

52 a post hoc analysis of a randomized trial of inhaled aztreonam.

53 was protective against a lethal challenge of inhaled bacillus spores at 3 and 28 weeks after vaccinat

54 Inhalational anthrax, a disease caused by inhaling Bacillus anthracis spores, leads to respiratory

55 xed, and, with high efficiency, phagocytosed inhaled bacterial pathogens such as P. aeruginosa and S.

56 We hypothesized that the inhaled beta-adrenergic agonist albuterol would improve

57 at children using inhaled corticosteroids or inhaled beta-agonists might be at increased risk of type

58 Dispensed inhaled corticosteroids and inhaled beta-agonists were associated with an increased

59 The mainstay of treatment for COPD is inhaled bronchodilators, whereas the role of inhaled cor

60 roids were randomised to receive once daily, inhaled budesonide 400 mug (those aged <11 years 200 mug

61 The mean (+/-SD) dose of inhaled budesonide was 107+/-109 mug per day in the bude

62 Early treatment with inhaled budesonide/formoterol in patients at risk for ac

63 hesia reduced the cardiac reflexes evoked by inhaled but not injected AITC.

64 ical for Th2 cell development in the lung to inhaled but not systemically delivered allergens but is

65 cles (3) that survive in the air and (4) are inhaled by a susceptible individual (5) who may become i

66 panic white majority, but disproportionately inhaled by black and Hispanic minorities.

67 ages and causes severe bronchopneumonia when inhaled by susceptible foals.

68 m that generate a condensation aerosol to be inhaled by the user.

69 Visits attributable to inhaled cannabis are more frequent than those attributab

70 Visits attributable to inhaled cannabis were more likely to be for cannabinoid

71 crossover study comparing cough responses to inhaled capsaicin in patients with mild atopic asthma af

72 lished nongenetic, CF-like sheep model, ewes inhaled CFTR(inh)172 and neutrophil elastase for 3 days,

73 olonizing airways is consistently exposed to inhaled cigarette smoke.

74 lticenter, double-blinded, clinical trial of inhaled CO in patients with idiopathic pulmonary fibrosi

75 The ventilatory response to inhaled CO(2) of mice was markedly decreased in vivo aft

76 To enhance synaptic transmission, mice inhaled CO2 to induce an acidosis and activate acid sens

77 rved blunting of the ventilatory response to inhaled CO2Tac1-Pet1 neurons thus appear distinct from a

78 ended to deliver nicotine to the user via an inhaled complex aerosol formed by heating a liquid compo

79 6J male mice that were exposed to real-world inhaled, concentrated PM2.5 (~10 times ambient levels/~6

80 Extra-fine particle formulations of inhaled corticosteroid (ICS) are associated with improve

81 Although inhaled corticosteroid (ICS) medication is considered th

82 c asthma and the impact on the microbiota of inhaled corticosteroid (ICS) treatment administered to I

83 cerbations.Objectives: To understand whether inhaled corticosteroid (ICS) withdrawal affected IMPACT

84 endronate can reduce LABA-associated LOBP in inhaled corticosteroid (ICS)-treated patients.

85 nt guidelines were updated to recommend that inhaled corticosteroid (ICS)/long-acting beta (2)-adreno

86 The combination drug of inhaled corticosteroid (ICS)/long-acting beta2 agonist i

87 e previous year, and previously treated with inhaled corticosteroid (TRIMARAN: medium dose; TRIGGER:

88 xhaled nitric oxide (FeNO) in 17 studies for inhaled corticosteroid adherence.

89 ing beta(2) agonist therapy to a maintenance inhaled corticosteroid dose that causes the same magnitu

90 However, a fall in FeNO following supervised inhaled corticosteroid dosing could indicate previous po

91 nochemical domains of powders containing the inhaled corticosteroid fluticasone propionate and long-a

92 with two long-acting bronchodilators and an inhaled corticosteroid in chronic obstructive pulmonary

93 al corticosteroids and omalizumab to regular inhaled corticosteroid inhalation failed to relieve symp

94 nistic insight as to how adding a LABA to an inhaled corticosteroid may improve clinical outcomes in

95 on of a long-acting muscarinic antagonist to inhaled corticosteroid plus long-acting beta(2)-agonist

96 School-supervised use of a once-daily inhaled corticosteroid regimen (supervised therapy) can

97 the absence oral corticosteroid use while on inhaled corticosteroid therapy.

