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1 to reduce membrane potential during the slow inhibitory postsynaptic potential.
2 ten the AP afterdepolarization, and generate inhibitory postsynaptic potentials.
3 y inputs were often followed by long-latency inhibitory postsynaptic potentials.
4 cells was no longer balanced by synchronous inhibitory postsynaptic potentials.
5 excitability and the reversal potential for inhibitory postsynaptic potentials.
6 o significant change in evoked excitatory or inhibitory postsynaptic potentials, and intrinsic cellul
7 aled that the activation of h-currents by an inhibitory postsynaptic potential can cause a short-term
9 iunit spikes increased with age, whereas the inhibitory postsynaptic potential caused by Purkinje cel
11 d that ethanol had little effect on compound inhibitory postsynaptic potentials/currents (IPSP/Cs) co
12 ss nuclear cell spiking, whereas synchronous inhibitory postsynaptic potentials entrain nuclear cell
13 evoked an excitatory postsynaptic potential/inhibitory postsynaptic potential (EPSP/IPSP) sequence,
14 ionotropic receptors to cause excitatory or inhibitory postsynaptic potentials (EPSPs or IPSPs), res
16 tic transmission, DHPG induces depression of inhibitory postsynaptic potentials evoked by primary aff
18 otein, induces a long-term transformation of inhibitory postsynaptic potentials from basket interneur
20 calcium spikes in both cell types when peak inhibitory postsynaptic potential hyperpolarization was
21 of GABAergic interneurons produces a strong inhibitory postsynaptic potential in spiny neurons, the
22 comitantly, as the reversal potential of the inhibitory postsynaptic potential in VPN neurons became
23 (NE) causes an increase in the frequency of inhibitory postsynaptic potentials in CA1 pyramidal neur
25 N density, which was associated with reduced inhibitory postsynaptic potentials in layer 2/3 pyramida
26 lation of the brachium produced monosynaptic inhibitory postsynaptic potentials in morphologically id
28 LTS activity was closely correlated with inhibitory postsynaptic potentials in neighboring FS int
29 ular stimulation of an OC interneuron evokes inhibitory postsynaptic potentials in the B3 motoneurons
30 rectifying K+ channels (GIRK) generate slow inhibitory postsynaptic potentials in the brain via G(i/
33 ddition, nicotine reduced field monosynaptic inhibitory postsynaptic potentials in the presence of ML
34 precision and reliability of excitatory and inhibitory postsynaptic potentials, in contrast to Media
35 ions and reversed the basket interneuron-CA1 inhibitory postsynaptic potential into an excitatory pos
37 otentials in response to single stimuli, the inhibitory postsynaptic potential (IPSP) conductance and
39 nerator potentials, but had no affect on the inhibitory postsynaptic potential (IPSP) in the B cell t
42 f a gamma-aminobutyric acid type B (GABA(B)) inhibitory postsynaptic potential (IPSP), but in drug-ex
43 and GABAB receptor-mediated hyperpolarizing inhibitory postsynaptic potentials (IPSPAs and IPSPBs, r
45 synaptic perforant path-evoked fast and slow inhibitory postsynaptic potentials (IPSPs) (53% and 66%,
46 y hyperpolarizing the reversal potential for inhibitory postsynaptic potentials (IPSPs) after SCI.
47 ased GABA type A (GABA(A)) receptor-mediated inhibitory postsynaptic potentials (IPSPs) and currents
49 of ghrelin increased the amplitude of evoked inhibitory postsynaptic potentials (IPSPs) and the frequ
50 he amplitude of stimulus-evoked monosynaptic inhibitory postsynaptic potentials (IPSPs) between acute
51 cular thalamic (RE) neurons in vivo revealed inhibitory postsynaptic potentials (IPSPs) between RE ce
52 ished basal-evoked GABA(A) receptor-mediated inhibitory postsynaptic potentials (IPSPs) by decreasing
53 t mitral cell synchrony was mainly driven by inhibitory postsynaptic potentials (IPSPs) imposed by GA
54 and cLF, respectively) revealed monosynaptic inhibitory postsynaptic potentials (IPSPs) in 75% and 65
55 rneurons in the hippocampus, eliciting giant inhibitory postsynaptic potentials (IPSPs) in CA3 pyrami
56 s (NRGc) evoked large amplitude, glycinergic inhibitory postsynaptic potentials (IPSPs) in cat motone
58 cells at the central canal elicits GABAergic inhibitory postsynaptic potentials (IPSPs) in intraspina
59 and stimulation evokes bicuculline-sensitive inhibitory postsynaptic potentials (IPSPs) in motorneuro
60 this system is the amplitude and duration of inhibitory postsynaptic potentials (IPSPs) in thalamocor
61 ly inhibited 5-HT(1A) receptor-mediated slow inhibitory postsynaptic potentials (IPSPs) in the dorsal
62 amplitude, whereas basket cells evoke slower inhibitory postsynaptic potentials (IPSPs) in their post
64 sent report, we studied the arrival times of inhibitory postsynaptic potentials (IPSPs) observed in i
65 s exhibited large amplitude monophasic GABAB inhibitory postsynaptic potentials (IPSPs) synchronous w
67 equency of spontaneous bicuculline-sensitive inhibitory postsynaptic potentials (IPSPs) when recorded
68 n of sympathetic postganglionic axons evoked inhibitory postsynaptic potentials (IPSPs), and stimulat
69 nin hyperpolarizes the reversal potential of inhibitory postsynaptic potentials (IPSPs), E(IPSP), in
70 issues and play key roles in generating late inhibitory postsynaptic potentials (IPSPs), slowing hear
72 elicited by short, high-frequency trains of inhibitory postsynaptic potentials (IPSPs), which reliab
79 ver, is that LTD in layer IV is modulated by inhibitory postsynaptic potentials (IPSPs); postsynaptic
81 Cl- levels required for the hyperpolarizing inhibitory postsynaptic potentials mediated by ionotropi
82 In vitro, dynamically clamped asynchronous inhibitory postsynaptic potentials mimicking Purkinje af
84 vectors increased the amplitude of cortical inhibitory postsynaptic potentials, only viral expressio
85 e lateral rectus muscle completely abolishes inhibitory postsynaptic potentials onto abducens motoneu
86 ogical blockers reduced the amplitude of the inhibitory postsynaptic potential recorded from CA1 PNs
87 technique did not affect the GABAA-mediated inhibitory postsynaptic potentials, the membrane resista
88 of the gamma gamma-aminobutyric acid (GABA) inhibitory postsynaptic potentials was markedly decrease
90 ceive strong barrages of both excitatory and inhibitory postsynaptic potentials, with the inhibitory