ライフサイエンスコーパス: insecticide

1 ositive control (emamectin benzoate 'Attack' insecticide).

2 affordable products (diagnostics, drugs and insecticides).

3 sects, which suggests its possible use as an insecticide.

4 impact, particularly if used with a residual insecticide.

5 whether unsprayed or sprayed with botanical insecticide.

6 high-affinity binding of SAP2 to pyrethroid insecticides.

7 eonicotinoids are among the most widely used insecticides.

8 mary targets for a broad range of commercial insecticides.

9 oring human exposure to synthetic pyrethroid insecticides.

10 of high GSTe2-based metabolic resistance to insecticides.

11 luating the resistance of NCR populations to insecticides.

12 ystem services including providing botanical insecticides.

13 esticide risk, which was driven primarily by insecticides.

14 n of mating disruption and use of biological insecticides.

15 ary exposure (e.g., via leaves) for systemic insecticides.

16 a mix of insecticides, or a mix of all five insecticides.

17 by allowing resistant insects to metabolize insecticides.

18 gnostics and production of steroid drugs and insecticides.

19 ferential susceptibilities of the species to insecticides.

20 use of an ever decreasing number of approved insecticides.

21 Lambda-cyhalothrin was the main insecticide (97.5%) used by farmers, and Anopheles coluz

22 apevine moth (Lobesia botrana) outbreaks and insecticides across c.

23 ate matter (669,000 kg) could contain enough insecticides (active ingredient mass basis) to kill over

24 s a powerful tool for developing tailor-made insecticides against new target insects.

25 Insecticides allow control of agricultural pests and dis

26 cide, propiconazole (the synergist), and the insecticide, alpha-cypermethrin, on the mortality of the

27 onyl butoxide restored the susceptibility to insecticide among the highly resistant mosquitoes, confi

28 affects the mixture toxicity of a pyrethroid insecticide and azole fungicide.

29 P. elaeisis aiming the rational use of this insecticide and its compatibility with parasitoids.

30 The neonicotinoid insecticides and 2,4-dichlorophenoxyacetic acid, were fo

31 This pest has developed resistance to insecticides and adapted to crop rotation and may alread

32 rough a blend of active ingredients (such as insecticides and fungicides), polymers, waxes, fillers,

33 suggesting coverage of 200 m for the use of insecticides and innovative vector control tools.

34 e longevity of widely-employed neonicotinoid insecticides and of the ubiquitous ectoparasitic mite Va

35 organophosphate [OR=2.0 (95% CI: 1.3, 3.0)] insecticides and phenoxyacetate herbicides [OR=1.9 (95%

36 icide resistance are feasible using existing insecticides and production technology, and early implem

37 idual water or sediment concentrations of 32 insecticides and their metabolites in 644 US surface wat

38 hat has evolved resistance to all classes of insecticide, and costs the world economy an estimated US

39 onitoring systems, threshold-based selective insecticides, and biological control.

40 are providing the basis of novel medicines, insecticides, and even nucleic acid sequencing technolog

41 A safe waiting period of 17-18 d after final insecticide application needs to be maintained before ha

42 Similarly, insecticide applications doubled in vineyard-dominated l

43 the importance of external factors, such as insecticide applications, in mediating the outcome of in

44 complexity may mitigate pest populations and insecticide applications.

45 bee toxic load' (honey bee lethal doses) for insecticides applied in the US between 1997-2012, calcul

46 e hazardous to bees despite lower volumes of insecticides applied, raising concerns about insect cons

47 phates) and two pesticide types (herbicides, insecticides) applied at standardised environmental conc

48 is study is to determine the extent to which insecticides are also transported into the environment b

49 Each year, millions of kilograms of insecticides are applied to crops in the US.

50 ht against malaria for decades, and chemical insecticides are critical to the success of vector contr

51 While insecticides are essential for reducing cockroach popula

52 Pyrethroid insecticides are known to be highly toxic to most aquati

53 Given that other insecticides are known to generate oxidative stress, thi

54 Pyrethroid contact insecticides are mainstays of malaria control, but their

55 istance is a paradigm of microevolution, and insecticides are responsible for the strongest cases of

56 Insecticides are the main method of controlling lepidopt

57 Contact insecticides are transmitted to the insect when its feet

58 as restricted the use of three neonicotinoid insecticides as seed dressings on bee-attractive crops.

