ライフサイエンスコーパス: innate lymphoid cell

1 pport tissue homeostasis and repair, such as innate lymphoid cells.

2 ndirectly through IL-13 production by goup 2 innate lymphoid cells.

3 -CCR2 chemokine axis and by IL-13 expressing innate lymphoid cells.

4 volving eosinophils, mast cells, and group 3 innate lymphoid cells.

5 omato-C3aR(-), except some LP-derived type 3 innate lymphoid cells.

6 sets of T cells, NK cells, and other group 1 innate lymphoid cells.

7 toid DCs, IL1B+ monocytes, and fewer group 1 innate lymphoid cells.

8 CD56+ granulocytes; and reductions in type 3 innate lymphoid cells.

9 for proper development of adaptive, but not innate, lymphoid cells.

10 ler (NK), lymphoid tissue inducer (LTi), and innate lymphoid cell 1 (ILC1) cells, but not ILC2 or ILC

11 iency does not alter the balance of NK cell/ innate lymphoid cell 1 generation and slightly decreases

12 integrin beta1, distinguishes NK cells from innate lymphoid cells 1 and other leukocytes.

13 zation and straightforward identification of innate lymphoid cell 2 progenitor populations.

14 Finally, the discovery of a prominent innate lymphoid cell-2 cluster links the single-cell RNA

15 ed on studies that predated the discovery of innate lymphoid cells-2 cells.

16 hils, dendritic cells, natural killer cells, innate lymphoid cells-2, and CD (cluster of differentiat

17 suggesting a strong atheroprotective role of innate lymphoid cells-2.

18 We report that MAIT cells repress group 2 innate lymphoid cell activation and restrict allergen-in

19 Increased permeability, IL-33 levels, type 2 innate lymphoid cell activation, and T(h)2 cell differen

20 adelta T cells, classic NK cells, and type 1 innate lymphoid cells), all of which can produce large a

21 Type 2 innate lymphoid cells and basophils were scarce in BAL f

22 in) by colon tissues, which activated type 2 innate lymphoid cells and dendritic cells to promote dif

23 L-33 then induces IL-13 secretion by group 2 innate lymphoid cells and enteroid gene expression consi

24 the numbers of eosinophils, IL-13(+) type 2 innate lymphoid cells and IL-13(+)CD4(+) T cells and IL-

25 primarily on mast cells, Th2 cells, group 2 innate lymphoid cells and regulatory T cells, and to a l

26 helial cells and antigen-presenting cells to innate lymphoid cells and regulatory T cells.

27 During development innate lymphoid cells and specialized lymphocyte subsets

28 lls resulted in loss of AHR-dependent type 3 innate lymphoid cells and T helper 17 cells and increase

29 ugh we observed increased numbers of group 3 innate lymphoid cells and Th17 cells and enhanced transc

30 n the absence of other inflammatory stimuli, innate lymphoid cells, and adaptive immunity.

31 osinophils, mast cells, T(H)2 cells, group 2 innate lymphoid cells, and antigen-presenting cells; and

32 (ER) stress, required extrinsic signals from innate lymphoid cells, and limited bacterial disseminati

33 sed together with CD14(+) CD16(+) monocytes, innate lymphoid cells, and natural killer cells.

34 Conversely, eosinophils, Th2 T cells, type 2 innate lymphoid cells, and possibly Foxp3+ Tregs protect

35 IL-5 and IL-13 coming from Th2 cells, type 2 innate lymphoid cells, and probably mast cells.

36 airway epithelial cells, macrophages, type 2 innate lymphoid cells, and TH2 cells along with increase

37 NK cell developmental intermediates, non-NK innate lymphoid cells, and the capacity for NK cells to

38 Finally, we provide evidence that group 2 innate lymphoid cells are a source of IL-13, which promo

39 Innate lymphoid cells are functionally diverse subsets o

40 ng cells, including Th17, gamma/delta T, and innate lymphoid cells, are differentially distributed al

41 Adipose type 1 innate lymphoid cells (AT1-ILCs) promote pro-inflammator

42 Natural killer (NK) cells are innate lymphoid cells being explored as they engage tumo

43 , ST2 deletion led to an overall increase in innate lymphoid cells (CD45(+)lin(-)CD25(+) cells) and I

44 ells) and lymphoid (natural killer cells and innate lymphoid cells) cell populations of the innate im

45 ctivated myeloid dendritic cells and group 2 innate lymphoid cells compared with mature TSLP.

