ライフサイエンスコーパス: inhibitory synapse

1 nnections (i.e., gap junctions or excitatory/inhibitory synapses).

2 ediating the anchoring of GABAA receptors at inhibitory synapses.

3 s contributed more to bursting behavior than inhibitory synapses.

4 )/calmodulin-dependent protein kinase II) at inhibitory synapses.

5 ing the dynamics of GABAARs and stability of inhibitory synapses.

6 ected differently by VACCs at excitatory and inhibitory synapses.

7 and increasing the density of excitatory and inhibitory synapses.

8 esponding with high densities of glycinergic inhibitory synapses.

9 fference between NLGN2 and 3 specifically at inhibitory synapses.

10 neurons contain intermingled excitatory and inhibitory synapses.

11 raction and switches TEN function to specify inhibitory synapses.

12 e the formation and functional maturation of inhibitory synapses.

13 h release probability (Pr) vesicles at these inhibitory synapses.

14 rally balanced development of excitatory and inhibitory synapses.

15 ruitment of presynaptic neurotransmitters at inhibitory synapses.

16 tic clefts that exist at both excitatory and inhibitory synapses.

17 g of the mechanisms neurons use to construct inhibitory synapses.

18 napses while NLGN3 is at both excitatory and inhibitory synapses.

19 lagen XIX-in the formation of Parvalbumin(+) inhibitory synapses.

20 tail that are necessary for NLGN function at inhibitory synapses.

21 ded from, but was evenly distributed across, inhibitory synapses.

22 n the Schaffer collateral pathway-associated inhibitory synapses.

23 t, much less is known about Abeta effects at inhibitory synapses.

24 g this exquisite alignment at the C. elegans inhibitory synapses.

25 nteracts with subtypes of GABAA receptors in inhibitory synapses.

26 uires balanced development of excitatory and inhibitory synapses.

27 the trafficking and synthesis of GABAARs at inhibitory synapses.

28 for further proteomic and imaging studies of inhibitory synapses.

29 endent homeostatic regulation of hippocampal inhibitory synapses.

30 n and glycosylation, and its localization to inhibitory synapses.

31 odulation of SV clustering and plasticity of inhibitory synapses.

32 are essential for homeostatic plasticity at inhibitory synapses.

33 was caused by an action of extinction on BA inhibitory synapses.

34 on constraints on KCC2 at excitatory but not inhibitory synapses.

35 synaptic ribbons and impairs the function of inhibitory synapses.

36 with modifications in the number and size of inhibitory synapses.

37 se development with a unique selectivity for inhibitory synapses.

38 onnected to a hub neuron with electrical and inhibitory synapses.

39 ic long-term plasticity induced by mGluRs at inhibitory synapses.

40 ation can alter the functional properties of inhibitory synapses.

41 e postsynaptic density), a characteristic of inhibitory synapses.

42 sters in the vicinity of both excitatory and inhibitory synapses.

43 in NLG-1, itself a postsynaptic organizer of inhibitory synapses.

44 sticity rules across multiple excitatory and inhibitory synapses.

45 o be determined whether this pathway affects inhibitory synapses.

46 reveal a previously unknown role for MET at inhibitory synapses.

47 further investigation of the role of MET at inhibitory synapses.

48 In all layers, we observe the loss of large inhibitory synapses.

49 reby decreased the confinement of GABAARs at inhibitory synapses.

50 the accumulation of GABAARs and gephyrin at inhibitory synapses.

51 nisms of neurotransmission at excitatory and inhibitory synapses.

52 nifying mechanism for development of diverse inhibitory synapses.

53 ermit rapid crosstalk between excitatory and inhibitory synapses.

54 atory and inhibitory synapses and facilitate inhibitory synapse adaptations.

55 Although inhibitory synapses also transmit signals spontaneously

56 Inhibitory synapse and feed-forward circuit defects are

57 ults support the dysregulation of the DGC at inhibitory synapses and altered neuronal network activit

58 sion arises from both an increased number of inhibitory synapses and an enhancement of presynaptic GA

59 ntified type-A GABA receptors (GABA(A)Rs) in inhibitory synapses and determined their in situ structu

60 can protect the adult brain by migrating to inhibitory synapses and displacing them from cortical ne

61 s providing new insight into the function of inhibitory synapses and extrasynaptic receptors in contr

62 oride transporter function at excitatory and inhibitory synapses and facilitate inhibitory synapse ad

63 nce of spontaneous release at excitatory and inhibitory synapses and heterogeneity of the mechanisms

64 ransmembrane protein, localizes at GABAergic inhibitory synapses and interacts with GABA(A)Rs.