59 s a key factor in the mode of action of this insecticide at low doses.

60 to strong selection with glucose-containing insecticide baits.

61 tained by controlling mosquito vectors using insecticide-based approaches.

62 technology that is compatible with existing insecticide-based control methods.

63 s crucial to prolonging the effectiveness of insecticide-based control tools including long lasting i

64 ndomised controlled trial evaluating a novel insecticide-based intervention in central Cote d'Ivoire,

65 Insecticide-based interventions have contributed to appr

66 cticide resistance poses a serious threat to insecticide-based interventions in Africa.

67 s detected here as increasing selection from insecticide-based interventions may change the dynamic i

68 ut PBO is a concern for the effectiveness of insecticide-based interventions, and action should be ta

69 er mass drug administration (MDA) for LF, or insecticide-based interventions.

70 of control programmes, which rely largely on insecticide-based interventions.

71 Traditional insecticide-based vector-control programmes have limited

72 rs were significantly depressed by synthetic insecticide but higher in other treatments whether unspr

73 rds are potentially exposed to neonicotinoid insecticides by ingestion of coated seeds during crop pl

74 in which novel, olfactory molecule-specific insecticides can be developed.

75 Excessive use of herbicide and insecticide causes bioaccumulation in the environment an

76 characterized as differentially expressed in insecticide-challenged mosquitoes, or increased mRNA exp

77 ulation that is completely naive to this new insecticide chemistry and it foreshadows a selective res

78 Production and use of the insecticide chlordecone has caused long-term environment

79 C DTF) magnified the negative impact of the insecticide chlorpyrifos (CPF) in larvae of low- and hig

80 , atrazine, cyanazine, and paraquat) and two insecticides (chlorpyrifos and chlordane).

81 Zika virus, understanding resistance to this insecticide class is of significant relevance.

82 r Here we use a naive population and a novel insecticide class to examine the ab initio genetic archi

83 ctors, which drive exposure for all relevant insecticide classes, and subsequently mapping these risk

84 e synergist piperonyl butoxide (PBO), or new insecticides, clothianidin and chlorfenapyr, were highly

85 we exposed monarch larvae to six pesticides (insecticide: clothianidin; herbicides: atrazine, S-metol

86 When insecticide concentrations observed in this study are pr

87 e regions, where the comparison of predicted insecticide concentrations with their RTLs indicate adve

88 chlorantraniliprole and the closely related insecticide cyantraniliprole.

89 oid insecticide thiamethoxam, the pyrethroid insecticide cypermethrin and the EBI fungicide tebuconaz

90 rpenes for fragrances, p-dichlorobenzene for insecticides, D4-siloxane for adhesives, para-chlorobenz

91 return to the notorious crystalline contact insecticide DDT.

92 exposed HPEEs, including two organochlorine insecticides (DDT and lindane) and four herbicides (alac

93 and effects on biological parameters of the insecticide deltamethrin, registered for the control of

94 B2 increases C. pomonella sensitivity to two insecticides, deltamethrin and azinphos methyl.

95 tion of the solid-state chemistry of contact insecticides, demonstrated here for DFDT and MFDT, is a

96 al as a new mode of action target for future insecticide development.

97 identifying channel blockers of interest to insecticide discovery or biosecurity.

98 d, and diverse synthetic compounds including insecticides exert JH-like effects.

99 2005 to 2017, with mean mortality following insecticide exposure declining from almost 100% to less

100 malaria control, its use contributes to high insecticide exposure in sprayed communities and raises c

101 We found no impact of insecticide exposure on colony weight gain, or the numbe

102 e strong evidence for honeydew as a route of insecticide exposure that may cause acute or chronic del

103 should not discount mosquitoes that survive insecticide exposure with fewer than six legs, as they m

104 stead, ABC transporters may be induced after insecticide exposure.