98 lation of IL12B and CORT are associated with inhaled corticosteroid treatment response in persistent

99 ious exacerbation frequency, smoking status, inhaled corticosteroid use at baseline, body-mass index

100 Secondary outcomes included inhaled corticosteroid use, outpatient physician's offic

101 ospitalization associated with asthma and/or inhaled corticosteroid use.

102 oportion of participants in whom the dose of inhaled corticosteroid was reduced halfway through the t

103 the proportion of participants whose dose of inhaled corticosteroid was reduced, or the cumulative fl

104 bination of beclometasone dipropionate (BDP; inhaled corticosteroid), formoterol fumarate (FF; long-a

105 d only if there is a progressive increase in inhaled corticosteroid/long-acting beta(2) agonist thera

106 FE vs. 30% IE vs. 13% WNE, P < .001) and use inhaled corticosteroids (77% FE vs. 15% IE vs. 18% WNE,

107 mechanisms underlying hyperinflation and how inhaled corticosteroids (ICS) affect this important aspe

108 e of exacerbations with therapies containing inhaled corticosteroids (ICS) and baseline blood eosinop

109 Inhaled corticosteroids (ICS) are a mainstay of treatmen

110 Rationale: Inhaled corticosteroids (ICS) are key treatments for con

111 Inhaled corticosteroids (ICS) are the most widely prescr

112 h difficult-to-control asthma who respond to inhaled corticosteroids (ICS) from refractory asthma is

113 Daily dose of inhaled corticosteroids (ICS) inversely correlated with

114 Increasing the dose of inhaled corticosteroids (ICS) is commonly used at early

115 ic obstructive pulmonary disease (COPD) with inhaled corticosteroids (ICS) is controversial, because

116 logy of "low," "medium," and "high" doses of inhaled corticosteroids (ICS) to define daily maintenanc

117 Use of inhaled corticosteroids (ICS) was associated with lower

118 In patients on high-dose inhaled corticosteroids (ICS) with type 2-high asthma (s

119 ase control, even while taking high doses of inhaled corticosteroids (ICS).

120 mendation for the use of triple therapy with inhaled corticosteroids (ICS)/LABA/LAMA over dual therap

121 Inhaled corticosteroids (ICSs) are widely used in COPD,

122 Variability in response to inhaled corticosteroids (ICSs) can result in less than o

123 s), while anti-inflammatory maintenance with inhaled corticosteroids (ICSs) has been reserved for pat

124 dynamic assessment of the systemic effect of inhaled corticosteroids (ICSs) is often done by measurin

125 sm of asthma in patients receiving high-dose inhaled corticosteroids (ICSs) is uncertain.

126 of higher-level medication: medium/high-dose inhaled corticosteroids (ICSs) or ICSs + add-on medicati

127 ed, including 208 (44%) prescribed high-dose inhaled corticosteroids and 122 (31%) with severe asthma

128 Early treatment with inhaled corticosteroids and beta agonists may reduce pro

129 Dispensed inhaled corticosteroids and inhaled beta-agonists were a

130 r to enrollment, despite receiving high-dose inhaled corticosteroids and other controller(s), plus om

131 th asthma aged 6 to 16 years taking low-dose inhaled corticosteroids and with serum 25-hydroxyvitamin

132 While bronchodilators and inhaled corticosteroids are the mainstay of asthma treat

133 The ongoing use of inhaled corticosteroids did not increase the risk of hos

134 safety of long-acting beta-agonists added to inhaled corticosteroids for the treatment of persistent

135 with dyspnea and cough had been treated with inhaled corticosteroids for X-15 years, but her symptoms

136 mportantly, investigators have reported that inhaled corticosteroids had a limited effect on airway i

137 Inhaled corticosteroids have been the foundation for ast

138 Inhaled corticosteroids have substantial risks, includin

139 They consider the indications for inhaled corticosteroids in COPD, when inhaled corticoste

140 might be important predictors of response to inhaled corticosteroids in preschool children, these rel

141 A methylation was analysed in individuals on inhaled corticosteroids in three independent and ethnica

142 Response to inhaled corticosteroids is highly variable, and the asso

143 inhaled bronchodilators, whereas the role of inhaled corticosteroids is less clear.

