ライフサイエンスコーパス: inner nuclear layer

1 een by SD-OCT were localized deeper than the inner nuclear layer.

2 oupled histologically with a thinning of the inner nuclear layer.

3 lion cells and some but not all cells of the inner nuclear layer.

4 retinal ganglion cells and the cells of the inner nuclear layer.

5 ssed in both the ganglion cell layer and the inner nuclear layer.

6 olar cell nuclei to the upper portion of the inner nuclear layer.

7 er driving retinal rhythms is located in the inner nuclear layer.

8 and Chx10-positive bipolar cells within the inner nuclear layer.

9 nuclei of ganglion cells and neurons in the inner nuclear layer.

10 s, when subtle changes were noted within the inner nuclear layer.

11 s of amacrine cell with somata occupying the inner nuclear layer.

12 etina and optic nerve, and some cells in the inner nuclear layer.

13 ture, and retarded apoptotic kinetics of the inner nuclear layer.

14 ying cells) have their soma displaced to the inner nuclear layer.

15 ant in the retina and, in particular, in the inner nuclear layer.

16 xiform layer and stopped at the level of the inner nuclear layer.

17 before bipolar cells begin to migrate to the inner nuclear layer.

18 anglion cell layer and the inner part of the inner nuclear layer.

19 P2 is expressed specifically by cells of the inner nuclear layer.

20 s in the retinal ganglion cell layer and the inner nuclear layer.

21 ared to be filled with interneurons from the inner nuclear layer.

22 situ hybridization to ganglion cells and the inner nuclear layer.

23 cells, presumed to be bipolar cells, in the inner nuclear layer.

24 nfrequent, labeling was also observed in the inner nuclear layer.

25 ted in the future bipolar-cell region of the inner nuclear layer.

26 ayer and in a small number of neurons in the inner nuclear layer.

27 oth ganglion cells and numerous cells in the inner nuclear layer.

28 and reticular fluorescence were noted in the inner nuclear layer.

29 GF mRNA in retinal ganglion cells and in the inner nuclear layer.

30 Histology demonstrated a preserved inner nuclear layer.

31 s well as in those of cells belonging to the inner nuclear layer.

32 ions demonstrated subsequent thinning of the inner nuclear layer.

33 ound homogeneous invisible tumors within the inner nuclear layer.

34 mal human retina exhibited positivity in the inner nuclear layer.

35 ement of ganglion cell, inner plexiform, and inner nuclear layers.

36 photoreceptors, occupy the ganglion cell or inner nuclear layers.

37 totic cells in the retinal ganglion cell and inner nuclear layers.

38 GF was produced by cells in the ganglion and inner nuclear layers.

39 p38 and phospho-Akt in the ganglion cell and inner nuclear layers.

40 In the inner nuclear layer, 48% of all cells residing in the am

41 nner plexiform layer, 86.2 vs. 103.4 microm; inner nuclear layer, 51.8 vs. 60.3 microm; photoreceptor

42 To our knowledge, microcystic inner nuclear layer abnormalities have not been investig

43 is delayed fusion of the optic fissure, and inner nuclear layer abnormalities indicate a cell-specif

44 eing expressed by Muller glial cells and the inner nuclear layer, additional expression was noted in

45 bitory neuron (RIN)--horizontal cells (HCs), inner nuclear layer amacrine cells (iACs) and displaced

46 crine cells), proximal parts of neuroblastic/inner nuclear layer (amacrine cells) and distal part of

47 of horizontal cells to inner aspects of the inner nuclear layer, among the retinal amacrine cells.

48 plexus were associated with thinning of the inner nuclear layer and abnormalities of both layers wer

49 ypically result in permanent thinning of the inner nuclear layer and are critical to identify in orde

50 Nearly one-half populate only the inner nuclear layer and are enriched in amacrine cells.

51 unoreactivity (LI) was found in somas in the inner nuclear layer and as punctate staining in the inne

52 yer, cell body size, and layering within the inner nuclear layer and by the morphology and stratifica

53 d small, dimly fluorescent (SD) cells in the inner nuclear layer and displaced (DIS) cells in the gan

54 gene expression of transferrin mainly to the inner nuclear layer and ferritin to both the inner and o

55 ccount for about 1% of amacrine cells in the inner nuclear layer and for up to 27% of displaced amacr

56 retina, represented by amacrine cells in the inner nuclear layer and ganglion cell layer (GCL).

