ライフサイエンスコーパス: inhibitory postsynaptic current

1 urs during DSI of the temporally overlapping inhibitory postsynaptic current.

2 ty, at least in part, by enhancing miniature inhibitory postsynaptic currents.

3 ing that they were GABA(A) receptor-mediated inhibitory postsynaptic currents.

4 gic neurotransmission by recording miniature inhibitory postsynaptic currents.

5 showed an increased frequency of spontaneous inhibitory postsynaptic currents.

6 ereas muscarinic agonists potently increased inhibitory postsynaptic currents.

7 ture inhibitory postsynaptic current; mIPSC) inhibitory postsynaptic currents.

8 togenic tissue exhibit decreased spontaneous inhibitory postsynaptic currents.

9 t produced a decrease in amplitude of evoked inhibitory postsynaptic currents.

10 ced PPD, suggesting the contribution of slow inhibitory postsynaptic currents.

11 ly to influence the kinetics and efficacy of inhibitory postsynaptic currents.

12 imetics did not affect bicuculline-sensitive inhibitory postsynaptic currents.

13 d comparable mixed GABAergic and glycinergic inhibitory postsynaptic currents.

14 s from the precise interaction of excitatory-inhibitory postsynaptic currents.

15 enced by an increased amplitude of miniature inhibitory postsynaptic currents.

16 icant increase in the amplitude of miniature inhibitory postsynaptic currents.

17 tion of GABA(A) receptor (GABA(A)R)-mediated inhibitory postsynaptic currents.

18 elevated frequency of GABAergic spontaneous inhibitory postsynaptic currents.

19 GABA(A)R number and reduces the amplitude of inhibitory postsynaptic currents.

20 litude of subsequent GABAA receptor-mediated inhibitory postsynaptic currents.

21 slices by recording 5-HT1A receptor-mediated inhibitory postsynaptic currents (5-HT1A -IPSCs) generat

22 s revealed altered 5-HT1A receptor-dependent inhibitory postsynaptic currents (5-HT1A-IPSCs) in femal

23 es and a lower rate of spontaneous miniature inhibitory postsynaptic current activity.

24 NPY also depressed the GABAergic inhibitory postsynaptic current, although leptin had no

25 me in null mice, with no change in miniature inhibitory postsynaptic current amplitude or frequency.

26 vels, flurazepam treatment reduced miniature inhibitory postsynaptic current amplitude, which returne

27 showed that both spontaneous excitatory and inhibitory postsynaptic current amplitudes in the DG wer

28 ynaptic gephyrin clusters and mean miniature inhibitory postsynaptic current amplitudes, whereas a do

29 aptic current kinetics and reduced miniature inhibitory postsynaptic current amplitudes.

30 avioral phenotypes, decreased excitatory and inhibitory postsynaptic current and reduced c-Fos immuno

31 A GABAergic component was absent in evoked inhibitory postsynaptic currents and miniature events.

32 urons and time-locked synchronized bursts of inhibitory postsynaptic currents and phasic firing in BL

33 GABA-mediated inhibitory postsynaptic currents and potentials decayed

34 entration that abolished miniature GABAergic inhibitory postsynaptic currents and responses to exogen

35 both action potential-dependent (spontaneous inhibitory postsynaptic current) and -independent (minia

36 ude of spontaneous GABA(A) receptor-mediated inhibitory postsynaptic currents, and are blocked by gab

37 ogether with reduced interneuron maturation, inhibitory postsynaptic currents, and dentate progenitor

38 Cl(-) influx into the cytosol, a decrease of inhibitory postsynaptic currents, and ultimately a shift

39 rsibly inhibited both evoked and spontaneous inhibitory postsynaptic currents, as well as GABA applic

40 e glycine receptor enables the generation of inhibitory postsynaptic currents at synapses via neurotr

41 release site opened on the timescale of the inhibitory postsynaptic currents before desensitizing.