105 nd 170-fold more sensitive to the butenolide insecticide flupyradifurone than other managed bee polli

106 tion addressed by SPT, a well-known systemic insecticide for sucking pest control.

107 or formulating effective essential oil-based insecticides for bed bug IPM and conducting mode-of-acti

108 les gambiae, caused by the widespread use of insecticides for malaria control, to investigate the rol

109 rmination of neonicotinoids as well as other insecticides, fungicides, herbicides, acaricides, growth

110 However, resistance to insecticides has become widespread in Anopheles populati

111 f WCR field-evolved resistance to pyrethroid insecticides has been confirmed in the US western Corn B

112 cultural use of diazinon, an organophosphate insecticide, has raised serious environmental health con

113 easingly recognized, that some neonicotinoid insecticides have a negative impact on non-target organi

114 New insecticides, however, can be expensive to implement in

115 ecause of the greatly diminished use of this insecticide in the United States over the last 20-25 yea

116 Resistance to multiple insecticides in An. gambiae s.s. and An. coluzzii was de

117 Resistance to major public health insecticides in Cote d'Ivoire has intensified and now th

118 e important implications for the safe use of insecticides in crops where M. rotundata is used for pol

119 The spread of resistance to insecticides in disease-carrying mosquitoes poses a thre

120 ance and to track their spread as the use of insecticides in malaria endemic countries intensifies an

121 the genetic basis of metabolic resistance to insecticides in malaria vectors is crucial to prolonging

122 public health and agricultural pest control insecticides in nature, and thus the effect of specific

123 Widespread use of neonicotinoid insecticides in North America has led to frequent detect

124 cide resistance due to indiscriminate use of insecticides in public health and agricultural system fa

125 ogaster, an experimental organism exposed to insecticides in the field.

126 control may be compromised by resistance to insecticides in vector populations.

127 development of novel antiparasite agents and insecticides in veterinary medicine and agriculture.

128 The potency of bifenthrin (a pyrethroid insecticide) in topical bioassays was 72,000 times highe

129 dapted to confer intrinsic tolerance to some insecticides including certain neonicotinoids.

130 The organophosphate class of insecticides increased snail abundance (first intermedia

131 These experiments demonstrated that insecticide-induced leg loss had no significant effect u

132 ce ROS production in the brain recapitulates insecticide-induced phenotypes in the metabolic tissues,

133 tes are potent vectors of viruses, the virus-insecticide interaction had no significant impact.

134 Indoor residual spraying of insecticide (IRS) has been associated with reductions in

135 The best known limonoid insecticide is azadirachtin, produced by the neem tree (

136 ercially recommended one and therefore, this insecticide is not selective for this natural enemy.

137 Indoor residual spraying (IRS) of insecticides is a major vector control strategy for mala

138 on of alternative modes of action for future insecticides is paramount.

139 Metabolic resistance to insecticides is threatening malaria control in Africa.

140 ome on the market, especially those with new insecticides, it will be imperative to monitor their com

141 Toxicants such as organochlorine insecticides, lead ammunition, and veterinary drugs have

142 ines, and organophosphates) of public health insecticides limits effective control of malaria transmi

143 synthetic chemicals that dominate the global insecticide market.

144 Pyrethroids insecticides may constitute a major hazard to honeybees,

145 Alternative 'botanical insecticides' may also have non-target effects on pollin

146 Resistance mechanisms to synthetic insecticides often include point mutations and increased

147 ers, opening the way for deploying many more insecticides on bednets than is currently possible.

148 We show consistent effects of herbicides and insecticides on ecosystem function, and slightly less co

149 es have demonstrated impacts of low doses of insecticides on insect behavior, but have not elucidated

150 s in nature, and thus the effect of specific insecticides on rate of resistance emergency or resistan

151 The effects of insecticides on respiration and primary productivity of

152 iral discrimination during the uptake of the insecticide or when binding at the sodium channel, the p

153 sticide alone, a mix of herbicides, a mix of insecticides, or a mix of all five insecticides.