46 In natural killer (NK) cells and other innate lymphoid cells, competent enhancers are primed du

47 are crucial at several stages of T cell and innate lymphoid cell development and differentiation.

48 7(+/-)CD94(+)NKp80(-)) NKDIs promoted non-NK innate lymphoid cell differentiation at the expense of N

49 aluated whether AHR antagonism could promote innate lymphoid cell differentiation from hESCs.

50 During early pregnancy, decidual innate lymphoid cells (dILCs) interact with surrounding

51 eficiency increased IL-5 production, group 2 innate lymphoid cell expansion, and host resistance to t

52 odeoxycholic acid induced intestinal group 3 innate lymphoid cell IL-22 secretion through GATA bindin

53 response (Th2 cells, M2 macrophages, type 2 innate lymphoid cells, IL-33, IL-4, IL-13, and mucus) th

54 ier gene mutations, Langerhans cells, type 2 innate lymphoid cells, IL-33, TSLP) that have important

55 olar ECs to suppress excessive activation of innate lymphoid cell (ILC) 2 during allergic lung inflam

56 cells and in ex vivo CD27(-)CD4(+) cells and innate lymphoid cell (ILC) 2 from patients with grass po

57 They are also required for innate lymphoid cell (ILC) development and maintenance,

58 s regulate early T cell progenitor (ETP) and innate lymphoid cell (ILC) development remains unclear.

59 We investigated NK cell and innate lymphoid cell (ILC) dynamics and function in rhes

60 Commitment to the innate lymphoid cell (ILC) lineage is determined by Id2,

61 al for commitment to both the T cell and the innate lymphoid cell (ILC) lineages in mammals.

62 ntial progenitor population specified toward innate lymphoid cell (ILC) lineages, but their relations

63 ously reported the selective loss of group 3 innate lymphoid cell (ILC) number and function in a Dock

64 Little is known about innate lymphoid cell (ILC) populations in the human gut,

65 Using multiparameter flow cytometry human innate lymphoid cell (ILC) subsets can be detected in th

66 promoted the development of tissue-resident innate lymphoid cell (ILC) subsets.

67 Although innate lymphoid cells (ILC) and natural killer (NK) cell

68 Innate lymphoid cells (ILC) are a heterogeneous family o

69 Innate lymphoid cells (ILC) are lymphocytes that lack an

70 Natural killer (NK) cells are a subset of innate lymphoid cells (ILC) capable of recognizing stres

71 We assessed eosinophils, T cells, Tregs, and innate lymphoid cells (ILC) from peripheral blood using

72 The signals that maintain tissue-resident innate lymphoid cells (ILC) in different microenvironmen

73 Innate lymphoid cells (ILC) play critical roles in regul

74 ing viral infection regulates the balance of innate lymphoid cells (ILC), a diverse class of lymphocy

75 Recently, several groups of innate lymphoid cells (ILC), distinct from NK cells in d

76 Group 2 innate lymphoid cells (ILC-2s) regulate immune responses

77 as a critical negative regulator of group 2 innate lymphoid cells (ILC-2s).

78 ng NK cells are known to convert to a type 1 innate lymphoid cell (ILC1)-like phenotype in response t

79 Here, we show that tissue-resident type 1 innate lymphoid cells (ILC1) serve an essential early ro

80 on and activation of CD3(-) NK1.1(+) group 1 innate lymphoid cells (ILC1) within the FRT, essential f

81 Group 1 innate lymphoid cells (ILC1s) are effectors of innate im

82 Among the features that distinguish type 1 innate lymphoid cells (ILC1s) from natural killer (NK) c

83 Although type 1 innate lymphoid cells (ILC1s) have been originally found

84 permanently "time stamp" NK cells and type 1 innate lymphoid cells (ILC1s) to characterize the dynami