57 pic bipolar cell processes extended into the inner nuclear layer and ganglion cell layer by PNM3.5.

58 ished by the displacement of the soma to the inner nuclear layer and has morphological similarities w

59 e cell bodies in the ganglion cell layer and inner nuclear layer and immunoreactive processes in the

60 minofluorescein was present in somata in the inner nuclear layer and in synaptic boutons in the inner

61 oportion of calretinin-positive cells in the inner nuclear layer and in the ganglion cell layer is gl

62 t abundant in cells in the inner half of the inner nuclear layer and in the ganglion cell layer.

63 ls, whose perikarya are at the border of the inner nuclear layer and inner plexiform layer.

64 ve immunolocalized the InsP3 receptor to the inner nuclear layer and limiting membranes of the catfis

65 ng tdTomato fluorescence was detected in the inner nuclear layer and localized to type 1, 3b, and 4 O

66 Later disease stages were accompanied by inner nuclear layer and nerve fiber layer abnormalities.

67 cation using (1) no atrophy; (2) presence of inner nuclear layer and outer plexiform layer subsidence

68 cation using (1) no atrophy; (2) presence of inner nuclear layer and outer plexiform layer subsidence

69 Microscopically, the inner nuclear layer and outer plexiform layer were the m

70 he MAs identified in this study straddle the inner nuclear layer and outer plexiform layer.

71 mporal parafoveal thickness, presence of the inner nuclear layer and outer segment, gestational age a

72 f the DAPI-3 cells are found in the proximal inner nuclear layer and send their processes into two su

73 l cystoid spaces located at the level of the inner nuclear layer and sparing other retinal layers and

74 lNAcPTase receptor becomes restricted to the inner nuclear layer and the ganglion cell layer (as well

75 reactivity (IR) was present primarily in the inner nuclear layer and the ganglion cell layer.

76 ell bodies were located at the border of the inner nuclear layer and the IPL, and thin varicose proce

77 ized to horizontal cell bodies in the distal inner nuclear layer and their processes in the outer ple

78 PINC are continually born in the inner nuclear layer and then migrate to the outer nuclea

79 s) in cells located in the ganglion cell and inner nuclear layers and did not alter NMDA-induced PARP

80 ript and protein were also found in the RPE, inner nuclear layer, and ganglion cells.

81 TRPM1 mRNA is found in cells of the retinal inner nuclear layer, and immunofluorescent confocal micr

82 e ganglion cell layer, in the neurons of the inner nuclear layer, and in the optic nerve and optic tr

83 etinal ganglia cells, outer plexiform layer, inner nuclear layer, and outer nuclear layer and in peri

84 ar nerve fiber layer, ganglion cell complex, inner nuclear layer, and outer plexiform layer thickness

85 tina, especially in the outer nuclear layer, inner nuclear layer, and photoreceptors.

86 ganglion and amacrine layers, locate in the inner nuclear layer, and project processes across the re

87 ition, moderate labeling was detected in the inner nuclear layer, and the ganglion cell layer.

88 as well as in the outer plexiform layer, the inner nuclear layer, and the inner plexiform layer.

89 d first in the ganglion cell layer, then the inner nuclear layer, and then the outer nuclear layer, s

90 ithin the outer plexiform layer (OPL) or the inner nuclear layer, and while present in the mature ret

91 emia, dramatically decreased thinning of the inner nuclear layers, and decreased the percentage of TU

92 Some cells in the outer half of the inner nuclear layer are labeled, and there was some labe

93 t dying cells in the developing ganglion and inner nuclear layers are clustered spatially and that ga

94 glion cell layer, inner plexiform layer, and inner nuclear layer as compared to that of the sound eye

95 be used to identify A amacrine cells in the inner nuclear layer as well as widefield amacrine and sm

96 ated these lesions to be centered within the inner nuclear layer at the border of the inner plexiform

97 transient wave of Smad1/5/8 signaling in the inner nuclear layer at the end of the first postnatal we

98 ging, the lesions had resolved into areas of inner nuclear layer atrophy with persistence of scotomas

99 ction, followed by apoptosis in cells in the inner nuclear layer by day 1.