154 intermediate useful for the synthesis of the insecticide permethrin and other natural products.

155 t types of pesticides (i.e. organophosphorus insecticides, phenoxy-acid herbicides, and triazine herb

156 system is a compelling target for the novel insecticide pipeline.

157 The fate of the insecticide pirimiphos-methyl (PM) in farmed Atlantic sa

158 from 2014 to 2016 using the organophosphate insecticide pirimiphos-methyl.

159 ayed treatments (with botanical or synthetic insecticide positive control) while treatments with bare

160 , increasing mosquito resistance to chemical insecticides presents challenges to this approach.

161 The groups exposed to the neonicotinoid insecticide produced 40%-76% fewer queens than control c

162 ic aromatic hydrocarbons (PAHs), phthalates, insecticides, pyrethroids, and N,N-diethyl-meta-toluamid

163 le systems for rapid assays of herbicide and insecticide residues have attracted prominent interests.

164 Insecticide resistance across sub-Saharan Africa may imp

165 is has been attributed to the development of insecticide resistance and behavioural adaptations in ma

166 osquitoes is threatened by the appearance of insecticide resistance and therefore new control chemica

167 to identify the contribution of each CNV to insecticide resistance and to track their spread as the

168 Barrier bednets that overcome insecticide resistance are feasible using existing insec

169 P450 monooxygenases play a critical role in insecticide resistance by allowing resistant insects to

170 Synergists can counteract metabolic insecticide resistance by inhibiting detoxification enzy

171 The directional selection for insecticide resistance due to indiscriminate use of inse

172 h populations and improving health outcomes, insecticide resistance has been a consistent barrier to

173 Whilst insecticide resistance has been widely investigated, the

174 Mitigating the threat of insecticide resistance in African malaria vector populat

175 r efficacies are declining due to widespread insecticide resistance in Anopheles mosquito populations

176 e potential contribution of agrochemicals to insecticide resistance in Anopheles mosquitoes breeding

177 ntrol is now threatened by alarming rates of insecticide resistance in insect populations, prompting

178 production could influence the selection for insecticide resistance in malaria vectors.

179 Past investigations on the emergence of insecticide resistance in mosquitoes mostly relied on fi

180 pact of gene drives as well as the spread of insecticide resistance in the wild.

181 The increase of insecticide resistance in wild populations of Anopheles

182 ring of occurrence, levels and mechanisms of insecticide resistance informs effective management stra

183 ucted seminal field studies to compare three insecticide resistance intervention strategies for cockr

184 Insecticide resistance is a paradigm of microevolution,

185 nagement enables potato production, although insecticide resistance is becoming an issue.

186 Understanding the molecular basis of insecticide resistance is key to improve the surveillanc

187 asmodium falciparum infection, and molecular insecticide resistance mechanisms in Guinea.

188 ndicating that neither tissue is involved in insecticide resistance mediated by the candidate P450s e

189 n, suggests the involvement of copper in the insecticide resistance of malaria vectors; this, however

190 studies directly assessing the influence of insecticide resistance on mosquito fitness.

191 nalysis using the GAL4/UAS system to examine insecticide resistance phenotypes conferred by increased

192 Insecticide resistance poses a serious threat to insecti

193 The role of miRNAs in mediating insecticide resistance remains largely unknown, even for

194 ow of malaria vectors can help in combatting insecticide resistance spread and planning new vector co

195 den of malaria, information on bionomics and insecticide resistance status of malaria vectors is gros

196 ep towards a quantitative genotypic model of insecticide resistance that can be used to predict resis

197 tifies a previously undescribed mechanism of insecticide resistance that is likely to be highly relev

198 udy is a summary of the current level of the insecticide resistance to selected organophosphates, pyr

199 High levels of insecticide resistance were observed for five out of six

200 he proximity of genes relevant for immunity, insecticide resistance, and development.

201 detoxifying enzymes has been associated with insecticide resistance, but their direct functional vali

202 morphisms (SNPs) potentially associated with insecticide resistance, including three SNPs found in th

203 28 were in gene families linked to metabolic insecticide resistance, representing significant enrichm

204 stigate the role of CNVs in the evolution of insecticide resistance.