85 IL-33 drives colonic group 2 innate lymphoid cell (ILC2) activation during infection

86 P and IL-33 synergistically promoted group 2 innate lymphoid cell (ILC2) activation to induce innate

87 , recombinant HpARI abrogated IL-33, group 2 innate lymphoid cell (ILC2) and eosinophilic responses t

88 e.Methods: Cytokines, viral load, and type 2 innate lymphoid cell (ILC2) levels in nasal aspirates, c

89 ugments Alternaria-induced pulmonary group 2 innate lymphoid cell (ILC2) responses and IL-33 release.

90 we demonstrate that helminth-induced group 2 innate lymphoid cell (ILC2) responses are exaggerated in

91 ammatory gene expression, and TH and group 2 innate lymphoid cell (ILC2) responses.

92 Here we identify a group 2 innate lymphoid cell (ILC2) subpopulation that can conve

93 competent repairing AAMs, as well as group-2 innate lymphoid cell (ILC2)-mediated AAM activation.

94 Here, we report that tissue-resident group 2 innate lymphoid cells (ILC2) accumulate in the choroid p

95 Type-2 innate lymphoid cells (ILC2) are a prominent source of t

96 Group 2 innate lymphoid cells (ILC2) are stimulated by IL-33 to

97 Group 2 innate lymphoid cells (ILC2) are tissue-resident innate

98 Group 2 innate lymphoid cells (ILC2) are tissue-resident, long-l

99 L233 increased the proportion of ST2-bearing innate lymphoid cells (ILC2) in blood and kidneys, and a

100 Type 2 innate lymphoid cells (ILC2) mediate inflammatory immune

101 Type 2 innate lymphoid cells (ILC2) share cytokine and transcri

102 one molecule), potentiated Tregs and group 2 innate lymphoid cells (ILC2) to prevent renal injury.

103 6.C3(Cg)-Rorasg/sg mice deficient in group 2 innate lymphoid cells (ILC2), and C57BL/6 wild-type mice

104 Group-2 innate lymphoid cells (ILC2), type-2 cytokines, and eosi

105 ived suppressor cells (M-MDSCs), and group 2 innate lymphoid cells (ILC2).

106 ) T cells expressing CRTh2), eosinophils and innate lymphoid cells (ILC2).

107 It also caused a reduction in innate lymphoid cell, ILC2, and IL-9(+) and IL-13(+) ILC

108 its impact on the differentiation of Group 2 Innate -Lymphoid -Cells (ILC2s) in the pathogenesis of a

109 associated with the expansion of lung type 2 innate lymphoid cells (ILC2s) and are dependent on IL-13

110 with the expansion of IL-13-producing type 2 innate lymphoid cells (ILC2s) and dependent on IL-25 and

111 translocation and release, increased type 2 innate lymphoid cells (ILC2s) and monocyte-derived dendr

112 Group 2 innate lymphoid cells (ILC2s) and type 2 helper T cells

113 Type-2 innate lymphoid cells (ILC2s) are a potent source of T-h

114 Group 2 innate lymphoid cells (ILC2s) are a potential innate sou

115 Type-2 innate lymphoid cells (ILC2s) are a recently discovered

116 Group 2 innate lymphoid cells (ILC2s) are a recently identified

117 ulators of allergic inflammation, and type 2 innate lymphoid cells (ILC2s) are an important cell type