42 BDNF had no effect on spontaneous inhibitory postsynaptic currents but produced a decrease

43 in the amplitude and frequency of miniature inhibitory postsynaptic currents compared with those in

44 es revealed a significantly faster miniature inhibitory postsynaptic current decay time in null mice,

45 The IL-6-induced inhibitory postsynaptic currents decrease was inhibited

46 ntials were inhibited by tetrodotoxin (TTX), inhibitory postsynaptic currents decreased and currents

47 Furthermore, the duration of inhibitory postsynaptic current depression mirrors the t

48 of the NTS resulted in evoked excitatory and inhibitory postsynaptic currents (eEPSCs and eIPSCs) in

49 antly inhibited the peak amplitude of evoked inhibitory postsynaptic currents (eIPSCs) in all 11 labe

50 ally, 5-HT depressed photostimulation-evoked inhibitory postsynaptic currents (eIPSCs) in PVT neurons

51 GO) and met-enkephalin (ME) inhibited evoked inhibitory postsynaptic currents (eIPSCs) in the RGS-ins

52 ts a long-term depression of evoked GABA(A)R inhibitory postsynaptic currents (eIPSCs) onto hippocamp

53 transient suppression of evoked GABAA ergic inhibitory postsynaptic currents (eIPSCs) that follows b

54 Inhibitory postsynaptic currents (eIPSCs) were evoked af

55 amate receptor blockade increased PNZ-evoked inhibitory postsynaptic currents (eIPSCs) without affect

56 The medial NTS was stimulated to evoke GABA inhibitory postsynaptic currents (eIPSCs).

57 eased the amplitude of GABAA-mediated evoked inhibitory postsynaptic currents (eIPSCs).

58 rding was performed to obtain excitatory and inhibitory postsynaptic currents (EPSCs and IPSCs) of th

59 y the frequency dependence of excitatory and inhibitory postsynaptic currents (EPSCs, IPSCs) elicited

60 We recorded excitatory and inhibitory postsynaptic currents (EPSCs/IPSCs) in TC neu

61 howed a reduction of synchronous spontaneous inhibitory-postsynaptic-current events in mutants, which

62 onists selectively decreased the spontaneous inhibitory postsynaptic current frequency in the head-in

63 Inhibitory postsynaptic current frequency, measured on p

64 hat ethanol enhances GABAA receptor-mediated inhibitory postsynaptic currents (GABAA IPSCs) and reduc

65 DPDPE inhibition of GABAA receptor-mediated inhibitory postsynaptic currents (GABAAR IPSCs) is assoc

66 Normal-appearing inhibitory postsynaptic currents (human) and intrinsic n

67 associated with a reduction of GABA-mediated inhibitory postsynaptic current in neocortex and hippoca

68 d the frequency of spontaneous excitatory or inhibitory postsynaptic currents in a concentration-rela

69 Superfusion of CORT decreases evoked inhibitory postsynaptic currents in a mbGR- and CB1 cann

70 f optically-evoked GABA(A) receptor-mediated inhibitory postsynaptic currents in adolescent animals.

71 cordings revealed spontaneous excitatory and inhibitory postsynaptic currents in all neurons, regardl

72 served an increased frequency of spontaneous inhibitory postsynaptic currents in CA1 pyramidal cells,

73 mulation of ventrobasal complex cells evoked inhibitory postsynaptic currents in cells of the medial

74 ecordings revealed a diminished frequency of inhibitory postsynaptic currents in dentate granule cell

75 ing increase in the frequency of spontaneous inhibitory postsynaptic currents in dentate granule cell

76 tribution in pyramidal neurons and supported inhibitory postsynaptic currents in distal dendrites bet

77 Tac2 fibers in the BNSTpl exhibited enhanced inhibitory postsynaptic currents in males compared to fe

78 t1 extended glutamatergic interneuron-evoked inhibitory postsynaptic currents in motor neurons.