65 nduction of long-term plasticity at distinct inhibitory synapses and its regulation of hippocampal ne

66 how that Syt1 is weakly coexpressed at these inhibitory synapses and must be genetically inactivated

67 napses, with neuroligin 2 (NLGN2) limited to inhibitory synapses and neuroligin 1 (NLGN1) dominating

68 distal networks recruit both excitatory and inhibitory synapses and result in divisive normalization

69 relevant Ca(2+) sensor(s) at fast-releasing inhibitory synapses and shows that two major Syt isoform

70 y visual experience is necessary to maintain inhibitory synapses and stabilize RFs in adulthood (Carr

71 current concepts of relevant complexities of inhibitory synapses and the importance of synaptic inhib

72 The number of inhibitory synapses and the ratio of cholecystokinin to

73 selectively strengthened excitatory, but not inhibitory, synapses and facilitated SD in both the neoc

74 nd cell cycle genes to disrupted adult glia, inhibitory synapses, and behavior suggests a mechanism f

75 omeostatic plasticity in both excitatory and inhibitory synapses, and impairment of this overall proc

76 To clarify adenosine's role at inhibitory synapses, and in excitation-inhibition balanc

77 naptic release of GABA, trigger formation of inhibitory synapses, and promote axonal outgrowth in inh

78 ly important Ca(2+) sensor at fast-releasing inhibitory synapses, and show that Syt1 and Syt2 can red

79 al morphogenesis, assembly of excitatory and inhibitory synapses, and synaptic refinement.

80 pha to induce presynaptic differentiation of inhibitory synapses, and that mice lacking IgSF21 exhibi

81 tes the dynamics and function of perisomatic inhibitory synapses, and they identify a CaMKII-dependen

82 rotransmitter release at both excitatory and inhibitory synapses, and they serve a critical role in t

83 Recent studies indicate that inhibitory synapses are also plastic and are controlled

84 maintenance, and activity of excitatory and inhibitory synapses are essential for neuronal network f

85 When both excitatory and inhibitory synapses are examined together in the same ne

86 However, the properties of retinal inhibitory synapses are less well known.

87 Defects in inhibitory synapses are linked to neurodevelopmental dis

88 lar mechanisms regulating the development of inhibitory synapses are poorly understood.

89 propose a general mechanism by which mature inhibitory synapses are strengthened.

90 ptors, we conclude that only a subset of all inhibitory synapses are strengthened.

91 by the same neural system if excitatory and inhibitory synapses are tuned appropriately.

92 GABAA receptors and inhibitory synapses are vital components of brain functi

93 nd Nlgn2, ligands that occur specifically at inhibitory synapses, are incompletely understood.

94 aradoxically regulates both the formation of inhibitory synapses as well as the development of excita

95 of inhibition is mediated by changes at both inhibitory synapses, as well as excitatory synapses on i

96 Abnormal excitatory and inhibitory synapse assembly and elevated expression of t

97 ergic synapses in vivo without affecting the inhibitory synapses assessed by miniature excitatory pos

98 the A1-mediated suppression, at about 50% of inhibitory synapses at pyramidal neurons.

99 It is unknown whether inhibitory synapses at these two cellular compartments o

100 ns were generated by reciprocal excitatory / inhibitory synapses, at locations as early as the cone-h

101 are involved in the final maturation of GABA inhibitory synapses before birth.

102 rigger of spontaneous release at neocortical inhibitory synapses but not at excitatory synapses, sugg

103 activation profoundly depresses PAG and RMTg inhibitory synapses but prevents synaptic plasticity onl

104 e neuropeptide hormone, insulin, strengthens inhibitory synapses by recruiting alpha6-containing GABA

105 We show that homeostatic plasticity in inhibitory synapses can align the functional connectivit

106 tudy indicates that glutamate spillover onto inhibitory synapses can directly interact with glycine r

107 ction at excitatory synapses, however, RA at inhibitory synapses causes a loss instead of the gain of

108 he maturation and function of excitatory and inhibitory synapses, causing deficits in synaptic struct

109 ntially alters responses from excitatory and inhibitory synapses, causing the I/E ratio to change as

110 This inhibitory synapse combines the activity of an unusual c

111 s slowed down and confined at excitatory and inhibitory synapses compared with extrasynaptic regions.