100 ing from amacrine cells, were present in the inner nuclear layer by PNM9.5.

101 ctive plaquelike lesions at the level of the inner nuclear layer by spectral-domain OCT and showed co

102 genitors primarily affects the generation of inner nuclear layer cell types, resulting in complete lo

103 factors on the mitotic rate of proliferative inner nuclear layer cells (PINC).

104 and (5) STAT3 and NF-kappaB are activated in inner nuclear layer cells by 1 day of RD.

105 ad, we observed a subtle pan-retinal loss of inner nuclear layer cells in all diabetes cases (P < 0.0

106 nal ganglion cells (RGCs) and by a subset of inner nuclear layer cells.

107 eading to an accumulation of progenitors for inner nuclear layer cells.

108 m f-iPSCs had fewer amacrine cells and other inner nuclear layer cells.

109 m a reduced number of rod photoreceptors and inner nuclear layer cells.

110 xiform layers and increased thickness in the inner nuclear layer compared with healthy subjects (P <

111 a characteristic hyperreflective band in the inner nuclear layer consistent with PAMM in his left eye

112 the VEGFA mRNA signal was located within the inner nuclear layer corresponding to CRALBP-labeled Mull

113 scriminating controls from NPDR; and (c) the inner nuclear layer distinguishes best between controls

114 was observed in the ganglion cell layer and inner nuclear layer during development.

115 y margin cells, and a subset of cells in the inner nuclear layer expressed the BDNF gene.

116 the specific sublayers of the photoreceptor: inner nuclear layer-external limiting membrane (INL-ELM)

117 retina, where KLF15-LacZ was observed in the inner nuclear layer, ganglion cell layer, and pigmented

118 The cell bodies in the inner nuclear layer, however, were larger in the periphe

119 were observed to be predominantly within the inner nuclear layer in all 17 eyes.

120 expressed in a subpopulation of cells in the inner nuclear layer in both the light and the dark.

121 uency of microcystoid macular changes in the inner nuclear layer in eyes with concomitant epiretinal

122 SD-OCT was less accurate at detecting the inner nuclear layer in ouabain-damaged retinas, but accu

123 cance of microcystoid macular changes in the inner nuclear layer in patients with idiopathic epiretin

124 e carried out a quantitative analysis of the inner nuclear layer in the retina of the marmoset (Calli

125 albumin was localized to some neurons of the inner nuclear layer, in the inner plexiform layer, and a

126 layer, and in cells in the outer zone of the inner nuclear layer, in the region occupied by bipolar c

127 ted with inner retinal folding and thickened inner nuclear layer, indicating more severe ERM.

128 ll bodies formed a regular mosaic within the inner nuclear layer, indicating they represent a single

129 ased cell numbers in the middle third of the inner nuclear layer (INL) (23%).

130 erage outer nuclear layer (ONL) (p = 0.998), inner nuclear layer (INL) (p = 0.807), and ONL/INL ratio

131 ated in the ganglion cell layer and proximal inner nuclear layer (INL) adjacent to the IPL.

132 amacrine cell with their cell bodies in the inner nuclear layer (INL) and a dense layer in the middl

133 Confetti fluorescent cell bodies were in the inner nuclear layer (INL) and a few cell bodies were in

134 a restricted group of amacrine cells in the inner nuclear layer (INL) and ganglion cell layer (GCL)

135 amacrine and displaced amacrine cells in the inner nuclear layer (INL) and ganglion cell layer (GCL),

136 ive cell bodies were located in the proximal inner nuclear layer (INL) and ganglion cell layer (GCL),

137 syntaxin (HPC-1)-immunoreactive cells in the inner nuclear layer (INL) and GCL, consistent with their

138 as expressed at a moderate level only in the inner nuclear layer (INL) and GCL.

