ライフサイエンスコーパス: inhibitory protein

1 novel interacting partner Rkip (Raf-1 kinase inhibitory protein).

2 ver-activating protein), LAP, and LIP (liver-inhibitory protein).

3 ed to as LIP (liver-enriched transcriptional inhibitory protein).

4 interaction of Cbl with the Sprouty2 (Spry2) inhibitory protein.

5 iapoptotic regulator cellular caspase-8-like inhibitory protein.

6 presence of increasing concentrations of the inhibitory protein.

7 n the epidermis RNases are complexed with an inhibitory protein.

8 that the HCMV UL38 gene encodes a cell death inhibitory protein.

9 nriched activator protein and liver-enriched inhibitory protein.

10 ion of striatum-enriched HspBP1, a chaperone inhibitory protein.

11 slation factor, and 4EBP1 translation factor inhibitory protein.

12 hile a human FoxP2 mutant acts as a dominant-inhibitory protein.

13 oxO3a and FoxO-mediated expression of growth-inhibitory proteins.

14 complex is regulated by both activating and inhibitory proteins.

15 hich is predicated upon an arsenal of potent inhibitory proteins.

16 are sequestered in the cytoplasm by IkappaB inhibitory proteins.

17 bitor of apoptosis proteins, and viral FLICE-inhibitory proteins.

18 ct from that associated with differentiation-inhibitory proteins.

19 inactivated in human cancers overexpressing inhibitory proteins.

20 ate immunity by expressing a series of small inhibitory proteins.

21 mily zinc finger 1)] or MGE [Map3k12 binding inhibitory protein 1 (Mbip); zinc-finger, SWIM domain co

22 creased expression of modulatory calcineurin inhibitory protein 1 (MCIP1), a direct downstream target

23 d by 409 +/- 32%, and modulatory calcineurin inhibitory protein 1 by 315 +/- 28%, P < 0.01).

24 duces an up-regulation of neuronal apoptosis inhibitory protein-1 expression in neurons that promotes

25 osis proteins (IAPs), the neuronal apoptosis inhibitory protein-1.

26 Plasma macrophage inhibitory protein-1alpha and insulin-like growth factor

27 li; and vi) reduced expression of macrophage inhibitory protein-2 and transforming growth factor-beta

28 The invasion inhibitory protein 45 (IIp45) we recently identified was

29 st-translationally down-regulating the eIF4E inhibitory protein 4E-BP1.

30 NAIP5 (NLR family, apoptosis inhibitory protein 5) binds the bacterial protein flagel

31 logue of NLRC4, NAIP5 (NLR family, apoptosis inhibitory protein 5), has been implicated in activation

32 ligand for the NAIP5 (NLR family, apoptosis inhibitory protein 5)/NLRC4 (NLR family, CARD-domain-con

33 NAIP5/NLRC4 (neuronal apoptosis inhibitory protein 5/nucleotide oligomerization domain-l

34 wnregulates expression of the cellular FLICE inhibitory protein, a negative regulator of death recept

35 tion induced the synthesis of liver-enriched inhibitory protein, a potent mitogen and the dominant ne

36 We have shown that the NF-kappaB inhibitory protein A20 exerts vasculoprotective effects

37 The impact of NF-kappaB inhibitory protein A20 on IFNgamma signaling in vascular

38 ealed the differential mapping of Raf kinase inhibitory protein after T cell recall in vivo.

39 levels of EZHIP (Enhancer of Zeste Homologs Inhibitory Protein, also termed CXORF67).

40 ain-like interleukin 1beta-converting enzyme inhibitory protein), an apoptosis inhibitor abundantly p

41 ory and active isoforms, i.e. liver-enriched inhibitory protein and liver-enriched activating protein

42 Liver-enriched inhibitory protein and liver-enriched activating protein

43 omoted the phosphorylation of the Raf kinase inhibitory protein and the downregulation of carabin, al

44 activity is critical for processing IkappaB inhibitory proteins and activating the NF-kappaB signali

45 in mapping the interactions between amyloid-inhibitory proteins and amyloidogenic peptides.

46 s that EZH2 inhibition stimulates cell cycle inhibitory proteins and enhances the production of extra

47 trix components, as well as several TGF-beta inhibitory proteins and pro-peptides.

48 f FLIP (FADD-like IL-1beta-converting enzyme-inhibitory protein) and Bcl-2.