79 ncreased action potential-driven spontaneous inhibitory postsynaptic currents in nearby interneurons

80 activation elicited pairs of excitatory and inhibitory postsynaptic currents in PCs, excitation (E)

81 Also, unitary inhibitory postsynaptic currents in PNs were more reliab

82 Furthermore, morphine suppressed evoked inhibitory postsynaptic currents in postsynaptic Vgat- e

83 7 nAChRs on feedforward interneurons induced inhibitory postsynaptic currents in pyramidal cells that

84 Unitary inhibitory postsynaptic currents in pyramidal neurons ar

85 ated with reduced frequency of GABA-mediated inhibitory postsynaptic currents in pyramidal neurons, a

86 firing, which robustly increases spontaneous inhibitory postsynaptic currents in pyramidal neurons.

87 reased efficacy of spontaneous and miniature inhibitory postsynaptic currents in pyramidal neurons.

88 d with a diminished frequency of spontaneous inhibitory postsynaptic currents in SPNs, a parallel hyp

89 itude of excitatory postsynaptic currents or inhibitory postsynaptic currents in supraoptic nucleus n

90 sed amplitudes of both spontaneous EPSCs and inhibitory postsynaptic currents in the Astn2 KO animals

91 an increase in the frequency of spontaneous inhibitory postsynaptic currents in the dorsal horn of t

92 s and decreased the frequency of spontaneous inhibitory postsynaptic currents in the indirect pathway

93 ase in the number and amplitude of miniature inhibitory postsynaptic currents in these cells in a TTX

94 GABAA receptor (GABAAR)-mediated spontaneous inhibitory postsynaptic currents in WT mice, indicating

95 oth the amplitude and frequency of miniature inhibitory postsynaptic currents increased in neurons de

96 c currents decreased, while the frequency of inhibitory postsynaptic currents increased.

97 This inhibitory postsynaptic current (IPSC) erosion resulted

98 role of nitric oxide (NO) in modulating the inhibitory postsynaptic current (IPSC) evoked by focal s

99 ontrols, PAE increased basal spontaneous (s) inhibitory postsynaptic current (IPSC) frequency in the

100 cell evoked a GABA(A) receptor-mediated slow inhibitory postsynaptic current (IPSC) in a PN and an au

101 apses in olfactory bulb slices evokes a slow inhibitory postsynaptic current (IPSC) in granule cells

102 ease in the amplitude of evoked monosynaptic inhibitory postsynaptic current (IPSC) in layer V pyrami

103 endent decrease of GABA(A) receptor-mediated inhibitory postsynaptic current (IPSC) is explained by t

104 release produced a 5-HT1A receptor-mediated inhibitory postsynaptic current (IPSC) that resulted in

105 This report describes an inhibitory postsynaptic current (IPSC) that was elicited

106 or activation depresses the stimulus-evoked, inhibitory postsynaptic current (IPSC).

107 ea of the pharmacologically isolated GABA/B/-inhibitory postsynaptic current (IPSC).

108 vity modulates action potential duration and inhibitory postsynaptic current (IPSC).

109 OF increased depression of inhibitory postsynaptic currents (IPSCs) along IPSC trai

110 n with PPs had no effect on the amplitude of inhibitory postsynaptic currents (IPSCs) and a minimal e

111 ition, with increased frequency of miniature inhibitory postsynaptic currents (IPSCs) and amplitude o

112 ocaine exposure in vivo caused a decrease in inhibitory postsynaptic currents (IPSCs) and an increase

113 show that all mutations prolong the decay of inhibitory postsynaptic currents (IPSCs) and induce spon

114 ld each depress the frequency of spontaneous inhibitory postsynaptic currents (IPSCs) and the amplitu

115 ynaptic inhibition was recorded by isolating inhibitory postsynaptic currents (IPSCs) at a membrane p

116 1 Hz) electrical stimulation produced evoked inhibitory postsynaptic currents (IPSCs) at a relatively

117 cuculline-sensitive and strychnine-sensitive inhibitory postsynaptic currents (IPSCs) could be evoked

118 rge increase in the frequency of spontaneous inhibitory postsynaptic currents (IPSCs) driven by actio

119 ate the effects of 5-HT and TRH on GABAergic inhibitory postsynaptic currents (IPSCs) evoked by stimu