139 The MME was restricted to the inner nuclear layer (INL) and had a characteristic perif

140 crine cells in the ferret retina both in the inner nuclear layer (INL) and in the ganglion cell layer

141 yzed for DA-IPC/ON-BC contacts in the distal inner nuclear layer (INL) and inner plexiform layer (IPL

142 field monostratified cells with somas in the inner nuclear layer (INL) and medium-field monostratifie

143 1) subunit distributed on cell bodies in the inner nuclear layer (INL) and on processes within both t

144 th was evaluated by TUNEL and measurement of inner nuclear layer (INL) and outer nuclear layer (ONL)

145 gher baseline age was associated with faster inner nuclear layer (INL) and outer nuclear layer (ONL)

146 presence of ectopic neuronal clusters in the inner nuclear layer (INL) and regions of disrupted retin

147 lyses showed retinal gRgr message within the inner nuclear layer (INL) and retinal ganglion cell laye

148 lations of cholinergic amacrine cells in the inner nuclear layer (INL) and the ganglion cell layer (G

149 NPY-IR cells were present in two layers, the inner nuclear layer (INL) and the ganglion cell layer (G

150 munoreactive (-IR) cells were located in the inner nuclear layer (INL) and the ganglion cell layer (G

151 e adult goldfish retina is restricted to the inner nuclear layer (INL) and to postmitotic, differenti

152 uently in Henle's fiber layer (HFL) than the inner nuclear layer (INL) and was highly associated with

153 nd angiogenesis were detected in the retinal inner nuclear layer (INL) before morphologic neoplastic

154 mentation of the inner plexiform layer (IPL)/inner nuclear layer (INL) boundary was carried out with

155 Labeling of nuclei in the RGCL and the inner nuclear layer (INL) by TUNEL was noted between 8 a

156 A depletion of inner nuclear layer (INL) cells in the adult Bhlhb4 knoc

157 ng eyes with outer retinal changes, isolated inner nuclear layer (INL) cysts were found in 6 of 131 e

158 glion cell layer (GCL) and inner part of the inner nuclear layer (INL) from 3-9 dpf; after 14 dpf, it

159 rocystic macular oedema (MMO) of the retinal inner nuclear layer (INL) has been identified in patient

160 ng for macular splitting were present in the inner nuclear layer (INL) in all 11 eyes and in the oute

161 pressed in the ganglion cell layer (GCL) and inner nuclear layer (INL) in both amacrine cells and RGC

162 layer (GCL) and in a subset of cells in the inner nuclear layer (INL) in both the macula and periphe

163 rate that this gene is also expressed in the inner nuclear layer (INL) of the human and mouse retina

164 l birth and proliferation also occurs in the inner nuclear layer (INL) of the mature fish retina.

165 redundantly expressed in the neurons of the inner nuclear layer (INL) of the retina.

166 Neurons in the inner nuclear layer (INL) of the vertebrate retina under

167 These inner nuclear layer (INL) rods contained markers of earl

168 ea of hyporeflective spaces was lower in the inner nuclear layer (INL) than in the complex formed by

169 With Fourier-domain-OCT, there was apparent inner nuclear layer (INL) thickening in regions with ONL

170 A small degree of inner nuclear layer (INL) thickening occurred in MSON ey

171 Inner nuclear layer (INL) thickness was significantly lo

172 ollowed over 205 +/- 109 days, GCL, IPL, and inner nuclear layer (INL) thicknesses decreased (p < 0.0

173 weeks of age, outer nuclear layer (ONL) and inner nuclear layer (INL) thicknesses were measured.

