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1 s, each of which is postsynaptic to a single inner hair cell.
2 axons and their sensory endings beneath each inner hair cell.
3 ed from a single synaptic ribbon in a single inner hair cell.
4  are similar to those of developing cochlear inner hair cells.
5 channels at the synaptic ribbons of auditory inner hair cells.
6 outer hair cells more severely affected than inner hair cells.
7 1.3 channels and Ca(2+) homeostasis in mouse inner hair cells.
8 elease of glutamate at the ribbon synapse of inner hair cells.
9 th in transfected HEK293T cells and in mouse inner hair cells.
10 subtle structural changes in and surrounding inner hair cells.
11  ganglion cells and outer hair cells but not inner hair cells.
12 kout mice, the stereocilia were thickened in inner hair cells.
13 akes transient direct synaptic contacts with inner hair cells.
14 being higher in outer hair cells (OHCs) than inner hair cells.
15  responsible for encoding the SK channels of inner hair cells.
16 heir significance for Ca(v)1.3 regulation in inner hair cells.
17 litudes but no difference in degeneration of inner hair cells.
18 re extensive between supporting cells around inner hair cells.
19 Ca(2+) influx at presynaptic active zones in inner hair cells.
20  of fast activating outward current from the inner hair cells.
21 fibers terminating onto the afferents to the inner hair cells.
22 chanical signal, which is then transduced by inner hair cells.
23 characterized BK channel expression in mouse inner hair cells.
24 lls and to a small increase in the number of inner hair cells.
25 rows of outer hair cells and a single row of inner hair cells.
26 ture of the SGNs and their synapses with the inner hair cells.
27  I SGNs that synapse on the modiolar side of inner hair cells.
28 cells prevented the functional maturation of inner hair cells.
29 icted to the cell apical region) in cochlear inner hair cells.
30 (+) efflux, and subsequent depolarization of inner hair cells.
31 or normal calcium currents and exocytosis in inner hair cells.
32 erformed intracellular recordings from mouse inner hair cells across the lifespan and show that effer
33 deaf due to loss of glutamate release at the inner hair cell afferent synapse.
34 ereocilia of outer hair cells (OHCs) but not inner hair cells and affects interactions of stereocilia
35     Nevertheless, BK channels are present in inner hair cells and encode a fast activating outward cu
36 undary, producing unexpected duplications of inner hair cells and inner phalangeal cells.
37 individually dedicated to the homeostasis of inner hair cells and outer hair cells.
38 a novel function of BK channels in mammalian inner hair cells and provide a framework for future rese
39 ablation caused duplication of FGF8-positive inner hair cells and reduction of outer hair cells witho
40 ria for: (i) presynaptic disorders affecting inner hair cells and ribbon synapses; (ii) postsynaptic
41 crodissected immature (postnatal days 10-13) inner hair cells and spiral ganglion cells but not outer
42  onset of spontaneous correlated activity in inner hair cells and spiral ganglion neurons, which begi
43 mutational load expansion toward the apex in inner hair cells and spiral ganglion neurons.
44  bundle, structural aberrations in outer and inner hair cells and stria vascularis defects, leading t
45 ll differentiation and an over production of inner hair cells and that these effects are likely media
46  occur in the synapses between the cochlea's inner hair cells and the auditory nerve, effectively sev
47 e first time, successful transduction of all inner hair cells and the majority of outer hair cells in
48 fate mapping shows this region gives rise to inner hair cells and their associated inner phalangeal c
49 f presynaptic structures or processes of the inner hair cells and their input.
50 that form a boundary between a single row of inner hair cells and three rows of outer hair cells (OHC
51 ed region of the cochlea in which one row of inner hair cells and three rows of outer hair cells are
52 sional finite-element methods, we modeled an inner hair-cell and an outer hair-cell stereocilia bundl
53 or-image duplications of tunnel of Corti and inner hair cells, and expressing ectopic vestibular-like
54 r cells, with the former being comparable to inner hair cells, and the latter comparable to OHCs, bas
55 es can affect the coupling between outer and inner hair cells are discussed.
56 t displacement value, which implies that the inner hair cells are more sensitive to V(BM) than to BM
57                                    Mammalian inner hair cells are not electrically tuned and, yet, BK
58                        While hair bundles of inner hair cells are of linear shape, those of outer hai
59                                              Inner hair cells are responsible for transducing mechani
60  lends support to the recent hypothesis that inner hair cells are stimulated by a net flow, in additi
61                                              Inner hair cells are transduced in an apex-to-base gradi
62 rves, a measure of the tuning of the sensory inner hair cells, are also sharply tuned, but the thresh
63 ells and lateral OC (LOC) projections to the inner hair cell area in humans 0-89 years of age.