120 Inhibitory postsynaptic currents (IPSCs) evoked in CA1 p

121 we obtained whole-cell recordings of unitary inhibitory postsynaptic currents (IPSCs) evoked in ventr

122 nduced more pronounced initial depression of inhibitory postsynaptic currents (IPSCs) followed by mod

123 Inhibitory postsynaptic currents (IPSCs) generated by GA

124 ation-dependent increase in the frequency of inhibitory postsynaptic currents (IPSCs) in 5-HT neurons

125 ressing interneurons (PVs and SOMs) triggers inhibitory postsynaptic currents (IPSCs) in both regular

126 s at 5-50 Hz frequencies generated trains of inhibitory postsynaptic currents (IPSCs) in CA1 stratum

127 ethanol increases GABA(A) receptor-mediated inhibitory postsynaptic currents (IPSCs) in CeA neurons

128 ession, we evoked 100 Hz trains of GABAergic inhibitory postsynaptic currents (IPSCs) in cerebellar n

129 receptors and loss of D2 receptor-dependent inhibitory postsynaptic currents (IPSCs) in D2 receptor-

130 ordings of intrinsic membrane properties and inhibitory postsynaptic currents (IPSCs) in dentate gran

131 Stimulation of the NTS evoked inhibitory postsynaptic currents (IPSCs) in DMV neurones

132 dopamine terminals evoked robust D2-receptor inhibitory postsynaptic currents (IPSCs) in GIRK2-expres

133 of a study of the frequency potentiation of inhibitory postsynaptic currents (IPSCs) in hypoglossal

134 duced monosynaptically evoked GABAA-mediated inhibitory postsynaptic currents (IPSCs) in NRT and soma

135 re (SL-M) stimuli that activate GABA(A,slow) inhibitory postsynaptic currents (IPSCs) in pyramidal ce

136 afferents induces rhythmic, theta-frequency inhibitory postsynaptic currents (IPSCs) in pyramidal ce

137 Inhibitory postsynaptic currents (IPSCs) in RTN neurons

138 ion, we found that GABA(A)-receptor-mediated inhibitory postsynaptic currents (IPSCs) in the inhibito

139 eratrol potentiated GABAA and GABAB-mediated inhibitory postsynaptic currents (IPSCs) in VTA dopamine

140 0-1724 blocked I-LTD and acute depression of inhibitory postsynaptic currents (IPSCs) induced by D(2)

141 postsynaptic currents (EPSCs) and GABAergic inhibitory postsynaptic currents (IPSCs) induced by post

142 In addition, retigabine potentiated inhibitory postsynaptic currents (IPSCs) mediated by act

143 roduces a long-lasting enhancement of evoked inhibitory postsynaptic currents (IPSCs) mediated by D1-

144 ncy of glycinergic spontaneous and miniature inhibitory postsynaptic currents (IPSCs) of lamina II ne

145 c in the inhibition by morphine of GABAergic inhibitory postsynaptic currents (IPSCs) recorded from n

146 The time course of inhibitory postsynaptic currents (IPSCs) reflects GABA(A

147 Cs) facilitated strongly, whereas disynaptic inhibitory postsynaptic currents (IPSCs) remained stable

148 ly were the amplitudes of evoked glycinergic inhibitory postsynaptic currents (IPSCs) significantly l

149 losteric modulator (PAM) AZD7325 potentiates inhibitory postsynaptic currents (IPSCs) specifically in

150 uency and amplitude of spontaneous GABAergic inhibitory postsynaptic currents (IPSCs) than did wild t

151 Vagal stimulation also evoked inhibitory postsynaptic currents (IPSCs) that were rever

152 T(4) receptors on the amplitude of GABAergic inhibitory postsynaptic currents (IPSCs) to a reduction.

153 y drive onto VB neurons from multiple peaked inhibitory postsynaptic currents (IPSCs) to single peake

154 Here the duration of inhibitory postsynaptic currents (IPSCs) was dependent o

155 Evoked inhibitory postsynaptic currents (IPSCs) were induced in

156 r the frequency of GABA-mediated spontaneous inhibitory postsynaptic currents (IPSCs) were reduced in

157 Evoked GABAergic inhibitory postsynaptic currents (IPSCs) were reversibly

158 oltage-clamped GABA(A fast) and GABA(A slow) inhibitory postsynaptic currents (IPSCs) were selectivel

159 Inhibitory postsynaptic currents (IPSCs) were twice as p

160 eurons mediating fast GABA(A) (GABA(A,fast)) inhibitory postsynaptic currents (IPSCs), whereas theta

161 pharmacologically distinct from spontaneous inhibitory postsynaptic currents (IPSCs).

