戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (1語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 lbumin-expressing (PV) cells form the larger inhibitory system.
2 al horn neurons by activating the descending inhibitory system.
3 ts GTPCH by engaging the endogenous feedback inhibitory system.
4 modulation system from the LC and the opioid inhibitory system.
5 in the cortex may be especially dependent on inhibitory systems.
6 ation of central descending facilitatory and inhibitory systems.
7  key neurotransmitter of the endogenous pain inhibitory system, acutely inhibits nociceptive transmis
8                              Together, these inhibitory systems allow graded control of thalamic outp
9 otopic synapses is altered and suggests that inhibitory systems are enhanced in the malformed brain.
10 es in primates, we wanted to see whether the inhibitory systems are influenced by changes in cytoarch
11 nted that both the GABAergic and glycinergic inhibitory systems are up-regulated following complete s
12  BH4 and engages the GFRP-dependent feedback inhibitory system; at higher concentrations, DAHP compet
13  whether there is a decline of the GABAergic inhibitory system between middle and old age.
14 fic nodes within the PFC that interface with inhibitory systems can affect the negative bias, failure
15 emarkably robust and fast-acting glycinergic inhibitory system capable of suppressing calyceal transm
16 regulated dopaminergic system, a compromised inhibitory system, hypothalamic-pituitary-adrenal axis h
17 th controls, suggesting a reduced descending inhibitory system in CP.
18         The results suggestive of a specific inhibitory system in the control of drinking are unique.
19 ole of GABA in adaptive changes in a lateral inhibitory system in the tiger salamander retina.
20 gated the role of the vestibular commissural inhibitory system in vestibular compensation (VC, the be
21 mpare their relationship with excitatory and inhibitory systems in rhinal cortices.
22  direct interplay between the excitatory and inhibitory systems in the hippocampus.
23 to examine the excitability of two different inhibitory systems in the human motor cortex: short inte
24 he present study investigated the descending inhibitory system involvement in EA action.
25 ings indicate that an imbalanced commissural inhibitory system is a root cause of the severe oculomot
26  function during activation of the brainstem inhibitory system is related to both the descending inhi
27               While disruption of limbic HPA-inhibitory systems is implicated in stress-related psych
28                              Thus, these two inhibitory systems may complement each other by having d
29 -apoptotic effects via an endogenous caspase inhibitory system mediated by cellular inhibitor of apop
30 rs may enhance gamma-aminobutyric acid-ergic inhibitory systems or inhibit glutamatergic excitatory n
31 n inhibited by them, thus forming a mutually inhibitory system resembling what electrical engineers c
32 n the calyx of Held by a powerful, precision inhibitory system suggests that the relay function of th
33 s have highlighted an apparent difference in inhibitory systems; synaptic inhibition is thought to be
34 he data in this report, we conclude that the inhibitory system that mediates atonia during the state
35 e key elements of a brainstem-reticulospinal inhibitory system that participates in rapid eye movemen
36  of premotor cortex is weak, possibly due to inhibitory systems that can shape the flow of informatio
37             These neurons may be involved in inhibitory systems that normally suppress aberrant male-
38  Thalamic circuits are targeted by two major inhibitory systems: the thalamic reticular nucleus (TRN)
39 in, we explored the contribution of the GABA inhibitory system to A3AR-mediated analgesia using well-
40 romise of developing interventions targeting inhibitory systems to help slow cognitive declines in he
41    The co-ordination of these excitatory and inhibitory systems, together with intrinsic voltage-gate
42               An imbalance in the excitatory/inhibitory systems with abnormalities in the glutamaterg
43    PFC pathways that interface with distinct inhibitory systems within the cortex, the amygdala, or t