64 mmunostained efferent terminals in outer and inner hair cell areas.
65 y, in olivocochlear fibers in both outer and inner hair cell areas.
66 fespan and show that efferent innervation of inner hair cells arises in parallel with the loss of aff
67 ng is in outer hair cells at the apex and in inner hair cells as well as spiral ganglion neurons at t
68  the basilar membrane to optimally drive the inner hair cells at their best frequency.
69 rders that result from noise-induced loss of Inner Hair Cell - Auditory Nerve synaptic connections.
70  of synaptic ribbons associated with loss of Inner Hair Cell - Auditory Nerve synaptic connections.
71                                              Inner hair cells, auditory synapses and spiral ganglion
72 a novel mechanism for limiting the number of inner hair cells being produced.
73 d at the presynaptic ribbon synapse of adult inner hair cells both in wild-type and CaBP4(-/-) mice a
74 yosin-XVa and is disrupted in Myo15(sh2/sh2) inner hair cells, but not in Myo15(sh2/sh2) outer hair c
75 outer hair cells, whereas innervation of the inner hair cells by type I spiral ganglion neurons was n
76                Of multiple CaBPs detected in inner hair cells (CaBP1, CaBP2, CaBP4 and CaBP5), CaBP1
77                             Loss of gata3 in inner hair cells causes progressive hearing loss and acc
78 urther suggest that in the cochlea, neuronal-inner hair cell connections may dynamically reshape as p
79 ordings showed that ANFs contacting the same inner hair cell could have different SRs, with no correl
80               Moreover, Foxo3-knock-out (KO) inner hair cells do not display reductions in numbers of
81                               Myo15(sh2/sh2) inner hair cells do not have obliquely oriented tip link
82 e transient cochlear efferent innervation to inner hair cells during the critical period before the o
83 ells, followed by slower degeneration of the inner hair cells, during the first 3 weeks of life.
84 feedback loop that is positioned to regulate inner hair cell excitability and refine maturation of th
85  is a form of hearing loss in which cochlear inner hair cells fail to correctly encode or transmit ac
86                            In Myo15(sh2/sh2) inner hair cells, fast adaptation is disrupted and the t
87 at embryonic day (E)14.5, beginning with the inner hair cells, Fgf20 expression precedes hair cell di
88 endent inactivation is marginally greater in inner hair cells from CaBP4(-/-) than from wild-type mic
89 most sensitive range, is absent in outer and inner hair cells from homozygous Snell's waltzer mutant
90               Hair bundles of both outer and inner hair cells from mice were deflected by using sinew
91 o synaptic vesicle recycling in the auditory inner hair cells from the organ of Corti and to investig
92                                              Inner hair cells from TMIE mutant mice show altered post
93 s suggest that the effective stimulus to the inner hair cell hair bundles results not from a simple O
94 m the modiolar face and/or basal pole of the inner hair cell have larger ribbons and smaller receptor
95                       The number of cochlear inner hair cells, however, is almost doubled.
96  neurons and is likely to depend on cochlear inner hair cell (IHC) action potentials.
97 sly, in both naked and Damaraland mole rats, inner hair cell (IHC) afferent ribbon density was reduce
98     Spatial magnitude and phase profiles for inner hair cell (IHC) depolarization throughout the chin
99 pends on the transfer characteristics of the inner hair cell (IHC) ribbon synapse and its multiple co
100 riggering of neurotransmitter release at the inner hair cell (IHC) ribbon synapses by Ca(2)(+) entry
101  heminodes relative to excitatory input from inner hair cell (IHC) ribbon synapses continued until ap
102              Transmitter release at auditory inner hair cell (IHC) ribbon synapses involves exocytosi
103 g loss (ARHL) is associated with the loss of inner hair cell (IHC) ribbon synapses, lower hearing sen
104 n requires efficient resupply of vesicles at inner hair cell (IHC) ribbon synapses.
105                                          Yet inner hair cell (IHC) ribbons and auditory nerve respons
106    The model simulates fluid flow around the inner hair cell (IHC) stereocilia bundle.
107 triggers exocytosis of glutamate at cochlear inner hair cell (IHC) synapses.