112 asic alteration in expression of hippocampal inhibitory synapse components in AD.

113 Inhibitory synapses comprise only approximately 20% of t

114 isoforms, as NL1 can also induce functional inhibitory synapse connections when the presynaptic inte

115 servation by examining the role of two other inhibitory synapse constituents, vesicular GABA transpor

116 Inhibitory synapses dampen neuronal activity through pos

117 ndocannabinoid (eCB)-mediated suppression of inhibitory synapses, decreased it at excitatory synapses

118 GABAB receptor-mediated inhibitory synapse defects are circuit-specific and are

119 or gene expression, they cause a decrease in inhibitory synapse density onto excitatory hippocampal n

120 in cultured rat hippocampal neurons reduced inhibitory synapse density without altering excitatory s

121 During maturation, inhibitory synapse development requires both NL2 and Sli

122 turbation of the NL2-ST3 interaction impairs inhibitory synapse development with consequent disruptio

123 olecules have been identified to function in inhibitory synapse development, it remains unknown wheth

124 ting protein, can co-regulate excitatory and inhibitory synapse development, offering a putative evol

125 IgSF9(DeltaC/DeltaC) mice) had no defects in inhibitory synapse development, providing genetic eviden

126 esion and organization mechanisms underlying inhibitory synapse development.

127 ttle is known about the molecular control of inhibitory synapse development.

128 lic synaptic adhesion protein that regulates inhibitory synapse development.

129 l molecular mechanism underlying hippocampal inhibitory synapse development.

130 tic evidence for a specific role of IgSF9 in inhibitory synapse development/maintenance, presumably b

131 y which FGF22 and FGF7 induce excitatory and inhibitory synapse differentiation are unknown.

132 ted by a decrease in an outward current from inhibitory synapses (disinhibition) combined with an inc

133 onism of NK cells is an active phenomenon of inhibitory synapse disruption.

134 c ephrin-B1 to influence both excitatory and inhibitory synapses during development can potentially c

135 c ephrin-B1 to influence both excitatory and inhibitory synapses during development can potentially c

136 tablish a balanced network of excitatory and inhibitory synapses during development for the brain to

137 The starkest contrast between excitatory and inhibitory synapse dynamics is on dually innervated spin

138 Our results suggest that the inhibitory synapse dysfunction in the cortico-hippocampa

139 .g., depotentiation) or induce plasticity at inhibitory synapses (e.g., long-term potentiation) to su

140 and non-linear interaction of excitatory and inhibitory synapses enables the somatic voltage to encod

141 synaptic activity, affecting excitatory and inhibitory synapses equally.

142 TA-associated inhibitory synapses exhibited increase in paired-pulse r

143 Although many types of inhibitory synapses exist, these disorders have been str

144 These results indicate that dorsal horn inhibitory synapses follow different rules of organizati

145 ptimal ratio of the number of excitatory and inhibitory synapses for maximizing the encoding capacity

146 In this scenario, alpha1-GABARs resident to inhibitory synapses form the hardwiring of neuronal circ

147 ism by which the Cb(R290H) mutation perturbs inhibitory synapse formation and causes brain dysfunctio

148 yaluronan impacts the ratio of excitatory to inhibitory synapse formation and the resulting neural ac

149 ders, but the molecular processes underlying inhibitory synapse formation are not well understood.

150 stic understanding of precise excitatory and inhibitory synapse formation in the mammalian brain.

151 and FGF7 specifically promote excitatory or inhibitory synapse formation, respectively.

152 f excitatory synapses, while also decreasing inhibitory synapse formation.

153 3 interaction and impairs excitatory but not inhibitory synapse formation.

154 ells that regulates targeted axon growth and inhibitory synapse formation.

155 s novel insights into the dynamic process of inhibitory synapse formation.

156 naling molecule Semaphorin4D (Sema4D) during inhibitory synapse formation.