118 The recently discovered group 2 innate lymphoid cells (ILC2s) are capable of secreting c

119 Group 2 innate lymphoid cells (ILC2s) are effective producers of

120 Group 2 innate lymphoid cells (ILC2s) are effector cells of type

121 Group 2 innate lymphoid cells (ILC2s) are effector cells within

122 Current models propose that group 2 innate lymphoid cells (ILC2s) are generated in the bone

123 Human type 2 innate lymphoid cells (ILC2s) are identified by coupled

124 Type 2 Innate lymphoid cells (ILC2s) are implicated in helminth

125 Group 2 innate lymphoid cells (ILC2s) are implicated in host def

126 Group 2 innate lymphoid cells (ILC2s) are important effector cel

127 Group 2 innate lymphoid cells (ILC2s) are important mediators of

128 Group 2 innate lymphoid cells (ILC2s) are involved in human dise

129 Group 2 innate lymphoid cells (ILC2s) are involved in the initia

130 Group 2 innate lymphoid cells (ILC2s) are key regulators of type

131 Group 2 innate lymphoid cells (ILC2s) are rare innate immune cel

132 Group 2 innate lymphoid cells (ILC2s) are the dominant innate ly

133 Type 2 innate lymphoid cells (ILC2s) are tissue sentinel mediat

134 Group 2 innate lymphoid cells (ILC2s) are tissue-resident cells

135 entified interleukin (IL)-9-producing type 2 innate lymphoid cells (ILC2s) as the mediators of a mole

136 Type 2 innate lymphoid cells (ILC2s) both contribute to mucosal

137 ell established, the newly identified type 2 innate lymphoid cells (ILC2s) can also contribute to orc

138 RATIONALE: Newly characterized type 2 innate lymphoid cells (ILC2s) display potent type 2 effe

139 orally controlled lineage tracing of group 2 innate lymphoid cells (ILC2s) during this period remains

140 Group 2 innate lymphoid cells (ILC2s) expand in the lungs of mic

141 versely associated with the number of type 2 innate lymphoid cells (ILC2s) expressing IL-33Ralpha and

142 Group 2 innate lymphoid cells (ILC2s) have recently been shown t

143 Group 2 innate lymphoid cells (ILC2s) have tissue-resident compe

144 Ts induced eosinophilia and expanded group 2 innate lymphoid cells (ILC2s) in aspirin-exacerbated res

145 SA, histology, and real-time PCR; and type 2 innate lymphoid cells (ILC2s) in lung single-cell prepar

146 s also limited regarding the role of group 2 innate lymphoid cells (ILC2s) in regulating humoral immu

147 murine models of allogeneic BMT, that type 2 innate lymphoid cells (ILC2s) in the lower GI tract are

148 pendent IL-33-induced accumulation of type 2 innate lymphoid cells (ILC2s) in the lungs, which perpet

149 Group 2 innate lymphoid cells (ILC2s) mediate allergic immunity

150 our observations in prostate cancer.Group 2 innate lymphoid cells (ILC2s) modulate inflammatory and

151 und that activation of lung-resident group 2 innate lymphoid cells (ILC2s) orchestrated suppression o

152 Group 2 innate lymphoid cells (ILC2s) play a key role in the ini

153 The recently identified type 2 innate lymphoid cells (ILC2s) play significant roles in

154 Group 2 innate lymphoid cells (ILC2s) possess indispensable role

155 Group 2 innate lymphoid cells (ILC2s) produce type 2 cytokines,

156 omeostasis at mucosal barriers, with group 2 innate lymphoid cells (ILC2s) producing type 2 cytokines

157 In innate immunity, IL-33 and group 2 innate lymphoid cells (ILC2s) provide an essential axis

158 Group 2 innate lymphoid cells (ILC2s) regulate immunity, inflamm

159 Group 2 innate lymphoid cells (ILC2s) regulate inflammation and

160 Group 2 innate lymphoid cells (ILC2s) represent a subset of newl

161 Type 2 innate lymphoid cells (ILC2s) represent an important typ

162 Type 2 innate lymphoid cells (ILC2s) resemble TH2 cells and pro

163 Type 2 innate lymphoid cells (ILC2s) resemble type 2 helper (Th

164 Group 2 innate lymphoid cells (ILC2s) reside in multiple organs

165 Group 2 innate lymphoid cells (ILC2s) secrete high amounts of T(

166 ly potent in eliciting and activating type 2 innate lymphoid cells (ILC2s) that may act throughout al

167 aging to define the tissue niches of group 2 innate lymphoid cells (ILC2s), which are critical instig

168 Group 2 innate lymphoid cells (ILC2s), which promote tissue eosi

169 t cells results in the activation of group 2 innate lymphoid cells (ILC2s), which subsequently drive

170 by TH2 cells and innate responses by group 2 innate lymphoid cells (ILC2s), with these latter being w

171 plasia phenotype that is dependent on type 2 innate lymphoid cells (ILC2s).