174 Although type 1 lesions lead to inner nuclear layer (INL) thinning, type 2 lesions resul

175 our patients with intraretinal tumors of the inner nuclear layer (INL) underwent a combination of oph

176 n cell and inner plexiform layer (GCIPL) and inner nuclear layer (INL) volumes were tested for associ

177 e retinal ganglion cell layer (RGCL) and the inner nuclear layer (INL) was noted in a rat model of re

178 imately 2% to 6% of the CM-1-IR cells in the inner nuclear layer (INL) were double-labeled for TH imm

179 dly damage the ganglion cell layer (GCL) and inner nuclear layer (INL) with minimal photoreceptor cel

180 n cell layer, amacrine cells in the proximal inner nuclear layer (INL), and bipolar cells in the dist

181 dence of the outer plexiform layer (OPL) and inner nuclear layer (INL), and development of a hyporefl

182 ell nuclei in the outer nuclear layer (ONL), inner nuclear layer (INL), and ganglion cell layer (GCL)

183 ent in the ganglion cell layer (GCL), in the inner nuclear layer (INL), and in two distinct bands of

184 lly involving the outer nuclear layer (ONL), inner nuclear layer (INL), and nerve fiber layer (NFL).

185 and visual thresholds; total nuclear layer, inner nuclear layer (INL), and outer nuclear layer (ONL)

186 eath of cells in the ganglion cell (GCL) and inner nuclear layer (INL), and subsequent loss of NF-L-p

187 L) as well as the inner plexiform layer, the inner nuclear layer (INL), and the outer plexiform layer

188 tive (IR) cells were located in the proximal inner nuclear layer (INL), and very rarely they were fou

189 expression in the ganglion cell layer (GCL), inner nuclear layer (INL), outer nuclear layer (ONL), an

190 the inner segment (IS) layer, but not in the inner nuclear layer (INL), outer plexiform layer (OPL),

191 BCs) located in the distal and middle of the inner nuclear layer (INL), respectively; in type IIA and

192 7, around the time of peak cell death in the inner nuclear layer (INL), significantly fewer neurons i

193 In the inner nuclear layer (INL), T+ and pyknotic cells first a

194 nglion cell layer (GCL) and the other in the inner nuclear layer (INL), that together comprise approx

195 ganglion cells and a subset of cells in the inner nuclear layer (INL), whereas NT-3 expression was c

196 e inner plexiform layer (IPL), ~0.025 in the inner nuclear layer (INL), ~0.087 in the outer plexiform

197 nglion cell layer, with 23% displaced to the inner nuclear layer (INL).

198 lls are located in the outermost part of the inner nuclear layer (INL).

199 y in the inner plexiform layer (IPL) and the inner nuclear layer (INL).

200 m the IPL and entering either the GCL or the inner nuclear layer (INL).

201 mbryonic day (E)12 and was restricted to the inner nuclear layer (INL).

202 populations of slowly-dividing cells in the inner nuclear layer (INL).

203 Muller glia, and retinal progenitors in the inner nuclear layer (INL).

204 neuroblastic retina and later to the GCL and inner nuclear layer (INL).

205 (likely to be bipolar cells) of the retinal inner nuclear layer (INL).

206 napsing with the second-order neurons of the inner nuclear layer (INL).

207 and inner plexiform layers (RGCL+), and the inner nuclear layer (INL).

208 also in the inner plexiform layer (IPL) and inner nuclear layer (INL).

209 ons in the middle retina, extending from the inner nuclear layer (INL)/outer plexiform layer junction

210 on of the retina produces chronic hypoxia of inner nuclear-layer (INL) neurons and Muller glia.

211 [ONL] thinning, outer plexiform layer [OPL]/inner nuclear layer [INL] subsidence, and hyporeflective

212 located in the most distal or middle of the inner nuclear layer [INL], respectively), had their axon

213 d by extracting semiautomated thicknesses of inner nuclear layer, inner retina, and total retina at t

214 in were detected in the ganglion cell layer, inner nuclear layer, inner/outer plexiform layers, photo

215 outer plexiform layer irregularity (8%), and inner nuclear layer irregularity (6%).