49 ntify the conserved protein SciP (small CtrA inhibitory protein) and show that it accumulates during

50 echanisms: transcriptional regulation, small inhibitory proteins, and protein degradation.

51 ta-converting enzyme inhibitory protein-like inhibitory protein], and metastasis (vascular endothelia

52 facilitator (APOA5 [apolipoprotein A-V]) and inhibitory proteins (APOC3 [apolipoprotein C-III], ANGPT

53 actin assembly, the physiologic functions of inhibitory proteins are unclear.

54 In several well-studied cases the inhibitory proteins are water-soluble and bind at a chan

55 NAIPs (NLR family, apoptosis inhibitory proteins) are NLRs that appear to function as

56 In this study we identify Raf-1 kinase inhibitory protein as an essential mediator of ethanol-i

57 ation of CNS axons is the presence of growth-inhibitory proteins associated with myelin and the glial

58 ns cFLIP(L) (cellular caspase-8 (FLICE)-like inhibitory protein), Bcl-2, and Bcl-X(L) and inhibit the

59 TX attenuates ABI by converting inhibitory, protein-binding aggregates into nonbinding c

60 beta-Lactamase inhibitory protein (BLIP) binds and inhibits a diverse c

61 Beta-lactamase inhibitory protein (BLIP) binds and inhibits a diverse c

62 ers for 20 alanine mutants in beta-lactamase inhibitory protein (BLIP) for binding to TEM-1 beta-lact

63 ted contact residue mutants ofbeta-lactamase-inhibitory protein (BLIP) using isothermal titration cal

64 The interactions between beta-lactamase inhibitory proteins (BLIPs) and beta-lactamases have bee

65 nt here structures of a bacterial complement inhibitory protein, both free and bound to its complemen

66 role for Spry involving sequestration of the inhibitory protein c-Cbl.

67 Cellular FLICE-inhibitory protein (c-FLIP(L)) is a key regulator of the

68 or, and reduces the levels of cellular FLICE-inhibitory protein (c-FLIP) (both the long and short for

69 ediated stabilization of cellular FLICE-like inhibitory protein (c-FLIP) and Mcl-1.

70 domain (FADD)-like apoptosis regulator-like inhibitory protein (c-FLIP) and myeloid cell leukemia 1

71 g the antiapoptotic gene cellular FLICE-like inhibitory protein (C-FLIP) in myeloid cells, we have ge

72 or inhibitor cellular caspase 8 (FLICE)-like inhibitory protein (c-FLIP) is required for TNFalpha-ind

73 FAS-mediated apoptosis pathway by FLICE-like inhibitory protein (c-FLIP) may contribute to oncogenesi

74 ain-like interleukin-1beta-converting enzyme-inhibitory protein (c-FLIP) mediates the DISC assembly i

75 suggest that cellular caspase 8 (FLICE)-like inhibitory protein (c-FLIP) promotes cell survival in de

76 osis was associated with cellular FLICE-like inhibitory protein (c-FLIP) turnover and that gamma-secr

77 n-regulated the expression of cellular FLICE-inhibitory protein (c-FLIP), a major negative regulator

78 ction based on degradation of cellular FLICE-inhibitory protein (c-FLIP), a major regulator of the de

79 We show that cellular FLICE-like inhibitory protein (c-FLIP), a procaspase-8-like apoptot

80 Cellular FLICE-inhibitory protein (c-FLIP), an antioxidant and an impor

81 lls led to down-regulation of cellular FLICE inhibitory protein (c-FLIP), an inhibitor of apoptosis.

82 the caspase-8 regulator cellular FLICE-like inhibitory protein (c-FLIP), targeting it for proteasome

83 ain-like interleukin-1beta-converting enzyme-inhibitory protein (c-FLIP), thus releasing their inhibi

84 ike interleukin-1beta-converting enzyme-like inhibitory protein (c-FLIP), were expressed in basal con

85 ke interleukin-1beta-converting enzyme)-like inhibitory protein (c-FLIP).

86 associated with a decrease in cellular FLICE-inhibitory protein (c-FLIP).