172 xpansion and activation of intestinal type-2 innate lymphoid cells (ILC2s).

173 e to the inflammation, especially in group 2 innate lymphoid cells (ILC2s).

174 cytokine (IL-5, IL-13) production by type 2 innate lymphoid cells (ILC2s).

175 atory T (Treg) cells and the emerging type 2 innate lymphoid cells (ILC2s).

176 via the activity of islet-associated group 2 innate lymphoid cells (ILC2s).

177 type 2 inflammation, eosinophils and group 2 innate lymphoid cells (ILC2s).

178 e correlated with reduced numbers of group 2 innate lymphoid cells (ILC2s).

179 al effects of IL-35 were assessed on group 2 innate lymphoid cells (ILC2s); dendritic cells primed wi

180 locked cNK development and supported group 3 innate lymphoid cell (ILC3) differentiation.

181 promoting IL-22 production from the group 3 innate lymphoid cell (ILC3) in an aryl hydrocarbon recep

182 the right vagus decreased peritoneal group 3 innate lymphoid cell (ILC3) numbers and altered peritone

183 interleukin-22 (IL-22), produced by group 3 innate lymphoid cells (ILC3) and gammadelta T cells, is

184 of increased numbers of RORgammat(+) group 3 innate lymphoid cells (ILC3) correlates with an increase

185 ignificant and persistent decrease in type 3 innate lymphoid cells (ILC3) in the lamina propria.

186 Here we demonstrate that group 3 innate lymphoid cells (ILC3) regulate steady state inter

187 ady-state activation hubs containing group 3 innate lymphoid cells (ILC3) that continuously produce i

188 olic responses that are controlled by type-3 innate lymphoid cells (ILC3)(1-3).

189 Type 3 innate lymphoid cells (ILC3s) are critical for lung defe

190 Group 3 innate lymphoid cells (ILC3s) are important for intestin

191 Group 3 innate lymphoid cells (ILC3s) are important regulators o

192 Type 3 innate lymphoid cells (ILC3s) are involved in maintenanc

193 Group 3 innate lymphoid cells (ILC3s) are major regulators of in

194 CCR6- group 3 innate lymphoid cells (ILC3s) are mediators of intestina

195 vealed that, in the small intestine, group-3 innate lymphoid cells (ILC3s) are the dominant cellular

196 Group 3 innate lymphoid cells (ILC3s) are the innate counterpart

197 gamma-delta (gammadelta) T cells and Type 3 innate lymphoid cells (ILC3s) as important producers of

198 Group 3 innate lymphoid cells (ILC3s) have emerged as master reg

199 L-22 by T helper 17 (Th17) cells and group 3 innate lymphoid cells (ILC3s) in response to the gut mic

200 RORgammat(+) group 3 innate lymphoid cells (ILC3s) maintain intestinal homeos

201 results in depletion of intrathymic group 3 innate lymphoid cells (ILC3s) necessary for thymic regen

202 The IL-23-driven tissue-resident group 3 innate lymphoid cells (ILC3s) play essential roles in in

203 interleukin-22 (IL-22) production by group 3 innate lymphoid cells (ILC3s) protects against pathobion

204 Group 3 innate lymphoid cells (ILC3s) regulate intestinal immuni

205 Group 3 innate lymphoid cells (ILC3s) sense environmental signal

206 As such, group 3 innate lymphoid cells (ILC3s) that reside in the intesti

207 Group 3 innate lymphoid cells (ILC3s), but not ILC1s or ILC2s, w

208 (DCs), but not T cells, B cells nor group 3 innate lymphoid cells (ILC3s), is critical specifically

209 Innate lymphoid cells (ILCs) and CD4(+) T cells produce

210 essor Id2 is constitutively expressed in all innate lymphoid cells (ILCs) and is required for their d

211 patial compartmentalization of skin-resident innate lymphoid cells (ILCs) and modulation of sebaceous

212 Innate lymphoid cells (ILCs) are critical for host defen

213 Innate lymphoid cells (ILCs) are enriched at barrier sur

214 Innate lymphoid cells (ILCs) are generated early during

215 Innate lymphoid cells (ILCs) are important regulators in

216 cular pathways underlying the development of innate lymphoid cells (ILCs) are mostly unknown.