216 tinin-immunoreactive cells is located in the inner nuclear layer, is immunopositive for glycine trans

217 acular inner plexiform layer (mIPL), macular inner nuclear layer (mINL), macular outer plexiform laye

218 The inner nuclear layer/Muller cell localization of the key

219 t CB and PV expression in ganglion cells and inner nuclear layer neurons proceeds in parallel with th

220 (A2aR) mRNAs were present in photoreceptors, inner nuclear layer neurons, and ganglion cells in C57BL

221 utofluorescent, cystoid macular edema in the inner nuclear layer, no light rise in the electro-oculog

222 pulled glass pipettes and inserted into the inner nuclear layer of fixed whole-mount retinas.

223 lls of the developing neuroretina and in the inner nuclear layer of the mature retina.

224 icrocystic oedema predominantly involved the inner nuclear layer of the retina and tended to occur in

225 Microcystic abnormalities involving the inner nuclear layer of the retina occurs in a subset of

226 dom two-dimensional cellular patterns in the inner nuclear layer of the retina were investigated usin

227 ng retinal ganglion cells (RGCs) through the inner nuclear layer of the retina with glutamate, a prim

228 ha expression is dramatically induced in the inner nuclear layer of the retina, suggesting that PGC-1

229 electron microscopy in the choroid, RPE, and inner nuclear layer of the retina.

230 FR-1 and VEGFR-2 mRNAs were localized to the inner nuclear layer of the retina.

231 urons in the substantia nigra and within the inner nuclear layer of the retina.

232 tures found within the sclerad region of the inner nuclear layer of the retina.

233 litudes, and thinned the outer plexiform and inner nuclear layers of both WT and hWtEPOR 8-week-old m

234 for photoreceptors in the ganglion cell and inner nuclear layers of the developing retina, and a mor

235 cifically expressed in the ganglion cell and inner nuclear layers of the mouse retina.

236 tor inner segments and the ganglion cell and inner nuclear layers of the retina, and lesser amounts w

237 eferentially expressed in both the outer and inner nuclear layers of the retina.

238 further show that cells in the ganglion and inner-nuclear layers of the retina constitutively expres

239 fiber, ganglion cells, inner plexiform, and inner nuclear layers) of eyes with previous optic neurit

240 yperreflectance of outer plexiform layer and inner nuclear layer on spectral-domain OCT was observed

241 tive plaque-like lesions at the level of the inner nuclear layer on spectral-domain OCT, with no fluo

242 clei of cells in the ganglion cell layer and inner nuclear layer on whole-retinal sections.

243 iform layer (GCL+IPL), RNFL, outer plexiform/inner nuclear layers (OPL+INL), and outer nuclear/photor

244 nces were observed in the number of cells in inner nuclear layer or in ganglion cells at 12 months of

245 not at all correlated with thickness of the inner nuclear layer or total retina.

246 idual layers detected on OCT scans, with the inner nuclear layer (outer superior quadrant, B = -3.06;

247 Edema was identified by thickening of the inner nuclear layer, outer plexiform layer, or full reti

248 and in the number of apoptotic cells in the inner nuclear layer (P < 0.0001).

249 peripheral reduction in the thickness of the inner nuclear layer (P < 0.001), and a 23.4% reduction i

250 membrane, the inner plexiform layer, and the inner nuclear layer (P < 0.02).

251 ner segments of photoreceptors, cells in the inner nuclear layer, particularly amacrine cells, and re

252 Microcystic inner nuclear layer pathology occurs in a proportion of

253 Identification of microcystic inner nuclear layer pathology on spectral-domain optical

254 lion cell layer, the anterior portion of the inner nuclear layer, photoreceptors, and choroidal strom

255 ally to (i) the combined ganglion cell layer/inner nuclear layer plus the embedded retinal vessels, (

256 sociated with GCIP (r = -0.30; p = 0.02) and inner nuclear layer (r = -0.25; p = 0.04) atrophy rates.

257 ollows: r, 0.01 (95% CI, -0.07 to -0.27) for inner nuclear layer; r, 0.19 (95% CI, 0.01 to 0.35) for

258 in the thickness of the inner plexiform and inner nuclear layers, respectively (P < 0.001).