108                                          The inner hair cell (IHC) to SGN synapse is susceptible to g
109  dynamics in a phenomenological model of the inner hair cell (IHC)-auditory nerve (AN) synapse succes
110 gle ribbon-type active zone of a presynaptic inner hair cell (IHC).
111 by an individual active zone (AZ) of a given inner hair cell (IHC).
112                                          The inner hair cell (IHC)/auditory nerve fiber (ANF) synapse
113                         Afferent synapses on inner hair cells (IHC) transfer auditory information to
114 le ribbon synapse in a single receptor cell (inner hair cell; IHC).
115 , roughly 95% of which innervate exclusively inner hair cells (IHCs) [2-4].
116               Ribbon synapses formed between inner hair cells (IHCs) and afferent dendrites in the ma
117 taneous whole-cell recordings from mammalian inner hair cells (IHCs) and auditory nerve fiber termina
118 n at birth, with gradual localization to the inner hair cells (IHCs) and its supporting cells, inner
119 y (P) 2, the efferent arbors associated with inner hair cells (IHCs) and outer hair cells (OHCs) disp
120 ntains two types of mechanotransducer cells, inner hair cells (IHCs) and outer hair cells (OHCs).
121 ation and maintenance of stereocilia in both inner hair cells (IHCs) and outer hair cells (OHCs).
122            Heterogeneity of synapses between inner hair cells (IHCs) and SGNs is an attractive candid
123 ntiation of afferent ribbon synapses between inner hair cells (IHCs) and spiral ganglion neurons (SGN
124 t cell types but remained relatively high in inner hair cells (IHCs) and to a lesser extent in IHC su
125 ceive auditory information from the cochlear inner hair cells (IHCs) and transmit that information to
126 input from several ribbons, whereas cochlear inner hair cells (IHCs) are contacted by several individ
127             In the mature mammalian cochlea, inner hair cells (IHCs) are mainly innervated by afferen
128                           Mammalian cochlear inner hair cells (IHCs) are specialized for the dynamic
129                           Mammalian cochlear inner hair cells (IHCs) are specialized sensory receptor
130                           Mammalian cochlear inner hair cells (IHCs) are specialized to process devel
131                                              Inner hair cells (IHCs) are the primary receptors for he
132                                          The inner hair cells (IHCs) are the primary sensory receptor
133                                              Inner hair cells (IHCs) are the primary sensory receptor
134                                              Inner hair cells (IHCs) are the primary transducer for s
135                                              Inner hair cells (IHCs) are the true sensory receptors i
136           We compared age-related changes in inner hair cells (IHCs) between four mouse strains with
137               In the mature auditory system, inner hair cells (IHCs) convert sound-induced vibrations
138                                     Cochlear inner hair cells (IHCs) convert sounds into receptor pot
139                          Wrb-deficient mouse inner hair cells (IHCs) displayed normal numbers of affe
140  (BK) current is prominent, and in mammalian inner hair cells (IHCs) displays unusual properties.
141                      This activity occurs in inner hair cells (IHCs) during the first postnatal week,
142        In the mammalian cochlea, the sensory inner hair cells (IHCs) encode auditory information.
143 ion of Ca(2+)SIGNIFICANCE STATEMENT Auditory inner hair cells (IHCs) encode sounds into nerve impulse
144                 From just after birth, mouse inner hair cells (IHCs) expressed a Ca(2+)-activated K(+
145 ing whole-cell voltage-clamp recordings from inner hair cells (IHCs) in acutely excised apical turns
146 he two types of supporting cells surrounding inner hair cells (IHCs) in mice in vivo.
147                                              Inner hair cells (IHCs) in the cochlea are the mammalian
148                                              Inner hair cells (IHCs) in the mammalian cochlea are abl
149                                              Inner hair cells (IHCs) in the middle turn of the cochle
150 s and prevented the normal synaptogenesis in inner hair cells (IHCs) in the newly identified mouse mu
151 ory system, spontaneous activity of cochlear inner hair cells (IHCs) is initiated by the release of A
152 (2+)-activated K(+) currents were studied in inner hair cells (IHCs) of mature mice.