162 concentrations that barely affect GABAergic inhibitory postsynaptic currents (IPSCs).

163 ete, strychnine-sensitive, chloride-mediated inhibitory postsynaptic currents (IPSCs).

164 des of both glycinergic and GABAergic evoked inhibitory postsynaptic currents (IPSCs).

165 show that besides blocking GABA(A)-mediated inhibitory postsynaptic currents (IPSCs, I(phasic)), the

166 creased spontaneous inhibitory transmission (inhibitory postsynaptic currents, IPSCs) in the DR.

167 The magnitude of the depression of the inhibitory postsynaptic current is dependent on the freq

168 nvergence ratios ( approximately 40:1), fast inhibitory postsynaptic current kinetics (tau(decay) = 2

169 subunit altered the time course of miniature inhibitory postsynaptic current kinetics and reduced min

170 -evoked inhibition, we recorded light-evoked inhibitory postsynaptic currents (L-IPSCs) from rod bipo

171 ted inputs to rod BCs prolonged light-evoked inhibitory postsynaptic currents (L-IPSCs), while smalle

172 DCG IV (1 microM) had no effect on inhibitory postsynaptic currents mediated by GABA.

173 e frequency and the amplitude of spontaneous inhibitory postsynaptic currents mediated by gamma-amino

174 This study describes spontaneous miniature inhibitory postsynaptic currents mediated by vesicular d

175 apses, causing clear reductions in miniature inhibitory postsynaptic current (mIPSC) amplitude and fr

176 tion of Gad1 significantly reduced miniature inhibitory postsynaptic current (mIPSC) amplitudes and G

177 on did not exhibit increased basal miniature inhibitory postsynaptic current (mIPSC) frequency but sh

178 minimally altered sIPSC amplitude, miniature inhibitory postsynaptic current (mIPSC) frequency, and m

179 tion of transmitter fluctuation to miniature inhibitory postsynaptic current (mIPSC) variability.

180 apse markers and decreased frequency of mini inhibitory postsynaptic currents (mIPSC) in the NAc of s

181 ptic vesicles and the frequency of miniature inhibitory postsynaptic current(mIPSC), but increase of

182 ynaptic current) and -independent (miniature inhibitory postsynaptic current; mIPSC) inhibitory posts

183 ptible to DSI, while TTX-resistant miniature inhibitory postsynaptic current (mIPSCs) were not.

184 e activates rapid GABA(A) receptor miniature inhibitory postsynaptic currents (mIPSCs) (predominant d

185 We recorded miniature inhibitory postsynaptic currents (mIPSCs) from hippocamp

186 sed the frequency of GABA-mediated miniature inhibitory postsynaptic currents (mIPSCs) in all nine la

187 t the decay time of GABAR-mediated miniature inhibitory postsynaptic currents (mIPSCs) in CA1 pyramid

188 uency of glycinergic and GABAergic miniature inhibitory postsynaptic currents (mIPSCs) in cardiac vag

189 endent increase in the frequency ofminiature inhibitory postsynaptic currents (mIPSCs) in primary cul

190 ptor agonist baclofen, potentiated miniature inhibitory postsynaptic currents (mIPSCs) in pyramidal n

191 re excitatory postsynaptic currents (mEPSCs)/inhibitory postsynaptic currents (mIPSCs) in the Mecp2-m

192 brain development the duration of miniature inhibitory postsynaptic currents (mIPSCs) mediated by GA

193 brain development the duration of miniature inhibitory postsynaptic currents (mIPSCs) mediated by GA