87 Overexpression of cellular FLICE-like inhibitory protein (c-FLIP-s) or knockdown of CD95 suppr

88 D95 or overexpression of cellular FLICE-like inhibitory protein (c-FLIP-s).

89 C/EBPbeta-liver-enriched inhibitory protein (C/EBPbeta-LIP), a truncated C/EBPbet

90 These inhibitory proteins can act both directly by targeting s

91 T cell expression of inhibitory proteins can be a critical component for the

92 d expression of Pim1 overcame the amino acid inhibitory protein cascade and activated mTORC1.

93 uzumab binding, minimal change in complement inhibitory protein (CD46) expression and no complement-d

94 on endothelial cell expression of complement inhibitory proteins CD59 and decay-accelerating factor.

95 SP8, the long isoform of cellular FLICE-like inhibitory protein (cFLIP(L)), in the regulation of this

96 , but not collagen, expressed cellular FLICE inhibitory protein (cFLIP) and TNFalpha stimulation of l

97 al an important role for cellular FLICE-like inhibitory protein (cFLIP) in the regulation of the infl

98 ppaB activation, and cellular caspase-8-like inhibitory protein (cFLIP) inhibited activation of NF-ka

99 Overexpression of cellular FLICE-like inhibitory protein (cFLIP) is reported to confer chemore

100 The cellular FLICE-like inhibitory protein (cFLIP) is reported to inhibit IFNalp

101 r permanent) with deletion of cellular FLICE-inhibitory protein (cFlip) or caspase-8 in the intestina

102 Cellular FLICE-like inhibitory protein (cFLIP) overexpression and caspase-8

103 ceptors and the antiapoptotic cellular FLICE-inhibitory protein (cFLIP) was inconsistent across the c

104 uncovered down-regulation of cellular FLICE inhibitory protein (cFLIP).

105 of the antiapoptotic molecule cellular FLICE inhibitory protein (cFLIP).

106 conditions and in the presence of the growth-inhibitory proteins chondroitin sulfate proteoglycan, my

107 (Cdk), (iii) increased expression of the Cdk inhibitory proteins (Cip1/p21, Kip1/p27), enhanced bindi

108 ent expression levels of CD38 and complement-inhibitory proteins (CIPs) are associated with response

109 The epithelial complement inhibitory proteins (CIPs) cluster of differentiation 46

110 To determine whether stimulation by the inhibitory protein coevolved with resistance or whether

111 MC38-FasL/FLICE-like inhibitory protein colon cancer cells induce apoptosis i

112 Expression of FLICE-like inhibitory protein confers apoptosis resistance, increas

113 permitted infection of cells expressing the inhibitory protein CPSF6-358.

114 ement receptor 2 (CR2) to a mouse complement-inhibitory protein, Crry.

115 ledge) signaling complex with the checkpoint inhibitory protein CTLA-4 in regulatory T cells (Tregs).

116 consequence of deficiency of the complement inhibitory proteins decay accelerating factor (DAF, CD55

117 d in wild-type-infected cells expressing the inhibitory proteins (DeltaH).

118 Increases in liver-enriched inhibitory protein directly correlated with proliferatin

119 The expression of the inhibitory protein elevated the strain's fitness signifi

120 n kinase, RNA activated), and viperin (virus inhibitory protein, endoplasmic reticulum associated, in

121 Virus-inhibitory protein, endoplasmic reticulum-associated, in

122 antiviral radical SAM enzyme viperin (virus-inhibitory protein, endoplasmic reticulum-associated, in

123 Complement-inhibitory proteins expressed on cancer cells can provid

124 itors (Y-27632, LY294002, PF271 and PP2) and inhibitory protein expression (FAK-related nonkinase).

125 lls, suggesting that the modulation of Raf-1 inhibitory protein expression may have future therapeuti

126 show that either suppression of Raf-1 kinase inhibitory protein expression using short hairpin RNA or

127 over, the analog is not stabilized unless an inhibitory protein factor binds to the enzyme.

128 e recruit the alternative pathway complement inhibitory protein factor H (fH) to their surfaces to ev

129 lls by mediating the degradation of the TRAF inhibitory protein, Fbxl2.