157 g fibers on Purkinje cells in cerebellum and inhibitory synapses formed by parvalbumin- or somatostat

158 ders have been strongly linked to defects in inhibitory synapses formed by Parvalbumin-expressing int

159 namics is on dually innervated spines, where inhibitory synapses frequently recur while excitatory sy

160 The inhibitory synapses from different pattern-generating ne

161 Here, we show that inhibitory synapses from parvalbumin and somatostatin ex

162 mate receptor transporter vGluT2 and receive inhibitory synapses from striatal neurons, and many also

163 1 adenosine receptor activation and enhanced inhibitory synapses from the lateral subdivision of the

164 s that control the balance of excitatory and inhibitory synapse function remain poorly understood; no

165 Defects in excitatory and inhibitory synapses have been implicated in schizophreni

166 the IgSF9 gene resulted in fewer functional inhibitory synapses; however, the strength of the remain

167 tion and size of gephyrin-tagged clusters at inhibitory synapses identified by correlated confocal el

168 itter levels dynamically set the strength of inhibitory synapses in a release-independent manner.

169 the structural and functional alterations of inhibitory synapses in AD are less well characterized.

170 est that mGlu7 serves as a heteroreceptor at inhibitory synapses in area CA1 and that the predominant

171 Inhibitory synapses in brain also present a particular c

172 o-regulate the development of excitatory and inhibitory synapses in cortical pyramidal neurons in viv

173 se and cocaine-stimulated 2-AG-modulation of inhibitory synapses in DA neurons.

174 breakdown of anandamide, suppressed >50% of inhibitory synapses in females with no effect in males,

175 subunits were reduced and less clustered at inhibitory synapses in Gabrb3+/- knockout mice.

176 on and distribution of proteins specific for inhibitory synapses in hippocampal areas of APPPS1 mice

177 an organizer of ligand-gated ion channels at inhibitory synapses in hippocampus CA1 and dentate gyrus

178 activity-evoked release at two types of fast inhibitory synapses in mouse brain.

179 gephyrin reorganization during plasticity of inhibitory synapses in mouse hippocampal cultured neuron

180 ified YFP signal localizing appropriately to inhibitory synapses in multiple brain regions including

181 tatory synapses, but the Ca(2+) sensor(s) at inhibitory synapses in native brain tissue are not well

182 Cadherin-10 localizes to both excitatory and inhibitory synapses in neocortex, where it is organized

183 e, GABAA receptor composition was altered at inhibitory synapses in Np(-/-) neurons as the alpha1 to

184 rols E/I ratios by regulating excitatory and inhibitory synapses in opposing directions.SIGNIFICANCE

185 We found that NLGN3 function at inhibitory synapses in rat CA1 depends on the presence o

186 n the Schaffer collateral pathway-associated inhibitory synapses in stratum radiatum.

187 n2 is required for continuous maintenance of inhibitory synapses in the adult mPFC, and that chronic

188 2 (Robo2) is critical for the maintenance of inhibitory synapses in the adult ventral tegmental area

189 Whether TRPV1 also regulates inhibitory synapses in the brain is unclear.

190 dulating the strength of both excitatory and inhibitory synapses in the brain, but by different mecha

191 f neuromuscular synapses, is also present at inhibitory synapses in the central nervous system.

192 o led to an imbalance between excitatory and inhibitory synapses in the cerebral cortex.

193 f CB1 receptor-immunopositive excitatory and inhibitory synapses in the inner one-third of the dentat

194 c factor (BDNF) in the assembly of GABAergic inhibitory synapses in the mouse cerebellar cortex.

195 GABA(B)R)-dependent short-term depression of inhibitory synapses in the nucleus reticularis thalami,

196 infiltration and preferential elimination of inhibitory synapses in the ventral thalamus, which lead

197 The results show that: 1) the strength of inhibitory synapses in vivo can be enhanced by increasin

198 tion of neuroligin-2, a key component of the inhibitory synapse, in the NAc that modifies behavioral

199 ately 50% of glutamatergic synapses, but not inhibitory synapses, in culture.

200 ver, the Ca(2+) sensor(s) used by identified inhibitory synapses, including the output synapses of pa

201 Such reciprocal inhibitory synapses increased the LGMD's selectivity for

202 How differences in the spatial targeting of inhibitory synapses interact with intracellular chloride

203 Conceptually, plasticity of inhibitory synapses is an attractive mechanism to allow

204 In fact, perturbing inhibitory synapses is associated with complex brain dis

205 has a role in the establishment of afferent inhibitory synapses is debated.

206 We show that LTD at inhibitory synapses is dependent on downstream cAMP/prot

207 regarding receptor mobility and function at inhibitory synapses is derived indirectly from using rec

208 Coordinated development of excitatory and inhibitory synapses is essential for higher brain functi

209 tate their targeting to and stabilization at inhibitory synapses is rudimentary.