205 plementation of new technologies that manage insecticide resistance.

206 to identify candidate genes associated with insecticide resistance.

207 tially mitigate the global health effects of insecticide resistance.

208 xisting chemical control strategies to avert insecticide resistance.

209 entation of new technologies that circumvent insecticide resistance.

210 ated with speciation, ecological habitat and insecticide resistance.

211 ystem favors an increase in the frequency of insecticide-resistant alleles in the natural populations

212 Genome-wide association studies contrasting insecticide-resistant and susceptible strains identify h

213 efficacy of Metarhizium against wild-caught, insecticide-resistant anophelines.

214 sed fungal lethality and the likelihood that insecticide-resistant mosquitoes would be eliminated fro

215 Expression of SAP2 is increased in insecticide-resistant populations and is further induced

216 pplied fluralaner and permethrin in all five insecticide-resistant strains tested.

217 Results are presented for the selection of insecticide-resistant vectors and the spread of resistan

218 ed to be 2 and 5microgkg(-1) for the studied insecticides, respectively, with the exception of imidac

219 en its feet contact the solid surface of the insecticide, resulting in absorption of the active agent

220 nagement programs largely depend on chemical insecticides, resulting in high economic and environment

221 face waters to determine the factors driving insecticide risks, that is, exceedance of regulatory thr

222 neonicotinoid pharmacophore not only confers insecticide selectivity but also impacts sorption behavi

223 phosphates and hence that the usage of these insecticides should be reconsidered.

224 However, if the costs of insecticide spraying increase, it can be supplemented (a

225 ee injection, elimination of infected trees, insecticide spraying), we determined that elimination of

226 ronmental and safety hazards associated with insecticide sprays that had previously targeted the pink

227 ol policy in India restricts use of residual insecticide sprays to domestic dwellings.

228 ingiensis (Bt) can suppress pests and reduce insecticide sprays, but their efficacy is reduced when p

229 ue to Cyp6m2 gives credence to proposed dual-insecticide strategies to overcome pyrethroid resistance

230 Increased incidence of insecticides such as cyfluthrin, permethrin and cyhaloth

231 ual spraying results in elevated exposure to insecticides such as dichlorodiphenyltrichloroethane (DD

232 ices led to widespread aerial application of insecticides, such as dichlorodiphenyltrichloroethane (D

233 The evolution of resistance to insecticides, such as organophosphates (OPs), is a serio

234 effects of imidacloprid (IMD) and the novel insecticide sulfoxaflor (SFX) on visual motion-detection

235 Insecticide-susceptible horn flies and stable flies were

236 amat (SPT) is used as phloem-mobile systemic insecticide targeting acetyl-CoA carboxylase (ACC) of pe

237 resistance were observed for five out of six insecticides tested, with the lowest mortality (0.97%) r

238 on the effects of neonicotinoids, a class of insecticide that has gained attention for non-target eff

239 iological control and as source of botanical insecticides that are relatively benign to natural enemi

240 anism, which could affect the performance of insecticides that are structurally related to pyrethroid

241 key variables related to pest outbreaks and insecticides that drive management decisions.

242 ven the worldwide emergence of resistance to insecticides, the current mainstay for vector control, i

243 However, in comparison to herbicides and insecticides, the exposure to and effects of fungicides

244 t parasite Nosema ceranae, the neonicotinoid insecticide thiamethoxam, the pyrethroid insecticide cyp

245 However, increasing resistance to insecticides threatens to undermine these efforts.

246 Pyrethroids remain one of the most used insecticides to control Aedes mosquitoes, despite the de

247 istance to the most important vector control insecticides to inform a Bayesian geostatistical ensembl

248 he capacity of imidacloprid (a neonicotinoid insecticide) to photoinduce the nitration and nitrosatio

249 arriers, to the formulation of cosmetics and insecticides, to the fabrication of nanostructured mater

250 of resistance through repeated deployment of insecticide treated nets (ITNs), in addition to scenario

251 in the eco-engineering plots compared to the insecticide-treated and control plots.