144 These findings suggest that children using inhaled corticosteroids or inhaled beta-agonists might b

145 previous year despite dual inhaled therapy (inhaled corticosteroids plus long-acting beta(2)-agonist

146 inic antagonists) or triple inhaled therapy (inhaled corticosteroids plus long-acting beta(2)-agonist

147 that remains uncontrolled despite high-dose inhaled corticosteroids plus other controller medication

148 ns for inhaled corticosteroids in COPD, when inhaled corticosteroids should be withdrawn, and what ot

149 Inhaled corticosteroids were measured directly in the bl

150 Similarly, the use of inhaled corticosteroids with or without systemic cortico

151 practice (ie, inhaled salbutamol, or oral or inhaled corticosteroids) for infants with bronchiolitis

152 nd diffuse panbronchiolitis and treated with inhaled corticosteroids, a long-acting beta agonist, and

153 of long-acting maintenance bronchodilators, inhaled corticosteroids, and pulmonary rehabilitation de

154 magnesium sulfate, anti-inflammatory agents, inhaled corticosteroids, and short-acting bronchodilator

155 lergic inflammation that was unresponsive to inhaled corticosteroids, but responsive to systemic cort

156 improvement despite treatment with high dose inhaled corticosteroids, oral corticosteroids and azathi

157 acerbations may benefit from the addition of inhaled corticosteroids, particularly those with elevate

158 st, and 5.6% (3.1-9.9) had been treated with inhaled corticosteroids.

159 may partly explain their reduced response to inhaled corticosteroids.

160 r rapid relief of symptoms, and daily use of inhaled corticosteroids.

161 e asthma that are treated with high doses of inhaled corticosteroids.

162 Inhaled delivery of IVT-mRNA has not yet been demonstrat

163 mediately following a positive diagnosis via inhaled delivery of mAbs with vibrating mesh nebulizers

164 The current CF therapy relies on inhaled deoxyribonuclease and hypertonic saline but does

165 Lung inhaled DEPs increase susceptibility to pneumococcal dis

166 dy was to evaluate methods of estimating the inhaled dose of air pollution and understand variability

167 SARS-CoV2 is transmitted through inhaled droplets and aerosol particles, thus posing an i

168 t may influence the pulmonary disposition of inhaled drugs and contribute to variability in therapeut

169 which may impact the localized metabolism of inhaled drugs and tobacco smoke procarcinogens.

170 open novel effective delivery strategies for inhaled drugs.

171 y epithelium forms the interface between the inhaled environment and the lung.

172 e host from the potential harming effects of inhaled environmental particles and to guarantee mainten

173 l mucus velocity and mucociliary clearance), inhaled ETX001 was able to accelerate clearance both whe

174 We demonstrate that inhaled exposure to DEPs disrupts asymptomatic nasophary

175 ing an inhalation simulator we measured peak inhaled flow (PIF), peak inhaled pressure (PIP) and amou

176 Inhaled flows at pressure of -15cmH(2)O were 14L/min in

177 cts with mild atopic asthma before and after inhaled fluticasone treatment, 18 atopic subjects withou

178 pattern recognition receptors to respond to inhaled foreign substances and pathogens.

179 ed the addition of a LABA (salmeterol) to an inhaled glucocorticoid (fluticasone propionate), a step-

180 or response to an increase in the dose of an inhaled glucocorticoid and almost half had a superior re

181 r a clinically directive trial to compare an inhaled glucocorticoid with other treatments in patients

182 persistent asthma to receive mometasone (an inhaled glucocorticoid), tiotropium (a long-acting musca

183 The increased dose of inhaled glucocorticoids was associated with a decrease i

184 at was inadequately controlled with low-dose inhaled glucocorticoids.

185 ng beta-agonists and medium-to-high doses of inhaled glucocorticoids.

186 508 patients (255 in the Amikacin Inhale group and 253 in the placebo group) were assessed

187 one dose of study drug (354 in the Amikacin Inhale group and 358 in the placebo group), although one

188 survival: 191 (75%) patients in the Amikacin Inhale group versus 196 (77%) patients in the placebo gr

189 t (295 [84%] of 353 patients in the Amikacin Inhale group vs 303 [84%] of 359 patients in the placebo

190 These studies highlight the potential of inhaled GSK3008348 as an anti-fibrotic therapy.

191 ajor part of the adaptive immune response to inhaled HDM allergen, particularly when the amount of in

192 typical and pathogenic IFN-gamma response to inhaled HDM.

193 Repeat dosing of inhaled hPBAE-mRNA generates consistent protein producti

194 Though the average human inhales hundreds of conidia daily, A. fumigatus invasive

195 Background MRI with inhaled hyperpolarized helium 3 ((3)He) allows for funct

196 Inhaled hypertonic saline (HTS) treatment is used to imp

197 Inhaled hypertonic saline enhances mucociliary clearance

198 Inhaled hypertonic saline improved the LCI(2.5) in child

199 We aimed to assess the effect of inhaled hypertonic saline on the lung clearance index (L

200 To date, studies of inhaled IFN-beta treatment have not demonstrated a signi

201 ntial subgroups for further investigation of inhaled IFN-beta1a.