194 rites in macaque monkeys, measured miniature inhibitory postsynaptic currents (mIPSCs) of granule cel

195 pare the properties of 'tonic' and miniature inhibitory postsynaptic currents (mIPSCs) recorded from

196 neous action potential independent miniature inhibitory postsynaptic currents (mIPSCs) was significan

197 postsynaptic currents (mEPSCs) and miniature inhibitory postsynaptic currents (mIPSCs) were recorded

198 croscopically, and the spontaneous miniature inhibitory postsynaptic currents (mIPSCs) were recorded

199 plitude and frequency of GABAergic miniature inhibitory postsynaptic currents (mIPSCs) were reduced i

200 n frequency and charge transfer of miniature inhibitory postsynaptic currents (mIPSCs) were significa

201 creased the frequency of miniature GABAergic inhibitory postsynaptic currents (mIPSCs) without changi

202 ynaptic currents (mEPSCs), but not miniature inhibitory postsynaptic currents (mIPSCs), increase in a

203 A currents and in the frequency of miniature inhibitory postsynaptic currents (mIPSCs).

204 as the frequency and amplitude of miniature inhibitory postsynaptic currents (mIPSCs).

205 or GABAergic markers and recording miniature inhibitory postsynaptic currents (mIPSCs).

206 as well as increased frequency of miniature inhibitory postsynaptic currents (mIPSCs).

207 tials (IPSPs) and the frequency of miniature inhibitory postsynaptic currents (mIPSCs).

208 g the neurons lost, and a marked decrease in inhibitory postsynaptic currents of lamina II neurons co

209 erneurons in the CA1 hippocampus to increase inhibitory postsynaptic currents on CA1 pyramidal cells.

210 activity during nesting and sleep, inducing inhibitory postsynaptic currents on diverse cells in the

211 excitability and enhancing the frequency of inhibitory postsynaptic currents on pyramidal neurons in

212 K-801 decreased the frequency of spontaneous inhibitory postsynaptic currents onto layer V pyramidal

213 amplitude but not the frequency of miniature inhibitory postsynaptic currents or expression of the gl

214 layer VI cortical neurons exhibited reduced inhibitory postsynaptic current peak amplitudes, prolong

215 the strength of the GABAa receptor-mediated inhibitory postsynaptic currents received by regular- vs

216 blunted membrane excitability and divergent inhibitory postsynaptic current responses to CRF applica

217 Analysis of the kinetics of inhibitory postsynaptic current revealed no modification

218 aracteristics of sound-evoked excitatory and inhibitory postsynaptic currents (seEPSCs and seIPSCs, r

219 the frequency of spontaneous excitatory and inhibitory postsynaptic currents (sEPSCs and sIPSCs).

220 pmol/L GLP-1 increased both the spontaneous inhibitory postsynaptic current (sIPSC) amplitudes and f

221 chol significantly increased the spontaneous inhibitory postsynaptic current (sIPSC) frequency and am

222 mutation had no clear effect on spontaneous inhibitory postsynaptic current (sIPSC) frequency at IN-

223 s displayed an elevated baseline spontaneous inhibitory postsynaptic current (sIPSC) frequency compar

224 ngs revealed large reductions in spontaneous inhibitory postsynaptic current (sIPSC) frequency in bot

225 cotine (mean increased spontaneous GABAergic inhibitory postsynaptic current (sIPSC) frequency was ap

226 pyramidal neuron also causes LTP of the slow inhibitory postsynaptic current (sIPSC) mediated by meta

227 Spontaneous and miniature inhibitory postsynaptic currents (sIPSCS and mIPSCs) wer

228 nger suppression (versus N40) of spontaneous inhibitory postsynaptic currents (sIPSCs) across multipl

229 ontrol of synaptic GABA release, spontaneous inhibitory postsynaptic currents (sIPSCs) and miniature

230 neurons presented frequencies of spontaneous inhibitory postsynaptic currents (sIPSCs) and spontaneou

231 fects on GABAA receptor-mediated spontaneous inhibitory postsynaptic currents (sIPSCs) and spontaneou