130 he degradation of the long form of the FLICE-inhibitory protein (FLIP(L)), an inhibitor of death-indu

131 isoform of the caspase-8 inhibitor, c-FLICE inhibitory protein (FLIP(S)), and that FLIP(S) expressio

132 n-1-beta-converting enzyme [FLICE/caspase 8]-inhibitory protein (FLIP) activates NF-kappaB more poten

133 down-regulated cellular levels of FLICE-like inhibitory protein (FLIP) and X-chromosome-linked inhibi

134 FLICE-inhibitory protein (FLIP) blocks death receptor-mediated

135 O and concomitant decrease in cellular FLICE inhibitory protein (FLIP) expression without significant

136 The viral FLICE-like inhibitory protein (FLIP) protein from Kaposi sarcoma-as

137 ein-like interleukin-1beta-converting enzyme inhibitory protein (FLIP) were all down-regulated by ind

138 rleukin-1beta-converting enzyme (FLICE)-like inhibitory protein (FLIP), and reduced FLIP expression p

139 feration when the caspase 8 inhibitor, FLICE-inhibitory protein (FLIP), is active.

140 interleukin-1beta-converting enzyme (FLICE) inhibitory protein (FLIP), leading to apoptosis.

141 in the presence of CDDP through a FLICE-like inhibitory protein (FLIP)-Itch interaction.

142 ates with decreased expression of FLICE-like inhibitory protein (FLIP).

143 ROS) generation and down-regulation of FLICE inhibitory protein (FLIP); however, the relationship bet

144 rylation of AKT and activation of FLICE-like-inhibitory-protein (FLIP).

145 FLICE-inhibitory proteins (FLIPs) are a family of viral (poxvi

146 Caspase-8/10 and FLICE/caspase-8 inhibitory proteins (FLIPs) that inhibit caspase activat

147 C20) but not of protein kinase C-potentiated inhibitory protein for myosin phosphatase of 17 kDa (CPI

148 Inhibitory proteins from the serum were determined as ne

149 atment caused a rapid abrogation of the ATGL inhibitory protein G0S2.

150 We find that GPR151 couples to the G-alpha inhibitory protein Galpha(o1) to reduce cyclic adenosine

151 protein (cpIAP) or cellular FLIP (FLICE-like inhibitory protein) gene restored the WT latency reactiv

152 Glucokinase (GK) and its inhibitory protein, GK regulatory protein (GKRP), were c

153 hus, the novel activity was termed GlcNAcT-I inhibitory protein (GnT1IP).

154 epelled by cornea, and presence of Robo-Slit inhibitory protein had no effect.

155 and therapy with various systemic complement-inhibitory proteins has been investigated in this model

156 atory and nuclear factor kappa B (NF-kappaB) inhibitory protein, has established pro-proliferative pr

157 beta-Lactamase inhibitory protein-II (BLIP-II) is a potent proteinaceou

158 (a degradation-resistant mutant of NF-kappaB inhibitory protein IkappaB alpha) in myotubes blocked th

159 nhibit NF-kappaB activity by stabilizing the inhibitory protein IkappaB alpha, raising the possibilit

160 nd by silencing and overexpressing NF-kappaB inhibitory protein IkappaB expression, we demonstrate th

161 t proceeded through specific isoforms of the inhibitory protein IkappaB mediated these diverse respon

162 Expression of the NF-kappaB inhibitory protein, IkappaB alpha, was increased in old

163 e phosphorylation and destabilization of its inhibitory protein, IkappaB-alpha.

164 d degradation, and reduced mRNA level of the inhibitory protein IkappaBA followed by the translationa

165 lB dissociates from its interaction with the inhibitory protein IkappaBalpha and binds to the promote

166 ctor NF-kappaB involves its release from the inhibitory protein IkappaBalpha in the cytoplasm and sub

167 ylation of nuclear factor-kappaB (NF-kappaB) inhibitory protein IkappaBalpha, (c) phosphorylation of

168 ociated with the enhanced degradation of the inhibitory proteins IkappaBalpha and IkappaBbeta but not

169 fect on the phosphorylation of the NF-kappaB inhibitory protein, IkappaBalpha; on the nuclear translo

170 Finally, the specific role of the cRel/p65 inhibitory protein IkappaBbeta was evaluated.

171 e distinctive physiological roles of the two inhibitory proteins IkappaBbeta and IkappaBalpha.

172 r investigated the use of a novel complement inhibitory protein in a therapeutic paradigm.