202 a pivotal role in protecting the lungs from inhaled infectious agents.

203 ted water supplies allows the bacteria to be inhaled into the human lung, where L. pneumophila can be

204 sive pulmonary aspergillosis when spores are inhaled into the respiratory tract and invade airway or

205 genicity varies greatly between ingested and inhaled invertebrate sources.

206 thelium is the first body surface to contact inhaled irritants and report danger.

207 Anaesthesia (inhaled isoflurane) inhibited the cardiac reflexes evoke

208 , aerosol particulates are highly toxic when inhaled, leading to millions of premature deaths per yea

209 In 2 studies, AGMs were challenged with inhaled lethal doses of Yersinia pestis.

210 We investigated the safety and efficacy of inhaled liposomal ciprofloxacin (ARD-3150) in two phase

211 lammation and acute neutrophilic response to inhaled LPS challenge in volunteers with asthma.

212 of neutrophilic inflammation in response to inhaled LPS, with additional control mechanisms arising

213 Inhaled LTD(4) blocked LTC(4)-mediated potentiation of o

214 ses, including airway hyperresponsiveness to inhaled methacholine, were assessed.

215 ing airways is the primary tissue exposed to inhaled microorganisms, allergens and pollutants.

216 Humans inhale mold conidia daily and typically experience lifel

217 iers of neutrophil effector activity against inhaled mold conidia.

218 in the opposite direction (ie, absorption of inhaled molecules) has not been increased in patients wi

219 People with higher OC exposure are likely inhaling more fresh OC2 and OC3, since secondary OC4 and

220 ere not driven by potential confounds of pre-inhale motor preparation or power changes.

221 Boosting with aerosol-inhaled MVA85A enhanced the intradermal primed responses

222 For decades, inhaled N-acetylcysteine (NAC) has been used as a mucoly

223 ly influence the pharmacokinetic behavior of inhaled nanoparticle systems and their payloads.

224 Finally, the feasibility of developing an inhaled nanoparticle-in-microparticle powder formulation

225 c assessment and were then randomized 1:1 to inhaled, nebulized albuterol or placebo.

226 ith longer, twice-daily regimens of oral and inhaled neuraminidase inhibitors.

227 Pregnant rats exposed to episodic inhaled nicotine via a novel lung alveolar region-target

228 smokers underwent 3.0-T MRI before and after inhaling nicotine-free e-cigarette aerosol.

229 Conclusion Inhaling nicotine-free electronic cigarette aerosol tran

230 ous neuromuscular blockade, corticosteroids, inhaled nitric oxide (iNO), prone positioning, high-freq

231 Placebo (nitrogen) or inhaled nitric oxide initiated at 20 ppm was decreased t

232 or use, extracorporeal membrane oxygenation, inhaled nitric oxide use, infection, estimated glomerula

233 Inhaled nitric oxide, initiated at 20 ppm on postnatal d

234 ntilation, paralysis, prone positioning, and inhaled nitric oxide.

235 ns, extracorporeal membrane oxygenation, and inhaled nitric oxide; Pao2:Fio2 ratio measured daily fro

236 rther increase exhaled NO concentration over inhaled nitrite alone in pigs exposed to alveolar hypoxi

237 ogy for treatment of health conditions where inhaled NO is beneficial, such as pulmonary hypertension

238 disease burdens worldwide and are caused by inhaled noxious agents including tobacco smoke.

239 ed to one tumor-bearing lung synergizes with inhaled NP-cGAMP, eliciting systemic anticancer immunity

240 Two inhales of FP/FM (50/5mug) at one time, twice daily were

241 ross cell types and can cause rapid death if inhaled or ingested.

242 current smoking, body mass index, diabetes, inhaled or oral corticosteroid use, study site and clini

243 in some asthmatics, even those on high-dose inhaled or oral corticosteroids.

244 However, inhaled or systemic corticosteroids are ineffective trea

245 creased duration of action commensurate with inhaled or topical delivery resulted in potent pan-JAK i

246 system to receive 400 mg amikacin (Amikacin Inhale) or saline placebo, both of which were aerosolise

247 Inhaled oxidative toxicants present in ambient air cause

248 as a limiting factor for bioaccessibility of inhaled particulate NPAHs and OPAHs, without considerabl

249 vements in the accuracy and precision of the inhaled pathogen dose were achieved through enhanced cor

250 ing potent and rapid protection against this inhaled pathogen.