153    The systematically varying innervation of inner hair cells (IHCs) of the cochlea provides a model
154                                              Inner hair cells (IHCs) of the cochlea use ribbon synaps
155 nsory epithelium and then diverge to contact inner hair cells (IHCs) or outer hair cells (OHCs), resp
156 opment before the onset of hearing, cochlear inner hair cells (IHCs) present spontaneous Ca(2+) actio
157 OHCs) in the mature mammalian cochlea and on inner hair cells (IHCs) prior to the onset of hearing.
158  onset of hearing at postnatal day 12, mouse inner hair cells (IHCs) produce spontaneous and evoked a
159 TRACT: Just before the onset of hearing, the inner hair cells (IHCs) receive inhibitory efferent inpu
160 ansient and sustained exocytosis in auditory inner hair cells (IHCs) remain largely unknown.
161           In the developing auditory system, inner hair cells (IHCs) spontaneously fire Ca(2+) spikes
162                                 In contrast, inner hair cells (IHCs) survive normally but they fail t
163                  Ribbon synapses of cochlear inner hair cells (IHCs) undergo molecular assembly and e
164                                              Inner hair cells (IHCs) were not significantly affected
165 k Ca(2+) and Ba(2+) currents of mature du/du inner hair cells (IHCs) were reduced by 30-40%, respecti
166 ogical membrane properties of mouse cochlear inner hair cells (IHCs) were studied from just after ter
167                 The GFP reporter showed that inner hair cells (IHCs) were transfected throughout the
168 + entry and exocytosis were studied in mouse inner hair cells (IHCs) which, together with the afferen
169          While most conventional AAVs infect inner hair cells (IHCs) with various efficiencies, they
170 ed by cochlear hair cells, preferentially in inner hair cells (IHCs), and was lacking from the postsy
171 cient mice, outer hair cells (OHCs), but not inner hair cells (IHCs), began to lose their third row o
172 on-type active zones (AZs) of mouse auditory inner hair cells (IHCs), but its modes and molecular reg
173 (AAV1) leads to transgene expression in only inner hair cells (IHCs), despite broader viral uptake.
174 s affecting the afferent dendrites under the inner hair cells (IHCs), however, little is known about
175  periodically release ATP, which depolarizes inner hair cells (IHCs), leading to bursts of action pot
176 diate neurotransmitter release from auditory inner hair cells (IHCs), pancreatic insulin secretion, a
177 f spiral ganglion neurons (SGNs) on cochlear inner hair cells (IHCs), resulting in loss of synapses,
178                      Significance statement: Inner hair cells (IHCs), the auditory sensory cells of t
179                                     Cochlear inner hair cells (IHCs), the mammalian auditory sensory
180 Similar to the primary sensory receptor, the inner hair cells (IHCs), the mature functional character
181  sensitivity of the synaptic machinery allow inner hair cells (IHCs), the primary auditory receptors,
182 (2+) current (I(Ca)) in prehearing and adult inner hair cells (IHCs), the primary sensory receptors o
183                                              Inner hair cells (IHCs), the primary sensory receptors o
184 natal day 7 (P7), before the primary sensory inner hair cells (IHCs), which become competent at about
185 s calcium action potentials are generated by inner hair cells (IHCs), which form the primary sensory
186  potential (AP) activity in immature sensory inner hair cells (IHCs), which is crucial for the refine
187 ction potential activity in immature sensory inner hair cells (IHCs), which is crucial for the refine
188 ltaC mouse), MYO7A is severely diminished in inner hair cells (IHCs), while expression in outer hair
189 tosis, and sound encoding at the synapses of inner hair cells (IHCs).
190 CaV1.3 Ca(2+) channels at the AZs of sensory inner hair cells (IHCs).
191 nputs: the presynaptic active zones (AZs) of inner hair cells (IHCs).
192 ase from the primary cochlear receptors, the inner hair cells (IHCs).
193 f loss of input from their target cells, the inner hair cells (IHCs).
194 at the ribbon synapses of sensory cells, the inner hair cells (IHCs).
195 cells prevented the functional maturation of inner hair cells (IHCs).
196 rt provided by their peripheral targets, the inner hair cells (IHCs): following ototoxic drugs or aco
197 ings of spiral ganglion cells in females and inner hair cells in males.
198  of glutamatergic transmission from cochlear inner hair cells in mice lacking the vesicular glutamate
199  Kcnq4_v3 variant in the spiral ganglion and inner hair cells in the basal hook region.
200 terns around ribbons from serial sections of inner hair cells in the cat, and compared data from low
201               Development of an extra row of inner hair cells in the cochlea and an approximate doubl
202  by action potentials (APs) arising from the inner hair cells in the developing cochlea.