217 Innate lymphoid cells (ILCs) are rapidly-responding cell

218 Innate lymphoid cells (ILCs) are strategically positione

219 Innate lymphoid cells (ILCs) are the most recently ident

220 Innate lymphoid cells (ILCs) are tissue-resident "first

221 Innate lymphoid cells (ILCs) are tissue-resident lymphoc

222 Innate lymphoid cells (ILCs) are tissue-resident lymphoc

223 Innate lymphoid cells (ILCs) are tissue-resident sentine

224 Innate lymphoid cells (ILCs) are tuned to quickly respon

225 Innate lymphoid cells (ILCs) communicate with other haem

226 Innate lymphoid cells (ILCs) consist of a heterogeneous

227 Intestinal innate lymphoid cells (ILCs) contribute to the protectiv

228 Although innate lymphoid cells (ILCs) functionally analogous to T

229 Innate lymphoid cells (ILCs) guard epithelial tissue int

230 In particular, the discovery of innate lymphoid cells (ILCs) has opened entirely new ave

231 Innate lymphoid cells (ILCs) have an important role in t

232 Innate lymphoid cells (ILCs) have been classified into "

233 e central importance of an emerging group of innate lymphoid cells (ILCs) in health and disease.

234 However, the involvement of innate lymphoid cells (ILCs) in immune responses to infe

235 e thymus and they control the development of innate lymphoid cells (ILCs) in the bone marrow.

236 a cardinal role of T-bet-dependent NKp46(+) innate lymphoid cells (ILCs) in the initiation of CD4(+)

237 cytokine production by CRTH2(-)IL7Ralpha(-) innate lymphoid cells (ILCs) is unknown.

238 crosis factor alpha [TNF-alpha]) produced by innate lymphoid cells (ILCs) located in the colon second

239 Depleting group I innate lymphoid cells (ILCs) or infecting Trail(-/-) mic

240 Innate lymphoid cells (ILCs) patrol environmental interf

241 Innate lymphoid cells (ILCs) play critical roles in immu

242 Although innate lymphoid cells (ILCs) play fundamental roles in m

243 Innate lymphoid cells (ILCs) play important functions in

244 Innate lymphoid cells (ILCs) play important roles in hos

245 er cells (Th cells), and recently identified innate lymphoid cells (ILCs) play important roles in hos

246 gated for the first time what roles, if any, innate lymphoid cells (ILCs) play in HSV-1 infection.

247 Innate lymphoid cells (ILCs) play strategic roles in tis

248 Innate lymphoid cells (ILCs) promote tissue homeostasis

249 Recent findings that innate lymphoid cells (ILCs) regulate adaptive T cell re

250 Innate lymphoid cells (ILCs) represent innate versions o

251 tural killer (NK) cells are cytotoxic type 1 innate lymphoid cells (ILCs) that defend against viruses

252 In two independent cohorts, we found that innate lymphoid cells (ILCs) were depleted in the blood

253 Innate lymphoid cells (ILCs) were originally classified

254 nated by macrophages, dendritic cells (DCs), innate lymphoid cells (ILCs), and natural killer (NK) ce

255 th a rapid depletion of all three subsets of innate lymphoid cells (ILCs), ILC1s, ILC2s and ILC3s, wh

256 ng the GR is selectively deleted in NKp46(+) innate lymphoid cells (ILCs), we demonstrated a major ro

257 ection rescued IL-22 production from group 3 innate lymphoid cells (ILCs), whereas IL-6 administratio

258 the unique characteristics of skin-resident innate lymphoid cells (ILCs).

259 ucer (LTi) cells are regarded as a subset of innate lymphoid cells (ILCs).

260 mmunity, demonstrating an important role for innate lymphoid cells (ILCs).