259 ness includes photoreceptor layer thickness: inner nuclear layer-retinal pigment epithelium (INL-RPE)

260 not prevent or alter the timing of outer and inner nuclear layer separation, but it inhibited phototr

261 The inner nuclear layer shows a low reflectivity; the proper

262 yer and ganglion cell layer, but also in the inner nuclear layer, suggesting that retinal injury is m

263 , inner plexiform layer (S6 and N6 sectors), inner nuclear layer (T6 and N6 sectors), and outer plexi

264 dle layers of the retina at the level of the inner nuclear layer that may develop in response to isch

265 of the DAPI-3 cells are the only ones in the inner nuclear layer that stain strongly for either the a

266 constitutive expression of VEGF mRNA in the inner nuclear layer that was increased 6 hours after the

267 Cells in the ganglion cell and inner nuclear layers that labeled for GABA or glycine we

268 ers of mitotic nuclei remaining in the basal inner nuclear layer, the region where Muller glia typica

269 consisted of nerve fiber, ganglion cell, and inner nuclear layer; the hypointense layer 2, the outer

270 retina in several cell layers, including the inner nuclear layer; they are present in primary mouse M

271 At the fovea, retinal and inner nuclear layer thickness increased with edema sever

272 At the fovea, retinal and inner nuclear layer thickness increased with edema sever

273 of only the GCL, inner plexiform layer, and inner nuclear layer thickness with rates of -0.11 mum/ye

274 Inner nuclear layer thickness, also in eyes without a hi

275 ence of CME, central foveal thickness (CFT), inner nuclear layer thickness, and foveal-to-parafoveal

276 eye development, whereas ganglion cell loss, inner nuclear layer thinness, and early onset of glaucom

277 MM lesions were identified as focal areas of inner nuclear layer thinning over an anteriorly displace

278 ronic or resolved PAMM lesion was defined as inner nuclear layer thinning with outer plexiform layer

279 egeneration with severe GCL loss, borderline inner nuclear layer thinning, and less prominent photore

280 and in rare neurites that extend across the inner nuclear layer to the inner plexiform layer.

281 ations with hyperreflective walls within the inner nuclear layer (type 1, n = 4) and vascular outpouc

282 tation mostly influences the function of the inner nuclear layer, unexpectedly the chy mutant phenoty

283 least one cell that had a large soma in the inner nuclear layer, varicose axons and dendrites with a

284 glion cell layer, ganglion cell complex, and inner nuclear layer volumes showed an inverse relationsh

285 Among the intraretinal layers, the inner nuclear layer was identified as the best indicator

286 The inner nuclear layer was normal or had borderline thinnin

287 ng including en face OCT segmentation of the inner nuclear layer was performed in all patients.

288 ar regression model showed that SSPiM in the inner nuclear layer was related to treatment response (P

289 n cell layer, but such neurons remain in the inner nuclear layer well into the first postnatal month.

290 me cases, the retinal pigment epithelium and inner nuclear layer were also affected.

291 er, and horizontal cells of all cells in the inner nuclear layer were comparable in central and perip

292 Microcystoid macular changes in the inner nuclear layer were diagnosed in 52 out of 264 eyes

293 eviously observed dopaminergic plexus in the inner nuclear layer were observed to contact the somata

294 ells residing in the outermost lamina of the inner nuclear layer where horizontal cells are typically

295 n was distributed in horizontal cells of the inner nuclear layer, whereas the mRNA was expressed in a

296 in RGCs and cells of the inner aspect of the inner nuclear layer, which, by double staining with anti

297 of apoE-deficient mice revealed cells of the inner nuclear layer with condensation of nuclear chromat

298 electron microscopy illustrated degenerated inner nuclear layer with disintegration of cells and los

299 Neuronal loss was noted in the inner nuclear layer with focal reduction in cell density

300 Substantial cell loss occurred in the inner nuclear layer, with a smaller reduction in photore