173 Acm1 was reported to be a Cdh1-binding and -inhibitory protein in budding yeast.

174 otic effectors such as Bcl-2, p53, and FLICE inhibitory protein in cancer cell anoikis is also discus

175 main protein-like IL-1beta-converting enzyme-inhibitory protein in wild-type MEF but not in PKR-/- ce

176 ve stress, the role of the IkappaB family of inhibitory proteins in modulating this activity remains

177 following injury; regeneration is blocked by inhibitory proteins in myelin, such as myelin-associated

178 saliva and support important roles for these inhibitory proteins in the modulation of PMN function in

179 ory (Th2i) cells that express high levels of inhibitory proteins including IL-10, CTLA-4, and granzym

180 mechanisms such as the repression of growth-inhibitory proteins, including JunD.

181 ed herpesvirus (KSHV) K13/vFLIP (viral Flice-inhibitory protein) induces transcription of numerous ge

182 Treatment with locostatin, an Raf kinase inhibitory protein inhibitor, induced T cell anergy by b

183 ovo H protein synthesis, suggesting that the inhibitory proteins interfere with the wild-type H prote

184 stimulation induced expression of the IRAK-1 inhibitory protein IRAK-M.

185 Phosphorylation of the inhibitory proteins is mediated by an IkappaB kinase (IK

186 c-FLIP (cellular FLICE inhibitory protein) is an enzymatically inactive relativ

187 ain-like interleukin 1beta-converting enzyme inhibitory protein) is an inhibitor of death receptor-me

188 Cellular FLIP (Flice-like inhibitory protein) is critical for the protection again

189 Loss of the liver-enriched inhibitory protein isoform results in increased ATF4 mRN

190 C cells express primarily the liver-enriched inhibitory protein isoform.

191 iated herpesvirus (KSHV)-encoded viral FLICE inhibitory protein K13 interacts with a cytosolic Ikappa

192 main protein-like IL-1beta-converting enzyme inhibitory protein K13 is sufficient to induce spindle c

193 h the expression of KSHV-encoded viral FLICE inhibitory protein K13.

194 tion or on ectopic expression of viral FLICE inhibitory protein K13.

195 idence was provided that PP2A counteracts an inhibitory protein kinase that phosphorylates and inacti

196 (also called ITGB4BP or p27BBP), a ribosome inhibitory protein known to prevent productive assembly

197 ein-like interleukin-1beta-converting enzyme inhibitory protein-like inhibitory protein], and metasta

198 te gene expression, while the liver-enriched inhibitory protein (LIP) isoform negatively regulates la

199 esponse to UV stress, and the liver-enriched inhibitory protein (LIP) isoform of C/EBPbeta, but not t

200 hat PIAS1 interacted with the liver-enriched inhibitory protein (LIP) region of C/EBPbeta.

201 Of the C/EBPbeta isoforms, liver inhibitory protein (LIP) was notably induced and liver-a

202 fied an isoform of C/EBPbeta, liver-enriched inhibitory protein (LIP), as a previously unrecognized t

203 eferred to as liver-enriched transcriptional inhibitory protein (LIP).

204 e dominant-negative C/EBPbeta isoform, liver inhibitory protein (LIP).

205 oteolytically active complex with FLICE-like inhibitory protein long (FLIP(L), also known as CFLAR),

206 at the antiapoptotic molecule cellular-FLICE-inhibitory protein long isoform [c-FLIP(L)] is necessary

207 ation and proteasomal degradation of the Myc inhibitory protein Mad1.

208 y decreases the expression of the complement inhibitory protein MCP on quiescent EC, but does not ind

209 lways accompanied by upregulation of the p53-inhibitory protein MDM2 and/or phosphorylation of MDM2 a

210 nt phosphorylation of migration and invasion inhibitory protein (MIIP) at Ser303; this phosphorylatio

211 phil-activating peptide (ENA)-78, macrophage inhibitory protein (MIP)-1alpha, MIP-1beta, monocyte che

212 rophage-colony-stimulating factor, migratory inhibitory protein [MIP]-1 alpha , MIP-2, MIP-3 alpha ,

213 , only the phosphorylation of the cell cycle inhibitory proteins MOBKL1A/B is lost entirely in TCR-st

214 analyzed ligand recognition by NLR apoptosis inhibitory protein (NAIP) inflammasomes.

215 Murine NLR family, apoptosis inhibitory protein (Naip)1, Naip2, and Naip5/6 are host

216 ine-rich repeat-containing family, apoptosis inhibitory proteins (NAIPs) activate the nucleotide-bind

217 The NLR family apoptosis inhibitory proteins (NAIPs) bind conserved bacterial lig

218 P-2, -9, and -14 and decreased levels of the inhibitory proteins neurocan and CD44 within the retina.

219 ichiometrically complexed with the NF-kappaB inhibitory protein, NF-kappaB1 p105, and the ubiquitin-b

220 mes, such as the NAIP (NLR family, apoptosis inhibitory protein)/NLRC4 inflammasomes.