203 d progressive degeneration of both outer and inner hair cells in the organ of Corti, following two re
204 f pre- and post-synaptic markers on cochlear inner hair cells, in guinea pigs surviving from 1 day to
205 ysical maturation, growth and innervation of inner hair cells; in contrast, it is required only for t
206       The strength of efferent inhibition of inner hair cells increases with hearing threshold elevat
207 rthermore, transmitter secretion of cochlear inner hair cells is compromised in mice lacking otoferli
208 al functional differentiation of the sensory inner hair cells is less clear.
209 al functional differentiation of the sensory inner hair cells is less clear.
210 nstrate that an Fgf8 signal arising from the inner hair cells is the key component in an inductive pa
211  type-I afferents by glutamate released from inner hair cells) is silenced [5, 6].
212 to was observed just below and medial to the inner hair cell layer.
213  supporting cell on the neural aspect of the inner hair cell layer.
214 and cell bodies in the cochlear apex without inner hair cell loss.
215 nocopies JLNS, with deafness associated with inner hair cell malfunction.
216 the endogenous release of glutamate from the inner hair cells may increase the strength of efferent i
217 hat CRFR1 has a developmental role affecting inner hair cell morphology and afferent and efferent syn
218 age than low-frequency cells; high-frequency inner hair cells must have a low Ca(2+) buffer capacity
219 tory nucleus.SIGNIFICANCE STATEMENT Cochlear inner hair cells of altricial mammals display spontaneou
220             HA immunopuncta also occurred in inner hair cells of pre-hearing (P7) but not in adult mi
221      In addition, VGLUT3 is expressed in the inner hair cells of the auditory system.
222 ere we present voltage-clamp recordings from inner hair cells of the C57BL/6J mouse model of age-rela
223 nces in afferent neurons and, in the case of inner hair cells of the cochlea, vulnerability to damage
224                                 In contrast, inner hair cells of the mammalian cochlea are extrinsica
225                                              Inner hair cells of the mammalian cochlea translate acou
226  (P21) to P120 mice that is most apparent in inner hair cells of the middle turn.
227 ral ganglion cells, but not with the loss of inner hair cells or a reduced thickness of the stria vas
228 pparatus in the inner ear and is composed of inner hair cells, outer hair cells, and highly specializ
229                                              Inner hair cells possess calcium channels that are essen
230          ErbB2 null mice retain afferents to inner hair cells possibly because of the prominent expre
231 notransduction and the acquisition of mature inner hair cell potassium currents.
232                       Fade-out of whirlin in inner hair cells precedes that of outer hair cells, cons
233                                              Inner hair cells primarily function as afferent sensory
234           We show that burst firing of mouse inner hair cells prior to hearing onset requires P2RY1 a
235          Notably, a single ribbon synapse of inner hair cells provides the entire input to each cochl
236               These efferent neurons inhibit inner hair cells, raising the possibility that they play
237                          Solution effects on inner hair cells reduced auditory nerve compound action
238 hly disorganized efferent innervation to the inner hair cell region.
239                      In connexin knock-outs, inner hair cells remained stuck at a prehearing stage of
240 ng the limited autoregulation of Ca(v)1.3 in inner hair cells remains a mystery.
241    Overexpression of twinfilin 2 in cochlear inner hair cells resulted in a significant reduction of
242           Genetic loss of VGLUT3 in cochlear inner hair cells results in profound deafness.
243 he mechanical responses of hair bundles from inner hair cells revealed a characteristic resonance and
244 ical recordings from postnatal (P) days P8-9 inner hair cells revealed ACh-gated currents in alpha10(
245                            Exocytosis at the inner hair cell ribbon synapse is achieved through the f
246                                 The solitary inner hair cell ribbon synapse uses multivesicular relea
247 y neuropathy owing to disordered function of inner hair cell ribbon synapses (temperature-sensitive a
248  blocker, would reduce noise-induced loss of Inner Hair Cell ribbons.
249 the compound action potentials, a measure of inner hair cell sensitivity, are significantly elevated.