252 osure to Anopheles bites varied according to insecticide-treated bed net use (P = .006).

253 iables including age, sex, household wealth, insecticide-treated bed net use, and vaccination status.

254 ia incidence and mortality due to the use of insecticide-treated bed nets and artemisinin combination

255 these parasites via the mass distribution of insecticide-treated bed nets have been extremely success

256 ons (MMPs), and includes improving access to insecticide-treated bed nets in the Myanmar artemisinin

257 Insecticide-treated bed nets reduce malaria transmission

258 hygiene practices, increased utilization of insecticide-treated bed nets, and greater participation

259 outdoors, thus reducing the effectiveness of insecticide-treated bed nets.

260 Royal Guard is a new insecticide-treated bed-net incorporated with a mixture

261 Insecticide-treated bednets (ITNs) are effective in prev

262 ehold-level mosquito exposure and individual insecticide-treated net (ITN) use on relative risk (RR)

263 36.6% reported sleeping under a long-lasting insecticide-treated net (LLIN) the previous night.

264 terval, 1.34-21.0; P = .02) and not using an insecticide-treated net during travel (18.0% for no use

265 In comparison, insecticide-treated net use was associated with 16%-17%

266 protected pregnancies (i.e., those not using insecticide-treated nets [ITNs]) leading to live births

267 es (RR/RR) significantly survive exposure to insecticide-treated nets and successfully blood feed mor

268 and their impact on the efficacy of various insecticide-treated nets established.

269 Population coverage of insecticide-treated nets increased by 8.34 percentage po

270 dditively exacerbate the reduced efficacy of insecticide-treated nets.

271 a control interventions, particularly use of insecticide-treated nets.

272 ls, the species co-existed on cabbage before insecticide treatments began, but with T. tabaci being t

273 Widespread and intensive insecticide usage has resulted in increased resistance i

274 Environmental contamination by intense insecticide usage is consistently proposed as a signific

275 vealed that toxicity-normalized agricultural insecticide use (i.e. use divided by toxicity) was the m

276 his substantially eliminated organophosphate insecticide use by 2001, replacing it with pest monitori

277 ins equal yields while reducing the need for insecticide use in crop fields.

278 Further, we find insecticide use increases with increasing field size.

279 While insecticide use supports food, fuel, and fiber productio

280 relation to the women's and their husbands' insecticide use using Cox proportional hazards regressio

281 mers and their wives provided information on insecticide use, demographics, and reproductive history

282 bollworm and facilitated an 82% reduction in insecticides used against all cotton pests in Arizona.

283 where mosquito populations are resistant to insecticides used in bed nets, but no association was fo

284 res are an attractive alternative to current insecticides used to control disease vectors.

285 neurons and mimicking the mode of action of insecticides used to control phloem-feeding insects.

286 ing the relative merits of sequential use of insecticides versus their deployment as a mixture to min

287 The residual effect of the insecticide was assessed monthly by exposing susceptible

288 itional cost associated with using botanical insecticides was not justified by greater levels of pest

289 various metrics of floral/nesting resources, insecticides, weather, and honey bee (Apis mellifera) ab

290 is highest, bednets treated with pyrethroid insecticide were highly effective in preventing mosquito

291 the doses of 0.64 and 1.40 mg a.i./L of the insecticide were lower.

292 at higher geographical elevations and use of insecticide were protective.

293 ile vapors of dichlorvos (an organophosphate insecticide) were 445 times more potent than thymol.

294 ulture is mainly based on the application of insecticides, which may impact nontarget beneficial orga

295 Spinosad is an insecticide widely used for the control of insect pest s

296 ro-2,2-bis(4-chlorophenyl)ethane), a contact insecticide with a rich and controversial history since

297 nterval is needed when the rotational use of insecticides with different modes of action is considere

298 oposed to overcome this challenge, including insecticides with new modes of action.

299 e selection pressure, and are effective when insecticides with no/low resistance are used.

300 Neonicotinoids are the most widely used insecticides worldwide and are typically deployed as see