261 tracellular bacterial mutant, is produced by innate lymphoid cells (ILCs).

262 ly changed with the discovery of a family of innate lymphoid cells (ILCs): ILC1s, ILC2s, and ILC3s.

263 expression of IL-33, a stimulator of type II innate lymphoid cells, in lung epithelial cells was asso

264 cell-derived cytokines that activate group 2 innate lymphoid cells, induce migration and activation o

265 disease pathology, mucus production, group 2 innate lymphoid cell infiltration, IL-5 and IL-13 produc

266 er resident leukocyte populations, including innate lymphoid cells, macrophages, natural killer and n

267 Surprisingly, besides CD4(+) T cells and innate lymphoid cells, mast cells are a source of GM-CSF

268 aligned with pathways associated with type 2 innate lymphoid cells, monocytes, neutrophil trafficking

269 e lineages, including natural killer T cell, innate lymphoid cell, mucosal-associated invariant T cel

270 (macrophages, neutrophils, dendritic cells, innate lymphoid cells, myeloid-derived suppressor cells,

271 ing novel mechanistic roles for macrophages, innate lymphoid cells, natural killer cells, innate gamm

272 sident population in other organs, including innate lymphoid cells, natural killer cells, natural kil

273 They either differentiate into innate lymphoid cells or migrate to the thymus to give r

274 nate lymphoid cells (ILC2s) are the dominant innate lymphoid cell population in the lungs at steady s

275 erentiation and function of mature T, B, and innate lymphoid cell populations including natural kille

276 e presence of mast cells, natural killer and innate lymphoid cell precursors in the yolk sac.

277 lin-induced IL-22 expression, which required innate lymphoid cells, prevented microbiota encroachment

278 f HIV that chronic SIV-infected gut contains innate lymphoid cells producing inflammatory cytokines.

279 Type 3 innate lymphoid cells producing predominantly GM-CSF are

280 d expressed on natural killer (NK) cells and innate lymphoid cells, recognizes PDGF-DD.

281 controlled by the microbiota through group 3 innate lymphoid cells, STAT3 (signal transducer and acti

282 PRKCQ gene expression was assessed in innate lymphoid cell subsets purified from human PBMCs a

283 tion of dendritic cell, mast cell, basophil, innate lymphoid cell, T-cell, and B-cell responses to al

284 Natural killer cells constitute potent innate lymphoid cells that play a major role in both tum

285 ion in T(reg) cells, which activated group 2 innate lymphoid cells to provide a feed-forward mechanis

286 tween epithelial cells, dendritic cells, and innate lymphoid cells translates to T-cell outcomes, wit

287 In obese mice, ozone increased lung IL-13+ innate lymphoid cells type 2 (ILC2) and IL-13+ gammadelt

288 -5 and IL-13 from T helper 2 (Th2) cells and innate lymphoid cells type 2 (ILCs), and increased airwa

289 s; these cytokines, especially IL-33, target innate lymphoid cells type 2 to produce type 2 cytokines

290 n to depend on IL-22, a cytokine produced by innate lymphoid cells type 3 (ILC3) during Enterobacteri

291 Unexpectedly, innate lymphoid cells were found to have a potent influe

292 tract (vagina and cervix), whereas APCs and innate lymphoid cells were mainly located in the upper t

293 airway inflammation, mast cells, and group 3 innate lymphoid cells were more enriched in adult-onset

294 Natural killer (NK) cells are innate lymphoid cells which mediate resistance against p

295 functions of specific subsets of T cells and innate lymphoid cells, which are key drivers of inflamma

296 osinophils and mast cells and the intriguing innate lymphoid cells, which are present in the circulat

297 acrophages and interleukin (IL)-22-producing innate lymphoid cells, which in turn promote pSTAT3 sign

298 ng tissue injury in sepsis, activates type 2 innate lymphoid cells, which promote polarization of M2

299 ere the focus of the 2(nd)EMBO Conference on Innate Lymphoid Cells, which took place from November 30

300 l sources of alloantigens, the cross talk of innate lymphoid cells with damaged epithelia and with th