210 lar events that take place when formation of inhibitory synapses is triggered by a specific signaling

211 phyrin, the principal scaffolding protein at inhibitory synapses, is essential for postsynaptic clust

212 o destabilization of GABAARs and gephyrin at inhibitory synapses, leading to reductions in the effica

213 sensory input-dependent plasticity, shortens inhibitory synapse lifetimes and lengthens intervals to

214 2), a postsynaptic cell-adhesion molecule of inhibitory synapses linked to neuropsychiatric disorders

215 The number of inhibitory synapses made by CCK(+)VGlut3(+) basket cells

216 5-GABA(A)R is preferentially targeted to the inhibitory synapses made by the vasoactive intestinal pe

217 c spines, whereas GABAA receptors cluster at inhibitory synapses mainly on the soma and dendritic sha

218 -10 forms nanoscale puncta at excitatory and inhibitory synapses, maintains excitatory and inhibitory

219 gene deletion enhances anatomical changes of inhibitory synapse markers after extinction training.

220 ated MDD patients is paralleled by decreased inhibitory synapse markers and decreased frequency of mi

221 were correlated with decreased expression of inhibitory synapse markers in IgSF9(-/-) mice, providing

222 ng activity of Cb and a consequent defect in inhibitory synapse maturation represent a likely molecul

223 Therefore, we hypothesized that inhibitory synapses may have different organizing princi

224 that the different effects on excitatory and inhibitory synapses may have resulted from off-target ac

225 system, we investigated the dynamics of the inhibitory synapse mediated by an inhibitory receptor, p

226 pendent transcription factor NPAS4 regulates inhibitory synapse number and function in cell culture,

227 this collagen results not only in decreased inhibitory synapse number, but also in the acquisition o

228 of presynaptic LRP4 reduces excitatory (not inhibitory) synapse number, impairs active zone architec

229 ndritic complexity, decreased excitatory and inhibitory synapse numbers, decreased intrinsic excitabi

230 CL1 selectively increases excitatory but not inhibitory synapse numbers, enhances excitatory but not

231 iding anatomical evidence for a reduction in inhibitory synapse numbers, whereas excitatory synapse d

232 GABA release is reduced in TA-associated inhibitory synapses of Fmr1 KO mice in a GABAB receptor-

233 We find that dynamic inhibitory synapses often disappear and reappear again i

234 onversely, presynaptic inhibition by KORs of inhibitory synapses on D2 MSNs enhances integration of e

235 The proportion of putative inhibitory synapses on dendritic shafts in the right MeP

236 tic sites (nociceptive afferent terminals vs inhibitory synapses on dorsal horn neurons).

237 Striatal medium spiny neurons (MSNs) form inhibitory synapses on neighboring striatal neurons thro

238 ve loss of striatal neurons and reduction of inhibitory synapses on pallidal neurons that serve as th

239 Motifs C4 and C5 form halos of 60-100 inhibitory synapses on proximal dendrites of AI gammaACs

240 or shafts (8%), with 2% symmetrical, likely inhibitory, synapses on shafts and spines.

241 , which was determined by the strength of an inhibitory synapse onto this neuron.

242 S-cone amacrine cell makes highly selective inhibitory synapses onto ipRGCs, resulting in a blue-OFF

243 spike-timing-dependent plasticity (STDP) of inhibitory synapses onto layer 5 neurons in slices of mo

244 ry reduced the strength of dynorphin-lineage inhibitory synapses onto mature lamina I spinoparabrachi

245 ncaging confirms MNTB neurons as a source of inhibitory synapses onto MOC neurons.

246 es of interneurons make functionally diverse inhibitory synapses onto principal neurons.

247 target-specific remodeling of BA perisomatic inhibitory synapses originating from parvalbumin and cho

248 activity-dependent adaptation of perisomatic inhibitory synapses over prolonged periods of time in hi

249 50% decrease of neurotransmitter release at inhibitory synapses, paralleled by a reduction in releas

250 This inhibitory synapse parallels IPS's inhibition of the sam

251 This inhibitory synapse parallels IRS's inhibition of the mic

252 Mullner et al. (2015) show that single inhibitory synapses placed in the right location on the

253 through which excitatory activity can affect inhibitory synapse plasticity.