221 ng antibodies against the nerve-fiber growth inhibitory protein Nogo-A applied to rats with severe SC

222 ng antibodies against the nerve-fiber growth inhibitory protein Nogo-A applied to rats with severe sp

223 fter stroke is neutralization of the neurite inhibitory protein Nogo-A.

224 R1) is a promiscuous receptor for the myelin inhibitory proteins Nogo/Nogo-66, myelin-associated glyc

225 d phosphorylation of nuclear factor-kappaB's inhibitory protein, nuclear factor-kappaB translocation

226 Protein kinase C (PKC)-potentiated inhibitory protein of 17 kDa (CPI-17) inhibits myosin li

227 and IKKbeta) that phosphorylate IkappaB, an inhibitory protein of NF-kappaB.

228 hosphorylation of the 17-kDa PKC-potentiated inhibitory protein of type 1 protein phosphatase (CPI-17

229 expression and overexpression of complement inhibitory proteins on MM target cells as well as DARA-i

230 Mitochondrial-targeted CaMKII inhibitory protein or cyclosporin A, an mPTP antagonist

231 ition of activin A by follistatin, a natural inhibitory protein, or by a specific blocking Ab, result

232 xpression of calpastatin, a specific calpain-inhibitory protein, or small interfering RNA-mediated kn

233 Phosphorylation of cell cycle-inhibitory protein p21(cip1), one of the downstream targ

234 ranscriptional stimulation of the cell-cycle inhibitory protein p21(Waf1/Cip1) Consistently, Foxp1 ac

235 the nucleus, and down-regulation of the CDK2 inhibitory protein p27 in the nucleus.

236 yampholytic C-terminal IDR of the cell cycle inhibitory protein p27(Kip1) (p27).

237 isordered region (p27 IDR) of the cell cycle inhibitory protein p27(Kip1).

238 a tolerant state that is enforced by the CDK inhibitory protein p27kip1.

239 The MAD2 inhibitory protein p31(comet) promotes checkpoint inacti

240 ia mutated (ATM) as models to isolate growth inhibitory proteins, peptides and antisense RNAs, and te

241 nine, the allosteric inhibitor, 2) prevented inhibitory protein phosphorylation, or 3) mimicked allos

242 uces protein kinase C-dependent Raf-1 kinase inhibitory protein phosphorylation, sensitization of cho

243 Knockdown of cellular FLICE inhibitory protein potentiates the caspase-dependent pat

244 domain-like interleukin-1-converting enzyme inhibitory protein preventing Fas-mediated apoptosis.

245 in-like interleukin-1 beta-converting enzyme-inhibitory protein), proliferation (c-myc, cyclin D1, an

246 ocks the action of several myelin-associated inhibitory proteins promotes relatively unrestricted reg

247 IL R1, Fas-associated death domain and FLICE-inhibitory protein proteins.

248 tions, we tried to identify the sequences of inhibitory proteins purified from venoms by searching th

249 its properties, we have re-named ORF145 RNAP Inhibitory Protein (RIP).

250 Raf kinase inhibitory protein (RKIP or PEBP) is an inhibitor of the

251 iously showed that the suppressor Raf kinase inhibitory protein (RKIP) inhibits breast tumour metasta

252 Raf kinase inhibitory protein (RKIP) is a physiologic inhibitor of

253 Raf kinase inhibitory protein (RKIP) negatively regulates the MAP k

254 of metastasis suppressors such as Raf kinase inhibitory protein (RKIP), which inhibits tumor invasive

255 urveillance cancer gene product Raf-1 kinase inhibitory protein (RKIP).

256 gered swap of protein partners by Raf Kinase Inhibitory Protein (RKIP).

257 ork for the metastasis suppressor Raf kinase inhibitory protein (RKIP).