250 that make simple one-to-one connections with inner hair cell sensory receptors, it has an elaborate o
251                             Remarkably, each inner hair cell serves as the sole input for 10-30 indiv
252  POINTS: Spontaneous activity of the sensory inner hair cells shapes maturation of the developing asc
253 ir role in this regard, Ca(v)1.3 channels in inner hair cells show little Ca(2+)-dependent inactivati
254 ter gene results in binaural transduction of inner hair cells, spiral ganglion neurons and vestibular
255 neurons originating in the brainstem inhibit inner hair cell spontaneous activity and may further ref
256 ed by conspicuous elongations and fusions of inner hair cell stereocilia and progressive degeneration
257 labeled mouse cochleas, we demonstrated that inner hair cell stereocilia developed in specific stages
258 s, consistent with the earlier maturation of inner hair cell stereocilia.
259  older Lmo7 KO mice develop abnormalities in inner hair cell stereocilia.
260 hich manifests with shortening and fusion of inner hair cells stereocilia and progressive degeneratio
261        Modulating the amount of free PIP2 in inner hair-cell stereocilia resulted in the following: (
262 the tectorial membrane directly overlies the inner hair cell stereociliary bundles, these data provid
263 mice suggest roles for the ALC in regulating inner hair cell stereociliary growth and differentiation
264 with Otoa mutations is caused by a defect in inner hair cell stimulation, and reveal the limbal attac
265 e inner ear, mainly outer hair cells (OHCs), inner hair cells, stria vascularis, spiral ganglia, and
266 ntials was within the normal range found for inner hair cell summating receptor potentials.
267  The results were observed first in afferent inner hair cell synapse of type I neurites, followed by
268                 Sound coding at the auditory inner hair cell synapse requires graded changes in neuro
269 in and Ca(v)1.3 Ca(2+) channels at the mouse inner hair cell synapse, which limits channel availabili
270  cellular mechanism known for HHL is loss of inner hair cell synapses (synaptopathy).
271 function follows a conserved mechanism where inner hair cell synapses are lost, termed cochlear synap
272 hannels and the exocytosis calcium sensor at inner hair cell synapses changes along the mammalian coc
273 on of AMPKalpha in OHCs, reduced the loss of inner hair cell synaptic ribbons and OHCs, and protected
274 nduced hearing loss by mediating the loss of inner hair cell synaptic ribbons and outer hair cells.
275 a(2+) currents and membrane capacitance from inner hair cells that clarin-1 is not essential for form
276 use they extend a peripheral axon beyond the inner hair cells that subsequently makes a distinct 90 d
277 sure and active outer hair cell force to the inner hair cells that synapse on afferent nerves.
278 ry epithelium after death (especially of the inner hair cells), the tectorial membrane appeared to be
279                                    For every inner hair cell, there are approximately three outer hai
280             Afferent synapses were lost from inner hair cells throughout the aged cochlea, together w
281 ontaneously release ATP, which causes nearby inner hair cells to depolarize and release glutamate, tr
282 95% of the cochlear nerve and contact single inner hair cells to provide acoustic analysis as we know
283 nously released ATP in the cochlea activates inner hair cells to trigger bursts of action potentials
284 lecules at the afferent synapses of cochlear inner hair cells to trigger exocytosis of neurotransmitt
285                                              Inner hair cells transmit auditory information to the br
286 earing relies on two types of sensory cells: inner hair cells transmit the auditory stimulus to the b
287 ential to maintain synaptic Ca(2+) influx in inner hair cells via fueling Ca(2+)-ATPases to avoid an
288 of strial marginal cells and is augmented in inner hair cells vs. outer hair cells.
289  primary sensory fibers innervating a single inner hair cell, we quantified the sizes of presynaptic
290 channel alpha and beta subunits in mammalian inner hair cells, we analyzed the morphology, physiology
291                            By P9, almost all inner hair cells were absent and by P16 all inner and ou
292                        Fibers underneath the inner hair cells were also MOR immunoreactive.
293 mycin injections (from P8 to P16), surviving inner hair cells were evident at P16 but absent by P19,
294                  Stereocilia of St3gal5(-/-) inner hair cells were fused by P17, and protein tyrosine
295                                          The inner hair cells were of normal number with preserved mo
296 t in young adult animals, the stereocilia of inner hair cells were reduced in width and length.
297 t send a single dendrite to contact a single inner hair cell, whereas unmyelinated type II afferents
298 ling synchronizes the output of neighbouring inner hair cells, which may help refine tonotopic maps i
299 in background have selective degeneration of inner hair cells while outer hair cells remain structura
300 ochondrial NADH in outer hair cells, but not inner hair cells, within minutes of administration.

 
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