232 aclofen reduced the frequency of spontaneous inhibitory postsynaptic currents (sIPSCs) but did not al

233 sEPSCs were isolated from the spontaneous inhibitory postsynaptic currents (sIPSCs) by application

234 AA receptor-mediated spontaneous monoquantal inhibitory postsynaptic currents (sIPSCs) from rat supra

235 1% of recordings and facilitated spontaneous inhibitory postsynaptic currents (sIPSCs) in 20% of reco

236 iPT produced robust increases in spontaneous inhibitory postsynaptic currents (sIPSCs) in basolateral

237 ed amplitudes and frequencies of spontaneous inhibitory postsynaptic currents (sIPSCs) in CA1 pyramid

238 and analogs on the properties of spontaneous inhibitory postsynaptic currents (sIPSCs) in cultured ra

239 ) subunit expression and reduced spontaneous inhibitory postsynaptic currents (sIPSCs) in D1-type, bu

240 conductance, but did not affect spontaneous inhibitory postsynaptic currents (sIPSCs) in dentate gra

241 ncy of GABA(A) receptor-mediated spontaneous inhibitory postsynaptic currents (sIPSCs) in dlBnST targ

242 lease increased the frequency of spontaneous inhibitory postsynaptic currents (sIPSCs) in downstream

243 of action potential-dependent, spontaneous, inhibitory postsynaptic currents (sIPSCs) in hippocampus

244 uency of spontaneous GABAA receptor-mediated inhibitory postsynaptic currents (sIPSCs) in RT (by 60 +

245 croM) increased the frequency of spontaneous inhibitory postsynaptic currents (sIPSCs) in the majorit

246 chemoconvulsant-induced burst of spontaneous inhibitory postsynaptic currents (sIPSCs) on CA1 pyramid

247 rate and net charge transfer of spontaneous inhibitory postsynaptic currents (sIPSCs) recorded from

248 The frequency of spontaneous inhibitory postsynaptic currents (sIPSCs) was similar in

249 Spontaneous inhibitory postsynaptic currents (sIPSCs) were not diffe

250 Under these conditions spontaneous GABAergic inhibitory postsynaptic currents (sIPSCs) were seen as i

251 h the frequency and amplitude of spontaneous inhibitory postsynaptic currents (sIPSCs) were significa

252 postsynaptic currents (sEPSCs), spontaneous inhibitory postsynaptic currents (sIPSCs), and bidirecti

253 rrents (sEPSCs) and decreases in spontaneous inhibitory postsynaptic currents (sIPSCs).

254 res the amplitude and frequency of miniature inhibitory postsynaptic currents to wild-type levels.

255 ecordings from PVs and PNs show that unitary inhibitory postsynaptic currents (uIPSCs) are larger in

256 By recording unitary inhibitory postsynaptic currents (uIPSCs) from cell pair

257 Unitary inhibitory postsynaptic currents (uIPSCs) were recorded

258 ability of synaptic failures in spike-evoked inhibitory postsynaptic currents (unitary IPSCs) in CA1

259 er, the frequency of GABAA receptor-mediated inhibitory postsynaptic currents was depressed in medium

260 of opioids because alpha(2)-AR-mediated slow inhibitory postsynaptic currents were depressed in wt bu

261 ss, and whole-cell patch clamp recordings of inhibitory postsynaptic currents were performed from ven

262 The effects of PS on GABAA receptor-mediated inhibitory postsynaptic currents were studied in culture

263 small decrease in the frequency of miniature inhibitory postsynaptic currents, which was not affected

264 duced the frequency of spontaneous miniature inhibitory postsynaptic currents while having little eff

265 vely and reversibly reduced the amplitude of inhibitory postsynaptic currents with a postsynaptic eff

266 ctively increases the frequency of miniature inhibitory postsynaptic currents with no effect on ampli

267 eveal a >20-fold increase in nicotine-evoked inhibitory postsynaptic currents with no effect on excit

268 Here we combine quantal analysis of evoked inhibitory postsynaptic currents with quantitative immun