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1  recordings in knockout mice devoid of tonic inhibitory current.
2  frequencies above 10 Hz evoked steady-state inhibitory currents.
3 hat contain delta subunits and mediate tonic inhibitory currents.
4 sponses comprised clusters of excitatory and inhibitory currents.
5  GABAergic interneurons and the reduction of inhibitory currents.
6 larizing sodium currents and hyperpolarizing inhibitory currents.
7 play of somatic sodium currents and synaptic inhibitory currents.
8 lar Dystrophy with ~ 50% reduction in phasic inhibitory currents.
9 he delta-GABA(A)Rs receptors produce a tonic inhibitory current and are widely distributed in the bra
10 rs results in a loss of alpha1-mediated fast inhibitory currents and a marked reduction in density of
11 t in hippocampal interneurons, mediate tonic inhibitory currents and are highly sensitive to low conc
12  we simultaneously measured phasic and tonic inhibitory currents and assessed their correlation.
13                      Tau reduction increased inhibitory currents and normalized excitation/inhibition
14             First, possible contributions of inhibitory currents and postsynaptic spiking to the anir
15 ry sites, reduced the frequency of miniature inhibitory currents, and increased excitability in the h
16 eous and evoked excitatory currents, reduced inhibitory currents, and NMDA receptor dysfunction.
17                                     When the inhibitory currents are activated or blocked, the model
18                            Despite this, the inhibitory currents are potent in modifying the firing p
19 duced numbers of Pv(+) neurons and decreased inhibitory currents, as well as alterations in event-rel
20 equency and amplitude of phasic IPSCs, tonic inhibitory currents, as well as in the number of inhibit
21 iadifenolide on action potential-independent inhibitory currents at GABAergic synapses, using (+)-bil
22 atal rat ventricular cardiomyocytes produced inhibitory currents at less than one-thousandth of the l
23 re consistent with most of hippocampal tonic inhibitory current being mediated by GABA released from
24 cker rTertiapin-Q diminished the NECA-evoked inhibitory current by 56 +/- 12%, whereas the SK channel
25  and that the balance between excitatory and inhibitory currents controls network stability and sensi
26  The cell type-specific differences in tonic inhibitory currents could potentially be used to selecti
27 tor evoked DA-dependent D2 receptor-mediated inhibitory currents (D2ICs).
28 actor neural network, taking into account an inhibitory current delivered to the network once a decis
29 Wong and Wang (2006), taking into account an inhibitory current delivered to the network once a decis
30                    Voltage-clamp analysis of inhibitory currents demonstrated that activity blockade
31 y a decrease in the frequency of spontaneous inhibitory currents due to a cell-specific reduction in
32                         Total excitatory and inhibitory currents during sensory-evoked swimming were
33                                  We measured inhibitory currents evoked by the tutor song throughout
34 roscopy to record spontaneous excitatory and inhibitory currents from birthdated DGCs.
35 he guinea pig (Cavia porcellus) by recording inhibitory currents from RGCs in the presence of ionotro
36 he guinea pig (Cavia porcellus) by recording inhibitory currents from RGCs in the presence of ionotro
37 table anesthetic etomidate increased a tonic inhibitory current generated by alpha5 subunit-containin
38  example, the balance between excitatory and inhibitory currents has been identified as a central com
39 regimes that produce balanced excitatory and inhibitory currents have specific advantages over other
40 w that GABA fails to evoke fast postsynaptic inhibitory currents in apical developing inner and outer
41 ology to record post-synaptic excitatory and inhibitory currents in complex horizontal connections in
42 onal application of GABA evoked large, rapid inhibitory currents in concordance with electron microsc
43 er was correlated with increased spontaneous inhibitory currents in excitatory neurons, suggesting th
44 and anticonvulsant treatment may alter tonic inhibitory currents in human neurons.
45 ing channelrhodopsin-2 evoked fast GABAergic inhibitory currents in mitral cells but failed to activa
46  Following removal of PNNs, the frequency of inhibitory currents in mPFC pyramidal neurons was decrea
47 onset and termination of "slow" postsynaptic inhibitory currents in neurons and atrial cells, RGS pro
48 ich contain delta subunits and mediate tonic inhibitory currents in neurons.
49       These data reveal an expanded range of inhibitory currents in PCs which may be critical to regu
50           The data revealed that perisomatic inhibitory currents in pyramidal cells generated the maj
51  hypothesized that these features potentiate inhibitory currents in RCs.
52 ivation of cholinergic interneurons produced inhibitory currents in SPNs.
53  marker expression, and perturbed excitatory/inhibitory currents in the hippocampus.
54 cts of isoflurane on properties of GABAergic inhibitory currents in the reticular thalamic nucleus (n
55  but reveal both excitatory and monosynaptic inhibitory currents in the ventral pallidum and lateral
56                                              Inhibitory currents in XII motoneurones were potentiated
57  demonstrate that pharmacologically reducing inhibitory currents increases correlated excitatory acti
58 he mIPSC frequency, NA also potentiated GABA inhibitory currents induced by direct stimulation of gra
59 s from nearby pyramidal cells, we found that inhibitory currents (IPSCs) are more correlated than exc
60 in PCs, the presence and mechanisms of tonic inhibitory currents, low amplitude currents resulting fr
61 ysiological findings suggest a disruption in inhibitory current may play a role in these changes.
62                             Phasic and tonic inhibitory currents of hippocampal pyramidal neurons exh
63 ed in part by weaker excitatory and stronger inhibitory currents onto MCs than onto TCs.
64 ristics and suggested that properties of the inhibitory currents rather than motoneuron size are resp
65 aneous inhibitory inputs and enhanced evoked inhibitory currents recorded from principal neurons of t
66 cytosis and protein kinase C to reduce GABAR inhibitory currents, surface GABAR alpha1 expression, an
67 citatory cells, overlapping with feedforward inhibitory currents that suppress action potentials.
68 ferential desensitization causing shift from inhibitory current to excitatory current with repeated c
69  individual contributions of AMPA, NMDA, and inhibitory currents to light responses of each cell type
70 ting a dynamic rebalancing of excitatory and inhibitory currents to maximize SNR.SIGNIFICANCE STATEME
71 on trains of ten of these excitatory and ten inhibitory current transients were combined with an addi
72           The mean reversal potential of the inhibitory current was -78 +/- 7 mV and inhibitions were
73 neous and sensory-evoked swimming, the total inhibitory current was more than threefold larger than t
74 induced CPP, both frequency and amplitude of inhibitory currents were decreased.
75     Patch-clamp recordings of excitatory and inhibitory currents were performed, then dendritic struc
76 rs to be driven by presynaptic activation of inhibitory currents, whereas the hSyn-HM4D-mediated incr
77 rin clusters and mixed glycinergic/GABAergic inhibitory currents with large peak amplitudes and long