251 the primary physical airway defense against inhaled pathogens and irritants.

252 ese cells play an important role in clearing inhaled pathogens and regulating the inflammatory enviro

253 For protection from inhaled pathogens many strategies have evolved in the ai

254 ght to determine whether mucosal exposure to inhaled pathogens stimulates a defensive swarm of microb

255 at cystic fibrosis airways do not respond to inhaled pathogens, thus favoring infection and inflammat

256 among the first immune cells that respond to inhaled pathogens.

257 fluid homeostasis and helps flush mucus and inhaled pathogens/toxicants out of the lung.

258 mental adjuvants to promote sensitization to inhaled peanut in mice.

259 ind, randomized controlled trial, once-daily inhaled placebo, fluticasone furoate (FF; 100 mug), vila

260 or we measured peak inhaled flow (PIF), peak inhaled pressure (PIP) and amount of the drug release fr

261 apical responses and increase sensitivity to inhaled proteases.

262 ment to initiate rescue treatments including inhaled pulmonary vasodilators, prone positioning, or ex

263 nternal organ-specific radiation dose due to inhaled radioactive aerosols has largely relied on exper

264 Oral and inhaled RBV appear to be well tolerated in LTRs, and our

265 ar mortality was significantly higher in the inhaled RBV group (24.1% versus 7.1% [oral RBV], P = 0.0

266 ts in the oral RBV group and 29 of 32 in the inhaled RBV group had symptomatic RSV infection.

267 (HR) for death and oral RBV use (compared to inhaled RBV), accounting for oxygen requirement and need

268 d they occur in small enough sizes, which if inhaled, reach the central airway and distal lung.

269 ), although one patient assigned to Amikacin Inhale received placebo in error and was included in the

270 gh catalytic generation of ROS in vivo after inhaling redox-active PM species (oxidative potential, O

271 the use of oral RBV as a safe alternative to inhaled ribavirin in LTRs.

272 uced rat model of airway inflammation by the inhaled route.

273 for the first time to explore the effects of inhaled SA on strength, variability, and entropy of func

274 lobal variation in prescribing practice (ie, inhaled salbutamol, or oral or inhaled corticosteroids)

275 The control condition was inhaled saline after filtered air.

276 icient method of preparing the nonsteroidal, inhaled selective glucocorticoid receptor modulator AZD7

277 Humans inhale several hundred to several thousand Aspergillus c

278 ll SR evidence on the efficacy and safety of inhaled short-acting bronchodilators to treat asthma and

279 DAS181, an inhaled sialidase, is undergoing clinical development fo

280 n of airway fluid and/or tissue generated by inhaled SO(2) .

281 lts suggested that the stimulatory effect of inhaled SO(2) on bronchopulmonary C-fibres was generated

282 s measured by exhaled gas concentration from inhaled sodium nitrite were not increased by acetazolami

283 helium acts as an initial defence barrier to inhaled spores, orchestrating an inflammatory response a

284 he in-vivo pharmacokinetic study showed that inhaled spray dried hydrogel microparticles (M6) formula

285 ten results in uncertainties associated with inhaled steroid therapy prescription.

286 to 5 or asthma diagnosis, bronchodilator or inhaled steroids, or unscheduled clinical evaluation for

287 e-to-severe asthma that is not controlled by inhaled steroids.

288 ptom control, despite lower dose maintenance inhaled steroids.

289 e furoate-umeclidinium-vilanterol) with dual inhaled therapy (fluticasone furoate-vilanterol or umecl

290 acerbation in the previous year despite dual inhaled therapy (inhaled corticosteroids plus long-actin

291 ong-acting muscarinic antagonists) or triple inhaled therapy (inhaled corticosteroids plus long-actin

292 Notwithstanding potential neurotoxicity of inhaled titanium dioxide nanoparticles (TiO2 NPs), the t

293 Users of inhaled tobramycin and azithromycin had FEV(1)% predicte

294 ed azithromycin use in cohorts using chronic inhaled tobramycin or aztreonam.Methods: This retrospect

295 ung cancer risk and tissue damage from other inhaled toxins.

296 ining topical treatment using PC945, a novel inhaled triazole, with systemic treatment using known tr

297 according to the route of allergen exposure (inhaled vs food allergens).

298 INHALE was a prospective, double-blind, randomised, plac

299 The proposed method images the uptake of inhaled xenon gas to the extravascular brain tissue comp

300 ondary objective was to assess the effect of inhaled xenon on myocardial ischemic damage in the same