254 Dynamic changes of the strength of inhibitory synapses play a crucial role in processing ne

255 ectively, these data reveal novel defects in inhibitory synapse protein expression during critical pe

256 BARs, which are ordinarily extrasynaptic, to inhibitory synapses, quashing further NMDA receptor acti

257 y temporarily expressing GFE3 we showed that inhibitory synapses regrow following ablation.

258 We show that the strength of inhibitory synapses relative to excitatory synapses can

259 can be imaged with a fluorescent cell fill, inhibitory synapses require a molecular tag.

260 The formation of excitatory and inhibitory synapses requires precise molecular control.

261 specific functional roles in excitatory and inhibitory synapses, respectively, but the molecular bas

262 lding proteins as proxies for excitatory and inhibitory synapses, respectively, to quantify the numbe

263 -side pairs results from a greater number of inhibitory synapses, revealing that an asymmetry in syna

264 K269Q-SynI expressing inhibitory synapses showed increased synaptic strength d

265 d reveal a novel role for presynaptic MET at inhibitory synapses.SIGNIFICANCE STATEMENT GABAergic syn

266 cadherin-10 reduces excitatory but increases inhibitory synapse size and strength, altering the E/I r

267 One such component is neuroligin-2 (NL2), an inhibitory synapse-specific cell surface protein that fu

268 t/stellate-cell synapse functions, such that inhibitory synapse-specific neuroligin-2 was unexpectedl

269 At inhibitory synapses, stochastic openings of VACCs trigge

270 od; no proteins that regulate excitatory and inhibitory synapse strength in a coordinated reciprocal

271 At fast inhibitory synapses, strengthening is thought to occur b

272 ocytotic cycle of vesicles at excitatory and inhibitory synapses that accounts for all modes of vesic

273 Inhibitory synapses that form on pyramidal neuron dendri

274 indicating a reduction in the number of NAc inhibitory synapses that is correlated with depression-l

275 a-Nrxn affect transmission at excitatory and inhibitory synapses, the contribution of neurexophilin-1

276 At both excitatory and inhibitory synapses, the magnitude of A1R-mediated suppr

277 At inhibitory synapses, the theory suggests a statistically

278 namic regulation of GABA(A)R accumulation at inhibitory synapses, thereby regulating the strength of

279 inhibitory interneurons and the capacity of inhibitory synapses to be plastic make them ideal regula

280 hat, when fused to GFP, allow excitatory and inhibitory synapses to be visualized in living neurons.

281 uncta, a postsynaptic scaffolding protein at inhibitory synapses used here as a proxy for the granule

282 , on the relative location of excitatory and inhibitory synapses, voltage-dependent and -independent

283 n of presynaptic and postsynaptic markers of inhibitory synapses was markedly increased in the DG of

284 The increase in inhibitory synapses was prevented by inhibiting the inse

285 de of 2-AG-dependent long-term depression of inhibitory synapses was reduced.

286 Np(+/+) neurons, the ratio of excitatory to inhibitory synapses was significantly lower in Np(-/-) c

287 ols to glycine neurotransmitter receptors at inhibitory synapses, we find that gephyrin scaffolds act

288 e CB1 receptor-immunopositive excitatory and inhibitory synapses were Glu-CB1 -RS, 21.89% (glutamater

289 When Cx36 was deleted, inhibitory synapses were more numerous, although both di

290 ephyrin is the major instructive molecule at inhibitory synapses, where it clusters glycine as well a

291 Correspondingly, we find specific defects at inhibitory synapses, where NONO regulates synaptic trans

292 ay involves crosstalk between excitatory and inhibitory synapses whereby Ca(2+)-entering through post

293 e and triggers an increase in the density of inhibitory synapses, which is accompanied by enhanced ax

294 NLGN2 is only at inhibitory synapses while NLGN3 is at both excitatory an

295 regulates in vivo postsynaptic maturation of inhibitory synapses with contrasting modes of action spe

296 primary afferents, and form GABA-A-mediated inhibitory synapses with the host.

297 teral pyloric (LP) neuron makes reciprocally inhibitory synapses with the pacemakers.

298 Select pairs promote excitatory and inhibitory synapses, with neuroligin 2 (NLGN2) limited t

299 s arbors mature, they acquire excitatory and inhibitory synapses, with the latter forming first and b

300 etion of astrocytic NRCAM reduces numbers of inhibitory synapses without altering glutamatergic synap