258 Raf kinase inhibitory protein (RKIP/PEBP1), a member of the phospha

259 ts of the metastasis suppressor Raf-1 kinase inhibitory protein (RKIP/PEBP1), we utilized an integrat

260 Raf kinase inhibitory protein (RKIP; also known as phosphatidyletha

261 ar FLICE (FADD-like IL-1b-converting enzyme)-inhibitory protein short, protected cells from the drug

262 in)-like interleukin-1beta-converting enzyme inhibitory protein, short form (c-FLIP-s) blocked enhanc

263 Expression of cellular FLICE-like inhibitory protein-short did not significantly inhibit c

264 CS family genes encoding the STAT signalling inhibitory proteins SOCS1, SOCS3 and CISH were marked by

265 and overrides the accumulation of cell cycle inhibitory proteins, such as p16INK4a.

266 -kappaB and phosphorylation of IkappaB alpha inhibitory protein suggesting that PAP2 signaling involv

267 of a novel, relatively small, broad-acting C inhibitory protein (termed OmCI) from the soft tick Orni

268 ) genomes encode a homolog of cellular FLICE-inhibitory proteins (termed v-FLIP) that activates NF-ka

269 ergoes alternative translation to produce an inhibitory protein that blocks TREK channels, leading to

270 f mast cells by downregulating Srcin1, a Src-inhibitory protein that counteracts FcvarepsilonRI signa

271 ingly, these mutations convert FLASH into an inhibitory protein that reduces in vitro processing effi

272 NF-kappaB is restricted to the cytoplasm by inhibitory proteins that are degraded when specifically

273 its the critical period for learning through inhibitory proteins that suppress axon sprouting and syn

274 activation through increased binding of the inhibitory protein thioredoxin (TXN/TRX-1/Trx), resultin

275 numbers of CD8(+) T cells that expressed the inhibitory protein TIGIT; these effects were not observe

276 NF-kappaB pathway involves degradation of an inhibitory protein, TNF receptor-associated factor 3 (TR

277 st that the wild-type protein transports the inhibitory protein to the pathway.

278 the identified genes, MBIP (MAP3K12 binding inhibitory protein), towards driving tumor invasion and

279 mmon strategy entails the transfer of growth-inhibitory protein toxins between competing cells.

280 is mediated through regulation of the mTORC1-inhibitory proteins, TSC1 and TSC2.

281 Here, we tested whether a defect in LYN, an inhibitory protein tyrosine kinase that is implicated in

282 is regulated by the KSHV-encoded viral FLICE inhibitory protein (vFLIP) and by viral IFN regulatory f

283 in-like interleukin-1beta-converting enzyme) inhibitory protein (vFLIP) caused efficient apoptosis in

284 iated herpesvirus (KSHV)-encoded viral FLICE inhibitory protein (vFLIP) enhances IRF4-mediated gene i

285 re, we have revealed the role of viral FLICE-inhibitory protein (vFLIP) in the initiation of PEL and

286 ain-like interleukin-1beta-converting enzyme-inhibitory protein (vFLIP) increased SQSTM1 expression a

287 ted death domain-like IL-1-converting enzyme inhibitory protein (vFLIP) is one of the few viral prote

288 terized the role of KSHV-encoded viral FLICE inhibitory protein (vFLIP) K13 in the modulation of anti

289 iated herpesvirus (KSHV)-encoded viral FLICE inhibitory protein (vFLIP) K13 is a potent activator of

290 a-associated herpesvirus-encoded viral FLICE inhibitory protein (vFLIP) K13 was originally believed t

291 ath domain-like IL- beta 1-converting enzyme inhibitory protein (vFLIP) K13.

292 l interleukin-6 (vIL-6) and viral FLICE-like inhibitory protein (vFLIP) transcripts.

293 herpesvirus 8 (HHV-8) encodes a viral FLICE inhibitory protein (vFLIP), called K13, with homology to

294 of B lymphocytes by KSHV-encoded viral FLICE-inhibitory protein (vFLIP).

295 ,5'-oligoadenylate synthetase (OAS-1), Virus inhibitory protein (viperin), ISG15 and ISG56.

296 The level of Socs3, the endogenous Stat3 inhibitory protein, was higher in Gprc5a(+/+) than in Gp

297 eron induced mortality-19 (GRIM-19), a STAT3-inhibitory protein, was isolated as a growth-suppressive

298 change and size exclusion chromatography, an inhibitory protein which exhibits significant similarity

299 upted by Rho guanine nucleotide dissociation inhibitory proteins, which are negative regulators of Cd

300 w that increased expression of the apoptosis inhibitory protein XIAP contributes to anoikis resistanc